Search results for "Oxidants"

showing 10 items of 878 documents

Pulsed electric field (PEF) recovery of biomolecules from Chlorella: Extract efficiency, nutrient relative value, and algae morphology analysis

2023

This study investigated the effects of pulsed electric field (PEF) (3 kV/cm, 44 pulses, 99 kJ/kg), solvent (H2O or 50 % DMSO) and time (0, 10, 20, 30, 60, 90, 120 and 180 min) on the extraction of Chlorella antioxidant biomolecules and minerals. The results showed that PEF treatment increased the biomolecules recovery. For the extraction time of 120 min, more proteins and polyphenols were obtained using water, while more chlorophyll a and b, and carotenoids were obtained using 50 % DMSO as the extraction solvent. The extracts mineral concentration (PEF vs control) were analysed including Mg, P, Ca, Fe and Zn, and the Relative Nutrient Values results indicated that Chlorella H2O-extracts cou…

MineralsChlorophyll AChlorellaNutrientsGeneral MedicinePulsed electric fields (PEF)AntioxidantsAnalytical ChemistryAlguesSolventsMicroalgaeAlgues d'aigua dolçaDimethyl SulfoxideNutritive ValueMicrostructureFood ScienceFood Chemistry
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Mitochondria as sources and targets of damage in cellular aging.

2011

Mitochondria are considered as the most important cellular sources and targets of free radicals. They are also a source of signalling molecules that regulate cell cycle, proliferation, and apoptosis. Denham Harman postulated the free radical theory of aging in 1956. Previously Rebecca Gershman showed that radiation toxicity could be attributed to free radical damage. Subsequently, Jaime Miquel formulated the mitochondrial free radical theory of aging. We have shown that mitochondrial size, membrane potential, inner membrane mass and peroxide production is altered inside cells in old animals. These result in an increase in the oxidative damage to mitochondrial DNA with aging that can be prev…

Mitochondrial DNAFree RadicalsDNA damageBiochemistry (medical)Clinical BiochemistryGeneral MedicineMitochondrionBiologyMitochondrial Sizemedicine.disease_causeAntioxidantsCell biologyMitochondriaOxidative StressMitochondrial biogenesisApoptosismedicineAnimalsHumansOxidative stressCellular SenescenceFree-radical theory of agingDNA DamageClinical chemistry and laboratory medicine
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Oxidant antioxidants and adaptive responses to exercise.

2015

The extensive damage produced by unaccustomed (acute) exercise and the health benefits of regular physical activity are well-known phenomena as well as the role played in them by reactive oxygen species (ROS). The present issue reports some interesting studies showing that the Janus faced effects of exercise-induced ROS in skeletal muscle. Most studies dealing with ROS contribution to acute exercise-induced tissue damage determine the levels of markers of oxidative damage to specific substances but they do not take into account total redox status of an individual before and after exercise. In their research article D. Stagos et al. used markers measuring plasma static (sORP) and capacity (c…

Mitochondrial ROSAgingmedicine.medical_specialtyArticle SubjectPhysical exerciseOxidative phosphorylationBiologymedicine.disease_causeBiochemistryAntioxidantsLipid peroxidationchemistry.chemical_compoundInternal medicinemedicineHumanslcsh:QH573-671Exercisechemistry.chemical_classificationReactive oxygen specieslcsh:CytologyCell BiologyGeneral MedicineOxidantsGlutathioneMitochondrial respiratory chainEndocrinologyEditorialchemistryBiochemistryExercise intensityReactive Oxygen SpeciesOxidative stressOxidative medicine and cellular longevity
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ALDH-2 deficiency increases cardiovascular oxidative stress--evidence for indirect antioxidative properties.

2007

Abstract Mitochondrial aldehyde dehydrogenase (ALDH-2) reduces reactive oxygen species (ROS) formation related to toxic aldehydes; additionally, it provides a bioactivating pathway for nitroglycerin. Since acetaldehyde, nitroglycerin, and doxorubicin treatment provoke mitochondrial oxidative stress, we used ALDH-2−/− mice and purified recombinant human ALDH-2 to test the hypothesis that ALDH-2 has an indirect antioxidant function in mitochondria. Antioxidant capacity of purified ALDH-2 was comparable to equimolar doses of glutathione, cysteine, and dithiothreitol; mitochondrial oxidative stress was comparable in C57Bl6 and ALDH-2−/− mice after acute challenges with nitroglycerin or doxorubi…

