Search results for "Oxidants"

showing 10 items of 878 documents

Mitochondrial glutathione oxidation correlates with age-associated oxidative damage to mitochondrial DNA

1996

Mitochondria may be primary targets of free radical damage associated with aging. We have found that mitochondrial glutathione is markedly oxidized with aging in rats and mice. The oxidized to reduced glutathione ratio rises with aging in the liver, kidney, and brain. The magnitude of these changes is much higher than that previously found in whole cells of any species previously studied. In the liver, this ratio (expressing GSSG as a percent of GSH) changed from 0.77 +/- 0.19% (n=5) in young rats to 2.47 +/- 1.25% (n=5) in old ones, i.e., 320% of the controls. In the brain and kidney, values for old rats were, respectively, 600 and 540% higher than those of young rats. A marked oxidation o…

Malemedicine.medical_specialtyMitochondrial DNAAgingAntioxidantmedicine.medical_treatmentMitochondrionmedicine.disease_causeBiochemistryDNA MitochondrialAntioxidantschemistry.chemical_compoundMiceInternal medicineGeneticsmedicineDeoxyguanosineAnimalsRats WistarMolecular BiologyFree-radical theory of agingKidneyGlutathione DisulfideChemistryDeoxyguanosineGlutathioneGlutathioneRatsMice Inbred C57BLOxidative StressEndocrinologymedicine.anatomical_structure8-Hydroxy-2'-DeoxyguanosineRabbitsOxidation-ReductionOxidative stressBiotechnologyDNA Damage
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Mitochondrial oxidant generation is involved in determining why females live longer than males

2006

Females live longer than males in many mammalian species, including humans. This natural phenomenon can be explained on the basis of the mitochondrial theory of aging. Mitochondria are a major source of free radicals in cells. Mitochondria from female rats generate half the amount of hydrogen peroxide than those of males and have higher levels of mitochondrial reduced glutathione. The latter is due to females behaving as double transgenic in over-expressing antioxidant enzymes. Estrogens bind to the estrogen receptors and subsequently activate the mitogen activated protein (MAP) kinase and nuclear factor kappa B (NFkappaB) signalling pathways, resulting in an upregulation of antioxidant enz…

Malemedicine.medical_specialtyMitochondrial DNALongevityEstrogen receptorMitochondrionBiologymedicine.disease_causechemistry.chemical_compoundDownregulation and upregulationInternal medicinemedicineAnimalsHumansSex CharacteristicsEstrogensGlutathioneOxidantsMitochondriaOxidative StressEndocrinologychemistryFemalePhytoestrogensSignal transductionOxidative stressSignal TransductionFrontiers in Bioscience
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Resveratrol Reverses Endothelial Nitric-Oxide Synthase Uncoupling in Apolipoprotein E Knockout Mice

2010

A crucial cause of the decreased bioactivity of nitric oxide (NO) in cardiovascular diseases is the uncoupling of the endothelial NO synthase (eNOS) caused by the oxidative stress-mediated deficiency of the NOS cofactor tetrahydrobiopterin (BH(4)). The reversal of eNOS uncoupling might represent a novel therapeutic approach. The treatment of apolipoprotein E knockout (ApoE-KO) mice with resveratrol resulted in the up-regulation of superoxide dismutase (SOD) isoforms (SOD1-SOD3), glutathione peroxidase 1 (GPx1), and catalase and the down-regulation of NADPH oxidases NOX2 and NOX4 in the hearts of ApoE-KO mice. This was associated with reductions in superoxide, 3-nitrotyrosine, and malondiald…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIISOD3SOD2ResveratrolAntioxidantsSuperoxide dismutaseMicechemistry.chemical_compoundApolipoproteins ESuperoxidesEnosMalondialdehydeInternal medicineStilbenesmedicineAnimalsGTP CyclohydrolaseMice KnockoutPharmacologychemistry.chemical_classificationReactive oxygen speciesbiologyReverse Transcriptase Polymerase Chain ReactionSuperoxide DismutaseChemistrySuperoxideMyocardiumTetrahydrobiopterinbiology.organism_classificationBiopterinIsoenzymesOxidative StressEndocrinologyBiochemistryResveratrolbiology.proteinRNATyrosineMolecular Medicinemedicine.drugJournal of Pharmacology and Experimental Therapeutics
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Postischemic application of lipid peroxidation inhibitor U-101033E reduces neuronal damage after global cerebral ischemia in rats.

