Search results for "Pathogenesis"

showing 10 items of 761 documents

Serum and fecal pancreatic enzymes in beta-thalassemia major

1997

This study, using indirect tests, demonstrated that exocrine pancreatic function is impaired in a proportion of patients with beta-thalassemia major (TM), though this impairment is generally mild or moderate.Impaired structure and function of the exocrine pancreas has been reported in patients with Beta-thalassemia major.In this study we measured fecal fats and serum and fecal pancreatic enzymes in 30 patients (13 M, 17 F) with TM, mean age 22.1 yr (range 14-39) and compared them with those of a matched group of healthy controls. Results were correlated with age, serum ferritin, blood transfusion, and various nutritional parameters. Enzymes assays included: serum pancreatic amylase (PA), li…

AdultMaleHemolytic anemiamedicine.medical_specialtyPancreatic diseaseAdolescentHydrolasesGastroenterologyFatsPathogenesisFecesEndocrinologyInternal medicinemedicineChymotrypsinHumansTrypsinFeceschemistry.chemical_classificationPancreatic Elastasebusiness.industrybeta-ThalassemiaGastroenterologyBeta thalassemiaLipasemedicine.diseaseHemoglobinopathyEnzymeEndocrinologyOncologychemistryAmylasesExocrine Pancreatic InsufficiencyFemalebusinessPancreatic enzymesInternational Journal of Pancreatology
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Sequential transcriptome analysis of human liver cancer indicates late stage acquisition of malignant traits

2014

Background & Aims Human hepatocarcinogenesis is as a multi-step process starting from dysplastic lesions to early carcinomas (eHCC) that ultimately progress to HCC (pHCC). However, the sequential molecular alterations driving malignant transformation of the pre-neoplastic lesions are not clearly defined. This lack of information represents a major challenge in the clinical management of patients at risk. Methods We applied next-generation transcriptome sequencing to tumor-free surrounding liver (n=7), low- (n=4) and high-grade (n=9) dysplastic lesions, eHCC (n=5) and pHCC (n=3) from 8 HCC patients with hepatitis B infection. Integrative analyses of genetic and transcriptomic changes were pe…

AdultMaleHepatocarcinogenesisCarcinoma HepatocellularCarcinogenesisBiologyBioinformaticsmedicine.disease_causePolymorphism Single NucleotideArticleMalignant transformationTranscriptomeCarcinomamedicineTumor MicroenvironmentHumansMolecular pathogenesisRNA NeoplasmGeneAgedTumor microenvironmentHepatologyGene Expression ProfilingLiver NeoplasmsWnt signaling pathwayRNA sequencingMiddle Agedmedicine.diseaseGene expression profilingCell Transformation NeoplasticMutationCancer researchDisease ProgressionFemaleCarcinogenesis
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Genetic control of immune response in carriers of the 8.1 ancestral haplotype: correlation with levels of IgG subclasses: its relevance in the pathog…

2007

Ancestral haplotype (AH) 8.1(HLA-A1, Cw7, B8, TNFAB*a2b3, TNFN*S, C2*C, Bf*s, C4A*Q0, C4B*1, DRB1*0301, DRB3*0101, DQA1*0501, DQB1*0201) seems to be associated with susceptibility to autoimmune diseases. Different mechanisms are probably involved in increasing autoimmunity, such as unbalanced cytokine production and the lack of C4A protein. So AH 8.1 modifies immune response in many ways. In this study we demonstrate that IgG2 serum levels were significantly lower in 8.1 AH carriers than in 8.1 AH non-carriers. On the contrary, as regards IgG1, IgG3, IgG4 serum levels, no significant differences were observed between the two groups. In AH 8.1 carriers low IgG2 levels might take to slower cl…

AdultMaleHeterozygoteAH 8.1autoimmune dis-easemedicine.medical_treatmentBiologymedicine.disease_causeimmune responseGeneral Biochemistry Genetics and Molecular BiologyAutoimmunityAutoimmune DiseasesHLA-B8 AntigenPathogenesisImmune systemHLA-DR3 AntigenHistory and Philosophy of ScienceAntigenmedicineHumansGenetic Predisposition to DiseaseGeneral NeuroscienceHaplotypeIgG subclasseC4AAutoantibodyHLA-B8DR3Middle AgedCytokineHaplotypesImmunoglobulin GImmunologyFemaleAnnals of the New York Academy of Sciences
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High prevalence of human cytomegalovirus in a population of periodontally healthy subjects.

