Search results for "PHAGOCYTOSIS"

showing 10 items of 169 documents

Imaging Noncanonical Autophagy and LC3-Associated Phagocytosis in Cultured Cells

2019

International audience; Monitoring of ATG8 proteins by western blotting and immunofluorescence microscopy are the most common methods to monitor the autophagy pathway. However, it has recently been shown that ATG8 proteins can be lipidated to non-autophagosome, single-membrane compartments through a noncanonical autophagy pathway. This is commonly found to occur during macro-endocytic processes such as phagocytosis, where it has been termed LC3-associated phagocytosis, and upon lysosomotropic drug treatment. Therefore, care is required when interpreting data based on ATG8 in order to conclude whether a signal relates to the canonical or noncanonical pathway. Here we provide methods to monit…

0303 health sciencesChemistryATG8PhagocytosisAutophagyImmunofluorescence MicroscopyATG8 Proteins3. Good healthCell biologyBlot03 medical and health sciencesDrug treatment0302 clinical medicinePhagocytosisLAPFluorescence microscopeLC3Noncanonical autophagy[SDV.BBM]Life Sciences [q-bio]/Biochemistry Molecular BiologyATG8030217 neurology & neurosurgery030304 developmental biology
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Essential omega-3 fatty acids tune microglial phagocytosis of synaptic elements in the developing brain

2019

SUMMARYOmega-3 fatty acids (n-3 polyunsaturated fatty acids; n-3 PUFAs) are essential for the functional maturation of the brain. Westernization of dietary habits in both developed and developing countries is accompanied by a progressive reduction in dietary intake of n-3 PUFAs. Low maternal intake of n-3 PUFAs has been linked to neurodevelopmental diseases in epidemiological studies, but the mechanisms by which a n-3 PUFA dietary imbalance affects CNS development are poorly understood. Active microglial engulfment of synaptic elements is an important process for normal brain development and altered synapse refinement is a hallmark of several neurodevelopmental disorders. Here, we identify …

2. Zero hungerchemistry.chemical_classification0303 health sciencesmedicine.medical_specialtybiologyOffspringDietary intakePhagocytosisHippocampusHippocampal formationSynapse03 medical and health sciencesLipoxygenase0302 clinical medicineEndocrinologychemistryInternal medicinemedicinebiology.proteinlipids (amino acids peptides and proteins)[SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC][SDV.NEU] Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]030217 neurology & neurosurgery030304 developmental biologyPolyunsaturated fatty acid
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Control of the production of oxygen intermediates of human polymorphonuclear leukocytes and monocytes by beta-adrenergic receptors.

1983

The control by beta-adrenergic receptors of the production of oxygen radicals by zymosan-stimulated human polymorphonuclear leukocytes (PMN) and monocytes (M phi) was studied in vitro by means of chemiluminescence. In addition we asked whether PMN and M phi exhibit differential sensitivity to beta-adrenergic stimulation. For beta-adrenergic stimulation we applied fenoterol ranging from 10(-5) to 10(-9) M x 2.7. We found a dose-dependent suppression of the production of oxygen radicals, the ID50 being approximately 10(-6) M both for PMN and M phi. By assessment of lactic dehydrogenase release a cytotoxic effect of the drug could be ruled out. When incubated together with the beta-adrenergic …

Adrenergic receptorFree RadicalsNeutrophilsImmunologychemistry.chemical_elementStimulationPropranololPharmacologyToxicologyOxygenMonocytesOxygen ConsumptionPhagocytosisReceptors Adrenergic betamedicineHumansReceptorFenoterolFenoterolPharmacologyAntagonistZymosanPropranololIn vitroOxygenchemistryBiochemistryLuminescent Measurementsmedicine.drugJournal of immunopharmacology
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Effects of ethanol and acetaldehyde on phagocytic functions

1985

Although a number of skin diseases are characterized by the presence of an increased number of phagocytes in their lesions, the effects of alcohol on phagocytic functions are not clearly understood. Therefore, we measured the influence of ethanol and acetaldehyde on the generation of oxygen radicals, chemotaxis and the release of lysosomal enzymes from human phagocytes. We added 0.03%-3% ethanol and 0.005%-0.25% acetaldehyde to cell cultures. We found that both ethanol and acetaldehyde suppressed the generation of oxygen radicals from granulocytes and monocytes; the ID50 was achieved at concentrations of approximately 0.25% for ethanol and 0.03% for acetaldehyde. A significant inhibition of…

AdultEthanolAdolescentEthanolNeutrophilsAcetaldehydeAlcoholAcetaldehydeDermatologyGeneral MedicineMonocytesRespiratory burstChemotaxis LeukocyteKineticschemistry.chemical_compoundPhagocytosischemistryBiochemistryLactate dehydrogenaseLuminescent MeasurementsHumansLymphocytesGranulocyte chemotaxisLysozymeEthanol metabolismArchives of Dermatological Research
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Hawthorn extract inhibits human isolated neutrophil functions.

