Search results for "ORIGINAL ARTICLES"

showing 10 items of 474 documents

Identity Formation in Adulthood: A Longitudinal Study from Age 27 to 50.

2016

Longitudinal patterns of identity formation were analyzed in a representative cohort group of Finnish men and women born in 1959 across ages 27, 36, 42, and 50. The data were drawn from the Jyväskylä Longitudinal Study of Personality. Identity status (diffused, moratorium, foreclosed, achieved) from all four ages was available for 172 participants (54% females). Marcia’s Identity Status Interview used in this research included five domains: religious beliefs, political identity, occupational career, intimate relationships, and lifestyle. The findings indicated great variability in identity status across domains at each age level, and the identity trajectories fluctuated from age 27 to 50. T…

Longitudinal studySociology and Political Sciencemedia_common.quotation_subjectsalience050109 social psychologyidentity developmentReligious identityArticleDevelopmental psychology5. Gender equalityidentity domainsfollow-upPersonality0501 psychology and cognitive sciences10. No inequalityta515media_commonAdult developmentcongruence05 social sciencesOriginal ArticlesMiddle ageCongruenceIdentity developmentoverall identityidentity statusCohortmiddle-agePsychology (miscellaneous)PsychologyIdentity formation050104 developmental & child psychologyIdentity (Mahwah, N.J.)
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Loss of life expectancy from air pollution compared to other risk factors: a worldwide perspective

2020

Abstract Aims Long-term exposure of humans to air pollution enhances the risk of cardiovascular and respiratory diseases. A novel Global Exposure Mortality Model (GEMM) has been derived from many cohort studies, providing much-improved coverage of the exposure to fine particulate matter (PM2.5). We applied the GEMM to assess excess mortality attributable to ambient air pollution on a global scale and compare to other risk factors. Methods and results We used a data-informed atmospheric model to calculate worldwide exposure to PM2.5 and ozone pollution, which was combined with the GEMM to estimate disease-specific excess mortality and loss of life expectancy (LLE) in 2015. Using this model, …

Lung DiseasesMaleFine particulate matterTime Factors010504 meteorology & atmospheric sciencesPhysiologyAnthropogenic emissionsFossil fuel emissionsAir pollution010501 environmental sciencesGlobal Healthmedicine.disease_cause01 natural sciencesRisk FactorsGlobal healthAcademicSubjects/MED00200Childmedia_commonAged 80 and overExposure to ViolenceExpectancy theoryAir PollutantsMortality rateMiddle AgedParticulatesCardiovascular DiseasesChild PreschoolPublic health risksFemaleCardiology and Cardiovascular MedicineLoss of lifeAdultPollutionAdolescentRisk in Cardiovascular Diseasemedia_common.quotation_subjectAir pollutionViolenceRisk AssessmentYoung AdultOzoneLife ExpectancyPhysiology (medical)Environmental healthTobacco SmokingmedicineHumansReview Series from the Naples 2019 Joint Meeting of the ESC Working Groups on Myocardial Function and Cellular Biology of the HeartAged0105 earth and related environmental sciencesInfant NewbornLoss of life expectancyInfantEnvironmental ExposureOriginal ArticlesNatural emissionsEditor's ChoiceLife expectancyEnvironmental scienceParticulate MatterTobacco Smoke PollutionCardiovascular Research
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mTORC1 activation in B cells confers impairment of marginal zone microarchitecture by exaggerating cathepsin activity

2018

Mammalian target of rapamycin complex 1 (mTORC1) is a key regulator of cell metabolism and lymphocyte proliferation. It is inhibited by the tuberous sclerosis complex (TSC), a heterodimer of TSC1 and TSC2. Deletion of either gene results in robust activation of mTORC1. Mature B cells reside in the spleen at two major anatomical locations, the marginal zone (MZ) and follicles. The MZ constitutes the first line of humoral response against blood‐borne pathogens and undergoes atrophy in chronic inflammation. In previous work, we showed that mice deleted for TSC1 in their B cells (TSC1(BKO)) have almost no MZ B cells, whereas follicular B cells are minimally affected. To explore potential underl…

Lymphotoxin-beta0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesImmunologyMice TransgenicSpleenCHO CellsmTORC1Lymphocyte proliferationMechanistic Target of Rapamycin Complex 1Tuberous Sclerosis Complex 1 ProteinCathepsin BCell LineMice03 medical and health sciencesCricetulus0302 clinical medicineLymphotoxin beta ReceptorTuberous Sclerosis Complex 2 ProteinmedicineAnimalsImmunology and AllergyReceptorLymphotoxin-alphaSirolimusCathepsinB-LymphocytesChemistryOriginal ArticlesMarginal zoneCathepsinsCell biology030104 developmental biologymedicine.anatomical_structureLymphotoxinSpleen030215 immunologyImmunology
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Cigarette smoke increases Toll-like receptor 4 and modifies lipopolysaccharide-mediated responses in airway epithelial cells.

