Search results for "OBESITY"

showing 10 items of 1833 documents

Molecular dynamics, dynamic site mapping, and highthroughput virtual screening on leptin and the Ob receptor as anti-obesity target.

2014

Body weight control is a mechanism finely regulated by several hormonal, metabolic, and nervous pathways. The leptin receptor (Ob-R) is crucial for energy homeostasis and regulation of food uptake. Leptin is a 16 kDa hormone that is mainly secreted by fat cells into the bloodstream, and under normal circumstances, circulating levels are proportionate to the fat body mass. Sensing of elevated leptin levels by the hypothalamic neurocircutry activates a negative feedback loop resulting in reduced food intake and increased energy expenditure. Decreased concentrations lead to opposite effects. Therefore rational design of leptin agonists constitute an appealing challenge in the battle against ob…

Leptinmedicine.medical_specialtyProtein ConformationAdipose tissueDrug designBiologyMolecular Dynamics SimulationDynamic SiteMapping HTVS Leptin Molecular Dynamics Obesity Protein/protein docking Multivariate analysis Ob ReceptorCatalysisEnergy homeostasisInorganic ChemistryStructure-Activity RelationshipInternal medicinemedicineMolecular Targeted TherapyPhysical and Theoretical ChemistryReceptorVirtual screeningLeptin receptorBinding SitesMolecular StructureLeptindigestive oral and skin physiologyOrganic ChemistryHydrogen BondingSettore CHIM/08 - Chimica FarmaceuticaComputer Science ApplicationsHigh-Throughput Screening AssaysMolecular Docking SimulationEndocrinologyComputational Theory and MathematicsDocking (molecular)Drug DesignMultivariate AnalysisComputer-Aided DesignReceptors LeptinAnti-Obesity AgentsHydrophobic and Hydrophilic InteractionsProtein BindingJournal of molecular modeling
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Obesity, type 2 diabetes and risk of digestive cancer.

2010

The frequency of obesity has been increasing worldwide for 20 years. Many epidemiological studies support a correlation between obesity and increased risk of cancer, particularly digestive cancers in both genders, and gynaecological cancer in women. Currently, about 5% of cancers could be directly related to overweight. Carcinogenesis mechanisms induced by obesity involve insulin resistance, adipokine and angiogenic factor secretions, and inflammation. Experimental and clinical evidence suggest that insulin resistance plays a major role in carcinogenesis. Insulin and non-protein banded IGF-1, whose levels are increased in type 2 diabetes, stimulate cellular growth and inhibit apoptosis. Abn…

Leptinmedicine.medical_specialtySerine Proteinase Inhibitorsmedicine.medical_treatmentAdipokineType 2 diabetesOverweightBioinformaticsGlobal HealthRisk AssessmentBody Mass IndexDiabetes ComplicationsBiological FactorsInsulin resistanceAdipokinesRisk FactorsInternal medicineDiabetes mellitusPlasminogen Activator Inhibitor 1PrevalenceMedicineHumansInsulinAdiponectin secretionObesityInsulin-Like Growth Factor IGastrointestinal NeoplasmsEvidence-Based Medicinebusiness.industryInsulinIncidenceGastroenterologyCancerGeneral Medicinemedicine.diseasePrognosisEndocrinologyAdipose TissueDiabetes Mellitus Type 2MetalloproteasesFrancemedicine.symptomInsulin ResistancebusinessGastroenterologie clinique et biologique
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Changes of inflammatory mediators in obese patients after laparoscopic cholecystectomy.

2010

Background Obesity is associated with the impairment of immunological functions. The aim of this study was to analyze some inflammatory mediators in obese subjects who underwent laparoscopic cholecystectomy. Methods Seventeen consecutive female patients with a BMI ranging from 35 to 45 kg/m2 (obese) and 17 consecutive female patients with BMI ranging from 20 to 25 kg/m2 (nonobese) were included in the study. All patients were affected by symptomatic gallbladder stone disease and underwent laparoscopic cholecystectomy. Changes in levels of leukocytes, neutrophils, IL-6, IL-10, leptin, and adiponectin were evaluated. Results We observed a significant increase in leukocyte and neutrophil level…

