Search results for "PATHOGENESIS"

showing 10 items of 761 documents

Cytomegalovirus-Associated Inhibition of Hematopoiesis Is Preventable by Cytoimmunotherapy With Antiviral CD8 T Cells

2020

Reactivation of latent cytomegalovirus (CMV) in recipients of hematopoietic cell transplantation (HCT) not only results in severe organ manifestations, but can also cause "graft failure" resulting in bone marrow (BM) aplasia. This inhibition of hematopoietic stem and progenitor cell engraftment is a manifestation of CMV infection that is long known in clinical hematology as "myelosuppression." Previous studies in a murine model of sex-chromosome mismatched but otherwise syngeneic HCT and infection with murine CMV have shown that transplanted hematopoietic cells (HC) initially home to the BM stroma of recipients but then fail to further divide and differentiate. Data from this model were in …

Male0301 basic medicineMicrobiology (medical)Stromal cellmurine cytomegalovirusgraft failuremedicine.medical_treatment030106 microbiologyImmunologylcsh:QR1-502CytomegalovirusCD8-Positive T-LymphocytesAntiviral AgentsMicrobiologylcsh:Microbiologybone marrow stromaProgenitor Cell Engraftmenthematopoietic (stem) cell transplantation (HCT HSCT)Mice03 medical and health sciencesCellular and Infection MicrobiologymedicineAnimalsCytotoxic T cellmyelosuppressionbusiness.industryhematopoietic reconstitutionImmunotherapyBrief Research Reportcytomegalovirus pathogenesisHematopoiesisTransplantationHaematopoiesis030104 developmental biologyInfectious Diseasesmedicine.anatomical_structureCytomegalovirus InfectionsImmunologyFemaleimmunotherapyBone marrowbusinessCD8Frontiers in Cellular and Infection Microbiology
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Potential Involvement of Osteopontin in Inflammatory and Fibrotic Processes in Pulmonary Embolism and Chronic Thromboembolic Pulmonary Hypertension

2019

Background Inflammation and incomplete thrombus resolution leading to obstructive fibrotic remodelling are considered critical mechanisms for the development of chronic thromboembolic pulmonary hypertension (CTEPH) after pulmonary embolism (PE). Osteopontin (OPN) is involved in a variety of biological processes including inflammation and tissue fibrosis. Methods OPN plasma concentrations were measured in 70 CTEPH and 119 PE patients. Tissue material from 6 CTEPH patients removed during pulmonary endarterectomy and murine venous thrombi induced by subtotal ligation of the inferior vena cava in C57BL/6 mice were analysed by (immuno)histochemistry. Results CTEPH patients had higher OPN plasma…

Male0301 basic medicinePathologymedicine.medical_specialtyHypertension Pulmonarymedicine.medical_treatmentInflammationEndarterectomy030204 cardiovascular system & hematologyInferior vena cavaTranslational Research BiomedicalPathogenesisMice03 medical and health sciences0302 clinical medicinestomatognathic systemFibrosisThromboembolismmedicineAnimalsHumansProspective StudiesOsteopontinThrombusMyofibroblastsAgedEndarterectomyInflammationbiologybusiness.industryThrombosisHematologyMiddle AgedPrognosismedicine.diseaseFibrosisPulmonary embolismMice Inbred C57BL030104 developmental biologymedicine.veinChronic Diseasebiology.proteinFemaleOsteopontinmedicine.symptomPulmonary EmbolismbusinessBiomarkersThrombosis and Haemostasis
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Comprehensive platelet phenotyping supports the role of platelets in the pathogenesis of acute venous thromboembolism - results from clinical observa…

2020

Background: The pathogenesis of arterial and venous thrombosis is in large part interlaced. How much platelet phenotype relates to acute venous thromboembolism (VTE) independent of the underlying cardiovascular profile is presently poorly investigated.Methods: Platelet count and mean platelet volume (MPV), platelet aggregation in whole blood and platelet rich plasma (PRP), platelet-dependent thrombin generation (TG) and platelet surface activation markers were measured under standardized conditions. Machine learning was applied to identify the most relevant characteristics associated with VTE from a large array (N = 58) of clinical and plateletrelated variables.Findings: VTE cases (N = 159)…

