Search results for "P3"

showing 10 items of 786 documents

Human brain ages with hierarchy-selective attenuation of prediction errors

2020

Abstract From the perspective of predictive coding, our brain embodies a hierarchical generative model to realize perception, which proactively predicts the statistical structure of sensory inputs. How are these predictive processes modified as we age? Recent research suggested that aging leads to decreased weighting of sensory inputs and increased reliance on predictions. Here we investigated whether this age-related shift from sensorium to predictions occurs at all levels of hierarchical message passing. We recorded the electroencephalography responses with an auditory local–global paradigm in a cohort of 108 healthy participants from 3 groups: seniors, adults, and adolescents. The detect…

MaleMismatch negativityAudiologyElectroencephalographyRandom Allocation[SCCO]Cognitive scienceCognition0302 clinical medicineprediction errorsEEGelectroencephalography (EEG)predictive codingmedia_commonAged 80 and overmedicine.diagnostic_testAcademicSubjects/SCI0187005 social sciencesBrainElectroencephalographyMiddle Agedhavaintopsykologiaauditory perceptionEvoked Potentials AuditoryOriginal ArticleFemalePsychologyAdultAuditory perceptionmedicine.medical_specialtyCognitive Neurosciencemedia_common.quotation_subjectSensory systemkuulohavainnot050105 experimental psychologyYoung Adult03 medical and health sciencesCellular and Molecular NeuroscienceP3aPerceptionP3bmedicineHumans0501 psychology and cognitive sciencesAcademicSubjects/MED00385AgedWorking memory[SCCO.NEUR]Cognitive science/NeuroscienceaginghavainnotikääntyminenAcoustic StimulationAcademicSubjects/MED00310Psychomotor Performance030217 neurology & neurosurgeryForecasting
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Modulatory effects of nitric oxide-active drugs on the anticonvulsant activity of lamotrigine in an experimental model of partial complex epilepsy in…

2007

Abstract Background The effects induced by administering the anticonvulsant lamotrigine, the preferential inhibitor of neuronal nitric oxide synthase 7-nitroindazole and the precursor of NO synthesis L-arginine, alone or in combination, on an experimental model of partial complex seizures (maximal dentate gyrus activation) were studied in urethane anaesthetized rats. The epileptic activity of the dentate gyrus was obtained through the repetitive stimulation of the angular bundle and maximal dentate gyrus activation latency, duration and post-stimulus afterdischarge duration were evaluated. Results Either Lamotrigine (10 mg kg-1) or 7-nitroindazole (75 mg kg-1) i.p. administration had an ant…

MalePARTIAL COMPLEX EPILEPSYIndazolesArgininemedicine.medical_treatmentLamotriginePharmacologyArginineLamotrigineNitric OxideSettore BIO/09 - Fisiologialcsh:RC321-571Nitric oxideCellular and Molecular Neurosciencechemistry.chemical_compoundEpilepsy Complex PartialmedicineAnimalsDrug InteractionsEnzyme InhibitorsRats Wistarlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryNitric oxide Lamotrigine epilepsy controlbiologyTriazinesExperimental modelGeneral NeuroscienceDentate gyruslcsh:QP351-495BrainElectric StimulationRatsNitric oxide synthaseDisease Models Animallcsh:Neurophysiology and neuropsychologyAnticonvulsantnervous systemchemistryDentate Gyrusbiology.proteinAnticonvulsantsNitric Oxide SynthaseResearch Articlemedicine.drugBMC Neuroscience
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Arterial and Venous Endothelia Display Differential Functional Fractalkine (CX 3 CL1) Expression by Angiotensin-II

2012

Objective— Angiotensin-II (Ang-II) promotes the interaction of mononuclear cells with arterioles and neutrophils with postcapillary venules. To investigate the mechanisms underlying this dissimilar response, the involvement of fractalkine (CX 3 CL1) was explored. Methods and Results— Enhanced CX 3 CL1 expression was detected in both cremasteric arterioles and postcapillary venules 24 hours after Ang-II intrascrotal injection. Arteriolar leukocyte adhesion was the unique parameter significantly reduced (83%) in animals lacking CX 3 CL1 receptor (CX 3 CR1). Human umbilical arterial and venous endothelial cell stimulation with 1 μmol/L Ang-II increased CX 3 CL1 expression, yet neutralization …

