Search results for "Offspring"

showing 10 items of 395 documents

Mother knows best: dominant females determine offspring dispersal in red foxes (Vulpes vulpes).

2011

Background: Relatedness between group members is central to understanding the causes of animal dispersal. In many group-living mammals this can be complicated as extra-pair copulations result in offspring having varying levels of relatedness to the dominant animals, leading to a potential conflict between male and female dominants over offspring dispersal strategies. To avoid resource competition and inbreeding, dominant males might be expected to evict unrelated males and related females, whereas the reverse strategy would be expected for dominant females. Methodology/Principal Findings: We used microsatellites and long-term data from an urban fox (Vulpes vulpes) population to compare disp…

MaleVulpesOffspringmedia_common.quotation_subjectPopulationZoologyFoxesMotherslcsh:MedicineCompetition (biology)Conflict PsychologicalFathersBehavioral EcologyInbreeding avoidanceAnimalsUrban Ecologyeducationlcsh:ScienceBiologymedia_commoneducation.field_of_studyEvolutionary BiologyMultidisciplinarybiologyEcologyAnimal BehaviorEcologyC182 Evolutionlcsh:Rbiology.organism_classificationSocial DominanceEvolutionary EcologyD300 Animal Scienceta1181Biological dispersalPhilopatryFemalelcsh:QInbreedingMicrosatellite RepeatsResearch ArticlePLoS ONE
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A multi-generational study on low-dose BPA exposure in Wistar rats: Effects on maternal behavior, flavor intake and development

2012

Bisphenol A (BPA) is a common endocrine disruptor found as an environmental and food contaminant. It exerts both developmental and behavioral effects, mainly when exposure occurs in early life. The aim of this study was to determine the multi-generational effects of chronic, human-relevant low-dose exposure to BPA on development, maternal behavior and flavor preference in Wistar rats. BPA was orally administered at a daily dose of 5 mu g/kg body weight to FO pregnant dams from the first day of gestation (GD 1) until the last day of lactation (LD 21), and then to Fl offspring from weaning (PND 21) to adulthood (PND 100). F2 offspring were not exposed. Development and clinical signs of toxici…

Male[ SDV.AEN ] Life Sciences [q-bio]/Food and Nutrition[ SDV.TOX ] Life Sciences [q-bio]/ToxicologyToxicology[ SDV.BA ] Life Sciences [q-bio]/Animal biologyEatingPregnancyLactationBirth RateMaternal BehaviorPerinatal ExposureChemistryTaste preferencesBISPHENOL-A EXPOSURE[SDV.BA]Life Sciences [q-bio]/Animal biologyAnogenital distanceAge FactorsDIETARY EXPOSUREmedicine.anatomical_structureEndocrine disruptorEndocrine disruptorPrenatal Exposure Delayed EffectsENVIRONMENTALLY RELEVANT LEVELS[SDV.TOX]Life Sciences [q-bio]/ToxicologyToxicityMalformationsFemaleCD-1 MICEReproductive toxicityPERINATAL EXPOSUREmedicine.medical_specialtyendocrine systemSEX-DIFFERENCESOffspringGestational AgeAir Pollutants OccupationalREPRODUCTIVE TOXICITYSEXUALLY DIMORPHIC BEHAVIORSFood PreferencesCellular and Molecular NeurosciencePhenolsDevelopmental NeuroscienceInternal medicinemedicineAnimalsWeaningSex RatioBenzhydryl CompoundsRats WistarSPRAGUE-DAWLEY RATSOFFSPRING TOXICITYBody WeightRatsFlavoring AgentsEndocrinologyAnimals NewbornF2 body weight change[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Prenatal cocaine alters later responses to morphine in adult male mice.

2006

Mice prenatally exposed to cocaine (25 mg/kg), physiological saline or non-treated during the last 6 days of pregnancy were evaluated as adults for the rewarding properties of 2 mg/kg of morphine, using the conditioned place preference (CPP) procedure. Likewise, isolated animals underwent a social interaction test with conspecifics after receiving the same morphine dose. Unlike control or animals pre-treated with saline, subjects prenatally treated with cocaine did not develop CPP with this dose of morphine. Only cocaine-exposed animals showed increased threat, avoidance and fleeing during the social encounter. No differences in motor effects of morphine were observed. Analysis of monoamine…

Malemedicine.medical_specialtyBiogenic AminesOffspringmedicine.medical_treatmentPharmacologyMotor ActivityMiceCocaineRewardPregnancyInternal medicinemedicineAnimalsInterpersonal RelationsSalineBiological PsychiatryPharmacologyBrain ChemistryPregnancyBehavior AnimalMorphinePrenatal cocaine exposuremedicine.diseaseConditioned place preferenceAggressionAnalgesics OpioidMonoamine neurotransmitterEndocrinologyPrenatal Exposure Delayed EffectsToxicityMorphineExploratory BehaviorConditioning OperantFemalePsychologymedicine.drugProgress in neuro-psychopharmacologybiological psychiatry
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Prenatal and neonatal risk factors for the development of enamel defects in low birth weight children

