Search results for "PROLIFERATION"

showing 10 items of 1193 documents

Role of apparent diffusion coefficient values in prostate diseases characterization on diffusion-weighted magnetic resonance imaging.

2019

BACKGROUND: To evaluate if normal and pathological prostate tissue can be distinguished by using apparent diffusion coefficient (ADC) values on magnetic resonance imaging (MRI) and to understand if it is possible to differentiate among pathological prostate tissues using ADC values.METHODS:Our population consisted in 81 patients (mean age 65.4 years) in which 84 suspicious areas were identified. Regions of interest were placed over suspicious areas, detected on MRI, and over areas with normal appearance, and ADC values were recorded. Statistical differences between ADC values of suspicious and normal areas were evaluated. Histopathological diagnosis, obtained from targeted biopsy using MRI-…

Image-Guided BiopsyMaleProstatic DiseasesProstate DiseasesUrologymedicine.medical_treatmentPopulation030232 urology & nephrologyurologic and male genital diseases030218 nuclear medicine & medical imagingDiffusion03 medical and health sciencesProstate cancer0302 clinical medicineProstateImage Processing Computer-AssistedmedicineHumansEffective diffusion coefficienteducationAgedRetrospective StudiesAged 80 and overProstate MRI Diffusion Weighted ImagingAtypical small acinar proliferationeducation.field_of_studymedicine.diagnostic_testbusiness.industryProstatectomyProstateProstatic NeoplasmsMagnetic resonance imagingMiddle Agedmedicine.diseasebody regionsDiffusion Magnetic Resonance Imagingmedicine.anatomical_structureNephrologyNeoplasm GradingbusinessNuclear medicineAlgorithms
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Recombinant virus-like particles as a carrier of B- and T-cell epitopes of hepatitis C virus (HCV)

2005

The major aim of the project was the development of virus-like particles (VLP) displaying B- and T-cell epitopes of hepatitis C virus (HCV) proteins. To this end, hepatitis B virus core (HBc) particles were used as a carrier of HCV epitopes. Fragments of HCV genes encoding core (aa 98) and NS3 (aa 155) proteins were fused to the 3' terminus of the truncated HBV core gene. All recombinant plasmids led to relatively high levels of expression of chimeric proteins in E. coli, which resulted in the formation of complete "mature" VLP. Chimeric HBc/HCV VLPs were purified by combination of gel filtration and sucrose gradient centrifugation, and used for immunogenicity studies in mice. All variants …

ImmunogenT-LymphocytesvirusesHepacivirusBiologyRecombinant virusEpitopeVirusEpitopesMiceVirus-like particleAnimalsCell ProliferationB-LymphocytesMice Inbred BALB CNS3General VeterinaryGeneral Immunology and MicrobiologyImmunogenicityVirionPublic Health Environmental and Occupational Healthvirus diseasesVirologyMolecular biologydigestive system diseasesHBcAgInfectious DiseasesMolecular MedicineElectrophoresis Polyacrylamide GelFemaleVaccine
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Mast cells enhance proliferation of B lymphocytes and drive their differentiation toward IgA-secreting plasma cells.

2010

AbstractThe evidence of a tight spatial interaction between mast cells (MCs) and B lymphocytes in secondary lymphoid organs, along with the data regarding the abundance of MCs in several B-cell lymphoproliferative disorders prompted us to investigate whether MCs could affect the proliferation and differentiation of B cells. To this aim, we performed coculture assays using mouse splenic B cells and bone marrow–derived MCs. Both nonsensitized and activated MCs proved able to induce a significant inhibition of cell death and an increase in proliferation of naive B cells. Such proliferation was further enhanced in activated B cells. This effect relied on cell-cell contact and MC-derived interle…