MalePathologyTime Factorsp38 Mitogen-Activated Protein KinasesMiceVenulesLeukocytesEndothelial dysfunctionExtracellular Signal-Regulated MAP KinasesReceptorCells CulturedMice KnockoutMembrane GlycoproteinsAngiotensin IINF-kappa BArteriesEndothelial stem cellArteriolesNADPH Oxidase 5NADPH Oxidase 4NADPH Oxidase 2FemaleRNA InterferenceReceptors ChemokineTumor necrosis factor alphaCardiology and Cardiovascular MedicineSignal Transductionmedicine.medical_specialtyCX3C Chemokine Receptor 1BiologyTransfectionPeripheral blood mononuclear cellLosartanVeinsInterferon-gammaApolipoproteins EDownregulation and upregulationInternal medicineCell AdhesionHuman Umbilical Vein Endothelial CellsmedicineAnimalsHumansLeukocyte RollingCX3CL1Chemokine CX3CL1Tumor Necrosis Factor-alphaEndothelial CellsMembrane ProteinsNADPH OxidasesAtherosclerosismedicine.diseaseAngiotensin IIMice Inbred C57BLDisease Models AnimalEndocrinologyAngiotensin II Type 1 Receptor BlockersArteriosclerosis, Thrombosis, and Vascular Biology
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Effects of caffeine on neuromuscular function in a non‐fatigued state and during fatiguing exercise

2020

New findings What is the central question of the study? What are the effects of caffeine on neuromuscular function in a non-fatigued state and during fatiguing exercise? What is the main finding and its importance? In a non-fatigued state, caffeine decreased the duration of the silent period evoked by transcranial magnetic stimulation. Caffeine-induced reduction of inhibitory mechanisms in the central nervous system before exercise was associated with an increased performance. Individuals who benefit from caffeine ingestion may experience lower perception of effort during exercise and an accelerated recovery of M-wave amplitude postfatigue. This study elucidates the mechanisms of action of …

MalePhysiologyväsymysmedicine.medical_treatmentliikunta030204 cardiovascular system & hematologyRC1200H-Reflexchemistry.chemical_compound0302 clinical medicineTriceps surae muscletranscranial magnetic stimulationMedicinerasitusMotor NeuronsNutrition and DieteticsMotor CortexGeneral MedicineTranscranial Magnetic Stimulationhermo-lihastoimintamedicine.anatomical_structureNeuromuscular AgentsMuscle FatigueCaffeineMuscle ContractionAdultmedicine.medical_specialtyQP301.H75_Physiology._Sport.Postureperipheral fatiguePlacebo03 medical and health sciencesPhysical medicine and rehabilitationDouble-Blind MethodCaffeinePhysiology (medical)Humansrate of perceived exertionExercisesoleussuorituskykySoleus musclePyramidal tractsbusiness.industryEvoked Potentials MotorQPCrossover studycentral fatigueTranscranial magnetic stimulationchemistrykofeiiniSilent periodbusiness030217 neurology & neurosurgeryExperimental Physiology
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T Helper Cell Subsets in the Pleural Fluid of Tuberculous Patients Differentiate Patients With Non-Tuberculous Pleural Effusions

2021

BackgroundTuberculous pleural effusion (TPE) is one of the most common forms of extrapulmonary tuberculosis (Tb). Patients with TPE or malignant pleural effusions (MPE) frequently have a similar lymphocytic pleural fluid profile. Since the etiology of PE in various diseases is different, identifying the cellular components may provide diagnostic clues for understanding the pathogenesis.ObjectiveWe determined the frequency of T helper (Th) subtypes in the PEs for differentiation of Tb and non-Tb patients.MethodsThirty patients with TPE, 30 patients with MPE, 14 patients with empyema (EMP), and 14 patients with parapneumonic effusion (PPE) were enrolled between December 2018 and December 2019…

MalePleural effusionLYMPHOCYTESGastroenterologyParapneumonic effusionPathogenesistuberculouspleural effusionT-Lymphocyte Subsets1108 Medical MicrobiologyIMMUNE-RESPONSEImmunology and AllergyMedicineIL-2 receptorOriginal Researchmedicine.diagnostic_testMESOTHELIAL CELLSFOXP3Exudates and TransudatesT-Lymphocytes Helper-InducerTuberculosis PleuralT helper celldifferentiationMiddle Agedmedicine.anatomical_structure1107 ImmunologyfrequencyFemaleTH17 CELLSADENOSINE-DEAMINASELife Sciences & Biomedicinemedicine.medical_specialtyImmunologyDIAGNOSISFlow cytometryDiagnosis DifferentialPredictive Value of TestsInternal medicineT helperHumansLymphocyte CountScience & Technologybusiness.industryRC581-607medicine.diseaseEmpyemaROC CurveFeasibility StudiesImmunologic diseases. AllergybusinessFrontiers in Immunology
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Transport of the major myelin proteolipid protein is directed by VAMP3 and VAMP7.

