Search results for "Organ Specificity"

showing 10 items of 141 documents

Rat CNS cell culture. Enhancement of neuronal survival and delay of glial proliferation by serum from patients with multiple sclerosis. A morphologic…

1984

The addition of serum from multiple sclerosis (MS) patients to the culture medium of dissociated cells from cerebral hemispheres of rat embryos caused a delay in glial proliferation and an enhancement of neuronal survival. Sera from normal individuals and patients with other neurological diseases failed to show this effect. These morphological observations are interpreted as the outcome of inhibition of in vitro gliogenesis.

Central Nervous Systemmedicine.medical_specialtyPathologyNeurologyMultiple SclerosisDermatologyBiologyGliotoxinmedicineAnimalsCells CulturedGliogenesisNeuronsGeneral NeuroscienceMultiple sclerosisEmbryoCell DifferentiationGeneral MedicineMycotoxinsmedicine.diseaseEmbryo MammalianIn vitroRatsPsychiatry and Mental healthCell cultureOrgan SpecificityImmunologyNeurology (clinical)NeurogliaItalian journal of neurological sciences
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The angiopoietin-Tie2 pathway regulates Purkinje cell dendritic morphogenesis in a cell-autonomous manner.

2021

Neuro-vascular communication is essential to synchronize central nervous system development. Here, we identify angiopoietin/Tie2 as a neuro-vascular signaling axis involved in regulating dendritic morphogenesis of Purkinje cells (PCs). We show that in the developing cerebellum Tie2 expression is not restricted to blood vessels, but it is also present in PCs. Its ligands angiopoietin-1 (Ang1) and angiopoietin-2 (Ang2) are expressed in neural cells and endothelial cells (ECs), respectively. PC-specific deletion of Tie2 results in reduced dendritic arborization, which is recapitulated in neural-specific Ang1-knockout and Ang2 full-knockout mice. Mechanistically, RNA sequencing reveals that Tie…

CerebellumalphaCytoskeleton organizationAngiogenesisPurkinje cellprotocadherinsMorphogenesisneural progenitor cellsMice Transgenicself-avoidanceBiologyModels BiologicalGeneral Biochemistry Genetics and Molecular BiologyAngiopoietinAngiopoietin-2Purkinje Cellsddc:570CerebellumexpressionGene expressionmedicineAngiopoietin-1MorphogenesisAnimalsmouseMice KnockoutIntegrasessubventricular zonedifferentiationDendritesmtorc2Angiopoietin receptorReceptor TIE-2Cell biologyMice Inbred C57BLmedicine.anatomical_structuremessenger-rnaGene Expression RegulationOrgan Specificityembryonic structurescardiovascular systembiology.proteinGene DeletionSignal TransductionCell reports
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Alterations of regional cerebral blood flow and oxygen saturation in a rat sinus-vein thrombosis model.

1996

Background and Purpose The pathophysiology of sinus-vein thrombosis (SVT) in patients and experimental animals is still poorly understood. This study was designed to examine and further elucidate the pathophysiological sequence of events, especially the relationship between local and regional blood flow and hemoglobin oxygen saturation (HbSO 2 ) detected at identical locations. The use of both parameters as outcome indicators should be compared. Methods SVT was induced by ligation of the superior sagittal sinus (SSS) and slow injection of kaolin-cephalin suspension into the SSS in rats. Regional cerebral blood flow (rCBF) was assessed by laser-Doppler flowmetry together with regional HbSO …

Cerebral veinsMalemedicine.medical_specialtyPathologyTime FactorsUltrasonography Doppler TranscranialHemodynamicsMicrocirculationHemoglobinsInternal medicinemedicineAnimalsHumansFluorescein AngiographyRats WistarAdvanced and Specialized NursingCerebral Cortexbusiness.industryBrainBlood flowIntracranial Embolism and Thrombosismedicine.diseaseThrombosisCerebral VeinsRatsSSS*OxygenDisease Models AnimalCerebral blood flowOrgan SpecificityRegional Blood FlowCerebrovascular CirculationCardiologyNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessSuperior sagittal sinusStroke
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Diurnal variation of corticotropin-releasing factor binding sites in the rat brain and pituitary.