Leptinmedicine.medical_specialtySurgical stressmedicine.medical_treatmentInflammationGastroenterologyBody Mass IndexLeukocyte CountLaparoscopic cholecystectomyStress PhysiologicalInternal medicinemedicineHumansLaparoscopic cholecystectomy Obesity Inflammatory mediatorsObesitybusiness.industryObesity Laparoscopic Cholecystectomy Inflammatory MediatorsInterleukin-6GallbladderCholecystolithiasisVascular surgerySweet SyndromeInflammatory mediatorsCardiac surgeryInterleukin-10Settore MED/18 - Chirurgia Generalemedicine.anatomical_structureCholecystectomy LaparoscopicCardiothoracic surgerySurgeryCholecystectomyFemaleAdiponectinmedicine.symptomInflammation MediatorsbusinessAbdominal surgery
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Polymorphisms in FTO and near TMEM18 associate with type 2 diabetes and predispose to younger age at diagnosis of diabetes

2013

Variations in the FTO gene and near the TMEM18 gene are risk factors for common form of obesity, but have also been linked with type 2 diabetes (T2D). Our aim was to investigate the contribution of these variants to risk of T2D in a population in Latvia. Four single nucleotide polymorphisms (SNP) in the first and fourth intronic regions of FTO and one close to TMEM18 were genotyped in 987 patients with T2D and 1080 controls selected from the Latvian Genome Data Base (LGDB). We confirmed association of SNPs in the first intron (rs11642015, rs62048402 and rs9939609) of FTO and rs7561317 representing the TMEM18 locus with T2D. Association between SNP in FTO and T2D remained significant after c…

Linkage disequilibriumendocrine system diseasesPopulationAlpha-Ketoglutarate-Dependent Dioxygenase FTOSingle-nucleotide polymorphismLocus (genetics)Genome-wide association studyBiologyPolymorphism Single NucleotideFTO geneGeneticsmedicineHumansSNPAge of OnseteducationGeneticseducation.field_of_studyAge FactorsMembrane ProteinsProteinsnutritional and metabolic diseasesGeneral Medicinemedicine.diseaseObesityDiabetes Mellitus Type 2Gene
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Role of adipose tissue cannabinoid 1 receptor (CB1R) on lipolysis and consequences on lipid and carbohydrate metabolism in mice

2017

Role of adipose tissue cannabinoid receptor 1 (CB1R) on lipolysis and consequences on lipid and carbohydrate metabolism in miceObesity is accompanied with lipolysis deregulation responsible for excessive Free Fatty Acid (FFA) release, which are key actors in the implementation of peripheral insulin resistance. In parallel, it is now well established that obesity is associated with EndoCannabinoid System (ECS) activity dysfunction in several metabolic tissues. If it is well described that overactivation of this system in the Adipose Tissue (AT) is favourable to its expansion, the consequences on lipolysis remain to be elucidated.The main objective of this thesis work was to precise the role …

Lipolyse[SDV.MHEP] Life Sciences [q-bio]/Human health and pathologyEndocannabinoid systemLiverLipolysisInsulinorésistanceAdipose tissueTissu adipeuxInsulin resistanceSystème endocannabinoïdesObesityObésitéFoie
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Immune Cell Toll-like Receptor 4 Mediates the Development of Obesity- and Endotoxemia-Associated Adipose Tissue Fibrosis

2014

International audience; Adipose tissue fibrosis development blocks adipocyte hypertrophy and favors ectopic lipid accumulation. Here, we show that adipose tissue fibrosis is associated with obesity and insulin resistance in humans and mice. Kinetic studies in C3H mice fed a high-fat diet show activation of macrophages and progression of fibrosis along with adipocyte metabolic dysfunction and death. Adipose tissue fibrosis is attenuated by macrophage depletion. Impairment of Toll-like receptor 4 signaling protects mice from obesity-induced fibrosis. The presence of a functional Toll-like receptor 4 on adipose tissue hematopoietic cells is necessary for the initiation of adipose tissue fibros…

