Search results for "OXY"

showing 10 items of 11443 documents

Endothelial nitric oxide synthase in vascular disease: from marvel to menace.

2006

Nitric oxide (NO·) is an important protective molecule in the vasculature, and endothelial NO· synthase (eNOS) is responsible for most of the vascular NO· produced. A functional eNOS oxidizes its substrate l -arginine to l -citrulline and NO·. This normal function of eNOS requires dimerization of the enzyme, the presence of the substrate l -arginine, and the essential cofactor (6 R )-5,6,7,8-tetrahydro- l -biopterin (BH 4 ), one of the most potent naturally occurring reducing agents. Cardiovascular risk factors such as hypertension, hypercholesterolemia, diabetes mellitus, or chronic smoking stimulate the production of reactive oxygen species in the vascular wall. Nicotinamide adenine dinu…

medicine.medical_specialtyEndotheliumNitric Oxide Synthase Type IIINitric OxideNitric oxidechemistry.chemical_compoundEnosPhysiology (medical)Internal medicinemedicineAnimalsHumansVascular Diseaseschemistry.chemical_classificationReactive oxygen speciesbiologySuperoxidebusiness.industrybiology.organism_classificationNitric oxide synthaseOxidative Stressmedicine.anatomical_structureEndocrinologychemistrybiology.proteinEndothelium VascularCardiology and Cardiovascular MedicinebusinessReactive Oxygen SpeciesNicotinamide adenine dinucleotide phosphatePeroxynitriteCirculation
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Pathophysiology, diagnosis and prognostic implications of endothelial dysfunction

2008

Endothelial dysfunction (ED) in the setting of cardiovascular risk factors such as hypercholesterolemia, hypertension, diabetes mellitus, chronic smoking as well as in patients with heart failure has been shown to be at least in part dependent on the production of reactive oxygen species (ROS) such as superoxide and the subsequent decrease in vascular bioavailability of nitric oxide (NO). Methods to quantify endothelial dysfunction include forearm plethysmography, flow-dependent dilation of the brachial artery, finger-pulse plethysmography, pulse curve analysis, and quantitative coronary angiography after intracoronary administration of the endothelium-dependent vasodilator acetylcholine. S…

medicine.medical_specialtyEndotheliumPathogenesisRisk FactorsDiabetes mellitusInternal medicinemedicineAnimalsHumansEndothelial dysfunctionClinical Trials as Topicbusiness.industrynutritional and metabolic diseasesGeneral MedicinePrognosismedicine.diseaseAscorbic acidPathophysiologyPrimary PreventionEndothelial stem cellOxidative StressEndocrinologymedicine.anatomical_structureCardiovascular DiseasesHeart failureCardiologyEndothelium VascularReactive Oxygen SpeciesbusinessBiomarkersAnnals of Medicine
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Targeting Heme-Oxidized Soluble Guanylate Cyclase

2007

Increased peripheral vascular resistance is a hallmark of advanced chronic congestive heart failure (CHF) and contributes to the phenomenon of increased afterload that complicates that condition. Multiple factors have been proposed to contribute to this phenomenon, such as increased sodium water content of the vasculature, increased activation of neurohormonal vasoconstrictor forces, and intrinsic abnormalities of the vasculature. During the past decade, it has also been shown that CHF is associated with a severe degree of endothelial dysfunction in experimental animals, as well as in humans. Given that the endothelium, as well as endothelium-dependent vasodilation, plays a crucial role in …

medicine.medical_specialtyEndotheliumSuperoxideVasodilationmedicine.diseasemedicine.disease_causechemistry.chemical_compoundEndocrinologymedicine.anatomical_structurechemistryHeart failureInternal medicineInternal MedicinemedicineVascular resistanceEndothelial dysfunctionPeroxynitriteOxidative stressHypertension
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Splenic respiratory gas exchange and glucose uptake in patients with splenomegaly in hypersplenism and Hodgkin's disease.

1977

Blood samples are taken from the splenic artery, vein and pulp of patients suffering from Hodgkin's disease (n=10) or hypersplenism (n=7) and undergoing splenectomy. In these samples, the relevant parameters of the respiratory gas exchange as well as glucose and lactate concentrations are determined. In hypersplenism (mean splenic wet weight: 543 g) the mean oxygen consumption of the splenic tissue amounts to 0.9 ml O2/100 g/min taking into account a mean splenic blood flow of 80 ml/100 g/min. The glucose uptake and the lactate release are 9 mg/100 g/min and 5.5 mg/100 g/min, respectively. These values are in close agreement with the results obtained in the normal and undisturbed spleen in …

medicine.medical_specialtyErythrocytesGlucose uptakeRespiratory gas exchangechemistry.chemical_elementSpleenSplenic arteryOxygenHypersplenismVeinsOxygen Consumptionmedicine.arteryInternal medicineDrug DiscoverymedicineHumansIn patientGenetics (clinical)business.industryGeneral MedicineVenous bloodHydrogen-Ion ConcentrationHodgkin DiseaseSurgerymedicine.anatomical_structureEndocrinologyGlucosechemistrySplenic TissueSplenomegalyLactatesMolecular MedicinebusinessSplenic ArterySpleenKlinische Wochenschrift
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Physical Fitness in Postmenopausal Women with Fibromyalgia