Male0301 basic medicinePlatelet Aggregationlcsh:MedicineDETERMINANTSGastroenterologyMachine LearningPathogenesisACTIVATION0302 clinical medicineRisk FactorsPlateletWhole bloodlcsh:R5-920AspirinOUTCOMESThrombinVenous ThromboembolismGeneral MedicineMiddle AgedThrombosisVenous thrombosis030220 oncology & carcinogenesisAcute DiseaseFemaleDisease Susceptibilitylcsh:Medicine (General)Research Papermedicine.drugBlood Plateletsmedicine.medical_specialtyPlatelet Function TestsGeneral Biochemistry Genetics and Molecular BiologyImmunophenotyping03 medical and health sciencesACUTE PULMONARY-EMBOLISMRISK-FACTORInternal medicinemedicineHumansMean platelet volumeMETAANALYSISAgedPlatelet Countbusiness.industrylcsh:RPlatelet Activationmedicine.diseasePREVENTIONASPIRINTHROMBOSIS030104 developmental biologyPlatelet-rich plasmaVOLUMEbusinessBiomarkersEBioMedicine
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Toxoplasma gondii seroprevalence varies by cat breed.

2017

Toxoplasma gondii is a widespread zoonotic parasite that is relevant for veterinary and public health. The domestic cat, the definitive host species with the largest worldwide population, has become evolutionarily and epidemiologically the most important host of T. gondii. The outcome of T. gondii infection is influenced by congenital and acquired host characteristics. We detected differences in T. gondii seroprevalence by cat breed in our previous studies. The aims of this study were to estimate T. gondii seroprevalence in selected domestic cat breeds, and to evaluate whether being of a certain breed is associated with T. gondii seropositivity, when the age and lifestyle of the cat are tak…

Male0301 basic medicineVeterinary medicineHOSTPhysiologylcsh:MedicinePathogenesis413 Veterinary sciencePathology and Laboratory MedicineBiochemistryDISEASEToxoplasma Gondii0403 veterinary scienceAnimal ProductsSeroepidemiologic StudiesImmune PhysiologyDirect agglutination testMedicine and Health SciencesEthnicitieslcsh:ScienceMammalsProtozoanseducation.field_of_studyImmune System ProteinsMultidisciplinarybiologyFATAL TOXOPLASMOSISAge FactorsAgriculture04 agricultural and veterinary sciencesFINLANDSERIESBreedFELINE INFECTIOUS PERITONITISPREVALENCE3. Good healthVertebratesHost-Pathogen InteractionsFemaleOTOCOLOBUS-MANULToxoplasmaResearch ArticleMeatNorwegian People040301 veterinary sciencesAnimal TypesImmunologyPopulationbiology.animal_breedAntibodies03 medical and health sciencesParasite Groupsparasitic diseasesmedicineAnimalsSeroprevalenceDomestic AnimalseducationNutritionlcsh:ROrganismsOocystsBiology and Life SciencesProteinsToxoplasma gondiiNorwegian Forest catbiology.organism_classificationmedicine.diseaseParasitic ProtozoansFeline infectious peritonitisToxoplasmosisDietLogistic ModelsToxoplasmosis Animal030104 developmental biologyFoodAmniotesPeople and PlacesMultivariate AnalysisCatsPopulation GroupingsParasitologylcsh:QZoologyApicomplexaPLoS ONE
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Prognostic Implications of the Complement Protein C1q in Gliomas

2019

The contribution of the complement system in the pathophysiology of brain cancers has been recently considered in light of its well-known involvement in carcinogenesis. Complement system represents an important component of the inflammatory response, which acts as a functional bridge between the innate and adaptive immune response. C1q, the first recognition subcomponent of the complement classical pathway, has recently been shown to be involved in a range of pathophysiological functions that are not dependent on complement activation. C1q is expressed in the microenvironment of various types of human tumors, including melanoma, prostate, mesothelioma, and ovarian cancers, where it can exer…

Male0301 basic medicinemedicine.disease_causePathogenesisbioinformatics analysis; C1q complement; gliomas; prognostic significance of C1q; survival probability0302 clinical medicinegliomaTumor MicroenvironmentImmunology and AllergyComplement C1qbioinformatics analysiOriginal ResearchSettore MED/27 - NeurochirurgiaBrain NeoplasmsMelanomaBioinformatics analysiGliomaPrognosisAcquired immune systemNeoplasm ProteinsGene Expression Regulation NeoplasticBioinformatics analysisFemalePrognostic significance of C1q.Databases Nucleic Acidlcsh:Immunologic diseases. Allergybioinformatics analysisImmunologyprognostic significance of C1qBiology03 medical and health sciencesClassical complement pathwayC1q complementGliomaBiomarkers TumormedicineHumanssurvival probabilitySurvival probabilityGliomasC1q complementComplement C1qmedicine.diseaseComplement systemgliomas030104 developmental biologyCancer researchlcsh:RC581-607Carcinogenesis030215 immunologyFrontiers in Immunology
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Polymorphisms of Pro-Inflammatory IL-6 and IL-1β Cytokines in Ascending Aortic Aneurysms as Genetic Modifiers and Predictive and Prognostic Biomarkers

