Search results for "PATHOGENESIS"

showing 10 items of 761 documents

Could Chronic Idiopatic Intestinal Pseudo-Obstruction Be Related to Viral Infections?

2021

Chronic idiopathic intestinal pseudo-obstruction (CIIPO) is a disease characterized by symptoms and signs of small bowel obstruction in the absence of displayable mechanical obstruction. Due to the known neuropathic capacity of several viruses, and their localization in the intestine, it has been hypothesized that such viruses could be involved in the pathogenesis of CIIPO. The most frequently involved viruses are John Cunningham virus, Herpesviridae, Flaviviruses, Epstein–Barr virus and Citomegalovirus. Therefore, the present narrative review aims to sum up some new perspectives in the etiology and pathophysiology of CIIPO.

Intestinal pseudo-obstructionmedicine.medical_specialtyviruseslcsh:MedicineReviewvirusDiseasemedicine.disease_causeGastroenterologyHerpesviridaeVirusPathogenesis03 medical and health sciences0302 clinical medicineInternal medicinemedicinebusiness.industrylcsh:Rgastrointestinal motility disordersGeneral Medicinemedicine.diseasePathophysiologyBowel obstructionchronic idiopathic intestinal pseudo-obstruction030220 oncology & carcinogenesisEtiology030211 gastroenterology & hepatologybusinessJournal of Clinical Medicine
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Presence of Blastocystis in gut microbiota is associated with cognitive traits and decreased executive function.

2022

Growing evidence implicates the gut microbiome in cognition. Blastocystis is a common gut single-cell eukaryote parasite frequently detected in humans but its potential involvement in human pathophysiology has been poorly characterized. Here we describe how the presence of Blastocystis in the gut microbiome was associated with deficits in executive function and altered gut bacterial composition in a discovery (n = 114) and replication cohorts (n = 942). We also found that Blastocystis was linked to bacterial functions related to aromatic amino acids metabolism and folate-mediated pyrimidine and one-carbon metabolism. Blastocystis-associated shifts in bacterial functionality translated into …

Intestins--MicrobiologiaMicrobiologiaPathogenesisBlastocystis InfectionsMicrobiologyMETAGENOMICSMEMBERExecutive FunctionMicegut microbiota ; Blastocystis ; executive function ; gut microbiome-brain axis.CognitionAparell digestiuDiagnosisAnimalsHumansEcology Evolution Behavior and SystematicsBlastocistMEMORYDNAGastrointestinal MicrobiomeCognicióBlastocystisBlastocist -- InfeccióMicrobiomaBiomarkersThe ISME journal
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Current Concepts and Perspectives on Brain Arteriovenous Malformations: A Review of Pathogenesis and Multidisciplinary Treatment.

2021

Brain arteriovenous malformations (bAVMs) are unusual vascular pathologies characterized by the abnormal aggregation of dilated arteries and veins in the brain parenchyma and for which the absence of a normal vascular structure and capillary bed leads to direct connections between arteries and veins. Although bAVMs have long been believed to be congenital anomalies that develop during the prenatal period, current studies show that inflammation is associated with AVM genesis, growth, and rupture. Interventional treatment options include microsurgery, stereotactic radiosurgery, and endovascular embolization, and management often comprises a multidisciplinary combination of these modalities. T…

Intracranial Arteriovenous Malformationsmedicine.medical_specialtyMicrosurgerymedicine.medical_treatmentNeurosurgeryArteriovenous shuntNervous System MalformationsRadiosurgeryRadiosurgeryPathogenesisArteriovenous malformationMultidisciplinary approachmedicineHumansEmbolizationRetrospective StudiesNatural courseInterventional treatmentbusiness.industryBrainMicrosurgeryAVMEmbolization TherapeuticPathobiologyTreatment OutcomeMultimodalSurgeryNeurology (clinical)NeurosurgeryRadiologybusinessWorld neurosurgery
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European expert opinion on the management of invasive candidiasis in adults

2011

Item does not contain fulltext This report discusses the present status of antifungal therapy and treatment options for candidaemia, considered by experts in the field in Europe. A conference of 26 experts from 13 European countries was held to discuss strategies for the treatment and prevention of invasive candidiasis, with the aim of providing a review on optimal management strategies. Published and unpublished comparative trials on antifungal therapy were analysed and discussed. Commonly asked questions about the management of candidaemia were selected, and possible responses to these questions were discussed. Panellists were then asked to respond to each question by using a touchpad ans…