1996

1. Corticotropin-releasing factor (CRF) is thought to be involved in the regulation of the diurnal activity of the hypothalamus-pituitary-adrenal (HPA) axis and to act as a neurotransmitter in the brain. To date it is unknown whether the binding sites of the central CRF system are subject to diurnal variations. 2. We measured the number of CRF binding sites over the course of a complete 24-hr light-dark cycle in the pituitary, amygdala, bed nucleus of the stria terminalis (BNST), cingulate cortex, visceral cortex, paraventricular nucleus of the hypothalamus, hippocampus, and locus ceruleus of rats by in vitro receptor autoradiography with iodinated ovine CRF. A 24-hr time course was also es…

Cingulate cortexMaleendocrine systemmedicine.medical_specialtyLightCorticotropin-Releasing HormoneHippocampusAmygdalaReceptors Corticotropin-Releasing HormoneIodine RadioisotopesRats Sprague-Dawley03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compound0302 clinical medicineCorticosteroneInternal medicinemedicineAnimalsNeurotransmitter030304 developmental biology0303 health sciencesBinding SitesSheepLocus CeruleusBrainCell BiologyGeneral MedicineDarknessCircadian RhythmRatsStria terminalismedicine.anatomical_structureEndocrinologychemistryHypothalamusOrgan SpecificityPituitary GlandAutoradiographyCorticosteronehormones hormone substitutes and hormone antagonists030217 neurology & neurosurgeryCellular and molecular neurobiology
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Human NK cells selective targeting of colon cancer-initiating cells: a role for natural cytotoxicity receptors and MHC class I molecules

2013

Abstract Tumor cell populations have been recently proposed to be composed of two compartments: tumor-initiating cells characterized by a slow and asymmetrical growth, and the “differentiated” cancer cells with a fast and symmetrical growth. Cancer stem cells or cancer-initiating cells (CICs) play a crucial role in tumor recurrence. The resistance of CICs to drugs and irradiation often allows them to survive traditional therapy. NK cells are potent cytotoxic lymphocytes that can recognize tumor cells. In this study, we have analyzed the NK cell recognition of tumor target cells derived from the two cancer cell compartments of colon adenocarcinoma lesions. Our data demonstrate that freshly p…

Cytotoxicity ImmunologicNKImmunologyGene ExpressionCancer Stem CellMice SCIDBiologyAdenocarcinomaInterleukin 21MiceNK-92Cancer stem cellMice Inbred NODTumor Cells CulturedImmunology and AllergyCytotoxic T cellAnimalsHumansCell LineageSettore MED/04 - Patologia GeneraleLymphokine-activated killer cellMicroscopy ConfocalNatural Cytotoxicity Triggering Receptor 3Natural Cytotoxicity Triggering Receptor 2Janus kinase 3Histocompatibility Antigens Class Inessuna parola chiaveKiller Cells NaturalOrgan SpecificityImmunologyCancer cellColonic NeoplasmsCancer researchInterleukin 12Neoplastic Stem Cellsimmunotherapy
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Heterochromatin Networks: Topology, Dynamics, and Function (a Working Hypothesis)

2021

Open systems can only exist by self-organization as pulsing structures exchanging matter and energy with the outer world. This review is an attempt to reveal the organizational principles of the heterochromatin supra-intra-chromosomal network in terms of nonlinear thermodynamics. The accessibility of the linear information of the genetic code is regulated by constitutive heterochromatin (CHR) creating the positional information in a system of coordinates. These features include scale-free splitting-fusing of CHR with the boundary constraints of the nucleolus and nuclear envelope. The analysis of both the literature and our own data suggests a radial-concentric network as the main structural…

DNA Replication TimingQH301-705.5HeterochromatinEmbryonic DevelopmentReviewtranscriptional pulsingTopologyModels Biologicalpositional informationphysics of lifeCell Line TumorAnimalsHumansConstitutive heterochromatinNucleosomeEpigeneticsBiology (General)PhysicsReplication timingheterochromatincytoskeletonActomyosinGeneral MedicineGenetic codenucleolar boundaryRatsChromatinGene Expression RegulationOrgan SpecificitynetworksRNA splicingscale-free oscillationsChickensCell Nucleoluschromatin organizationCells
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Molecular cloning of rat G-protein-coupled receptor kinase 6 (GRK6) from brain tissue, and its mRNA expression in different brain regions and periphe…

1997

The rat G-protein-coupled receptor kinase 6 (GRK6) cDNA was cloned from rat brain tissue by a combination of reverse-transcription polymerase chain reactions (RT-PCR), based on homology to the cloned human GRK6, and rapid amplification of cDNA ends (RACE-PCR). We obtained a clone of 2817 bp with an open reading frame of 1731 bp encoding for a protein of 576 amino acids that is 96.7% identical and 97.9% similar to its human counterpart. mRNA was detectable in all brain areas examined. In addition, GRK6 was expressed in skeletal muscle, small intestine, aorta, liver, heart, lung, thymus, stomach, uterus and kidney.

