Search results for "PAM"

showing 10 items of 1768 documents

A longitudinal study on the information needs and preferences of patients after an acute coronary syndrome.

2016

Background Research has shown that the provision of pertinent health information to patients with cardiovascular disease is associated with better adherence to medical prescriptions, behavioral changes, and enhanced perception of control over the disease. Yet there is no clear knowledge on how to improve information pertinence. Identifying and meeting the information needs of patients and their preferences for sources of information is pivotal to developing patient-led services. This prospective, observational study was aimed at exploring the information needs and perceived relevance of different information sources for patients during the twenty-four months following an acute coronary synd…

MaleLongitudinal studyActivities of daily livingHealth BehaviorLongitudinal researchFriends030204 cardiovascular system & hematology0302 clinical medicineInformation seeking behaviorSurveys and Questionnaires030212 general & internal medicineLongitudinal StudiesProspective StudiesPatient PreferenceMiddle AgedCardiovascular diseaseNeeds assessmentFemaleTelevisionPeriodicals as TopicFamily PracticeInformation needs; Acute coronary syndrome; Cardiovascular disease; Health information sources; Longitudinal researchNeeds AssessmentSpecializationResearch ArticleSettore M-PSI/01 - Psicologia GeneraleAdultmedicine.medical_specialtyAcute coronary syndrome; Cardiovascular disease; Health information sources; Information needs; Longitudinal research; Adult; Aged; Family; Female; Friends; General Practitioners; Health Behavior; Humans; Internet; Longitudinal Studies; Male; Middle Aged; Needs Assessment; Pamphlets; Periodicals as Topic; Prospective Studies; Specialization; Surveys and Questionnaires; Television; Acute Coronary Syndrome; Information Seeking Behavior; Patient Education as Topic; Patient Preference; Family PracticeInformation Seeking BehaviorInformation needsHealth information sources03 medical and health sciencesPatient Education as TopicGeneral PractitionersmedicineHumansRelevance (information retrieval)FamilyAcute Coronary SyndromeAgedInternetInformation needbusiness.industryRepeated measures designInformation needsHealth information sourcePhysical therapyObservational studyPamphletsbusinessBMC family practice
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Social defeat-induced increase in the conditioned rewarding effects of cocaine: Role of CX3CL1

2019

Abstract Social stress is associated with higher vulnerability to drug use, as it enhances the reinforcing effects of psychostimulants in rodents. Furthermore, continued or severe stress induces a proinflammatory state of microglial activation and augmented cytokine production. The aim of the present work was to evaluate the role of fractalkine [C-X3-C motif ligand 1 (CX3CL1)], an inflammatory chemokine, in the increased conditioned rewarding effects of cocaine in animals exposed to social defeat stress. In addition, we measured the signaling cascade pathway of CX3CL1 in the hippocampus (HPC) (including p-ERK/ERK, p-p38/p38 MAPK, p-p65/p65 NFκB and p-CREB/CREB ratios). The glutamate recepto…

MaleMAPK/ERK pathwaymedicine.medical_specialtyCREBSocial DefeatSocial defeatMice03 medical and health sciences0302 clinical medicineCocaineDopamine Uptake InhibitorsRewardInternal medicineConditioning PsychologicalCX3CR1AnimalsMedicineCX3CL1Biological PsychiatryMice KnockoutPharmacologySocial stressbiologyChemokine CX3CL1business.industryGlutamate receptorConditioned place preference030227 psychiatryMice Inbred C57BLEndocrinologybiology.proteinbusinessProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Understanding Cannabinoid Psychoactivity with Mouse Genetic Models

2007

Marijuana and its main psychotropic ingredient Δ9-tetrahydrocannabinol (THC) exert a plethora of psychoactive effects through the activation of the neuronal cannabinoid receptor type 1 (CB1), which is expressed by different neuronal subpopulations in the central nervous system. The exact neuroanatomical substrates underlying each effect of THC are, however, not known. We tested locomotor, hypothermic, analgesic, and cataleptic effects of THC in conditional knockout mouse lines, which lack the expression of CB1 in different neuronal subpopulations, including principal brain neurons, GABAergic neurons (those that release γ aminobutyric acid), cortical glutamatergic neurons, and neurons expres…

