Search results for "Oncogene"

showing 10 items of 1005 documents

Effectiveness of Toki s criteria and determination of variables for identification of HPV L1 protein in oral lesions

2013

Objectives: To evaluate the effectiveness of Toki's criteria in identifying the HPV L1 protein in oral lesions with the use of immunohistochemistry (IHC) and to determine which criteria optimize such identification. Study Design: Retrospective study of 277 cases diagnosed as HPV lesions at 22 years. Tests of sensitivity, specificity, positive predictive value (PPV), negative predictive value (NPV), kappa coefficients, and chi 2 values, as well as two logistic regression analyses ( (p≤0.05), were conducted. Results: Of the lesions studied, 96.4% (267 of 277) were positive for HPV using Toki's criteria and 28.5% (79 of 277) were positive for L1 by IHC. Toki's criteria showed sensitivity=93.67…

AdultMalePathologymedicine.medical_specialtyAdolescentConcordanceOdontologíaPapillomatosisLogistic regressionSensitivity and SpecificityGastroenterologyYoung AdultInternal medicinemedicineHumansYoung adultChildGeneral DentistryAgedRetrospective StudiesAged 80 and overMouth neoplasmOral Medicine and Pathologybusiness.industryResearchPapillomavirus InfectionsMouth MucosaInfantRetrospective cohort studyRegression analysisOncogene Proteins ViralMiddle Aged:CIENCIAS MÉDICAS [UNESCO]Ciencias de la saludImmunohistochemistryOtorhinolaryngologyChild PreschoolUNESCO::CIENCIAS MÉDICASCapsid ProteinsFemaleMouth NeoplasmsSurgerymedicine.symptombusinessKappaMedicina Oral Patología Oral y Cirugia Bucal
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Oncogene overexpression in non-small-cell lung cancer tissue: prevalence and clinicopathological significance.

1994

In contrast to small-cell lung cancer, few data are available on the role of oncogene overexpression in non-small-cell lung cancers (NSCLC). To determine the prevalence and extent of the transcriptional activation of cancer genes in NSCLC we investigated the level of mRNA of the three important cellular oncogenes — erbB2, Ki-ras, and c-myc — in 39 surgically or endoscopically obtained tumor samples and 24 samples of normal bronchopulmonary tissue taken from the same patients. Tissue RNA was prepared and the specific mRNA analyzed by the highly sensitive nuclease S1 protection assay. Oncogene mRNA in the tumors was quantified by comparison with the homogeneously weak signals in normal lung t…

AdultMalePathologymedicine.medical_specialtyLung NeoplasmsAdolescentBiologyCarcinoma Non-Small-Cell LungDrug DiscoveryGene expressionmedicineCarcinomaHumansLung cancerGenetics (clinical)AgedAged 80 and overMessenger RNAOncogeneCancerOncogenesMiddle Agedmedicine.diseaseMolecular medicineGene Expression Regulation NeoplasticCancer researchMolecular MedicineAdenocarcinomaFemaleThe Clinical investigator
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DNA-fragmentation and apoptosis-related proteins of muscle cells in motor neuron disorders

2009

Apoptosis has been described as one of the mechanisms of muscle fiber loss in infantile spinal muscular atrophy. In order to investigate if muscle fiber-apoptosis plays a role in other denervating disorders as well, we studied DNA-fragmentation, a hallmark of apoptosis, by the TUNEL-method and, moreover, the expression patterns of apoptosis-related proteins in 2 patients suffering from ALS and in 6 patients with polyneuropathy. We identified DNA-cleavage in muscle fibers of all these patients. Furthermore, we found strong expression of bax and ICE promoting apoptosis in muscle fibers. However, also strong expression of the anti-apoptotic factor bcl-2 was found. Our findings indicate that de…

