Search results for "PROTEIN-SYNTHESIS"

showing 5 items of 5 documents

Treating cachexia using soluble ACVR2B improves survival, alters mTOR localization, and attenuates liver and spleen responses.

2018

Background Cancer cachexia increases morbidity and mortality, and blocking of activin receptor ligands has improved survival in experimental cancer. However, the underlying mechanisms have not yet been fully uncovered. Methods The effects of blocking activin receptor type 2 (ACVR2) ligands on both muscle and non‐muscle tissues were investigated in a preclinical model of cancer cachexia using a recombinant soluble ACVR2B (sACVR2B‐Fc). Treatment with sACVR2B‐Fc was applied either only before the tumour formation or with continued treatment both before and after tumour formation. The potential roles of muscle and non‐muscle tissues in cancer cachexia were investigated in order to understand th…

MaleTUMOR-BEARING MICElcsh:Diseases of the musculoskeletal systemCachexiaprotein synthesisActivin Receptors Type IIMDSCphysical activityAcute phase responseKaplan-Meier EstimateACTIVATIONActivinMiceNeoplasmsOrthopedics and Sports MedicineTOR Serine-Threonine Kinasesactivinlcsh:Human anatomyII RECEPTORSRecombinant ProteinsProtein TransportLivermyostatinPROTEIN-SYNTHESISSKELETAL-MUSCLECytokinessyöpätauditInflammation MediatorsACUTE-PHASE RESPONSE3122 CancersINHIBITIONlcsh:QM1-695acute phase responsePhysiology (medical)Cell Line TumorAnimalsHumansMuscle SkeletalActivin; Acute phase response; MDSC; Myostatin; Physical activity; Protein synthesis; Orthopedics and Sports Medicine; Physiology (medical)Physical activityMyeloid-Derived Suppressor CellsMyostatinXenograft Model Antitumor AssaysDisease Models AnimalACTIVIN-APHYSICAL-ACTIVITY3121 General medicine internal medicine and other clinical medicineproteiinitEXPERIMENTAL CANCER CACHEXIAlcsh:RC925-935Protein synthesislihassurkastumasairaudetBiomarkersSpleenJournal of cachexia, sarcopenia and muscle
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Effects of Leucine Administration in Sarcopenia: A Randomized and Placebo-controlled Clinical Trial

2020

Treating sarcopenia in older individuals remains a challenge, and nutritional interventions present promising approaches in individuals that perform limited physical exercise. We assessed the efficacy of leucine administration to evaluate whether the regular intake of this essential amino acid can improve muscle mass, muscle strength and functional performance and respiratory muscle function in institutionalized older individuals. The study was a placebo-controlled, randomized, double-blind design in fifty participants aged 65 and over (ClinicalTrials.gov identifier NCT03831399). The participants were randomized to a parallel group intervention of 13 weeks&rsquo

Malemedicine.medical_specialtyBODY-COMPOSITIONrespiratory muscles030209 endocrinology & metabolismPhysical exerciselcsh:TX341-641MASSPlaceboOBSTRUCTIVE PULMONARY-DISEASEelderlyArticlesarcopenia03 medical and health sciencesDOUBLE-BLIND0302 clinical medicineDouble-Blind MethodLeucineInternal medicineSTRENGTHRespiratory musclemedicineMUSCLE PROTEIN-SYNTHESISHumansAMINO-ACIDS030212 general & internal medicineVITAMIN-D SUPPLEMENTATIONEssential amino acidAgedchemistry.chemical_classificationAged 80 and overNutrition and Dieteticsbusiness.industryWHEY-PROTEINmedicine.diseaseClinical trialnutritionchemistrymuscle massSarcopeniaLean body massmuscle strengthFemaleLeucinePHYSICAL PERFORMANCEbusinesslcsh:Nutrition. Foods and food supplyFood ScienceNutrients
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Differentiation of Murine C2C12 Myoblasts Strongly Reduces the Effects of Myostatin on Intracellular Signaling