MaleMESH: Body TemperatureCannabinoid receptormedicine.medical_treatmentGene ExpressionMESH: Receptor Cannabinoid CB1NeocortexMESH: gamma-Aminobutyric AcidMESH: CatalepsyPharmacologyHippocampusMESH: Mice KnockoutMESH: Corpus StriatumBody TemperatureMESH: Autonomic Nervous SystemMESH: NeocortexMice0302 clinical medicineReceptor Cannabinoid CB1MESH: Behavior AnimalCannabinoid receptor type 1MESH: AnimalsMESH: Gene SilencingDronabinolMESH: NociceptorsBiology (General)gamma-Aminobutyric AcidMice Knockout0303 health sciencesBehavior Animalmusculoskeletal neural and ocular physiologyGeneral NeuroscienceMESH: Pain ThresholdNociceptorsMESH: Glutamic AcidMESH: InterneuronsMESH: Motor Activity3. Good healthGABAergicMESH: TetrahydrocannabinolGeneral Agricultural and Biological SciencesResearch Articlemedicine.drugPain ThresholdMESH: Gene ExpressionMESH: Psychotropic DrugsQH301-705.5Glutamic AcidMotor ActivityBiologyAutonomic Nervous SystemGeneral Biochemistry Genetics and Molecular Biologygamma-Aminobutyric acid03 medical and health sciencesGlutamatergicDopamine receptor D1InterneuronsCannabinoid Receptor Modulatorsmental disorders[SDV.BBM] Life Sciences [q-bio]/Biochemistry Molecular BiologymedicineAnimalsGenetic Predisposition to Disease[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyGene SilencingTetrahydrocannabinolMESH: MiceAnesthesiology and Pain Management030304 developmental biologyPharmacologyCatalepsyPsychotropic DrugsModels GeneticGeneral Immunology and MicrobiologyCannabinoidsIllicit Drugsorganic chemicalsMESH: MaleCorpus StriatumPrimerDisease Models Animalnervous systemCannabinoidNervous System Diseases030217 neurology & neurosurgeryNeurosciencePLoS Biology
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Clozapine: Strong antiaggressive effects with minimal motor impairment

1992

Abstract Clinical studies have shown clozapine to be effective in the treatment of schizophrenia and associated with an extremely low incidence of extrapiramidal side effects. Diverse studies indicate that clozapine is an atypical neuroleptic with a preferential activity on the mesolimbic structures and a lower affinity for striatal D2 receptors than the classical antipsychotics. The purpose of this study was to assess the behavioral properties of clozapine, especially its effects on aggressive and motor behaviors. Individually housed male mice of the OF1 strain were exposed to anosmic “standard opponents” 30 minutes after the last drug administration. One category of animals received a sin…

MaleMale miceExperimental and Cognitive PsychologyAtypical neurolepticMotor ActivityPharmacologyMiceBehavioral NeuroscienceDopamine receptor D2medicineAnimalsClozapineClozapineDose-Response Relationship DrugDrug administrationMotor impairmentmedicine.diseaseAggressionLower affinityMotor SkillsSchizophreniaAnesthesiaArousalPsychologyPsychomotor Performancemedicine.drugPhysiology & Behavior
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Effects of risperidone and SCH 23390 on isolation-induced aggression in male mice.