2008

Hawthorn extract is a popular herbal medicine given as adjunctive treatment for chronic heart failure. In contrast to the cardiac properties of hawthorn extract, its anti-inflammatory effect has been scarcely investigated. This study examines the effects of a dry extract of leaves and flowers of Crataegus laevigata on various functional outputs of human neutrophils in vitro. Incubation of human neutrophils obtained from peripheral blood of healthy donors with C. laevigata extract (0.75-250 microg/ml) inhibited N-formyl-Met-Leu-Phe (FMLP)-induced superoxide anion generation, elastase release and chemotactic migration with potency values of 43.6, 21.9, and 31.6 microg/ml, respectively. By con…

AdultLeukotriene B4NeutrophilsFlowersPharmacologyIn Vitro TechniquesLeukotriene B4Calcium in biologychemistry.chemical_compoundPhagocytosisExtracellularMedicineHumansRespiratory BurstPharmacologyCrataegusbiologyChemotactic FactorsSuperoxidebusiness.industryPlant ExtractsElastasebiology.organism_classificationRespiratory burstCrataegus laevigataN-Formylmethionine Leucyl-PhenylalaninePlant LeavesChemotaxis LeukocytechemistryBiochemistryAdjunctive treatmentCytokinesCalciumbusinessLeukocyte ElastasePharmacological research
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The monocytic population in chronic lymphocytic leukemia shows altered composition and deregulation of genes involved in phagocytosis and inflammatio…

2013

Macrophages reside in tissues infiltrated by chronic lymphocytic leukemia B cells and the extent of infiltration is associated with adverse prognostic factors. We studied blood monocyte population by flow cytometry and whole-genome microarrays. A mixed lymphocyte reaction was performed to evaluate proliferation of T cells in contact with monocytes from patients and normal donors. Migration and gene modulation in normal monocytes cultured with CLL cells were also evaluated. The absolute number of monocytes increased in chronic lymphocytic leukemia patients compared to the number in normal controls (792 +/- 86 cells/mu L versus 485 +/- 46 cells/mL, P=0.003). Higher numbers of non-classical CD…

AdultMaleCD14Chronic lymphocytic leukemiaPhagocytosisPopulationDown-RegulationInflammationMICROENVIRONMENTCD16BiologyTUMOR-ASSOCIATED MACROPHAGES; TIE2-EXPRESSING MONOCYTES; MICROENVIRONMENT; CLLMonocytesImmune systemPhagocytosismedicineHumanseducationCells CulturedAgedAged 80 and overInflammationeducation.field_of_studyMonocyteGene Expression ProfilingHematologyMiddle Agedmedicine.diseaseLeukemia Lymphocytic Chronic B-CellTIE2-EXPRESSING MONOCYTESGene Expression Regulation NeoplasticChronic Lymphocytic Leukemia; Monocyte; microenvironmentTUMOR-ASSOCIATED MACROPHAGESmedicine.anatomical_structureImmunologyFemalemedicine.symptomLymphocyte Culture Test MixedOriginal Articles and Brief ReportsCLLHaematologica
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Bone marrow biopsy in hemophagocytic syndrome.

2002

Abstract Aims. Hemophagocytic syndrome (HPS) is a severe and acute clinical event occurring with fever, hepatosplenomegaly, and pancytopenia due to uncontrolled phagocytosis of blood cells and precursors. Although HPS represents a secondary phenomenon, it can mask the underlying condition, generally a neoplastic or infective disease, thus making the patient management rather difficult. The aims of this study were to point out the main pathological features useful to highlight the primary disease and show the eventual discrepancies among the different cases. Methods and results. Bone-marrow biopsies (BMBs) of 26 patients with HPS were morphologically and immunophenotypically evaluated; the p…

AdultMalePathologymedicine.medical_specialtyAdolescentHistiocytosis Non-Langerhans-CellBiopsyT-LymphocytesHepatosplenomegalyPathology and Forensic MedicineBone MarrowBiopsymedicineBiomarkers TumorHumansMolecular BiologyHistiocyteAgedAged 80 and overmedicine.diagnostic_testbusiness.industryHistiocytesCell BiologyGeneral MedicineHyperplasiaMiddle Agedmedicine.diseasePancytopeniaImmunohistochemistryHistiocytosisLeukemiaVirus DiseasesHematologic NeoplasmsHemophagocytosismedicine.symptombusinessVirchows Archiv : an international journal of pathology
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A macrophage-suppressing 40-kD protein in a case of pulmonary alveolar proteinosis.