2008

Background: Human cytomegalovirus (HCMV) appears to be more frequent in periodontally affected patients than in healthy control groups. Based on this assumption, it has been suggested that HCMV may play a role in the pathogenesis of periodontal disease. Objective: The objective of this uncontrolled study was to assess the occurrence of HCMV in a large unselected population of periodontally healthy subjects. Study Design: Fifty consecutive periodontally healthy patients satisfied the inclusion criteria. Two samples of gingival crevicular fluids were taken from two non-bleeding on probing sites for each patient. Samples were collected from the anterior and the posterior area. Polymerase chain…

AdultMaleHuman cytomegalovirusmedicine.medical_specialtyvirusesPopulationGingivaCytomegalovirusDentistryGastroenterologyPathogenesisYoung AdultInternal medicinePrevalencemedicineHumansYoung adulteducationGeneral Dentistryeducation.field_of_studyHigh prevalencebusiness.industryHealthy subjectsvirus diseasesMiddle Agedbiochemical phenomena metabolism and nutrition:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseDrinking habitsOtorhinolaryngologyUNESCO::CIENCIAS MÉDICASGingival Crevicular FluidsFemaleSurgerybusiness
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Preferential splenic CD8+ T-cell activation in rituximab-nonresponder patients with immune thrombocytopenia

2013

The pathogenic role of B cells in immune thrombocytopenia (ITP) has justified the therapeutic use of anti-CD20 antibodies such as rituximab (RTX). However, 60% of ITP patients do not respond to RTX. To decipher the mechanisms implicated in the failure of RTX, and because the spleen plays a well-recognized role in ITP pathogenesis, 12 spleens from ITP patients who had been nonresponders to RTX therapy were compared with 11 spleens from RTX-untreated ITP patients and 9 controls. We here demonstrate that in RTX-nonresponder ITP patients, preferential Th1 and Tc1 T lymphocyte polarizations occur, associated with an increase in splenic effector memory CD8(+) T-cell frequency. Moreover, in the RT…

AdultMaleImmunologyDrug ResistanceSpleenCD8-Positive T-LymphocytesLymphocyte ActivationReal-Time Polymerase Chain ReactionBiochemistryPathogenesisAntibodies Monoclonal Murine-DerivedYoung Adultimmune system diseaseshemic and lymphatic diseasesmedicineHumansImmunologic FactorsCytotoxic T cellAgedAged 80 and overPurpura Thrombocytopenic Idiopathicbiologybusiness.industryCell BiologyHematologyT lymphocyteMiddle AgedImmunohistochemistrymedicine.anatomical_structureImmunologyMonoclonalbiology.proteinFemaleRituximabAntibodyRituximabbusinessSpleenCD8medicine.drugBlood
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Differential up-regulation of circulating soluble selectins and endothelial adhesion molecules in Sicilian patients with Boutonneuse fever

1999

SUMMARYIn 150 patients with Boutonneuse fever (BF), caused by Rickettsia conorii, we studied the plasma levels of soluble l-selectin (s l-selectin), vascular cell adhesion molecule-1 (sVCAM-1), intercellular adhesion molecule-1 (sICAM-1) and E-selectin (sE-selectin) in various phases of disease to clarify their role in disease evolution. Results indicate that during the acute phase of BF there is a significant increase in the serum levels of s l-selectin, sE-selectin, sVCAM-1 and sICAM-1. s l-selectin and sVCAM-1 returned to normal levels in the third week of disease, whereas sE-selectin and sICAM-1 persisted at significantly high levels even after the third week. The secretion of these sol…

AdultMaleImmunologyIntercellular Adhesion Molecule-1Vascular Cell Adhesion Molecule-1BiologyBoutonneuse FeverPathogenesisLeukocyte CountmedicineHumansImmunology and AllergyL-SelectinCell adhesionAgedAnalysis of VarianceCell adhesion moleculeMiddle AgedIntercellular Adhesion Molecule-1medicine.diseasebiology.organism_classificationUp-RegulationEndothelial stem cellBoutonneuse feverSolubilityImmunologyLinear ModelsCytokinesFemaleOriginal ArticleE-SelectinRickettsia conoriiSelectinClinical and Experimental Immunology
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Promyelocytic leukemia (PML) gene expression is a prognostic factor in ampullary cancer patients

2008

Background: Promyelocytic leukemia (PML) tumor suppressor gene plays a key role in acute PML pathogenesis but its involvement in pathogenesis and prognosis of solid cancers has not been defined yet. Patients and methods: In all, 62 ampullary adenocarcinoma patients who underwent curative surgery between 1996 and 2005 were included. Expression analysis of PML was carried out by immunohistochemical staining and correlated with disease-free survival (DFS) and overall survival (OS). Results: In 24 tumor specimens (38.7%), PML was classified as absent, in 16 (25.8%) as focally expressed and in 22 (35.5%) as diffusely expressed. By univariate analysis, DFS was significantly influenced by patholog…