2008

Airway epithelium is emerging as a regulator of innate immune responses to a variety of insults including cigarette smoke. The main goal of this study was to explore the effects of cigarette smoke extracts (CSE) on Toll-like receptor (TLR) expression and activation in a human bronchial epithelial cell line (16-HBE). The CSE increased the expression of TLR4 and the lipopolysaccharide (LPS) binding, the nuclear factor-kappaB (NF-kappaB) activation, the release of interleukin-8 (IL-8) and the chemotactic activity toward neutrophils. It did not induce TLR2 expression or extracellular signal-regulated signal kinase 1/2 (ERK1/2) activation. The LPS increased the expression of TLR4 and induced bot…

MAPK/ERK pathwayLipopolysaccharidesLipopolysaccharideNeutrophilsImmunologyBronchiRespiratory Mucosachemistry.chemical_compoundSmokeTobaccoImmunology and AllergyHumansImmunity MucosalCell Line TransformedMitogen-Activated Protein Kinase 1Toll-like receptorMitogen-Activated Protein Kinase 3Interleukin-8NF-kappa BChemotaxisEpithelial CellsOriginal ArticlesCell biologyChemokine CXCL10Toll-Like Receptor 4TLR2Chemotaxis LeukocytechemistryImmunologyTLR4Respiratory epitheliumSignal transductionSignal TransductionImmunology
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Effect of simultaneous inhibition of TNF-α production and xanthine oxidase in experimental acute pancreatitis: The role of mitogen activated protein …

2004

Javier Pereda et al.

MAPK/ERK pathwayMalemedicine.medical_specialtyXanthine OxidaseOxypurinolPharmacologychemistry.chemical_compoundInternal medicineMedicineAnimalsEnzyme InhibitorsPentoxifyllinePhosphorylationRats WistarXanthine oxidaseProtein kinase ALungPancreasPeroxidasebiologybusiness.industryKinasePancreatitis Acute NecrotizingTumor Necrosis Factor-alphaAscitesOriginal Articlesmedicine.diseaseRatsEnzyme ActivationOxidative StressEndocrinologychemistryMitogen-activated protein kinasebiology.proteinAcute pancreatitisPancreatitisSurgeryTumor necrosis factor alphaInflammation MediatorsMitogen-Activated Protein Kinasesbusiness
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a1D-Adrenoceptors are responsible for the high sensitivity and the slow time-course of noradrenaline-mediated contraction in conductance arteries.

2013

The objective of this study was to determine whether the different time-course characteristics of α1-adrenoceptor-mediated contraction in arteries can be related to the subtypes involved. Contractile responses to noradrenaline (NA) were compared with inositol phosphate accumulation and extracellular signal-regulated kinase (ERK)1/2 phosphorylation after α1-agonist stimuli in the same vessels in the presence or absence of α1-antagonists in rat or in α1-subtype knockout (KO) mice. Aorta, where α1D-AR is the main functional subtype, had higher sensitivity to NA (in respect of inositol phosphate [IP], pERK1/2, and contractile response) than tail artery, where the α1A-adrenoceptor subtype is pre…

MAPK/ERK pathwaychemistry.chemical_classificationAgonistmedicine.medical_specialtyAortaContraction (grammar)business.industryKinasemedicine.drug_classcontraction time-courseconductance and resistance vesselsOriginal ArticlesEndocrinologyNeurologychemistryInternal medicinemedicine.arterymedicineExtracellularPhosphorylationGeneral Pharmacology Toxicology and Pharmaceuticsconductance and resistance vessels contraction time-course a1A-adrenoceptorsα1A-adrenoceptorsInositol phosphatebusiness
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Morphology and evolution of the oral shield in marsupial neonates including the newborn monito del monte (Dromiciops gliroides, Marsupialia Microbiot…

2017

International audience; Newborn marsupials can be arranged into three grades of developmental complexity based on their external form, as well as based on their organ systems and their cytology. The dasyurids are considered the least developed marsupials at birth, while didelphids and peramelids are intermediate, and macropods are the most developed. Currently there is still little information on caenolestid and microbiotherid development at birth. Developmental stages can be graded as G1, G2 and G3, with G1 being the least developed at birth, and G3 the most developed. Marsupials are also characterized by having an extremely developed craniofacial region at birth compared with placentals. …

Male0106 biological sciences0301 basic medicineHistologyZoology[ SDV.BA ] Life Sciences [q-bio]/Animal biology010603 evolutionary biology01 natural sciences03 medical and health sciencesDromiciops gliroidesnewbornAnimalsMolecular BiologyEcology Evolution Behavior and SystematicsMicrobiotheriaMarsupialMouthoral shieldbiology[SDV.BA]Life Sciences [q-bio]/Animal biologyOriginal ArticlesCell Biologymarsupialbiology.organism_classificationBiological EvolutionRhinariumAltricialstomatognathic diseasesMarsupialia030104 developmental biologyAnimals NewbornEmbryologyFemaleMonotremataAnatomyPouchmonotremeDevelopmental BiologyMonito del monte
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Natural selection mediated by seasonal time constraints increases the alignment between evolvability and developmental plasticity