LipopolysaccharidesMESH: Signal TransductionMESH: InflammationMESH : Toll-Like Receptor 4Adipose tissueMESH : AdipocytesMESH : LipopolysaccharidesMicechemistry.chemical_compoundFibrosisAdipocyteAdipocytes[ SDV.IMM ] Life Sciences [q-bio]/ImmunologyMESH: ObesityMESH: Animalslcsh:QH301-705.5Mice Inbred C3HToll-like receptorMESH : Diet High-FatMESH: Toll-Like Receptor 43. Good healthMESH: Insulin ResistanceAdipose TissueMESH: FibrosisMESH : Fibrosis[SDV.IMM]Life Sciences [q-bio]/ImmunologyMESH : ObesityMESH : Insulin ResistanceMESH: Adipose TissueSignal Transductionmedicine.medical_specialty[SDV.IMM] Life Sciences [q-bio]/ImmunologyAdipose tissue macrophagesBiologyDiet High-FatMESH : Adipose TissueGeneral Biochemistry Genetics and Molecular BiologyImmune systemMESH : Mice Inbred C3HInternal medicineMESH : MicemedicineAnimalsHumansObesityMESH: Mice Inbred C3HMESH: MiceMESH: AdipocytesInflammationMESH : Signal TransductionMESH : InflammationMESH: HumansMESH : EndotoxemiaMESH : Humans3T3-L1medicine.diseaseFibrosisMESH : Disease Models AnimalEndotoxemiaToll-Like Receptor 4Disease Models AnimalMESH: Diet High-FatEndocrinologylcsh:Biology (General)chemistryMESH: EndotoxemiaMESH : AnimalsInsulin ResistanceMESH: Disease Models AnimalMESH: LipopolysaccharidesAdipocyte hypertrophyCell Reports
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Miglioramenti della qualità di vita correlata alla salute (HRQoL) con Liraglutide 3 mg rispetto a placebo per la gestione del peso

2015

Obiettivo: L'obesità ha un impatto negativo sulla qualità di vita correlata alla salute (HRQoL), la cui gravità dipende dalla severità dell’obesità. Lo studio SCALE (Satiety and Clinical Adiposity - Liraglutide Evidence) Obesity and Prediabetes ha valutato l’efficacia di Liraglutide 3 mg in aggiunta a dieta ed esercizio fisico sulla HRQoL in soggetti con sovrappeso o obesità, con o senza prediabete. Metodi: I soggetti (BMI ≥ 27 kg/m2 con ≥ 1 comorbidità o ≥ 30 kg/m2) sono stati randomizzati (2:1), a ricevere liraglutide 3 mg (n = 2487) o placebo (n = 1244) una volate al giorno s.c., in aggiunta ad un programma di intervento sullo stile di vita (dieta ipocalorica con deficit di 500 Kcal/die …

Liraglutide quality of life obesity
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A new definition for metabolic dysfunction-associated fatty liver disease: An international expert consensus statement

2020

The exclusion of other chronic liver diseases including “excess” alcohol intake has until now been necessary to establish a diagnosis of metabolic dysfunction-associated fatty liver disease (MAFLD). However, given our current understanding of the pathogenesis of MAFLD and its rising prevalence, “positive criteria” to diagnose the disease are required. In this work, a panel of international experts from 22 countries propose a new definition for the diagnosis of MAFLD that is both comprehensive and simple, and is independent of other liver diseases. The criteria are based on evidence of hepatic steatosis, in addition to one of the following three criteria, namely overweight/obesity, presence …

Liver Cirrhosis0301 basic medicineCirrhosisDiagnostic criteriaCirrhosis; Diabetes; Diagnostic criteria; MAFLD; Metabolic; NAFLD; Obesity; Steatohepatitis[SDV]Life Sciences [q-bio]HISTOLOGIC FEATURESPROGRESSIONDiseaseTerminology0302 clinical medicineMedicine10. No inequalitySteatohepatitisNONALCOHOLIC STEATOHEPATITISFatty liverHIGH BLOOD-PRESSUREDiabetesHEALTHY OBESE3. Good healthPREVALENCECausalityCirrhosisDisease Progression030211 gastroenterology & hepatologymedicine.medical_specialtyConsensusMAFLDDIAGNOSIS03 medical and health sciencesMetabolic DiseasesTerminology as TopicDiabetes mellitusNAFLDMANAGEMENTHumansObesityIntensive care medicineHepatologybusiness.industryType 2 Diabetes MellitusNATURAL-HISTORYmedicine.diseaseFatty LiverClinical trial030104 developmental biologyDiabetes Mellitus Type 2MetabolicSteatohepatitisbusiness
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Serum ferritin is a discriminant marker for both fibrosis and inflammation in histologically proven non-alcoholic fatty liver disease patients