Mitochondrial ROSAntioxidantmedicine.medical_treatmentBiophysicsAldehyde dehydrogenaseAcetaldehydeMitochondrionPharmacologymedicine.disease_causeBiochemistryCardiovascular SystemModels BiologicalAntioxidantschemistry.chemical_compoundMiceNitroglycerinmedicineAnimalsHumansCysteineMolecular Biologychemistry.chemical_classificationReactive oxygen speciesbiologyDose-Response Relationship DrugAldehyde Dehydrogenase MitochondrialAcetaldehydeCell BiologyGlutathioneAldehyde DehydrogenaseGlutathioneMitochondriaMice Inbred C57BLDithiothreitolOxidative StresschemistryBiochemistryDoxorubicincardiovascular systembiology.proteinReactive Oxygen SpeciesOxidative stressBiochemical and biophysical research communications
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Evidence for a relationship between mitochondrial Complex I activity and mitochondrial aldehyde dehydrogenase during nitroglycerin tolerance: effects…

2012

The medical use of nitroglycerin (GTN) is limited by patient tolerance. The present study evaluated the role of mitochondrial Complex I in GIN biotransformation and the therapeutic effect of mitochondrial antioxidants. The development of GIN tolerance (in rat and human vessels) produced a decrease in mitochondrial 02 consumption. Co-incubation with the mitochondria-targeted antioxidant mitoquinone (MQ 10(-6) mol/L) or with glutathione ester (GEE, 10(-4) mol/L) blocked GTN tolerance and the effects of GTN on mitochondrial respiration and aldehyde dehydrogenase 2 (ALDH-2) activity. Biotransformation of GTN depended on the mitochondria being functionally active, particularly mitochondrial Comp…

Mitochondrial ROSMaleAntioxidantmedicine.medical_treatmentAldehyde dehydrogenaseMitochondrionmedicine.disease_causeBiochemistryAntioxidantsRats Sprague-Dawleychemistry.chemical_compoundMiceNitroglycerinCyclic GMPAortaBiotransformationbiologyDrug ToleranceGlutathioneMitochondriaVasodilationBiochemistrycardiovascular systemAntioxidantcirculatory and respiratory physiologyBiophysicsIn Vitro TechniquesALDH-2Nitric oxideCell LineOxygen ConsumptionRotenoneRespirationmedicineHuman Umbilical Vein Endothelial CellsAnimalsHumansElectron Transport Complex IDose-Response Relationship DrugNitric oxideGlutathioneCell BiologyAldehyde DehydrogenaseRatschemistryOxidative stressMutationbiology.proteinReactive Oxygen SpeciesOxidative stressBiochimica et biophysica acta
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Mitochondrial Oxidative Stress, Mitochondrial DNA Damage and Their Role in Age-Related Vascular Dysfunction

2015

The prevalence of cardiovascular diseases is significantly increased in the older population. Risk factors and predictors of future cardiovascular events such as hypertension, atherosclerosis, or diabetes are observed with higher frequency in elderly individuals. A major determinant of vascular aging is endothelial dysfunction, characterized by impaired endothelium-dependent signaling processes. Increased production of reactive oxygen species (ROS) leads to oxidative stress, loss of nitric oxide (•NO) signaling, loss of endothelial barrier function and infiltration of leukocytes to the vascular wall, explaining the low-grade inflammation characteristic for the aged vasculature. We here disc…

Mitochondrial ROSmedicine.medical_specialtyMitochondrial DNADNA RepairInflammationReviewBiologyMitochondrionmedicine.disease_causeDNA MitochondrialCatalysisAntioxidantsNitric oxideInorganic Chemistrylcsh:Chemistrychemistry.chemical_compoundInternal medicinemedicineAnimalsHumansPhysical and Theoretical ChemistryEndothelial dysfunctionMolecular Biologylcsh:QH301-705.5Spectroscopychemistry.chemical_classificationReactive oxygen speciesmitochondrial oxidative stressOrganic Chemistryagingmitochondrial DNA damageGeneral Medicinevascular dysfunctionmedicine.diseaseComputer Science ApplicationsMitochondriaOxidative StressEndocrinologyBiochemistrychemistrylcsh:Biology (General)lcsh:QD1-999Cardiovascular Diseasesmedicine.symptomReactive Oxygen SpeciesOxidative stressDNA DamageInternational Journal of Molecular Sciences
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Pathophysiological role of oxidative stress in systolic and diastolic heart failure and its therapeutic implications

2015

Abstract Systolic and diastolic myocardial dysfunction has been demonstrated to be associated with an activation of the circulating and local renin–angiotensin–aldosterone system (RAAS), and with a subsequent inappropriately increased production of reactive oxygen species (ROS). While, at low concentrations, ROS modulate important physiological functions through changes in cellular signalling and gene expression, overproduction of ROS may adversely alter cardiac mechanics, leading to further worsening of systolic and diastolic function. In addition, vascular endothelial dysfunction due to uncoupling of the nitric oxide synthase, activation of vascular and phagocytic membrane oxidases or mit…