2021

Background: Previous studies have demonstrated that polymorphisms involved in immune genes can affect the risk, pathogenesis, and outcome of thoracic ascending aortic aneurysms (TAAA). Here, we explored the potential associations of five functional promoter polymorphisms in interleukin-6 (IL-6), IL-1B, IL-1A, IL-18, and Tumor necrosis factor (TNF)A genes with TAAA. Methods: 144 TAAA patients and 150 age/gender matched controls were typed using KASPar assays. Effects on telomere length and levels of TAAA related histopathological and serological markers were analyzed. Results: Significant associations with TAAA risk were obtained for IL-6 rs1800795G&gt

Male0301 basic medicinemedicine.medical_specialtyInterleukin-1betamedial cell apoptosisSingle-nucleotide polymorphism030204 cardiovascular system & hematologyrs16944MicrobiologyPolymorphism Single NucleotideBiochemistryGastroenterologyArticleProinflammatory cytokinePathogenesis03 medical and health sciences0302 clinical medicineAneurysmInternal medicineGenotypeelastic fragmentationmedicinetelomere lengthHumansInterleukin 6thoracic ascending aortic aneurysmsMolecular BiologyAgedAortic Aneurysm ThoracicbiologyInterleukin-6business.industryMMP9Prognosismedicine.diseasers1800795QR1-502Settore MED/23030104 developmental biologyproinflammatory cytokinescystic medial changesbiology.proteinCytokinesBiomarker (medicine)FemaleTumor necrosis factor alphaInflammation MediatorsbusinessBiomarkersBiomolecules
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Mediterranean Diet Adherence and Congestive Heart Failure: Relationship With Clinical Severity and Ischemic Pathogenesis

2020

Abstract Objectives To our knowledge, no study has addressed the relationship between adherence to a Mediterranean diet style and severity of heart failure. The aim of this retrospective study was to evaluate the relationship between adherence to the Mediterranean diet assessed using the calculation of Mediterranean diet score and congestive heart failure (CHF), its severity, and pathogenesis. Methods We analyzed charts and collected data of all consecutive patients with a diagnosis of CHF at admission to our Internal Medicine Ward from 2008 to 2014. Results We analyzed 209 patients with CHF and 200 controls. Patients with CHF showed a significantly lower mean MeDi score than controls. At r…

Male0301 basic medicinemedicine.medical_specialtySettore MED/09 - Medicina InternaMediterranean dietEndocrinology Diabetes and MetabolismMyocardial Ischemia030209 endocrinology & metabolismDiseaseDiet MediterraneanSeverity of Illness IndexNew york heart associationPathogenesis03 medical and health sciences0302 clinical medicineInternal medicineMediterranean dietmedicineHumansClinical severityCADcardiovascular diseasesAgedRetrospective StudiesAged 80 and overHeart Failure030109 nutrition & dieteticsNutrition and DieteticsReceiver operating characteristicNYHA.business.industryRetrospective cohort studyMiddle Agedmedicine.diseasehumanitiesAdherenceHeart failurePatient ComplianceFemalebusinessCHF
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Evaluation of low dose anaphylatoxic peptides in the pathogenesis of the adult respiratory distress syndrome (ARDS). Monitoring of early C5a effects …

1986

A guinea-pig in vivo model is presented that allows the infusion of purified C5a via a central vein catheter and the monitoring of its effects on granulocytes and platelets, the most important cells in the pathogenesis of several lung disorders, e.g. shock lung. After the infusion of C5a, which was adjusted to a quantity that caused slight and transient alterations of lung physiology, granulocytes disappeared from circulation within 1 min. Simultaneously the granulocyte content of the lung increased about three-fold as judged by histological evaluations. Morphologic destructions were not observed. After the drop a rebound of circulating Polymorpho-nuclear leucocytes (PMN) occurred, which wa…

