Search results for "PEPTIDE"

showing 10 items of 4589 documents

Obstructive sleep apnea and Alzheimer’s disease-related cerebrospinal fluid biomarkers in mild cognitive impairment

2020

Abstract Previous studies have demonstrated that sleep-breathing disorders, and especially obstructive sleep apnea (OSA), can be observed in patients with a higher risk of progression to Alzheimer’s disease (AD). Recent evidence indicates that cerebrospinal fluid (CSF) AD-biomarkers are associated with OSA. In this study, we investigated these associations in a sample of patients with mild cognitive impairment (MCI), a condition that is considered the first clinical phase of AD, when patients showed biomarkers consistent with AD pathology. A total of 57 patients (mean age = 66.19; SD = 7.13) with MCI were included in the study. An overnight polysomnography recording was used to assess objec…

medicine.medical_specialtyHeart diseaseRapid eye movement sleeptau ProteinsPolysomnography03 medical and health sciences0302 clinical medicineAlzheimer DiseasePhysiology (medical)Internal medicinemedicineHumansCognitive DysfunctionAgedSleep Apnea ObstructiveAmyloid beta-Peptidesmedicine.diagnostic_testbusiness.industryApneamedicine.diseaseSleep in non-human animalsPeptide FragmentsObstructive sleep apnea030228 respiratory systemCardiologyNeurology (clinical)medicine.symptombusinessHypopneaBody mass indexBiomarkers030217 neurology & neurosurgerySleep
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A human hepatocellular in vitro model to investigate steatosis

2006

The present study was designed to define an experimental model of hepatocellular steatosis with a fat overaccumulation profile in which the metabolic and cytotoxic/apoptotic effects could be separated. This was accomplished by defining the experimental conditions of lipid exposure that lead to significant intracellular fat accumulation in the absence of overt cytotoxicity, therefore allowing to differentiate between cytotoxic and apoptotic effects. Palmitic (C16:0) and oleic (Cl 8: 1) acids are the most abundant fatty acids (FFAs) in liver triglycerides in both normal subjects and patients with nonalcoholic fatty liver disease (NAFLD). Therefore, human hepatocytes and HepG2 cells were incub…

medicine.medical_specialtyHepG2Carcinoma HepatocellularCell SurvivalPalmitic AcidApoptosisBiologyFatty Acids NonesterifiedIn Vitro TechniquesToxicologyfatty acidscellular steatosisPalmitic acidchemistry.chemical_compoundInternal medicineCell Line TumorNonalcoholic fatty liver diseasemedicineHumansCytotoxicityDose-Response Relationship DrugapoptosisGeneral Medicinemedicine.diseaseFatty LiverDose–response relationshipmedicine.anatomical_structureEndocrinologychemistryBiochemistryApoptosisNeutral RedHepatocyteHepatocyteslipids (amino acids peptides and proteins)hepatocytesSteatosisIntracellularOleic Acid
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Telaprevir drug monitoring during antiviral therapy of hepatitis C graft infection after liver transplantation

2014

Background & Aims Recurrence of hepatitis C virus (HCV) infection after orthotopical liver transplantation (OLT) is common and associated with reduced graft and patient survival. The protease inhibitor telaprevir may enhance virological response rates in patients after OLT in combination with pegylated interferon-alfa and ribavirin. Pharmacokinetic studies have shown significant drug–drug interactions between telaprevir and immunosuppression (IS), but telaprevir pharmacokinetics in OLT patients with IS are unknown. Aim of the present study was to analyse telaprevir plasma concentrations in patients with HCV genotype 1 infection after OLT in comparison to patients without OLT and IS. Methods…

medicine.medical_specialtyHepatitis C virusmedicine.medical_treatmentPharmacologyLiver transplantationmedicine.disease_causeAntiviral AgentsGastroenterologyStatistics NonparametricTacrolimusPolyethylene GlycolsTelaprevirchemistry.chemical_compoundRecurrenceTandem Mass SpectrometryInternal medicineRibavirinmedicineHumansProspective StudiesImmunosuppression TherapyHepatologybusiness.industryRibavirinInterferon-alphaHepatitis Cmedicine.diseaseHepatitis CRecombinant ProteinsTacrolimusLiver TransplantationTransplantationsurgical procedures operativechemistryDrug Therapy CombinationDrug MonitoringbusinessViral hepatitisOligopeptidesChromatography Liquidmedicine.drugLiver International
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Prostanoid release of cultured liver sinusoidal endothelial cells in response to endotoxin and tumor necrosis factor