Immunoglobulin AMAST CELL B LYMPHOCITESCellular differentiationImmunologyNaive B cellCD40 LigandPlasma CellsCell CommunicationImmunoglobulin ELymphocyte ActivationBiochemistryMast cellMiceImmune systemIg isotype switchmedicineAnimalsHumansMast CellsCD40 AntigensCell ProliferationIG-A.B cellB cellsMast cell; B cells; Differentiation; Ig isotype switchCD40biologyCell DeathInterleukin-6Cell DifferentiationCell BiologyHematologyMast cellhumanitiesCell biologyImmunity HumoralImmunoglobulin Amedicine.anatomical_structureGene Expression RegulationDifferentiationImmunologybiology.proteinMAST CELL B LYMPHOCITES; IG-A.Syndecan-1AntibodyBlood
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A20 deficiency in B cells enhances B-cell proliferation and results in the development of autoantibodies.

2011

A20/TNFAIP3 is an ubiquitin-editing enzyme, important for the regulation of the NF-κB pathway. Mutations in the TNFAIP3 gene have been linked to different human autoimmune disorders. In human B-cell lymphomas, the inactivation of A20 results in constitutive NF-κB activation. Recent studies demonstrate that in mice the germline inactivation of A20 leads to early lethality, due to inflammation in multiple organs of the body. In this report, we describe a new mouse strain allowing for the tissue-specific deletion of A20. We show that B-cell-specific deletion of A20 results in a dramatic reduction in marginal zone B cells. Furthermore, A20-deficient B cells display a hyperactive phenotype repre…

ImmunologyB-Lymphocyte SubsetsInflammationBiologymedicine.disease_causeLymphocyte ActivationGermlineAutoimmunityMiceimmune system diseaseshemic and lymphatic diseasesmedicineImmunology and AllergyAnimalsHumansTumor Necrosis Factor alpha-Induced Protein 3AutoantibodiesCell ProliferationMice KnockoutB-LymphocytesCell growthAutoantibodyIntracellular Signaling Peptides and ProteinsNF-kappa BMarginal zoneGerminal CenterMolecular biologyPhenotypeCell biologyCysteine EndopeptidasesModels Animalbiology.proteinmedicine.symptomAntibodySignal TransductionEuropean journal of immunology
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MicroRNAs play a central role in molecular dysfunctions linking inflammation with cancer.

2013

It is now largely admitted that a pro-inflammatory environment may curtail anti-tumor immunity and favor cancer initiation and progression. The discovery that small non-coding regulatory RNAs, namely microRNAs (miRNAs), regulate all aspects of cell proliferation, differentiation, and function has shed a new light on regulatory mechanisms linking inflammation and cancer. Thus, miRNAs such as miR-21, miR-125b, miR-155, miR-196, and miR-210 that are critical for the immune response or hypoxia are often overexpressed in cancers and leukemias. Given the high number of their target transcripts, their deregulation may have a number of deleterious consequences, depending on the cellular context. In…

ImmunologyBiologyBioinformaticsmedicine.disease_causemiR-155Transforming Growth Factor betaNeoplasmsmicroRNAmedicineImmunology and AllergyAnimalsHumansTranscription factorCell ProliferationRegulation of gene expressionInflammationImmunityBiological TherapyGene Expression Regulation NeoplasticMicroRNAsCell Transformation NeoplasticTumor EscapeCancer researchTumor EscapeSignal transductionCarcinogenesisTransforming growth factorSignal TransductionTranscription FactorsImmunological reviews
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Inhibition of human allergic T-helper type 2 immune responses by induced regulatory T cells requires the combination of interleukin-10-treated dendri…

2006

Summary Background In grass pollen-allergic individuals, T cell anergy can be induced by IL-10-treated dendritic cells (IL-10-DC) resulting in the suppression of T helper type 1 (Th1) as well as Th2 cells. This study was performed to analyse whether such IL-10-DC-treated T cells are able to act as regulatory T cells (Treg) suppressing the function of other T cells in the periphery. As transforming growth factor (TGF)-β is also a potential inducer of Treg, we additionally analysed the inhibitory capacity of TGF-β-treated T cells in this system. Materials and Methods Freshly isolated CD4+ or CD4+CD25− T cells from grass pollen-allergic donors were stimulated with autologous mature monocyte-de…