1998

In this study, the antiaggressive effects of risperidone and SCH 23390 have been explored. Using the paradigm of isolation-induced aggression, 150 albino male mice of the OF1 strain were allocated to control and experimental groups which received three doses of risperidone (0.01, 0.05 and 0.1 mg/kg) or two doses of SCH 23390 (0.05 and 0.1 mg/kg). Only the highest doses of risperidone decreased threat and attack behaviours but all doses significantly impaired motor behaviour. SCH 23390 decreased attack with the two doses used and also produced significant increases in immobility. Although both antipsychotics are antiaggressive, this action seems to be more specific in the case of risperidone…

MaleMale micePharmacologyNeurotransmissionMotor Activitychemistry.chemical_compoundMiceSexual Behavior AnimalDopaminemedicineAnimalsPharmacology (medical)Biological PsychiatryPharmacologySCH-23390RisperidoneAggressionReceptors Dopamine D1BenzazepinesRisperidoneGroomingAggressionPsychiatry and Mental healthDopamine D2 Receptor AntagonistsNeurologychemistryIsolation induced aggressionSocial IsolationDepression ChemicalExploratory BehaviorDopamine AntagonistsFemaleNeurology (clinical)Serotoninmedicine.symptomPsychologymedicine.drugEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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Acceleration of glutathione efflux and inhibition of gamma-glutamyltranspeptidase sensitize metastatic B16 melanoma cells to endothelium-induced cyto…

2005

Highly metastatic B16 melanoma (B16M)-F10 cells, as compared with the low metastatic B16M-F1 line, have higher GSH content and preferentially overexpress BCL-2. In addition to its anti-apoptotic properties, BCL-2 inhibits efflux of GSH from B16M-F10 cells and thereby may facilitate metastatic cell resistance against endothelium-induced oxidative/nitrosative stress. Thus, we investigated in B16M-F10 cells which molecular mechanisms channel GSH release and whether their modulation may influence metastatic activity. GSH efflux was abolished in multidrug resistance protein 1 knock-out (MRP-/-1) B16M-F10 transfected with the Bcl-2 gene or in MRP-/-1 B16M-F10 cells incubated with l-methionine, wh…

MaleMelanoma ExperimentalCystic Fibrosis Transmembrane Conductance RegulatorApoptosisBiochemistryOligodeoxyribonucleotides Antisensechemistry.chemical_compoundMiceCell AdhesionAnimalsEndotheliumNeoplasm MetastasisCytotoxicityCell adhesionMolecular BiologybiologyActivator (genetics)Cell BiologyGlutathioneTransfectiongamma-GlutamyltransferaseMolecular biologyGlutathioneCystic fibrosis transmembrane conductance regulatorMice Inbred C57BLKineticsOxidative StresschemistryProto-Oncogene Proteins c-bcl-2VerapamilApoptosisbiology.proteinEffluxMultidrug Resistance-Associated ProteinsThe Journal of biological chemistry
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Brain met-enkephalin immunostaining after subacute and subchronic exposure to benzene

1994

Benzene is used in a wide variety of domestic and occupational activities, and due to its lipophilic nature, it accumulates in lipid-rich tissues like the brain. In this sense, neurotoxic action has long been associated with organic solvent exposure and it has been shown that benzene, injected in a single dose or during a prolongued administration, modifies the content of dopamine, noradrenaline, serotonin and its main metabolite 5-hydroxy indolacetic acid, in several brain regions of the rat, then revealing a stimulating action on brain monoamine synthesis and turnover. However, information concerning neurotoxic action of benzene exposure in vivo on peptidergic neuromodulatory systems is s…

MaleMet-enkephalinmedicine.medical_specialtyTime FactorsEnkephalin MethionineHealth Toxicology and MutagenesisCentral nervous systemNeuropeptideBiologyToxicologyRats Sprague-Dawleychemistry.chemical_compoundDopamineInternal medicineImage Processing Computer-AssistedmedicineAnimalsBrain ChemistryStaining and LabelingProteolytic enzymesBrainBenzeneGeneral MedicinePollutionRatsEndocrinologyMonoamine neurotransmittermedicine.anatomical_structurechemistryImmunologic TechniquesSerotoninImmunostainingmedicine.drugBulletin of Environmental Contamination and Toxicology
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NMDA glutamate but not dopamine antagonists blocks drug-induced reinstatement of morphine place preference.