1990

Abstract Vascular endothelial cells release prostanoids, especially prostacyclin, when properly stimulated. In addition to short acting stimuli like thrombin and histamine an increased prostanoid release occurs in the presence of endotoxin, interleukin 1 or tumor necrosis factor (TNF). The response of sinusoidal endothelial liver cells to such stimuli — probably important in hepatic inflammatory disease — is unknown. Sinusoidal endothelial liver cells from the guinea pig were isolated by centrifugal elutriation and investigated as confluent monolayers. Their prostanoid release in response to endotoxin and human recombinant TNF was determined by radioimmunoassays and compared to that obtaine…

medicine.medical_specialtyHepatologyInterleukinProstanoidProstacyclinBiologyUmbilical veinEndothelial stem cellchemistry.chemical_compoundEndocrinologychemistryCell cultureInternal medicinecardiovascular systemmedicinelipids (amino acids peptides and proteins)Tumor necrosis factor alphaHistaminemedicine.drugJournal of Hepatology
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A double-blind, randomized, dose response study testing the pharmacological efficacy of synthetic porcine secretin

2000

Background: Biologically derived porcine secretin has been used as a diagnostic agent in clinical gastrointestinal practice for many years. Pure synthetic porcine secretin is now available for investigational clinical use. Aim: To compare the pharmacology of synthetic porcine secretin and biologically derived porcine secretin in healthy volunteers. Methods: Secretin stimulation tests were performed in 12 volunteer subjects in a double-blind, randomized, Latin square crossover design study comparing three doses of synthetic porcine secretin (0.05, 0.2, and 0.4 μg/kg) with a standard dose of biologically derived porcine secretin (1 CU/kg). Duodenal aspirates were analysed for total volume and…

medicine.medical_specialtyHepatologybusiness.industryBicarbonateGastroenterologyPeptide hormonedigestive systemCrossover studydigestive system diseasesDose Response StudySecretinchemistry.chemical_compoundfluids and secretionsEndocrinologymedicine.anatomical_structurechemistryGastrointestinal hormoneInternal medicinemedicinePharmacology (medical)businessPancreasVolunteerhormones hormone substitutes and hormone antagonistsAlimentary Pharmacology & Therapeutics
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Murine genetic deficiency of neuronal nitric oxide synthase (nNOS-/-) and interstitial cells of Cajal (W/Wv): Implications for achalasia?

2014

Background and aim Nitric oxide (NO) is an important inhibitory mediator of esophageal function, and its lack leads to typical features of achalasia. In contrast, the role of intramuscular interstitial cells of Cajal (ICC-IM) and vasoactive intestinal peptide (VIP) in lower esophageal sphincter (LES) function is still controversial. Therefore, we examined the function and morphology of the LES in vivo in NO-deficient (nNOS(-/-) ), ICC-IM-deficient (W/W(v) )-, and wild-type (WT) mice. Methods Esophageal manometry was performed with a micro-sized transducer catheter to quantify LES pressure, swallow evoked LES relaxation, and esophageal body motility. The LES morphology was examined by semiqu…

medicine.medical_specialtyHepatologybusiness.industryVasoactive intestinal peptideGastroenterologyMotilityAchalasiaInhibitory postsynaptic potentialmedicine.diseaseNitric oxideInterstitial cell of Cajalchemistry.chemical_compoundsymbols.namesakeEndocrinologychemistryIn vivoInternal medicineotorhinolaryngologic diseasesmedicinesymbolsbusinessNeuronal Nitric Oxide SynthaseJournal of Gastroenterology and Hepatology
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Hereditary angioneurotic oedema and blood-coagulation: interaction between C1-esterase-inhibitor and the activation factors of the proteolytic enzyme…

1983

C-1-inactivator (C-1-INA) does not only exert its important inhibitory functions in the complement system but also in the first step in the activation of the coagulation, fibrinolytic and kallikrein system. We therefore determined in nine patients with hereditary angioneurotic oedema (HANE) with obvious quantitative or functional defects of C-1-INA, and one further patient with Quincke-type oedema of different origin, the coagulation factors of the initial phase such as Hageman factor, plasma thromboplastin antecedent (PTA) and high molecular weight kininogen (HMWK). These factors were further correlated with the concentration as well as functional activity of C-1-INA. Nine of ten patients …

medicine.medical_specialtyHigh-molecular-weight kininogenInternal medicineDrug DiscoverymedicineHumansAngioedemaFactor XIBlood CoagulationGenetics (clinical)Factor XIFactor XIIComplement C1sChemistryKininogensProteolytic enzymesGeneral MedicineKallikreinMolecular medicineBlood Coagulation FactorsComplement systemEnzyme ActivationEndocrinologyCoagulationFactor XIIMolecular MedicinePeptide HydrolasesKlinische Wochenschrift
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The effect of oral hormone replacement therapy on lipoprotein profile, resistance of LDL to oxidation and LDL particle size