ImmunologyEnzyme-Linked Immunosorbent AssayCell CommunicationBiologyPoaceaeT-Lymphocytes RegulatoryInterleukin 21Interferon-gammaTh2 CellsAntigens CDTransforming Growth Factor betaHypersensitivityImmunology and AllergyCytotoxic T cellHumansCTLA-4 AntigenIL-2 receptorAntigen-presenting cellCells CulturedInterleukin 3Cell ProliferationDendritic cellDendritic CellsAllergensNatural killer T cellFlow CytometryAntigens DifferentiationCell biologyInterleukin-10ImmunologyInterleukin 12PollenImmunizationInterleukin-4Interleukin-5Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology
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Physico-chemical and mechanical characterization of in-situ forming xyloglucan gels incorporating a growth factor to promote cartilage reconstruction

2016

Abstract The development of growth factors is very promising in the field of tissue regeneration but specifically designed formulations have to be developed in order to enable such new biological entities (NBEs). In particular, the range of therapeutic concentrations is usually very low compared to other active proteins and the confinement in the target site can be of crucial importance. In-situ forming scaffolds are very promising solutions for minimally invasive intervention in cartilage reconstruction and targeting of NBEs. In this work injectable, in-situ forming gels of a temperature responsive partially degalactosylated xyloglucan (Deg-XG) incorporating the growth factor FGF-18 are fo…

In situInjectionFibroblast Growth FactorChemical Phenomenamedicine.medical_treatment02 engineering and technologyFibroblast growth factor01 natural sciencesViscositychemistry.chemical_compoundTissue ScaffoldSettore BIO/10 - BiochimicaComposite materialGlucansGelTissue ScaffoldsIn-situ forming gelsViscosityGrowth factor021001 nanoscience & nanotechnologyGlucanXyloglucanmedicine.anatomical_structureSettore ING-IND/22 - Scienza E Tecnologia Dei MaterialiMechanics of MaterialsXylansMaterials Science (all)0210 nano-technologyMaterials scienceMechanical PhenomenaInjectable scaffoldsBioengineeringCondensed Matter Physic010402 general chemistryInjectable scaffoldInjectionsBiomaterialsShear modulusXylanChondrocytesmedicineAnimalsMechanics of MaterialXyloglucanCartilage reconstructionCell ProliferationMechanical PhenomenaAnimalCartilageGrowth factorMechanical EngineeringIn-situ forming gelChondrocyte0104 chemical sciencesFibroblast Growth FactorsMolecular WeightCartilagechemistryBiophysicsCattleSettore CHIM/07 - Fondamenti Chimici Delle TecnologieTemperature-responsiveGels
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Synthesis and antiproliferative activity of 3-amino-N-phenyl-1H-indazole-1-carboxamides

2007

Abstract A series of new 3-amino-N-phenyl-1H-indazole-1-carboxamides 10 have been prepared from commercially available phenyl isocyanate precursors 8 and 3-aminoindazole 9. Some of the synthesized compounds were evaluated for their in vitro antineoplastic activity against 60 human cell lines derived from seven clinically isolated cancer types (lung, colon, melanoma, renal, ovarian, brain, and leukemia) according to the NCI standard protocol. The test results indicated that 3-amino-1H-indazole-1-carboxamides 10 were endowed with an interesting antiproliferative activity. The most active compounds of this series, 10d,e, were able to inhibit cell growth of many neoplastic cell lines at concent…