2011

CNS myelination by oligodendrocytes requires directed transport of myelin membrane components and a timely and spatially controlled membrane expansion. In this study, we show the functional involvement of the R-solubleN-ethylmaleimide-sensitive factor attachment protein receptor (R-SNARE) proteins VAMP3/cellubrevin and VAMP7/TI-VAMP in myelin membrane trafficking. VAMP3 and VAMP7 colocalize with the major myelin proteolipid protein (PLP) in recycling endosomes and late endosomes/lysosomes, respectively. Interference with VAMP3 or VAMP7 function using small interfering RNA-mediated silencing and exogenous expression of dominant-negative proteins diminished transport of PLP to the oligodendro…

MaleProteolipid protein 1Vesicle-Associated Membrane Protein 3MESH: Myelin SheathMESH: R-SNARE Proteins[SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/NeurobiologyR-SNARE ProteinsMiceMyelin0302 clinical medicineMESH: Microscopy ImmunoelectronMESH: Genetic VectorsImage Processing Computer-AssistedMESH: AnimalsMicroscopy ImmunoelectronMESH: Myelin Proteolipid ProteinCells CulturedMyelin SheathMESH: Vesicle-Associated Membrane Protein 3VAMP30303 health sciencesMESH: ExocytosisGeneral NeuroscienceMESH: Enzyme-Linked Immunosorbent AssayArticlesImmunohistochemistryMESH: Image Processing Computer-AssistedMyelin proteolipid proteinCell biologymedicine.anatomical_structureElectrophoresis Polyacrylamide GelFemaleRNA InterferenceMESH: Cells CulturedEndosomeGenetic VectorsMESH: RNA InterferenceBiological Transport ActiveEnzyme-Linked Immunosorbent AssayEndosomesBiologyTransfectionExocytosisExocytosis03 medical and health sciencesMESH: Mice Inbred C57BLmedicineAnimalsSecretionMyelin Proteolipid ProteinMESH: MiceSecretory pathway030304 developmental biologyMESH: TransfectionCell MembraneMESH: ImmunohistochemistryMESH: MaleMice Inbred C57BLnervous systemMESH: EndosomesMESH: Biological Transport ActiveLysosomesMESH: Female030217 neurology & neurosurgeryMESH: LysosomesMESH: Cell MembraneMESH: Electrophoresis Polyacrylamide Gel
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Decreased FOXP3 expression in small airways of smokers with COPD

2008

CD4+CD25+ FOXP3-positive T-regulatory cells have an important role in controlling immune and inflammatory reactions. The present authors hypothesise that these cells may be involved in the pathogenesis of chronic obstructive pulmonary disease (COPD). The aim of the present study was to characterise the expression of FOXP3 in large and small airways of nonsmokers, smokers with normal lung function and COPD patients. A total of 19 nonsmokers, 20 smokers with normal lung function and 20 smokers with moderate COPD, undergoing lung resection for a solitary peripheral nonsmall cell carcinoma, were enrolled in the study. Immunohistochemical methods were used to evaluate FOXP3 expression in large a…

MalePulmonary and Respiratory MedicineLung NeoplasmsBronchiAsymptomaticPathogenesisPulmonary Disease Chronic ObstructiveImmune systemCarcinoma Non-Small-Cell LungForced Expiratory VolumemedicineCarcinomaHumansIL-2 receptorAgedCOPDbusiness.industrySmokingCase-control studyFOXP3Forkhead Transcription FactorsMiddle Agedrespiratory systemmedicine.diseaserespiratory tract diseasesCase-Control StudiesImmunologyFemalemedicine.symptombusinessEuropean Respiratory Journal
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Phosphodiesterase 4 inhibition decreases MUC5AC expression induced by epidermal growth factor in human airway epithelial cells

2005

Background: A common pathological feature of chronic inflammatory airway diseases such as asthma and chronic obstructive pulmonary disease (COPD) is mucus hypersecretion. MUC5AC is the predominant mucin gene expressed in healthy airways and is increased in asthmatic and COPD patients. Recent clinical trials indicate that phosphodiesterase type 4 (PDE4) inhibitors may have therapeutic value for COPD and asthma. However, their direct effects on mucin expression have been scarcely investigated. Methods: MUC5AC mRNA and protein expression were examined in cultured human airway epithelial cells (A549) and in human isolated bronchial tissue stimulated with epidermal growth factor (EGF; 25 ng/ml).…