1998

Background and Purpose —The lipid peroxidation inhibitor U-101033E was examined for effects on cerebral blood flow (CBF), cortical tissue hemoglobin oxygen saturation (HbS o 2 ), and neuronal damage. Methods —Fifteen minutes of global cerebral ischemia was induced by two-vessel occlusion and hypobaric hypotension. Wistar rats (n=25) were randomized to receive vehicle (n=9) or 40 mg/kg U-101033E (n=9) intraperitoneally during 2 hours of reperfusion. A sham group (n=7) had neither ischemia nor therapy. Histology was evaluated 7 days after ischemia. Results —During late hyperperfusion (at 17 minutes), vehicle-treated animals had a higher ( P =0.044) cortical tissue HbS o 2 (72.0±1.4%) than di…

Malemedicine.medical_specialtyPyrrolidinesVasodilator AgentsIschemiaCell CountHippocampal formationAntioxidantsCentral nervous system diseaseLipid peroxidationchemistry.chemical_compoundCortex (anatomy)Internal medicinemedicineLaser-Doppler FlowmetryAnimalsRats WistarAdvanced and Specialized NursingNeuronsbusiness.industryCarbon Dioxidemedicine.diseaseSurgeryRatsEndocrinologymedicine.anatomical_structureNeuroprotective AgentsPyrimidinesCerebral blood flowchemistryCerebral cortexIschemic Attack TransientCerebrovascular CirculationOxyhemoglobinsNeurology (clinical)Lipid PeroxidationHypotensionCardiology and Cardiovascular MedicinebusinessReperfusion injuryStroke
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Chronic ethanol feeding induces cellular antioxidants decrease and oxidative stress in rat peripheral nerves. Effect of S-adenosyl-L-methionine and N…

1998

Chronic ethanol feeding promotes oxidative stress in rat peripheral nerve. Malondialdehyde, a lipid peroxidation product, content increases in sciatic nerves of rats fed an ethanol-containing diet, when compared with pair-fed animals. Moreover, glutathione content and glutathione peroxidase activity in this same tissue decrease in ethanol-fed vs. pair-fed rats. S- adenosyl-L-methionine and N-acetyl-L-cysteine, both with possible therapeutic action on alcoholism, were tested in this animal model. Only N-acetyl-L- cysteine was able to normalize malondialdehyde content and to restore glutathione content and glutathione peroxidase activity, to values not significantly different from those of sc…

Malemedicine.medical_specialtyS-AdenosylmethionineEthanol feedingmedicine.disease_causeBiochemistryAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundFree radicalPhysiology (medical)Internal medicineMalondialdehydemedicineAnimalsPeripheral Nerveschemistry.chemical_classificationGlutathione PeroxidaseEthanolS-adenosyl-L-methionineEthanolGlutathione peroxidaseN-acetyl-L- cysteineNerveGlutathioneMalondialdehydeGlutathionePeripheralAcetylcysteineRatsMicroscopy ElectronOxidative StressEndocrinologychemistryBiochemistryGlutathione peroxidaseLipid PeroxidationEthanoOxidative stressFree radical biologymedicine
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Vitamin C supplementation does not improve hypoxia-induced erythropoiesis.

2012

Martinez-Bello,Vladimir E., Fabian Sanchis-Gomar, Daniel Martinez-Bello, Gloria Olaso-Gonzalez, Mari Carmen Gomez-Cabrera, and Jose Viña. Vitamin C Supplementation Does Not Improve Hypoxia-Induced Erythropoiesis. High Alt Med Biol 13:269–274, 2012.—Hypoxia induces reactive oxygen species production. Supplements with antioxidant mixtures can compensate for the decline in red cell membrane stability following intermittent hypobaric hypoxia by decreasing protein and lipid oxidation. We aimed to determine whether supplementation with vitamin C is implicated in the regulation of erythropoiesis and in the oxygen-carrying capacity of the blood, and also whether antioxidant supplementation prevents…

Malemedicine.medical_specialtyScientific ArticlesAntioxidantPhysiologymedicine.medical_treatmentAscorbic AcidHematocritBiologyProtein oxidationAntioxidantsDrug Administration ScheduleRandom AllocationLipid oxidationInternal medicineMalondialdehydemedicineAnimalsErythropoiesisRats WistarHypoxiaHematologic Testsmedicine.diagnostic_testVitamin CPublic Health Environmental and Occupational HealthIntermittent hypoxiaGeneral MedicineBlood ProteinsHypoxia (medical)RatsOxidative StressEndocrinologyBiochemistryDietary SupplementsErythropoiesismedicine.symptomOxidation-ReductionBiomarkersHigh altitude medicinebiology
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Effect of aqueous extract of Ajuga iva supplementation on plasma lipid profile and tissue antioxidant status in rats fed a high-cholesterol diet

2006

Abstract The present study was designed to explore the possible antioxidant and hypolipidemic effects of the aqueous extract of Ajuga iva (0.5% in the diet) in rats fed a high-cholesterol (1%) diet (HCD). The results indicated that the HCD-Ai versus HCD treatment led to many changes in biochemical parameters. They showed a decrease of plasma total cholesterol (TC) and VLDL-cholesterol but an increase of HDL2-cholesterol. The triacylglycerol contents were reduced in plasma and in VLDL. The lipid peroxidation determined by TBARS was decreased by 75% in plasma. TBARS in liver, heart and kidneys were highly reduced excepted in the adipose tissue. Ajuga iva treatment enhanced superoxide dismutas…