2020

Alongside in vivo models, a simpler and more mechanistic approach is required to study the effects of myostatin on skeletal muscle because myostatin is an important negative regulator of muscle size. In this study, myostatin was administered to murine (C2C12) and human (CHQ) myoblasts and myotubes. Canonical and noncanonical signaling downstream to myostatin, related ligands, and their receptor were analyzed. The effects of tumorkines were analyzed after coculture of C2C12 and colon cancer-C26 cells. The effects of myostatin on canonical and noncanonical signaling were strongly reduced in C2C12 cells after differentiation. This may be explained by increased follistatin, an endogenous blocke…

Muscle Fibers Skeletallcsh:QR1-502lihaksetlcsh:MicrobiologyArticleTGF-BETA SUPERFAMILYCell LineMyoblastsMicetumorkineCell Line TumorfollistatinAnimalsHumansCANCER CACHEXIAskeletal muscleMUSCLE ATROPHYlihassolutSmadsoluviestintäRECEPTORCell DifferentiationIN-VITROMyostatinmusculoskeletal systemMAPKActivinsLEUKEMIA INHIBITORY FACTORACTIVIN-AinflammationCulture Media ConditionedCELLSPROTEIN-SYNTHESISmyotubeGROWTH1182 Biochemistry cell and molecular biologyproteiinit3111 BiomedicinecocultureSignal Transduction
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Treatment with soluble activin type IIB-receptor improves bone mass and strength in a mouse model of Duchenne muscular dystrophy

2017

Background: Inhibition of activin/myostatin pathway has emerged as a novel approach to increase muscle mass and bone strength. Duchenne muscular dystrophy (DMD) is a neuromuscular disorder that leads to progressive muscle degeneration and also high incidence of fractures. The aim of our study was to test whether inhibition of activin receptor IIB ligands with or without exercise could improve bone strength in the mdx mouse model for DMD. Methods: Thirty-two mdx mice were divided to running and non-running groups and to receive either PBS control or soluble activin type IIB-receptor (ActRIIB-Fc) once weekly for 7 weeks. Results: Treatment of mdx mice with ActRIIB-Fc resulted in significantly…

bone-muscle interactionsOXIDATIVE CAPACITYMDX MICEbone μCTexerciseBLOCKINGBone mu CTEXERCISEPREVENTS3126 Surgery anesthesiology intensive care radiologyMYOSTATINBone-muscle interactionsanimal modelsAnimal modelsDEFICIENCYTGF-βsDENSITY3121 General medicine internal medicine and other clinical medicineMUSCLE PROTEIN-SYNTHESISOrthopedics and Sports MedicineTGF-beta sMETAANALYSIS
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Influence of Oral Contraceptive Use on Adaptations to Resistance Training

2019

Introduction: The majority of young women use oral contraceptives (OCs). Use of OCs has been associated with lower myofibrillar protein and tendon collagen synthesis rates, but it is unknown whether OCs will limit the adaptive response of myotendinous tissue to resistance training. Design and Methods: Fourteen healthy untrained young regular OC users (24 +/- 1 years, fat% 32 +/- 1, 35 +/- 2 ml.min(-1).kg(-1)) and 14 NOC users (non-OC, controls) (24 +/- 1 years, fat% 32 +/- 2, 34 +/- 2 ml.min(-1).kg(-1)) performed a 10-week supervised lower extremity progressive resistance training program. Before and after the intervention biopsies from the vastus lateralis muscle and the patellar tendon we…

estrogeenitmedicine.medical_specialtyestradioliTESTOSTERONE LEVELStendonPhysiologyVastus lateralis muscleUrologyIsometric exerciselcsh:PhysiologyMuscle hypertrophy03 medical and health sciences0302 clinical medicinePhysiology (medical)estradiolmedicineestrogenmuscle hypertrophyIN-VIVOHUMAN ACHILLES-TENDONOriginal Researchlcsh:QP1-981ehkäisypilleritexerciseMUSCLE-FIBER SIZETENDON COLLAGEN-SYNTHESISbusiness.industryResistance training030229 sport sciencesMECHANICAL-PROPERTIESTendonmedicine.anatomical_structureContraceptive uselihasmassaBIOMECHANICAL PROPERTIESMuscle strengthmuscle strengthPROTEIN-SYNTHESISSKELETAL-MUSCLEvoimaharjoitteluwomenMENSTRUAL-CYCLEbusinessMyofibril030217 neurology & neurosurgerylihasvoima
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