MalePulmonary and Respiratory Medicinemedicine.medical_specialtyBlotting WesternBronchiEnzyme-Linked Immunosorbent AssayRespiratory MucosaMucin 5ACp38 Mitogen-Activated Protein KinasesWestern blotEpidermal growth factorInternal medicineGene expressionCyclic AMPmedicineHumansRNA MessengerPhosphotyrosineCells CulturedRoflumilastRolipramAgedA549 cellEpidermal Growth Factormedicine.diagnostic_testReverse Transcriptase Polymerase Chain Reactionbusiness.industryCilomilastMucinMucinsEpithelial CellsMiddle Agedrespiratory systemMolecular biologyRecombinant ProteinsCyclic Nucleotide Phosphodiesterases Type 4respiratory tract diseasesEndocrinology3'5'-Cyclic-AMP PhosphodiesterasesAirway BiologyFemalebusinessmedicine.drugThorax
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In vitro anti-inflammatory effects of AZD8999, a novel bifunctional muscarinic acetylcholine receptor antagonist /β2-adrenoceptor agonist (MABA) comp…

2019

Recent evidence indicates that AZD8999 (LAS190792), a novel muscarinic acetylcholine receptor antagonist and β2-adrenoceptor agonist (MABA) in development for chronic respiratory diseases, induces potent and sustained relaxant effects in human bronchi by adressing both muscarinic acetylcholine receptors and β2-adrenoceptor. However, the anti-inflammatory effects of the AZD8999 monotherapy or in combination with corticosteroids are unknown. This study investigates the anti-inflammatory effects of AZD8999 in monotherapy and combined with fluticasone propionate in neutrophils from healthy and chronic obstructive pulmonary disease (COPD) patients. Peripheral blood neutrophils from healthy and C…

MalePulmonologyNeutrophilsPhysiologyAnti-Inflammatory AgentsPharmacologyPathology and Laboratory MedicineBiochemistryPulmonary Disease Chronic ObstructiveWhite Blood CellsGlucocorticoid receptorAnimal CellsMuscarinic acetylcholine receptorMedicine and Health SciencesPost-Translational ModificationPhosphorylationReceptorImmune ResponseMultidisciplinaryPharmaceuticsQRDrug SynergismMiddle AgedReceptors MuscarinicHealthy VolunteersBody FluidsChemistryBloodPhysical SciencesQuinolinesMedicineDrug Therapy CombinationFemalemedicine.symptomCellular TypesAnatomymedicine.drugResearch ArticleSignal TransductionAgonistTransmembrane Receptorsmedicine.drug_classp38 mitogen-activated protein kinasesChronic Obstructive Pulmonary DiseaseImmune CellsScienceImmunologyInflammationMuscarinic AntagonistsThiophenesFluticasone propionateSigns and SymptomsDrug TherapyCyclohexanesDiagnostic MedicinemedicineHumansAdrenergic beta-2 Receptor AgonistsAgedInflammationBlood CellsDose-Response Relationship Drugbusiness.industryAntagonistChemical CompoundsBiology and Life SciencesProteinsCell BiologyAcetylcholine ReceptorsFluticasoneMuscarinic Acetylcholine ReceptorsReceptors Adrenergic beta-2PropionatesbusinessReceptor Antagonist TherapyPLoS ONE
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Epigenetic Control of the foxp3 Locus in Regulatory T Cells

2007

Compelling evidence suggests that the transcription factor Foxp3 acts as a master switch governing the development and function of CD4+ regulatory T cells (Tregs). However, whether transcriptional control of Foxp3 expression itself contributes to the development of a stable Treg lineage has thus far not been investigated. We here identified an evolutionarily conserved region within the foxp3 locus upstream of exon-1 possessing transcriptional activity. Bisulphite sequencing and chromatin immunoprecipitation revealed complete demethylation of CpG motifs as well as histone modifications within the conserved region in ex vivo isolated Foxp3+CD25+CD4+ Tregs, but not in naïve CD25−CD4+ T cells. …

MaleQH301-705.5Bisulfite sequencingImmunologyMolecular Sequence Datachemical and pharmacologic phenomenaCell SeparationThymus GlandBiologyT-Lymphocytes RegulatoryGeneral Biochemistry Genetics and Molecular BiologyEpigenesis GeneticMiceTranscriptional regulationAnimalsEpigeneticsBiology (General)Regulation of gene expressionMice Inbred BALB CGeneral Immunology and MicrobiologyBase SequenceGeneral NeuroscienceInterleukin-2 Receptor alpha SubunitFOXP3Homo (human)hemic and immune systemsForkhead Transcription FactorsDNA MethylationFlow CytometryMolecular biologyMus (mouse)Cell biologyIn VitroDNA demethylationGene Expression RegulationDNA methylationCpG IslandsGeneral Agricultural and Biological SciencesChromatin immunoprecipitationResearch ArticlePLoS Biology
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