1987

Pulmonary alveolar proteinosis (PAP) is a rare disease of unknown etiology. Macrophage dysfunctions are claimed to be involved in the pathogenesis. We investigated phagocytosis and oxidative metabolism of alveolar macrophages in a case of pulmonary alveolar proteinosis. These cells phagocytize normally and phagocytizable stimulants cause a normal oxidative burst. In response to the membrane signals phorbolmyristate acetate and aggregated immunoglobulin, however, no stimulated turnover of the oxidative metabolism can be observed. A 40-kD protein found in the lavage fluid mediates this macrophage-inhibiting effect. This phenomenon may contribute to the frequent opportunistic infections seen i…

AdultMalePathologymedicine.medical_specialtyPhagocytosisOpportunistic InfectionsPulmonary Alveolar ProteinosisPathogenesisPhagocytosisDrug DiscoverymedicineMacrophageHumansMacrophage Migration-Inhibitory FactorsGenetics (clinical)Lungmedicine.diagnostic_testbiologyMacrophagesfood and beveragesProteinsGeneral MedicineMacrophage Activationmedicine.diseaseRespiratory burstMolecular WeightPulmonary AlveoliBronchoalveolar lavagemedicine.anatomical_structureImmunologyLuminescent Measurementsbiology.proteinMolecular MedicineAntibodyPulmonary alveolar proteinosisEnergy MetabolismBronchoalveolar Lavage FluidKlinische Wochenschrift
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Differentiation, phenotype, and function of interleukin-17-producing human Vγ9Vδ2 T cells.

2011

Abstract In healthy adults, the major peripheral blood γδ T-cell subset expresses the Vγ9Vδ2 TCR and displays pleiotropic features. Here we report that coculture of naive Vγ9Vδ2 T cells with phosphoantigens and a cocktail of cytokines (IL-1-β, TGF-β, IL-6, and IL-23), leads to selective expression of the transcription factor RORγt and polarization toward IL-17 production. IL-17+ Vγ9Vδ2 T cells express the chemokine receptor CCR6 and produce IL-17 but neither IL-22 nor IFN-γ; they have a predominant terminally differentiated (CD27−CD45RA+) phenotype and express granzyme B, TRAIL, FasL, and CD161. On antigen activation, IL-17+ Vγ9Vδ2 T cells rapidly induce CXCL8-mediated migration and phagocy…

AdultMalebeta-DefensinsAdolescentNeutrophilsCellular differentiationT cellImmunologyC-C chemokine receptor type 6BiologyBiochemistryImmunophenotypingMeningitis BacterialImmune systemAntigenPhagocytosismedicineHumansCell LineageChildCells CulturedAntigens BacterialT-cell receptorInterleukin-17Interleukin-8Cell DifferentiationReceptors Antigen T-Cell gamma-deltaCell BiologyHematologyCoculture TechniquesGranzyme Bmedicine.anatomical_structureChild PreschoolImmunologyTh17 CellsFemaleInterleukin 17Blood
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The influence of high dose intravenous immunoglobulins on immunological and metabolic pattern in newly diagnosed type I diabetic patients

1990

In autoimmune disease the functional deficiency of T suppressor cells, also described in Type I diabetes, may be restored through immunoglobulin (Ig) infusion, which increases antigen phagocytosis, NK activity, cell clones and antibody anti-idiotype responses. Sixteen Type I diabetic patients were studied: eight were treated soon after the initial correction of disease-onset glycemic deterioration with intensive intravenous (i.v.) 7S Ig treatment (0.4 g/kg/BW) for 1 week and once per week for 6 months, whilst the remaining patients constituted the control group. All patients were evaluated during the study for metabolic and immunological parameters. A reduction in insulin requirement compar…

AdultMalemedicine.medical_specialtyAdolescentmedicine.medical_treatmentPhagocytosisImmunologyCellAutoimmune DiseasesRandom AllocationAntigenInternal medicinemedicineHumansInsulinImmunology and AllergyChildAutoantibodiesGlycemicAutoimmune diseaseC-Peptidebiologybusiness.industryInsulinImmunization PassiveReceptors Interleukin-2medicine.diseaseDiabetes Mellitus Type 1medicine.anatomical_structureEndocrinologyIntravenous ImmunoglobulinsChild Preschoolbiology.proteinFemaleAntibodybusinessJournal of Autoimmunity
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