AdultMalePathologymedicine.medical_specialtyMuscle Fibers SkeletalApoptosisCell Cycle ProteinsDNA FragmentationBiologyProto-Oncogene ProteinsGene expressionmedicineHumansMyocytefas ReceptorMotor Neuron DiseaseAmyotrophic lateral sclerosisMuscle SkeletalActinAgedReceptors Leukocyte-AdhesionAmyotrophic Lateral SclerosisPeripheral Nervous System DiseasesGeneral MedicineMiddle AgedMotor neuronmedicine.diseaseCell biologyCysteine Endopeptidasesmedicine.anatomical_structureNeurologyApoptosisNerve DegenerationDNA fragmentationFemaleNeurology (clinical)AtrophyPolyneuropathyActa Neurologica Scandinavica
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Atypical Pleomorphic Extraosseous Ewing Tumor/Peripheral Primitive Neuroectodermal Tumor with Unusual Phenotypic/Genotypic Profile

2002

A pleomorphic undifferentiated tumor primarily located in the retroperitoneum with a phenotype compatible with an extraosseous Ewing tumor/peripheral primitive neuroectodermal tumor (ET/pPNET) pattern and unusual molecular features is described. Immunohistochemically, HBA-71 (CD99/mic2) and several neural markers were intensively expressed together with scattered cells expressing carcinoembryonic antigen (CEA). Short-term culture showed biphasic neuroblastic and epithelioid cell populations, with the latter expressing germ cell markers (CEA, alpha-fetoprotein, and the beta-subunit of chorionic gonadotrophin). Conventional cytogenetics displayed several chromosomic rearrangements, especially…

AdultMalePathologymedicine.medical_specialtyOncogene Proteins FusionChromosomes Human Pair 22CD99Soft Tissue NeoplasmsChromosomal translocationSarcoma EwingBiologyTranslocation GeneticPathology and Forensic MedicineExonFatal OutcomeCarcinoembryonic antigenBiomarkers TumorTumor Cells CulturedmedicineHumansNeuroectodermal Tumors PrimitiveRetroperitoneal NeoplasmsMolecular BiologyGene Rearrangementmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionPeripheral Primitive Neuroectodermal TumorChromosomes Human Pair 11Neoplasms Second PrimaryDNA NeoplasmCell BiologyGenes p53Chromosome Bandingmedicine.anatomical_structureKaryotypingMutationbiology.proteinEpithelioid cellGerm cellFluorescence in situ hybridizationDiagnostic Molecular Pathology
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A poorly differentiated synovial sarcoma (SYT/SSX1) expresses neuroectodermal markers: a xenografts and in vitro culture study.

2004

Synovial sarcoma (SS) is a neoplasm that poses diagnostic problems, due to its histologic heterogeneity. The poorly differentiated variant, in particular, may be histologically indistinguishable from other small round cell tumors. Detection of the synovial sarcoma-associated t(X;18) or SYT-SSX fusion transcripts may be necessary to confirm the diagnosis of SS in difficult cases. Most of SS carry a t(X;18) in about one third of cases as the sole cytogenetic abnormality. We evaluated a case of poorly differentiated synovial sarcoma and their derived tumors in nude mice xenografts and cell cultures. We used a panel of antibodies (including those to intermediate filament, nerve-sheath associate…

AdultMalePathologymedicine.medical_specialtyOncogene Proteins FusionMice NudeNeuroectodermal TumorsPolymerase Chain ReactionPathology and Forensic MedicineDiagnosis DifferentialMiceSarcoma SynovialImmunophenotypingAntigenmedicineTumor Cells CulturedNeoplasmAnimalsHumansIntermediate filamentMolecular BiologyIn Situ HybridizationbiologyImmunochemistryCell DifferentiationCell Biologymedicine.diseaseXenograft Model Antitumor AssaysSynovial sarcomaIn vitroCell cultureKaryotypingbiology.proteinButtocksAntibodyBiomarkersDiagnostic molecular pathology : the American journal of surgical pathology, part B
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Ewing-like sarcoma with CIC-DUX4 gene fusion in a patient with neurofibromatosis type 1. A hitherto unreported association.