2007

The purpose of this study was to compare physical fitness and health-related quality-of-life between twenty-three postmenopausal women with fibromyalgia (age 58 +/- 3 years) and eleven healthy women (58 +/- 5 years). Aerobic fitness and isometric force of different muscle groups were measured. Functional performance was assessed by walking and stair-climbing times. Symptoms were assessed by visual analog scale and quality-of-life by RAND-36 questionnaire. Women with fibromyalgia had significantly lower isometric force in bilateral leg extensors (1285 vs. 1898 N), unilateral knee extensors (414 vs. 502 N) and flexors (197 vs. 235 N) than healthy women, but no differences were observed in for…

medicine.medical_specialtyFibromyalgiaVisual analogue scaleStrength trainingPhysical fitnessPhysical Therapy Sports Therapy and RehabilitationIsometric exerciseOxygen ConsumptionPhysical medicine and rehabilitationFibromyalgiamedicineHumansAerobic exerciseOrthopedics and Sports MedicineMuscle StrengthAgedElectromyographybusiness.industryVO2 maxMiddle Agedmedicine.diseaseTrunkPostmenopausePhysical FitnessExercise TestPhysical therapyFemalebusinessInternational Journal of Sports Medicine
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Effects of Concurrent Strength and Endurance Training on Physical Fitness and Symptoms in Postmenopausal Women With Fibromyalgia: A Randomized Contro…

2008

Abstract Valkeinen H, Alen M, Hakkinen A, Hannonen P, Kukkonen-Harjula K, Hakkinen K. Effects of concurrent strength and endurance training on physical fitness and symptoms in postmenopausal women with fibromyalgia: a randomized controlled trial. Objective To examine the effectiveness of concurrent strength and endurance training on muscle strength, aerobic and functional performance, and symptoms in postmenopausal women with fibromyalgia (FM). Design Randomized controlled trial. Setting Local gym and university research laboratory. Participants Twenty-six women with FM. Intervention Progressive and supervised 21-week concurrent strength and endurance training. Main Outcome Measures Muscle …

medicine.medical_specialtyFibromyalgiamedicine.medical_treatmentPhysical fitnessPhysical Therapy Sports Therapy and Rehabilitationlaw.inventionOxygen ConsumptionPhysical medicine and rehabilitationRandomized controlled triallawEndurance trainingSurveys and QuestionnairesFibromyalgiamedicineHumansMuscle StrengthAnalysis of VarianceRehabilitationbusiness.industryRehabilitationVO2 maxMiddle Agedmedicine.diseaseTrunkExercise TherapyPostmenopauseTreatment OutcomePhysical FitnessPhysical EndurancePhysical therapyFemaleAnalysis of variancebusinesshuman activitiesArchives of Physical Medicine and Rehabilitation
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Prooxidative toxicity and selenoprotein suppression by cerivastatin in muscle cells

2012

Statins are the most widely used drugs for the treatment of hypercholesterolemia. In spite of their overall favorable safety profile, they do possess serious myotoxic potential, whose molecular origin has remained equivocal. Here, we demonstrate in cultivated myoblasts and skeletal muscle cells that cerivastatin at nanomolar concentrations interferes with selenoprotein synthesis and evokes a heightened vulnerability of the cells toward oxidative stressors. A correspondingly increased vulnerability was found with atorvastatin, albeit at higher concentrations than with cerivastatin. In selenium-saturated cells, cerivastatin caused a largely indiscriminate suppression of selenoprotein biosynth…

medicine.medical_specialtyGPX1Cell SurvivalPyridinesMevalonic AcidMevalonic acidBiologyToxicologyCell LineMyoblastsMiceSeleniumchemistry.chemical_compoundInternal medicineAtorvastatinmedicineAnimalsMyocytePyrrolesSelenoproteinseducationchemistry.chemical_classificationeducation.field_of_studySelenoprotein NEbselenSkeletal muscleCerivastatinHydrogen PeroxideGeneral MedicineRatsOxidative StressEndocrinologymedicine.anatomical_structureGene Expression RegulationchemistryHeptanoic AcidsSelenoproteinHydroxymethylglutaryl-CoA Reductase Inhibitorsmedicine.drugToxicology Letters
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Statin-Induced Liver Injury Involves Cross-Talk between Cholesterol and Selenoprotein Biosynthetic Pathways