Invasive mycoses and compromised host Translational research [N4i 2]AdultMicrobiology (medical)medicine.medical_specialtyAntifungal Agentsmedicine.medical_treatmentPlacebo-controlled studyMEDLINEInvasive mycoses and compromised host Infection and autoimmunity [N4i 2]MicrobiologyMedicineHumanscandidaemiaCandidiasis InvasiveguidelinesIntensive care medicineWatchful Waitingtherapybusiness.industryTreatment optionsPathogenesis and modulation of inflammation Infection and autoimmunity [N4i 1]General MedicineInvasive candidiasisComparative trialAntibiotic Prophylaxismedicine.diseaseOptimal managementEuropeIntensive Care UnitsInfectious DiseasesconsensusExpert opinioncandidabusinessWatchful waitingClinical Microbiology and Infection
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Cicatrices chéloïdes de la tête et du cou

2012

A keloid scar is a benign proliferative lesion of dermic collagen. It is predominant in black skin patients. It is most commonly located on the head and neck. Skin trauma and a genetic predisposition may be responsible for the keloid scar. Nevertheless, the pathogenesis of keloid scar is still unclear, and no currently available treatment is 100% effective. The authors had for aim to review the current data on keloid scar pathogenesis and treatment for an optimal management of this condition.

Keloid scarsmedicine.medical_specialtybusiness.industryProliferative lesionPathogenicitymedicine.diseaseDermatologyOptimal managementSurgeryPathogenesisKeloidOtorhinolaryngologymedicineGenetic predispositionSurgeryOral Surgeryskin and connective tissue diseasesHead and neckbusinessRevue de Stomatologie et de Chirurgie Maxillo-faciale
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Characterization of disease-specific cellular abundance profiles of chronic inflammatory skin conditions from deconvolution of biopsy samples

2019

Background Psoriasis and atopic dermatitis are two inflammatory skin diseases with a high prevalence and a significant burden on the patients. Underlying molecular mechanisms include chronic inflammation and abnormal proliferation. However, the cell types contributing to these molecular mechanisms are much less understood. Recently, deconvolution methodologies have allowed the digital quantification of cell types in bulk tissue based on mRNA expression data from biopsies. Using these methods to study the cellular composition of the skin enables the rapid enumeration of multiple cell types, providing insight into the numerical changes of cell types associated with chronic inflammatory skin c…

Keratinocytes0301 basic medicinePathologyMicroarraysBiopsyPATHOGENESISTranscriptome0302 clinical medicineDatabases GeneticLeukocytesATOPIC-DERMATITISGenetics (clinical)SkinPSORIASISmedicine.diagnostic_testintegumentary systemAtopic dermatitisDermismedicine.anatomical_structureDIFFERENTIATION030220 oncology & carcinogenesisChronic inflammatory skin diseasesResearch ArticleEXPRESSIONlcsh:Internal medicinemedicine.medical_specialtyCell typeGENESlcsh:QH426-470BiologyDENDRITIC CELLSDermatitis AtopicFlow cytometryMECHANISMS03 medical and health sciencesDermisPsoriasisBiopsyGeneticsmedicineHumanslcsh:RC31-1245SIGNATURESInflammationIDENTIFICATIONReproducibility of Resultsmedicine.diseaselcsh:Genetics030104 developmental biologyGene Expression RegulationChronic DiseaseSkin biopsyGene expressionEpidermis
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Identification of avarol derivatives as potential antipsoriatic drugs using an in vitro model for keratinocyte growth and differentiation.

2006

Contains fulltext : 49512schalkwijk.pdf (Publisher’s version ) (Closed access) Avarol, a marine sesquiterpenoid hydroquinone, and 14 avarol derivatives have shown interesting anti-inflammatory properties in previous studies. In this study, avarol and derivatives were evaluated in high-throughput keratinocyte culture models using cytokeratin 10 and SKALP/Elafin expression as markers for respectively normal and psoriatic differentiation. Avarol and five of its derivatives (5, 10, 13, 14 and 15) were selected for further study. Only 10, 13, 14 and 15 were able to inhibit keratinocyte cell growth. Changes in expression levels of 22 genes were assessed by quantitative real time PCR (qPCR). From …