2011

INTRODUCTION: Differentiation between steatosis and non-alcoholic steatohepatitis (NASH) in non-alcoholic fatty liver disease (NAFLD) is important as NASH progress to cirrhosis. No specific laboratory/imaging technique exists either to diagnose NASH or to select patients for liver biopsy. PATIENTS AND METHODS: We evaluated serum ferritin and the features of metabolic syndrome with respect to histological inflammation and/or fibrosis in NAFLD patients. The Kleiner scoring system was used to classify NAFLD in consecutive liver biopsies. One hundred and eleven patients: median age 52.6, 64 males, obesity 62, diabetes mellitus (DM) 58, arterial hypertension 26 and hyperlipidaemia 40%. RESULTS: …

Liver CirrhosisAdultMaleBiopsyHyperlipidemiasFatty Liver/blood/diagnosis/etiology/pathologyRisk AssessmentSeverity of Illness IndexHepatitisBody Mass IndexDiabetes ComplicationsYoung AdultNon-alcoholic Fatty Liver DiseasePredictive Value of TestsRisk FactorsNAFLDLondonMetabolic Syndrome X/blood/*complicationsHumansAspartate AminotransferasesObesityBiological Markers/bloodliver fibrosisAgedMetabolic SyndromeInflammationFerritinChi-Square DistributionPatient SelectionNASHHepatitis/blood/complications/*diagnosisMiddle AgedFibrosisFatty LiverLiver Cirrhosis/blood/*diagnosis/etiologyNomogramsLogistic ModelsHyperlipidemias/blood/complicationsHypertension/blood/complicationsFerritinsHypertensionFerritin; Fibrosis; Inflammation; NAFLD; NASHAspartate Aminotransferases/bloodFemaleDiabetes Complications/blood/diagnosis/etiologyObesity/complicationsBiomarkersFerritins/*blood
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Sarcopenia is associated with severe liver fibrosis in patients with non-alcoholic fatty liver disease

2017

Background: Sarcopenia recognises insulin resistance and obesity as risk factors, and is frequently associated with cardiometabolic disorders, including non-alcoholic fatty liver disease (NAFLD). Aim: To test the prevalence of sarcopenia and its relation with the severity of fibrosis (main outcome) and the entire spectrum of liver histology in patients with NAFLD. Methods: We considered 225 consecutive patients with histological diagnosis of NAFLD (Kleiner score). The skeletal muscle index (%) (total appendicular skeletal muscle mass (kg)/weight (kg) × 100), a validated measure of sarcopenia, was assessed by bioelectrical impedance analysis. Sarcopenia was defined as a skeletal muscle mass…

Liver CirrhosisAdultMaleSarcopeniamedicine.medical_specialtyLiver CirrhosiSeverity of Illness IndexGastroenterology03 medical and health sciencesLiver disease0302 clinical medicineInsulin resistanceRisk FactorsNon-alcoholic Fatty Liver DiseaseFibrosisInternal medicineDiabetes mellitusSeverity of illnessmedicineHumansPharmacology (medical)Prospective StudiesObesityAgedCross-Sectional StudieHepatologybusiness.industryRisk FactorFatty liverGastroenterologyMiddle Agedmusculoskeletal systemmedicine.diseaseProspective StudieCross-Sectional StudiesEndocrinology030220 oncology & carcinogenesisSarcopeniaDisease ProgressionFemale030211 gastroenterology & hepatologyInsulin ResistanceSteatosisbusinesshuman activitiesHumanAlimentary Pharmacology & Therapeutics
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