2009

Oral Diseases (2010) 16, 257–262 Objective:  To analyse the influence of several prenatal and neonatal risk factors in the development of enamel defects in low birth weight children. Subjects and methods:  Children between 4 and 5 years of age (n = 102) were classified into: Group 1) 52 low birth weight (<2500 g); Group 2) 50 normal birth weight (≥2500 g). Medical history, prenatal and neonatal variables were collected. Enamel defects were evaluated with the modified Developmental Defects of Enamel Index. Results:  The prevalence of hypoplasia and average number of affected teeth were significantly higher in group 1 than in group 2 (59.6%vs 16% and 1.6 vs 0.3 respectively). Low gestational …

Malemedicine.medical_specialtyBirth weightGestational AgeMultiple Birth OffspringPregnancyRisk FactorsIntubation IntratrachealmedicineHumansGeneral DentistryPregnancyEnamel paintCesarean SectionObstetricsbusiness.industryInfant NewbornGestational ageInfant Low Birth WeightEnamel hypoplasiamedicine.diseaseLow birth weightOtorhinolaryngologyCase-Control StudiesChild PreschoolPrenatal Exposure Delayed Effectsvisual_artApgar Scorevisual_art.visual_art_mediumDental Enamel HypoplasiaFemaleApgar scoreMultiple birthmedicine.symptombusinessMaternal AgeOral Diseases
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Maternal Treatment of Spontaneously Hypertensive Rats With Pentaerythritol Tetranitrate Reduces Blood Pressure in Female Offspring

2014

Pentaerythritol tetranitrate is devoid of nitrate tolerance and shows no reproductive or developmental toxicity in animal studies. Recently, pentaerythritol tetranitrate has been demonstrated to reduce the risk of intrauterine growth restriction and the risk of preterm birth in women with abnormal placental perfusion. This study was conducted to test the perinatal programming effect of pentaerythritol tetranitrate in spontaneously hypertensive rats, a rat model of genetic hypertension. Parental spontaneously hypertensive rats were treated with pentaerythritol tetranitrate (50 mg/kg per day) during pregnancy and lactation periods; the offspring received standard chow without pentaerythritol …

Malemedicine.medical_specialtyGPX1Nitric Oxide Synthase Type IIIOffspringVasodilator AgentsDevelopmental toxicityBlood PressureVasodilationPentaerythritol tetranitratePentaerythritolchemistry.chemical_compoundPregnancyRats Inbred SHRInternal medicineInternal MedicinemedicineAnimalsPentaerythritol Tetranitratebusiness.industryGene Expression Regulation DevelopmentalDNARatsVasodilationHeme oxygenaseEndocrinologyBlood pressureAnimals NewbornchemistryMaternal ExposureHypertensionPregnancy AnimalFemaleEndothelium VascularbusinessHypertension
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Murine tissue factor disulfide mutation causes a bleeding phenotype with sex specific organ pathology and lethality.

2019

Tissue factor is highly expressed in sub-endothelial tissue. The extracellular allosteric disulfide bond Cys186-Cys209 of human tissue factor shows high evolutionary conservation and in vitro evidence suggests that it significantly contributes to tissue factor procoagulant activity. To investigate the role of this allosteric disulfide bond in vivo, we generated a C213G mutant tissue factor mouse by replacing Cys213 of the corresponding disulfide Cys190-Cys213 in murine tissue factor. A bleeding phenotype was prominent in homozygous C213G tissue factor mice. Pre-natal lethality of 1/3rd of homozygous offspring was observed between E9.5 and E14.5 associated with placental hemorrhages. After b…

Malemedicine.medical_specialtyOffspring610 Medicine & healthHemorrhage030204 cardiovascular system & hematologyBiologymedicine.disease_causeArticleThromboplastin11459 Center for Molecular Cardiology03 medical and health sciencesTissue factorArterial Thrombosis; Blood Coagulation and Fibrinolysis; Disorders of Coagulation and FibrinolysisMice0302 clinical medicineIn vivoPregnancyInternal medicinemedicineExtracellularAnimalsDisulfidesMutationHematologyPhenotypeIn vitroEndocrinologyPhenotype10036 Medical Clinic10076 Center for Integrative Human PhysiologyHemostasisMutation10209 Clinic for CardiologyFemale030215 immunologyHaematologica
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N-3 fatty acids modulate Th1 and Th2 dichotomy in diabetic pregnancy and macrosomia.