2021

Abstract Phenotypic plasticity can either hinder or promote adaptation to novel environments. Recent studies that have quantified alignments between plasticity, genetic variation, and divergence propose that such alignments may reflect constraints that bias future evolutionary trajectories. Here, we emphasize that such alignments may themselves be a result of natural selection and do not necessarily indicate constraints on adaptation. We estimated developmental plasticity and broad sense genetic covariance matrices (G) among damselfly populations situated along a latitudinal gradient in Europe. Damselflies were reared at photoperiod treatments that simulated the seasonal time constraints ex…

Male0106 biological sciences0301 basic medicinelife historyOdonata01 natural sciencesG‐matrixphenotypic plasticityDivergenceEvolutionsbiologiG&#8208time constraintsdevelopmental biassopeutuminenNatural selectionluonnonvalintalatitudeAdaptation PhysiologicalBiological EvolutionOriginal ArticleFemaleSeasonsGeneral Agricultural and Biological Sciencesympäristönmuutoksetgenetic constraintsPhotoperiodevoluutioLestesBiology010603 evolutionary biology03 medical and health sciencesGeneticsAnimalsSelection GeneticAdaptationEcology Evolution Behavior and SystematicsSelection (genetic algorithm)Phenotypic plasticityEvolutionary BiologyhentosudenkorennotGenetic VariationOriginal Articlesbiology.organism_classificationmatrixEvolvability030104 developmental biologyEvolutionary biologyDevelopmental plasticityfenotyyppiAdaptation
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Telmisartan cardioprotects from the ischaemic/hypoxic damage through a miR‐1‐dependent pathway

2019

The aim of this study was to investigate whether telmisartan protects the heart from the ischaemia/reperfusion damage through a local microRNA-1 modulation. Studies on the myocardial ischaemia/reperfusion injury in vivo and on the cardiomyocyte hypoxia/reoxygenation damage in vitro were done. In vivo, male Sprague-Dawley rats administered for 3 weeks with telmisartan 12 mg/kg/d by gastric gavage underwent ischaemia/reperfusion of the left descending coronary artery. In these rats, infarct size measurement, ELISA, immunohistochemistry (IHC) and reverse transcriptase real-time polymerase chain reaction showed that expressions of connexin 43, potassium voltage-gated channel subfamily Q member …

Male0301 basic medicineCell SurvivalMyocardial InfarctionIschemiaConnexinMyocardial Reperfusion InjuryPharmacologymiR‐1telmisartanCell Lineconnexin 43Rats Sprague-Dawleyhypoxic H9c2 cells03 medical and health sciences0302 clinical medicineIn vivomedicineAnimalsBcl-2Myocytes CardiacKCNQ1ChemistryBcl‐2Original ArticlesCell BiologyTransfectionHypoxia (medical)medicine.diseasemiR-1Cell HypoxiaIn vitroRatsMicroRNAsmyocardial ischaemia/reperfusion030104 developmental biologyProto-Oncogene Proteins c-bcl-2030220 oncology & carcinogenesisKCNQ1 Potassium ChannelMolecular Medicinehypoxic H9c2 cellOriginal Articlemedicine.symptomTelmisartanReperfusion injurymedicine.drugJournal of Cellular and Molecular Medicine
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Neural stem cell lineage-specific cannabinoid type-1 receptor regulates neurogenesis and plasticity in the adult mouse hippocampus

2018

Abstract Neural stem cells (NSCs) in the adult mouse hippocampus occur in a specific neurogenic niche, where a multitude of extracellular signaling molecules converges to regulate NSC proliferation as well as fate and functional integration. However, the underlying mechanisms how NSCs react to extrinsic signals and convert them to intracellular responses still remains elusive. NSCs contain a functional endocannabinoid system, including the cannabinoid type-1 receptor (CB1). To decipher whether CB1 regulates adult neurogenesis directly or indirectly in vivo, we performed NSC-specific conditional inactivation of CB1 by using triple-transgenic mice. Here, we show that lack of CB1 in NSCs is su…

Male0301 basic medicineCell signalingCannabinoid receptorNeurogenesisCognitive NeuroscienceLong-Term PotentiationMice Transgenicmouse hippocampus ; neural stem cells ; neurogenesis-dependent behavior ; CB1 ; adult neurogenesisHippocampal formationBiologyHippocampus03 medical and health sciencesCellular and Molecular Neurosciencemouse hippocampus0302 clinical medicineNeural Stem CellsReceptor Cannabinoid CB1Animalsreproductive and urinary physiologySpatial MemoryBehavior AnimalNeurogenesisLong-term potentiationOriginal ArticlesCB1Endocannabinoid systemneurogenesis-dependent behaviorNeural stem cellCell biologyadult neurogenesisMice Inbred C57BL030104 developmental biologynervous systemlipids (amino acids peptides and proteins)biological phenomena cell phenomena and immunityStem cell030217 neurology & neurosurgeryCerebral Cortex
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