2001

Abstract Objectives: To disclose if oral estradiol (E 2 ), alone or in combination with natural progesterone (P) or medroxyprogesterone acetate (MPA), may modify the oxidizability of low density lipoprotein (LDL), and if the effect is achieved at physiological dosages. LDL oxidizability was assessed by the resistance to oxidation by copper and by the particle size profile, since small particles have increased oxidation susceptibility. Methods: Thirty-three women received two consecutive, two-month length doses of 1 and 2 mg/day of oral E 2 . They were then randomly assigned to a fourteen-day treatment of 2 mg/day E 2 plus either 300 mg/day P or 5 mg/day MPA. A parallel group of experiments …

medicine.medical_specialtyHormone Replacement Therapymedicine.drug_classMedroxyprogesteroneAdministration OralMedroxyprogesterone AcetateGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundHigh-density lipoproteinOral administrationInternal medicinemedicineHumansMedroxyprogesterone acetateParticle SizeProgesteroneDiminutionDose-Response Relationship DrugEstradiolbusiness.industryObstetrics and GynecologyCholesterol LDLMiddle AgedPostmenopauseEndocrinologychemistryEstrogenLow-density lipoproteinFemalelipids (amino acids peptides and proteins)businessOxidation-Reductionmedicine.drugLipoproteinMaturitas
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Increased pain and neurogenic inflammation in mice deficient of neutral endopeptidase

2009

The complex regional pain syndrome (CRPS) is characterized by enhanced neurogenic inflammation, mediated by neuropeptides. Neutral endopeptidase (NEP) is a key enzyme in neuropeptide catabolism. We used NEP knock out (ko) mice to investigate whether NEP deficiency leads to increased pain behavior and signs of neurogenic inflammation after soft tissue trauma with and without nerve injury. After chronic constriction injury (CCI) of the right sciatic nerve, NEP ko mice were more sensitive to heat, to mechanical stimuli, and to cold than wild type mice. Tissue injury without nerve injury produced no differences between genotypes. After CCI, NEP ko mice showed increased hind paw edema but lower …

medicine.medical_specialtyHot TemperaturePainSubstance PEnzyme-Linked Immunosorbent AssayCalcitonin gene-related peptideSubstance PEndothelin 1lcsh:RC321-571chemistry.chemical_compoundMiceCGRP catabolismEdemaInternal medicinePhysical StimulationMedicineAnimalsEdemaMuscle SkeletalNeprilysinlcsh:Neurosciences. Biological psychiatry. NeuropsychiatryPain MeasurementSkinMice KnockoutNeurogenic inflammationEndothelin-1business.industryCCIfungiNerve injurymedicine.diseaseNeutral endopeptidaseEndothelin 1Sciatic NerveHindlimbCold TemperatureMice Inbred C57BLComplex regional pain syndromeEndocrinologyNeurologychemistryAnesthesiaNeprilysinmedicine.symptomNeurogenic InflammationbusinessSkin TemperaturePrimarily cold CRPSNeurobiology of Disease
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Selenoprotein synthesis and side-effects of statins.

2004

Statins are possibly the most effective drugs for the prevention and treatment of hypercholesterolaemia and coronary heart disease. They are generally well tolerated, however, they do cause some unusual side-effects with potentially severe consequences, most prominently myopathy or rhabdomyolysis and polyneuropathy. We noted that the pattern of side-effects associated with statins resembles the pathology of selenium deficiency, and postulated that the mechanism lay in a well established, but often overlooked, biochemical pathway--the isopentenylation of selenocysteine-tRNA([Ser]Sec). A negative effect of statins on selenoprotein synthesis does seem to explain many of the enigmatic effects a…

medicine.medical_specialtyHypercholesterolemiaCoronary DiseaseBioinformaticsModels BiologicalRhabdomyolysisPolyneuropathiesSeleniumMuscular DiseasesSelenium deficiencyInternal medicinemedicineHumanscardiovascular diseasesSelenium metabolismMyopathySelenoproteinschemistry.chemical_classificationbusiness.industrynutritional and metabolic diseasesProteinsGeneral Medicinemedicine.diseaseCoronary heart diseaseEndocrinologychemistryProteins metabolismProtein Biosynthesislipids (amino acids peptides and proteins)Selenoproteinmedicine.symptomHydroxymethylglutaryl-CoA Reductase InhibitorsbusinessRhabdomyolysisPolyneuropathyLancet (London, England)
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