2006

We assessed the implication of Th (helper)-cells and the modulation of the Th1/Th2 dichotomy by n-3 polyunsaturated fatty acids (PUFA) in type I diabetic pregnancy (DP) and macrosomia. Female gestant rats fed a standard diet or n-3 PUFA regimen were rendered diabetic by administration of five low doses of streptozotocin. The macrosomic (MAC) offspring were sacrificed at the age of 90 days. The mRNAs of IL-2 and IFN-gamma (Th1 cytokines) and IL-4 (Th2 cytokine) were downregulated in the pancreas and spleen of diabetic pregnant rats. The levels of IL-10 mRNA, another Th2 cytokine, were unchanged in the spleen or upregulated in the pancreas of these animals. Feeding an n-3 PUFA diet to rats wi…

Malemedicine.medical_specialtyOffspringImmunologyPregnancy in DiabeticsSpleenBiologyFetal MacrosomiaTh2 CellsDownregulation and upregulationPregnancyInternal medicineDiabetes mellitusFatty Acids Omega-3medicineImmunology and AllergyAnimalsRNA MessengerRats Wistarchemistry.chemical_classificationPregnancyFatty AcidsTh1 CellsStreptozotocinmedicine.diseaseDietRatsmedicine.anatomical_structureEndocrinologyCholesterolDiabetes Mellitus Type 1chemistryCytokinesFemalePancreasSpleenmedicine.drugPolyunsaturated fatty acidJournal of autoimmunity
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Post-weaning xenohormone intake affects adult rat submandibular gland in a sex-dependent manner

2018

International audience; OBJECTIVES: We previously reported that maternal exposure to genistein, vinclozolin, ingested alone or in combination, affects submandibular salivary glands of rats' offspring's. Here, we investigated the responsiveness of submandibular gland when such xenohormone exposure occurs later in life. MATERIALS AND METHODS: Chemicals were given orally to male and female Wistar rats (1mg/kg body weight per day), from weaning to adulthood. Submandibular glands and plasma were collected at postnatal day 100 for histological and molecular analysis. RESULTS: Whereas no effect was observed in females, increases in granular convoluted tubules area coupled with a modification of sa…

Malemedicine.medical_specialtyOffspringSubmandibular GlandPhytoestrogenssalivary glandWeaning03 medical and health scienceschemistry.chemical_compound0302 clinical medicineSex Factorsstomatognathic systemInternal medicineProgesterone receptormedicineWeaningAnimalsVinclozolinRNA MessengerCystatin CGeneral DentistryOxazolesbiologySalivary glandbusiness.industryMucinsex-dimorphismAndrogen Antagonists030206 dentistryTransforming Growth Factor alphaSubmandibular glandGenisteinRatsEndocrinologymedicine.anatomical_structureendocrine disruptorsepidermal growth factorOtorhinolaryngologyCystatin CchemistryReceptors Androgen030220 oncology & carcinogenesisbiology.proteinFemalebusiness[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
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Perinatal xenohormone exposure impacts sweet preference and submandibular development in male rats.

2013

Objective To determine the effect of perinatal exposure to low doses of genistein and/or vinclozolin on submandibular salivary gland (SSG) development in juvenile and adult male rats and to establish a link with sweet preference. Material and Methods Female rats received orally (1 mg kg−1 body weight/day) genistein and vinclozolin, alone or in combination, from the first gestational day up to weaning. Sweet preference was assessed at weaning and in adulthood in male offspring; submandibular glands were then collected to study the morphogenesis and mRNA expression of steroid receptors, growth factors and taste related proteins. Results Exposure to genistein and/or vinclozolin resulted in a h…

Malemedicine.medical_specialtyOffspringsalivary glandsSubmandibular Glandendocrine disruptor mixtureGenisteinPhytoestrogensBiology03 medical and health scienceschemistry.chemical_compoundFood Preferences0302 clinical medicineFetusSaccharinstomatognathic systemInternal medicineProgesterone receptormedicineWeaningEndocrine systemAnimalsVinclozolinRats WistarGeneral DentistryOxazoles030304 developmental biology0303 health sciencesPerinatal Exposuregrowth factorAndrogen AntagonistsSex hormone receptorGenisteinRatsEndocrinologyOtorhinolaryngologychemistryAnimals NewbornTastephytoestrogen[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryOral diseases
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Single, intense prenatal stress decreases emotionality and enhances learning performance in the adolescent rat offspring: Interaction with a brief, d…

2006

Perinatal manipulations can lead to neurobehavioural changes in the progeny. In this study we investigated, in adolescent male rat offspring, the consequences of a single, intense prenatal stress induced by a 120 min-maternal immobilization at gestational day 16, and of a daily, brief maternal separation from postnatal day 2 until 21, on: unconditioned fear/anxiety-like behaviour in open field and in elevated plus-maze; learning performance in the "Can test", a non-aversive spatial and tactile/visual task; corticosterone plasma levels under basal and stress-induced conditions. Our results indicate that both prenatal stress and maternal separation procedures decrease emotionality and enhance…

Malemedicine.medical_specialtySettore BIO/14 - FARMACOLOGIAOffspringPrenatal stress Maternal separation Adolescent ra tBehaviour CorticosteroneAnxietyStatistics NonparametricOpen fieldDiscrimination LearningRandom AllocationBehavioral Neurosciencechemistry.chemical_compoundPregnancyEmotionalityCorticosteroneInternal medicineAdaptation PsychologicalmedicineAnimalslearning performanceRats WistarAnalysis of VarianceMaternal deprivationPregnancyMaternal DeprivationAge Factorsmedicine.diseaseRatsbehaviourEndocrinologyPrenatal stresschemistryprenatal stressPrenatal Exposure Delayed EffectsExploratory BehaviorFemaleAnalysis of varianceCorticosteronePsychologyStress Psychological
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