Mitochondrial ROSmedicine.medical_specialtyXanthine OxidasePhosphodiesterase InhibitorsDiastoleAngiotensin-Converting Enzyme InhibitorsReviewmedicine.disease_causeNitric OxideCardiovascular SystemAntioxidantsInternal medicinemedicineHumansEndothelial dysfunctionHeart Failure DiastolicEjection fractionNitratesbusiness.industryDiastolic heart failureNADPH OxidasesStroke VolumeVitaminsHydralazinemedicine.diseaseHydralazineExercise TherapyMitochondriaOxidative StressHeart failureCardiologyDrug Therapy CombinationNitric Oxide SynthaseCardiology and Cardiovascular MedicinebusinessReactive Oxygen SpeciesOxidative stressmedicine.drugHeart Failure Systolic
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Revised structure of trans-resveratrol: Implications for its proposed antioxidant mechanism

2015

The crystal structure of trans-resveratrol has been redetermined by X-ray diffraction. The newly refined structure demonstrates that the previously reported, dynamically disordered hydrogen-bonding network is rather the superposition of two crystallographically independent molecules of trans-resveratrol. This latter arrangement possesses a well-defined hydrogen-bonding network in a unit cell of double the previously reported volume. While not meant as a criticism of the proposed antioxidant mechanism itself, the present studies clearly show that the X-ray diffraction data should no longer be used for its additional support.

Models MolecularDiffractionStereochemistryClinical BiochemistryStructure (category theory)Pharmaceutical ScienceCrystal structureCrystallography X-Ray010402 general chemistry01 natural sciencesBiochemistryAntioxidantsStructure-Activity RelationshipStilbenesDrug DiscoveryMoleculeMolecular BiologyMolecular StructureTrans-resveratrol010405 organic chemistryHydrogen bondChemistryOrganic ChemistryHydrogen Bondingdisorder0104 chemical sciencesCrystallographyResveratrolMechanism (philosophy)Molecular MedicineX-ray structuresuperstructureSuperstructure (condensed matter)Bioorganic & Medicinal Chemistry Letters
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Preparation of two sets of 5,6,7-trioxygenated dihydroflavonol derivatives as free radical scavengers and neuronal cell protectors to oxidative damag…

2009

An unusual class of 5,6,7-trioxygenated dihydroflavonols (3a-e and 4a-j) were designed and prepared. Their antioxidative properties were assessed by examining their capacities in several in vitro models, including superoxide anion and 1,1-diphenyl-2-picrylhydrazyl (DPPH) radical scavenging, rat liver homogenate lipid peroxidation inhibition, PC12 cells protection from oxidative damage, and xanthine oxidase inhibition. These dihydroflavonols displayed positive quenching abilities towards O(2)(-) and DPPH free radicals, in which the majority exhibited superior antioxidant properties to Vitamin C. cis-Configurated compound (+/-)-3e demonstrated remarkable inhibition to LPO with an IC(50) value…

Models MolecularXanthine OxidaseAntioxidantFlavonolsmedicine.drug_classDPPHmedicine.medical_treatmentClinical BiochemistryMolecular ConformationPharmaceutical Sciencemedicine.disease_causeBiochemistryPC12 CellsAntioxidantsLipid peroxidationRats Sprague-Dawleychemistry.chemical_compoundStructure-Activity RelationshipDrug DiscoverymedicineAnimalsXanthine oxidaseMolecular BiologyXanthine oxidase inhibitorNeuronsSuperoxideOrganic ChemistryFree Radical ScavengersFree radical scavengerRatschemistryBiochemistryMolecular MedicineLipid PeroxidationReactive Oxygen SpeciesOxidative stressBioorganicmedicinal chemistry
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Effects of elevated hydrogen peroxide 'strip' bleaching on surface and subsurface enamel including subsurface histomorphology, micro-chemical composi…

2007

Abstract Objectives This study examined the effects of elevated concentration hydrogen peroxide tooth whitening treatments on tooth surface and subsurface integrity. Methods Sound human molars were ground and polished to prepare an uniform substrate for bleaching treatments. A cycling treatment included alternating ex vivo human salivary exposures with bleaching treatments under conditions of controlled temperature and durations of treatment. Bleaching was carried out with prototype bleaching strips containing hydrogen peroxide gel at 13% and 16% concentrations. A non-bleached group was used as a control. Treatments included 28 h of total bleaching exposure in vitro . Surface color was meas…

MolarToothbrushingMaterials scienceTime Factorsgenetic structuresBleachDentistryColorSpectrum Analysis RamanPeroxideFluorescencechemistry.chemical_compoundstomatognathic systemHardnessMaterials TestingDentinmedicineTooth BleachingHumansHydrogen peroxideDental EnamelSalivaGeneral DentistryDentifricesTooth whiteningMicroscopy ConfocalEnamel paintbusiness.industryTemperatureTooth surfaceHydrogen PeroxideOxidantsstomatognathic diseasesmedicine.anatomical_structurechemistryvisual_artDentinvisual_art.visual_art_mediumMicroscopy Electron ScanningColorimetrysense organsbusinessNuclear chemistryJournal of dentistry
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