MaleARDSPathologymedicine.medical_specialtyGuinea PigsClinical BiochemistryComplement C5aGranulocyteBiochemistryPathogenesisLung DisorderLeukocyte CountIn vivomedicineAnimalsInfusions IntravenousLungRespiratory Distress SyndromeLungRespiratory distressPlatelet Countbusiness.industryComplement C5General Medicinerespiratory systemmedicine.diseaseDisease Models Animalmedicine.anatomical_structureFemaleBone marrowbusinessGranulocytesEuropean Journal of Clinical Investigation
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HLA association is different in children and adults with severe acquired aplastic anemia

2007

Background Severe aplastic anemia (SAA) is defined as pancytopenia caused by bone marrow failure. The pathogenesis of SAA is thought to involve autoimmune processes. Increased susceptibility to autoimmunity has been shown to be associated with several different HLA alleles. In SAA, few large studies based on data mainly from adults describe a positive HLA correlation with HLA-DR2 (DRB1*15) and HLA-B14. Procedure This study explored the HLA constitution of 181 children with SAA who were enrolled in the prospective multi-center study SAA94 between January 1994 and January 2002. The control group consisted of 303 healthy individuals of comparable demographic background. Allelic frequencies bet…

MaleAdolescentHuman leukocyte antigenmedicine.disease_causeAutoimmunityPathogenesisHLA-B14 AntigenHLA Antigenshemic and lymphatic diseasesmedicineHumansHLA-DR2 AntigenProspective StudiesChildAllelesbusiness.industryBone marrow failureAnemia AplasticInfantHematologymedicine.diseasePancytopeniaPathophysiologyExact testOncologyHLA-B AntigensChild PreschoolPediatrics Perinatology and Child HealthImmunologyCohortFemalebusinessPediatric Blood & Cancer
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Genome-wide Analyses Identify KIF5A as a Novel ALS Gene

2018

© 2018 Elsevier Inc.

MaleAls geneGenome-wide association studyFAMILIAL ALSALS; axonal transport; cargo; GWAS; KIF5A; WES; WGS0302 clinical medicine80 and overPsychologyGWASKIF5AAetiologycargoAged 80 and over0303 health sciencesFrench ALS ConsortiumKinesinKINESIN HEAVY-CHAINCognitive Sciencesaxonal transportHumanHereditary spastic paraplegiaNeuroscience(all)Single-nucleotide polymorphismTARGETED DISRUPTIONArticle03 medical and health sciencesGeneticsHumansAmino Acid SequenceLoss functionAgedHEXANUCLEOTIDE REPEATNeuroscience (all)MUTATIONSAmyotrophic Lateral Sclerosis3112 Neurosciences1702 Cognitive Sciencemedicine.diseaseITALSGEN ConsortiumAnswer ALS Foundation030104 developmental biologyALS Sequencing ConsortiumHuman medicine1109 Neurosciences030217 neurology & neurosurgery0301 basic medicineALS; GWAS; KIF5A; WES; WGS; axonal transport; cargo[SDV]Life Sciences [q-bio]KinesinsNeurodegenerativeGenetic analysisGenomeAMYOTROPHIC-LATERAL-SCLEROSIS3124 Neurology and psychiatryCohort StudiesPathogenesisLoss of Function MutationMissense mutation2.1 Biological and endogenous factorsAmyotrophic lateral sclerosisNYGC ALS ConsortiumGeneticsGeneral NeuroscienceALS axonal transport cargo GWAS KIF5A WES WGSMiddle AgedPhenotypeSettore MED/26 - NEUROLOGIANeurologicalProject MinE ALS Sequencing ConsortiumKinesinWESFemaleAdultBiologyGENOTYPE IMPUTATIONALS; axonal transport; cargo; GWAS; KIF5A; WES; WGS; Adult; Aged; Aged 80 and over; Amino Acid Sequence; Amyotrophic Lateral Sclerosis; Cohort Studies; Female; Genome-Wide Association Study; Humans; Kinesin; Loss of Function Mutation; Male; Middle Aged; Young AdultNOYoung AdultRare DiseasesmedicineSLAGEN ConsortiumGene030304 developmental biologyClinical Research in ALS and Related Disorders for Therapeutic Development (CReATe) ConsortiumNeurology & NeurosurgeryHuman GenomeNeurosciencesAXONAL-TRANSPORTBrain DisordersALS; axonal transport; cargo; GWAS; KIF5A; WES; WGS;Family memberDNA-DAMAGEMOTOR-NEURONS3111 BiomedicineCohort StudieALSGenomic Translation for ALS Care (GTAC) ConsortiumWGSAmyotrophic Lateral SclerosiGenome-Wide Association StudyALS; axonal transport; cargo; GWAS; KIF5A; WES; WGS; Neuroscience (all)
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