IndazolesAntineoplastic AgentsCrystallography X-RayRetinoblastoma Proteinchemistry.chemical_compoundStructure-Activity RelationshipCell Line TumorNeoplasmsDrug DiscoverymedicineHumansCell ProliferationG0-G1 arrestPharmacologyIndazoleMolecular StructureChemistryCell growthMelanomaOrganic ChemistryCell CycleCancer1H-Indazole-1-carboxamides; Crystallographic study; G0-G1 arrest; pRb1H-Indazole-1-carboxamideGeneral MedicineCell cyclemedicine.diseaseAmidesSettore CHIM/08 - Chimica FarmaceuticaIn vitroCrystallographyc studyLeukemiapRbBiochemistryNeoplastic cell
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Novel 4-(3-phenylpropionamido), 4-(2-phenoxyacetamido) and 4-(cinnamamido) substituted benzamides bearing the pyrazole or indazole nucleus: synthesis…

2019

Based on some common structural features of known compounds interfering with p53 pathways and our previously synthesized benzamides, we synthesized new ethyl 5-(4-substituted benzamido)-1-phenyl-1H-pyrazole-4-carboxylates 26a-c, ethyl 5-(4-substituted benzamido)-1-(pyridin-2-yl)-1H-pyrazole-4-carboxylates 27a-c and N-(1H-indazol-6-yl)-4-substituted benzamides 31a,b bearing in the 4 position of the benzamido moiety the 2-phenylpropanamido or 2-phenoxyacetamido or cinnamamido groups. A preliminary test to evaluate the antiproliferative activity against human lung carcinoma H292 cells highlighted how compound 26c showed the best activity. This last was therefore selected for further studies wi…

IndazolesStereochemistryAntineoplastic AgentsApoptosisTRAIL-receptorPyrazole01 natural sciencesBiochemistrychemistry.chemical_compoundDownregulation and upregulationCell Line Tumor2-(3-phenylpropanamido)benzamideDrug DiscoverymedicineHumansMoietyMolecular BiologyCell ProliferationBiological evaluationP53Indazole010405 organic chemistryDrug Discovery3003 Pharmaceutical Science2-cinnamamidobenzamideOrganic ChemistryApoptosi2-(2-phenoxyacetamido)benzamide0104 chemical sciences010404 medicinal & biomolecular chemistrymedicine.anatomical_structureProto-Oncogene Proteins c-bcl-2Mechanism of actionchemistryApoptosisBenzamidesPyrazolesDrug Screening Assays Antitumormedicine.symptomNucleusBioorganic Chemistry
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Synthesis of substituted 3-amino-N-phenyl-1H-indazole-1-carboxamides endowed with antiproliferative activity

2010

Abstract Several new N-phenyl-1H-indazole-1-carboxamides 1c–h and 4l,m were prepared by reacting phenyl isocyanate derivatives 3a,b with 3-amino-1H-indazole derivatives 2c,e,g or 1H-indazole 2l respectively. Chemical transformations of compounds 1a,b and 1g,h gave 3-acetamido-N-phenyl-1H-indazole-1-carboxamide derivatives 5a,b, and 3,5-diamino-N-phenyl-1H-indazole-1-carboxamide derivatives 4i, j respectively. Finally, 3,5-diacetamido-N-phenyl-1H-indazole-1-carboxamide derivatives 6a,b were prepared by acetylation of 4i, j. Some of synthesized compounds were evaluated for their in vitro antiproliferative activity against the full NCI tumor cell lines panel derived from nine clinically isolat…

IndazolesStereochemistryCellAntineoplastic AgentsRetinoblastoma Proteinchemistry.chemical_compoundCell Line TumorDrug DiscoveryG0–G1 arrestmedicineHumansCell ProliferationPharmacologyIndazoleCell growth3-amino-N-phenyl-1H-indazole-1-carboxamideMelanomaCell CycleOrganic ChemistryAntiproliferative agentsCancerGeneral Medicinemedicine.diseaseSettore CHIM/08 - Chimica FarmaceuticaIn vitropRbmedicine.anatomical_structurechemistryAcetylationK562 cellsEuropean Journal of Medicinal Chemistry
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