Malemedicine.medical_specialtyVery low-density lipoproteinAntioxidantLipoproteinsmedicine.medical_treatmentHypercholesterolemiaAdipose tissueAjugaThiobarbituric Acid Reactive SubstancesAntioxidantsAjugaCholesterol DietaryLipid peroxidationEatingchemistry.chemical_compoundInternal medicineDrug DiscoverymedicineTBARSAnimalsRats WistarPharmacologybiologyPlant ExtractsCholesterolBody WeightOrgan Sizebiology.organism_classificationLipidsDietEnzymesRatsEndocrinologychemistryDietary SupplementsIntestinal cholesterol absorptionlipids (amino acids peptides and proteins)Journal of Ethnopharmacology
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Tissue antioxidant status differs in spontaneously hypertensive rats fed fish protein or casein

2003

The present study was designed to determine whether changes in dietary protein source are related to changes in antioxidant status determined by enzyme activities of catalase, superoxide dismutase (SOD), gluthatione peroxidase (GSH-Px) and gluthatione reductase (GSSG-Red) and lipid peroxidation levels in various tissues. Spontaneously hypertensive rats (SHR; 5 wk old) were fed diets containing 20% casein or fish protein for 2 mo. Feeding the fish protein diet lowered blood pressure and reduced plasma total cholesterol levels and SOD activity in all tissues except muscle compared with the casein diet. Feeding fish protein also enhanced GSH level and GSH-Px activity in liver and heart, accomp…

Malemedicine.medical_specialtyVery low-density lipoproteinAntioxidantmedicine.medical_treatmentMedicine (miscellaneous)Blood PressureKidneyFish ProteinsAntioxidantsSuperoxide dismutaseLipid peroxidationchemistry.chemical_compoundRats Inbred SHRCaseinInternal medicineFish ProductsmedicineAnimalsNutrition and DieteticsbiologyCaseinsHeartCatalaseFish productsRatsCholesterolEndocrinologyAdipose TissueLiverchemistryCatalasebiology.proteinDietary ProteinsLipid Peroxidation
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Role of mitochondrial oxidative stress to explain the different longevity between genders. Protective effect of estrogens

2006

Females live longer than males. Work from our laboratory has shown that this may be due to the up-regulation of longevity-associated genes by estrogens. Estrogens bind to the estrogen receptors and subsequently activate the mitogen activated protein kinase and nuclear factor kappa B signalling pathways, resulting in an up-regulation of antioxidant enzymes. Estrogen administration, however, has serious undesirable effects and of course, cannot be administered to males because of its powerful feminizing effects. Thus, we tested the effect of genistein, a phytoestrogen of high nutritional importance whose structure is similar to estradiol, on the regulation of the expression of antioxidant, lo…

Malemedicine.medical_specialtymedicine.drug_classmedia_common.quotation_subjectLongevityEstrogen receptorGenisteinPhytoestrogensBiologymedicine.disease_causeBiochemistryAntioxidantschemistry.chemical_compoundSex FactorsDownregulation and upregulationInternal medicinemedicineAnimalsEstrogen Receptor betaHumansmedia_commonSuperoxide DismutaseLongevityEstrogensGeneral MedicineMitochondriaRatsUp-RegulationOxidative StressEndocrinologychemistryEstrogenFemalePhytoestrogensOxidative stressHormoneFree Radical Research
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Nitric oxide metabolites, leukocyte activation markers and oxidative status in dialyzed subjects.

2008

<i>Aims:</i> Our purpose was to evaluate, in a group of 42 end-stage renal disease patients who regularly undergo hemodialysis, some indexes of leukocyte activation, nitric oxide metabolites (NOx) and other parameters that reflect the oxidative stress before and after a standard hemodialysis session. <i>Methods:</i> Elastase and myeloperoxidase (MPO) were determined by means of ELISA. The NO production was evaluated by a micromethod which measures the concentration of NOx. The oxidation of polyunsaturated fatty acids was evaluated in plasma by detection of thiobarbituric acid-reactive substances (TBARS). Total antioxidant status (TAS) was obtained using spectrophotom…

Malemedicine.medical_treatmentActivation markersOxidative phosphorylationHemodialysis Oxidative status Nitric oxide Elastase MyeloperoxidasePharmacologyNitric OxideThiobarbituric Acid Reactive SubstancesAntioxidantsNitric oxidechemistry.chemical_compoundRenal DialysisLeukocytesmedicineHumansAgedPeroxidasechemistry.chemical_classificationbiologyElastaseHematologyGeneral MedicineMiddle AgedOxidative StressEnzymechemistryNephrologyMyeloperoxidaseImmunologybiology.proteinKidney Failure ChronicFemaleHemodialysisLeukocyte Elastase
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