KeratinocytesDrug Evaluation PreclinicalAntineoplastic AgentsEnzyme-Linked Immunosorbent AssayIn Vitro TechniquesBiologyGeneral Biochemistry Genetics and Molecular BiologyDownregulation and upregulationTranslational research [ONCOL 3]DysideaGene expressionDithranolmedicineAnimalsHumansPsoriasisRNA MessengerGeneral Pharmacology Toxicology and PharmaceuticsCells CulturedCell ProliferationChronic inflammation and autoimmunity [UMCN 4.2]Messenger RNATumor Necrosis Factor-alphaCell growthInterleukin-8Membrane ProteinsCell DifferentiationGeneral MedicineMolecular biologyElafinPathogenesis and modulation of inflammation [N4i 1]medicine.anatomical_structureMechanism of actionCyclooxygenase 2KeratinsClinical Pharmacology and physiology [CTR 2]medicine.symptomKeratinocyteSesquiterpenesInfection and autoimmunity [NCMLS 1]Elafinmedicine.drug
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Collagenolytic proteinases in keratoconus.

2006

To study the proteolytic phenomena contributing to the pathogenesis of keratoconus, corneal enzymes with potential to cleave fibrillar collagen were studied.Immunohistochemical labeling was undertaken of conventional and novel mammalian collagenases (MMP-1, -2, -8, -13, and -14) of the matrix metalloproteinase (MMP) family and other collagenolytic proteinases of the serine (human trypsin-2) and cysteine (cathepsin K) endoproteinase families. The results were analyzed using a semiquantitative scoring system.Labeling of MMP-8 was moderate in healthy controls, but weak in keratoconus. Moderate MMP-2 and weak MMP-14 expressions were similar in controls and keratoconus. MMP-1 was slightly overex…

KeratoconusPathologymedicine.medical_specialtygenetic structuresCathepsin KMatrix metalloproteinaseKeratoconusPathogenesisCorneaImmunoenzyme TechniquesCorneamedicineCathepsin KHumansTrypsinCollagenaseschemistry.chemical_classificationmedicine.diseaseCathepsinseye diseasesMatrix MetalloproteinasesOphthalmologyEnzymemedicine.anatomical_structurechemistryCollagenaseTrypsinogenImmunohistochemistrysense organsKeratoplasty Penetratingmedicine.drugCornea
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Renal tubular dysgenesis (RTD) - an important cause of the oligohydramnion-sequence

2000

Renal tubular dysgenesis (RTD) is a disorder characterized by neonatal renal failure and regular gross renal architecture, although the histological features of immature and shortened proximal tubules lead to neonatal death. The pathogenesis of this condition includes a congenital familial condition, a twin-twin transfusion syndrome, and an angiotensin-converting enzyme inhibitor intake by the mother. The clinical picture shows an association with oligohydramnia, pulmonary hypoplasia, and skull ossification defects. In the present paper, we report the occurrence of RTD in three infants of a consanguinous couple and compared our data with those of the literature. Our data confirm that late s…

KidneyPregnancyPathologymedicine.medical_specialtybusiness.industryCell BiologyConsanguinitymedicine.diseasePathology and Forensic MedicinePathogenesisPulmonary hypoplasiaSkullmedicine.anatomical_structuremedicineImmunohistochemistrybusinessSequence (medicine)Pathology - Research and Practice
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TD-09 IL-34 promotes macrophage-mediated lupus nephritis in MRL-Faslpr mice

2018

Background Nephritis is the major cause of mortality and morbidity in patients with lupus. Macrophages (Mo) are central to kidney destruction in lupus-prone mice and patients. CSF-1, and the newly identified IL-34, mediate Mo survival and proliferation. However, IL-34 and CSF-1 differ during development and disease. While CSF-1 and IL-34 share the CSF-1 receptor (cFMS), expressed by Mo, IL-34 binds to a second receptor, Protein-Tyrosine Phosphatase ζ (PTPRZ) in inflamed kidneys. Intra-renal IL-34, cFMS, and PTPRZ are increased during the progression of lupus nephritis in MRL-Faslpr mice. Therefore, we hypothesized that IL-34 is a potential therapeutic target for lupus nephritis. Methods and…

KidneySystemic lupus erythematosusbusiness.industryLupus nephritisMonocyte proliferationmedicine.diseasePathogenesismedicine.anatomical_structureImmunologymedicineBone marrowbusinessReceptorNephritisTissue Damage
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