DNA ComplementaryTranscription GeneticMolecular Sequence DataProtein Serine-Threonine KinasesMolecular cloningBiologyPolymerase Chain ReactionOpen Reading FramesCellular and Molecular NeuroscienceRapid amplification of cDNA endsGTP-Binding ProteinsComplementary DNAGene expressionAnimalsHumansAmino Acid SequenceRNA MessengerCloning MolecularProtein kinase AMolecular BiologyG protein-coupled receptor kinaseMessenger RNABase SequenceSequence Homology Amino AcidBrainReceptor Protein-Tyrosine KinasesG-Protein-Coupled Receptor KinasesMolecular biologyRatsOpen reading frameOrgan SpecificityFemaleSequence AlignmentMolecular Brain Research
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Dual roles of Aβ in proliferative processes in an amyloidogenic model of Alzheimer’s disease

2017

Alzheimer’s disease is a major neurodegenerative disorder that leads to severe cognitive deficits in the elderly population. Over the past two decades, multiple studies have focused on elucidating the causative factors underlying memory defects in Alzheimer’s patients. In this regard, new evidence linking Alzheimer’s disease-related pathology and neuronal stem cells suggests that hippocampal neurogenesis impairment is an important factor underlying these cognitive deficits. However, because of conflicting results, the impact of Aβ pathology on neurogenesis/gliogenesis remains unclear. Here, we investigated the effect of Aβ on neuronal and glial proliferation by using an APP/PS1 transgenic m…

Doublecortin Domain ProteinsMale0301 basic medicineCellular pathologyPathologymedicine.medical_specialtyNeurogenesisGene ExpressionHippocampuslcsh:MedicineMice TransgenicBiologyHippocampusArticleAmyloid beta-Protein PrecursorMice03 medical and health sciences0302 clinical medicineNeural Stem CellsAlzheimer DiseaseSpheroids CellularNeurospheremedicineAnimalsHumansProgenitor celllcsh:ScienceCells CulturedCell ProliferationGliogenesisNeuronsAmyloid beta-PeptidesMultidisciplinaryNeuropeptidesNeurogenesislcsh:RCell DifferentiationNeural stem cellDisease Models Animal030104 developmental biologynervous systemOrgan Specificitylcsh:QStem cellMicrotubule-Associated ProteinsNeurogliaNeuroscience030217 neurology & neurosurgery
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DNA-methylation dependent regulation of embryo-specific 5S ribosomal DNA cluster transcription in adult tissues of sea urchin Paracentrotus lividus

2013

Abstract We have previously reported a molecular and cytogenetic characterization of three different 5S rDNA clusters in the sea urchin Paracentrotus lividus and recently, demonstrated the presence of high heterogeneity in functional 5S rRNA. In this paper, we show some important distinctive data on 5S rRNA transcription for this organism. Using single strand conformation polymorphism (SSCP) analysis, we demonstrate the existence of two classes of 5S rRNA, one which is embryo-specific and encoded by the smallest (700 bp) cluster and the other which is expressed at every stage and encoded by longer clusters (900 and 950 bp). We also demonstrate that the embryo-specific class of 5S rRNA is ex…

Embryo NonmammalianTranscription GeneticBase pairDNA RibosomalParacentrotus lividusEpigenesis Geneticsea urchin5S ribosomal RNASingle-Strand conformation polymorphism (SSCP)Transcription (biology)biology.animalGeneticsAnimalsGene SilencingSea urchinRibosomal DNAPolymorphism Single-Stranded ConformationalGeneticsbiologyRNA Ribosomal 5SDNA Methylation5S ribosomal RNARibosomal RNAbiology.organism_classificationSettore BIO/18 - GeneticaOrgan Specificity5S ribosomal genesilencingDNA methylationAzacitidineOocytesParacentrotusFemaleepigeneticGenomics
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Mitochondrial localization and temporal expression of the Drosophila melanogaster DnaJ homologous tumor suppressor Tid50

1998

The Drosophila melanogaster tumor suppressor gene lethal(2)tumorous imaginal discs (tid) was identified as a homolog of all dnaJ-like genes known to date which have been well preserved in evolution. Homozygous D. melanogaster l(2)tid mutants l(2)tid1, l(2)tid2 and l(2)tid3 are characterized by neoplastic transformation of the adult integumental primordia, the imaginal discs, and the death at the time of puparium formation. The first part of this study is concerned with the identification and subcellular localization of the l(2)tid-encoded protein, Tid50. The second part examines its tissue specific expression during wild-type development and in tumorous imaginal discs. To specify the functi…

Embryo NonmammalianTumor suppressor geneMutantGenes InsectCell FractionationBiochemistryCell LineMitochondrial ProteinsMelanogasterAnimalsDrosophila ProteinsGenes Tumor SuppressorNeoplastic transformationRNA MessengerGeneHeat-Shock ProteinsbiologyPupaGene Expression Regulation DevelopmentalRNANeoplasms ExperimentalSequence Analysis DNAOriginal ArticlesCell BiologyHSP40 Heat-Shock Proteinsbiology.organism_classificationMolecular biologyMitochondriaGene Expression Regulation NeoplasticImaginal discDrosophila melanogasterOrgan SpecificityLarvaRabbitsDrosophila melanogasterCell Stress & Chaperones
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