2004

The effects of dopaminergic and glutamatergic antagonists on the drug-induced reinstatement of a previously extinguished morphine conditioned place preference (CPP) in mice were evaluated. Following extinction of a place preference induced by morphine (40 mg/kg), a non-contingent injection of the dopaminergic antagonists SCH 23390 (0.125, 0.5 mg/kg), raclopride (0.3, 1.2 mg/kg), haloperidol (0.1, 0.2 mg/kg) and the dopamine (DA) release inhibitor CGS 10746B (1, 10 mg/kg) or glutamatergic NMDA antagonists memantine (10, 20, 40 mg/kg) and MK-801 (0.1, 0.2, 0.3 mg/kg) alone or with 10 mg/kg morphine was given. Neither the dopaminergic nor the glutamatergic antagonists alone reinstated the plac…

MaleMice Inbred StrainsPharmacologyReceptors N-Methyl-D-AspartateExtinction PsychologicalGlutamatergicMiceDopaminemedicineHaloperidolAnimalsDrug InteractionsRacloprideAnalysis of VarianceBehavior AnimalDose-Response Relationship DrugMorphineChemistryGeneral NeuroscienceDopaminergicMemantineConditioned place preferenceAnalgesics OpioidNMDA receptorConditioning OperantDopamine AntagonistsExcitatory Amino Acid Antagonistsmedicine.drugBrain research bulletin
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7-nitroindazole protects striatal dopaminergic neurons against MPP+-induced degeneration: an in vivo microdialysis study.

2007

The neuropathological hallmark of Parkinson's disease (PD) is the selective degeneration of dopaminergic (DAergic) neurons in the substantia nigra pars compacta (SNc). In this study, using a microdialysis technique, we investigated whether an inhibitor of neuronal nitric oxide synthase (nNOS), 7-nitrindazole (7-NI), could protect against DAergic neuronal damage induced by in vivo infusion of 1-methyl-4-phenylpiridinium iodide (MPP(+)) in freely moving rats. Experiments were performed over 2 days in three groups of rats: (a) nonlesioned, (b) MPP(+)-lesioned, and (c) 7-NI pretreated MPP(+)-lesioned rats. On day 1, control rats were perfused with an artificial CSF, while 1 mM MPP(+) was infuse…

MaleMicrodialysis1-Methyl-4-phenylpyridinium7-NitroindazoleIndazolesDopamineMicrodialysisSubstantia nigraStriatumNitric Oxide Synthase Type IPharmacologyNeuroprotectionGeneral Biochemistry Genetics and Molecular BiologyRats Sprague-Dawleychemistry.chemical_compoundHistory and Philosophy of SciencemedicineAnimalsEnzyme InhibitorsNeuronsPars compactaChemistryGeneral NeuroscienceDopaminergicNeurotoxicityParkinson Diseasemedicine.diseaseRatsSubstantia NigraDisease Models AnimalNeuroprotective Agentsnervous systemNeuroscienceAnnals of the New York Academy of Sciences
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Induction of brain CYP2E1 changes the effects of ethanol on dopamine release in nucleus accumbens shell.

2009

CYP2E1 is an important enzyme involved in the brain metabolism of ethanol that can be induced by chronic consumption of alcohol. Recent works have highlighted the importance of this system in the context of the behavioural effects of ethanol. Unfortunately, the underlying neurochemical events for these behavioural changes, has not been yet explored. In this work, we have started this exploration by analyzing the possible changes in the neurochemical response of the mesolimbic system to ethanol after pharmacological induction of brain CYP2E1. We have used the dopamine extracellular levels in nucleus accumbens (NAc) core and shell, measured by means of microdialysis in vivo, as an index of th…

MaleMicrodialysisDopamineContext (language use)Nucleus accumbensPharmacologyToxicologyNucleus Accumbenschemistry.chemical_compoundNeurochemicalDopaminemedicineAnimalsPharmacology (medical)Rats WistarNeurotransmitterInfusions IntravenousPharmacologyEthanolEthanolBrainCytochrome P-450 CYP2E1RatsPsychiatry and Mental healthchemistryEnzyme InductionCatecholamineNeurosciencemedicine.drugDrug and alcohol dependence
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