2015

Sarcoma with CIC-DUX4 gene fusion is emerging as the most prevalent subset of Ewing-like undifferentiated small round cell sarcomas with around 50 cases published. We report hereby the case of a 40-year-old male who presented a CIC-DUX4 sarcoma in deep soft tissues in his thigh. He had been diagnosed with neurofibromatosis type 1 at age 19 and over the years underwent resection of multiple neural neoplasms, including two malignant peripheral nerve sheath tumors with classical spindle-cell histopathology. The CIC-DUX4 sarcoma was treated with surgical resection, radiation and chemotherapy, but lung and brain metastases developed and the patient died from the disease 14 months after diagnosis…

AdultMalePathologymedicine.medical_specialtySoft Tissue NeoplasmLung NeoplasmsNeurofibromatosis 1Oncogene Proteins Fusionmedicine.medical_treatmentSoft Tissue NeoplasmsThighBiologyPathology and Forensic MedicineFusion geneFatal OutcomemedicineHumansNeurofibromatosisChemotherapyBrain NeoplasmsSoft tissueCell Biologymedicine.diseasemedicine.anatomical_structureSarcoma Small CellHistopathologySarcomaPathology, research and practice
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Evaluation of prognostic factors and their capacity to predict biological behavior in gastrointestinal stromal tumors.

2011

Gastrointestinal stromal tumors (GISTs) are c-KIT-signaling-driven mesenchymal tumors of the human digestive tract, many of which have c-KIT or PDGFRα activating mutations. The authors studied the immunohistochemical markers, c-KIT and PDGFRα mutations, in GISTs and their association with the clinicopathological and clinical follow-up in 145 GISTs. Tumors were located mainly in the stomach, the median tumor size being 7.5 cm. The mitotic index was ≤5 mitoses per 50 high-power fields in 61% of cases, 96% expressed CD117, and c-KIT or PDGFRα mutations were detected in 68% of cases. The median follow-up of the series was 52 months (range = 1 to 244.9 months). Tumor size, cell morphology, mito…

AdultMalePathologymedicine.medical_specialtyStromal cellMitotic indexReceptor Platelet-Derived Growth Factor alphaGastrointestinal Stromal Tumorsmedicine.disease_causeCell morphologyDisease-Free SurvivalPathology and Forensic MedicineYoung AdultPredictive Value of TestsStomach NeoplasmsIntestinal NeoplasmsmedicineBiomarkers TumorMitotic IndexHumansAgedAged 80 and overMutationbiologyCD117StomachMesenchymal stem cellMiddle AgedPrognosisProto-Oncogene Proteins c-kitmedicine.anatomical_structureKi-67 AntigenMutationbiology.proteinImmunohistochemistrySurgeryFemaleAnatomyInternational journal of surgical pathology
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P53 and bcl-2 immunoexpression in patients with oral lichen planus a nd oral squamous cell carcinoma

2012

Objective: The aim of this study was to determine by immunohistochemistry the presence and significance of p53 and bcl-2 proteins in oral lichen planus (OLP) and oral squamous cell carcinoma (OSCC). Study Design: We used 21 cases diagnosed as OLP 16 diagnosed as OSCC and four normal gingival biopsies taken from healthy patients were used as controls. Slides were processed for immunohistochemistry using anti-p53 and anti-bcl-2 monoclonal antibodies. Results: We found p53 immunoexpression in 71.4� OLP cases and 6� .7� OSCC cases, with no immunoexpres: We found p53 immunoexpression in 71.4� OLP cases and 6� .7� OSCC cases, with no immunoexpresWe found p53 immunoexpression in 71.4� OLP cases an…