2009

Statins have become the mainstay of hypercholesterolemia treatment. Despite a seemingly clear rationale behind their use, the inhibition of HMG-CoA reductase, these compounds have been shown to elicit a variety of unanticipated and elusive effects and side effects in vivo. Among the most frequently noted side effects of statin treatment are elevations in liver enzymes. Here, we report our finding that atorvastatin, cerivastatin, and lovastatin at clinically common concentrations induce a selective, differential loss of selenoprotein expression in cultured human HepG2 hepatocytes. The primarily affected selenoprotein was glutathione peroxidase (GPx), whose biosynthesis, steady-state expressi…

medicine.medical_specialtyGPX1Thioredoxin-Disulfide ReductaseStatinPyridinesmedicine.drug_classAtorvastatinBiologyGPX4tert-ButylhydroperoxideCell Line TumorInternal medicineAtorvastatinmedicineHumansPyrrolesLovastatinSelenoproteinsPharmacologychemistry.chemical_classificationGlutathione Peroxidaseintegumentary systemCytotoxinsGlutathione peroxidaseCerivastatinIsoenzymesCholesterolEndocrinologychemistryHeptanoic AcidsHepatocytesMolecular MedicineLovastatinSelenoproteinHydroxymethylglutaryl-CoA Reductase InhibitorsReactive Oxygen SpeciesSignal Transductionmedicine.drugMolecular Pharmacology
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Genotype-driven pharmacokinetic simulations of warfarin levels in Puerto Ricans.

2020

Abstract Objectives The inter-individual variability of warfarin dosing has been linked to genetic polymorphisms. This study was aimed at performing genotype-driven pharmacokinetic (PK) simulations to predict warfarin levels in Puerto Ricans. Methods Analysis of each individual dataset was performed by one-compartmental modeling using WinNonlin®v6.4. The k e of warfarin given a cytochrome P450 2C9 (CYP2C9) genotype ranged from 0.0189 to 0.0075 h−1. K a and V d parameters were taken from literature. Data from 128 subjects were divided into two groups (i.e., wild-types and carriers) and statistical analyses of PK parameters were performed by unpaired t-tests. Results In the carrier group (n=6…

medicine.medical_specialtyGenotypeCmax030204 cardiovascular system & hematology030226 pharmacology & pharmacyGastroenterologyArticle03 medical and health sciences0302 clinical medicinePharmacokineticsVitamin K Epoxide ReductasesInternal medicineGenotypemedicineHumansPharmacology (medical)DosingGeneral Pharmacology Toxicology and PharmaceuticsCYP2C9GenotypingCytochrome P-450 CYP2C9business.industryWarfarinArea under the curveFarmàcia InvestigacióAnticoagulantsHispanic or LatinoAryl Hydrocarbon HydroxylasesWarfarinbusinessmedicine.drugDrug metabolism and personalized therapy
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How registers could enhance knowledge and characterization of genetic dyslipidaemias: The experience of the LIPIGEN in Italy and of other networks fo…

2020

Familial hypercholesterolemia (FH) is a common genetic disorder of lipid metabolism, still underdiagnosed and undertreated in the general population. Pathology registers could play a crucial role in the creation of a comprehensive and integrated global approach to cover all aspects of this disease. Systematic data collection of patients affected by FH has increased dramatically worldwide in the past few years. Moreover, results from registers already established for the longest time showed their potentialities in the implementation of the knowledge of FH, comparing country-specific approaches and providing real-world data about identification, management and treatment of FH individuals in t…

medicine.medical_specialtyGenotypeFamilial hypercholesterolemiaPopulationFamilial hypercholesterolemiaDisease030204 cardiovascular system & hematologyHyperlipoproteinemia Type II03 medical and health sciences0302 clinical medicineGenetic dyslipidaemiasFamilial hypercholesterolemia; Genetic dyslipidaemias; Pathology registersInternal MedicineHumansMedicineGenetic Predisposition to DiseaseRegistries030212 general & internal medicineeducationIntensive care medicineHypolipidemic Agentseducation.field_of_studybusiness.industryGenetic disorderDiagnostic algorithmsGeneral MedicinePathology registersmedicine.diseaseClinical PracticePhenotypeItalyCardiovascular DiseasesHeart Disease Risk FactorsDisease riskIdentification (biology)Hydroxymethylglutaryl-CoA Reductase InhibitorsCardiology and Cardiovascular MedicinebusinessAlgorithmsAtherosclerosis Supplements
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