AdultMalePathologymedicine.medical_specialtymedicine.drug_classMonoclonal antibodyMalignancyYoung Adultstomatognathic systemmedicineCarcinomaHumansIn patientBasal cellGeneral DentistryAgedMouth neoplasmOral Medicine and Pathologybusiness.industryMiddle Agedmedicine.disease:CIENCIAS MÉDICAS [UNESCO]stomatognathic diseasesProto-Oncogene Proteins c-bcl-2OtorhinolaryngologyUNESCO::CIENCIAS MÉDICASCarcinoma Squamous CellImmunohistochemistryResearch-ArticleFemaleMouth NeoplasmsSurgeryOral lichen planusTumor Suppressor Protein p53businessLichen Planus Oral
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Apoptosis resistance in epithelial tumors is mediated by tumor-cell-derived interleukin-4

2008

We investigated the mechanisms involved in the resistance to cell death observed in epithelial cancers. Here, we identify that primary epithelial cancer cells from colon, breast and lung carcinomas express high levels of the antiapoptotic proteins PED, cFLIP, Bcl-xL and Bcl-2. These cancer cells produced interleukin-4 (IL-4), which amplified the expression levels of these antiapoptotic proteins and prevented cell death induced upon exposure to TRAIL or other drug agents. IL-4 blockade resulted in a significant decrease in the growth rate of epithelial cancer cells and sensitized them, both in vitro and in vivo, to apoptosis induction by TRAIL and chemotherapy via downregulation of the antia…

AdultMaleProgrammed cell deathLung NeoplasmsTime Factorsapoptosis interleukin-4 cancer stem cells cancer chemiotherapy cytokinesCASP8 and FADD-Like Apoptosis Regulating Proteinbcl-X ProteinAntineoplastic AgentsApoptosisBreast NeoplasmsBiologyTNF-Related Apoptosis-Inducing LigandTumor Cells CulturedmedicineHumansAutocrine signallingMolecular BiologyInterleukin 4AgedCell ProliferationSettore MED/04 - Patologia GeneraleCell DeathDose-Response Relationship DrugCell growthCarcinomaIntracellular Signaling Peptides and ProteinsAntibodies MonoclonalInterleukin-4 Receptor alpha SubunitCorrectionCancerCell BiologyMiddle AgedPhosphoproteinsmedicine.diseaseUp-RegulationCell biologyAutocrine CommunicationProto-Oncogene Proteins c-bcl-2Drug Resistance NeoplasmApoptosisColonic NeoplasmsCancer cellFemaleInterleukin-4Interleukin-4 Cancer stem cellsSignal transductionApoptosis Regulatory ProteinsSignal Transduction
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Apoptosis-related Proteins in Skeletal Muscle Fibers of Spinal Muscular Atrophy

1997

There is evidence that apoptosis in spinal muscular atrophies (SMA) is not restricted to motor neurons but also affects muscle fibers. Studying the expression of several apoptosis-associated proteins we found constant expression of bax in muscle fibers, which promoted cell death. The expression of bax correlated with defective innervation of muscle fibers was also indicated by upregulation of N-CAM. While in early-onset SMA atrophic as well as normo- and hypertrophic muscle fibers displayed expression of bax, muscle fibers in late-onset SMA and peripheral neuropathies showed bax-expression only in atrophic fibers. Other investigated apoptosis-associated factors comprised interleukin-1 beta …

AdultMaleProgrammed cell deathPathologymedicine.medical_specialtyMuscle Fibers Skeletalbcl-X ProteinMuscle ProteinsApoptosisBiologyMicrofilamentPathology and Forensic MedicineMuscular Atrophy SpinalCellular and Molecular NeuroscienceReference ValuesProto-Oncogene ProteinsmedicineHumansMyocyteMuscle SkeletalActinAgedbcl-2-Associated X ProteinCaspase 1InfantPeripheral Nervous System DiseasesGeneral MedicineSpinal muscular atrophyMiddle AgedSMA*Spinal muscular atrophiesmedicine.diseaseCell biologyCysteine EndopeptidasesProto-Oncogene Proteins c-bcl-2NeurologyFemaleNeural cell adhesion moleculeNeurology (clinical)Journal of Neuropathology and Experimental Neurology
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