AdultMaleOncologyAmpulla of Vatermedicine.medical_specialtyPathology(PML)ampullary cancerTumor suppressor geneCommon Bile Duct NeoplasmsAdenocarcinomaPromyelocytic Leukemia ProteinsurvivalCohort StudiesPathogenesispromyelocytic leukemia gene expressionPromyelocytic leukemia proteinampulla of vater cancer; promyelocytic leukemia gene expression; prognosisInternal medicineBiomarkers TumorHumansMedicinePathologicalAgedRetrospective StudiesAged 80 and overUnivariate analysisPMLbiologybusiness.industryTumor Suppressor ProteinsNuclear Proteinsampullary cancerHematologyMiddle AgedPrognosismedicine.diseaseSurvival AnalysisGene Expression Regulation NeoplasticLeukemiaOncologyPML; ampullary cancer; survivalbiology.proteinImmunohistochemistryT-stageampulla of vater cancerFemalebusinessTranscription FactorsAnnals of Oncology
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GTF2I Mutation in Thymomas: Independence From Racial-Ethnic Backgrounds. An Indian/German Comparative Study

2021

Thymomas are the most frequent adult mediastinal cancers. Their etiology is unknown and their pathogenesis poorly understood. Racial, ethnic and environmental factors influence tumorigenesis in many cancers, but their role in thymomas remains unclear to date. In this study that included pretreatment thymoma cases from India and Germany (n = 37 and n = 77, respectively) we compared i) the prevalence of the thymoma-specific chromosome 7 c.74146970T > A mutation of the GTF2I gene in type A and AB thymomas; ii) epidemiological features; and iii) the frequency of myasthenia gravis (MG). Due to a known predominance of GTF2I mutation in A and AB histotypes, we included only a marginal numbe…

AdultMaleOncologymedicine.medical_specialtyCancer ResearchThymomaThymomaEthnic groupIndiaracial-ethnic factorsPathology and Forensic MedicinePathogenesisTranscription Factors TFII03 medical and health sciences0302 clinical medicineGermanyInternal medicineEpidemiologymedicineHumansOriginal ResearchAged030304 developmental biologyChromosome 7 (human)myasthenia gravis0303 health sciencesbusiness.industryPathology and Oncology ArchiveGTF2I mutationThymus NeoplasmsGeneral MedicineMiddle Agedmedicine.diseaseMyasthenia gravisRace Factors3. Good healthOncology030220 oncology & carcinogenesisMutationCohortEtiologyepidemiologyFemalebusinessPathology and Oncology Research
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Cellular Immunity and Retrobulbar Fibroblasts in Graves' Ophthalmopathy

1994

In Graves' ophthalmopathy (GO), retrobulbar connective tissue is infiltrated by T cells whose role in the pathogenesis of the disease was investigated in the present work. The aims included firstly to characterize subsets of blood lymphocytes and of sessile lymphocytes cloned from a retroorbital tissue specimen. Second, in counterstimulation assays, the ability of patients' T cells to influence cultivated retrobulbar fibroblasts and in turn the enhancement of lymphocyte proliferation by retrobulbar fibroblasts was investigated. Blood lymphocytes of 16 GO patients and 12 controls isolated by density gradient centrifugation and retrobulbar fibroblasts obtained from orbital decompression were …

AdultMalePathologymedicine.medical_specialtyCellular immunitygenetic structuresT-LymphocytesEndocrinology Diabetes and MetabolismFluorescent Antibody TechniqueConnective tissueEnzyme-Linked Immunosorbent AssayDiseaseEyeLymphocyte ActivationPathogenesisGraves' ophthalmopathyEndocrinologymedicineHumansCells CulturedAgedAged 80 and overbusiness.industryFibroblastsMiddle Agedmedicine.diseaseGraves Diseaseeye diseasesmedicine.anatomical_structureImmunologyFemalebusinessCell DivisionThyroid
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The Glomerular Lesions in Endotheliotropic Hemolytic Nephroangiopathy (Hemolytic Uremic Syndrome, Malignant Nephrosclerosis, Post Partal Renal Insuff…

1982

The term "Endotheliotropic Hemolytic Nephroangiopathy" (EHN) comprises various clinically or pathomorphologically defined disease states with severe renal lesions (e.g. hemolytic uremic syndrome, malignant nephrosclerosis, post partum renal insufficiency) which, to date, have been considered as different entities. We attempted to assign accompanying glomerular changes based upon light and electron microscopy to the above mentioned clinical pictures and their various stages. The accordingly classified glomerular lesions (G1--G3 and Ga) are of critical importance in pathohistological differential diagnosis. Since it is assumed that fibrin is a causing event in the pathogenesis of the vascular…

AdultMalePathologymedicine.medical_specialtyEndotheliumBiopsyKidney GlomerulusDiseaseFibrinPathology and Forensic MedicinePathogenesisPregnancyGlomerulopathyBiopsymedicineHumansChildAgedFibrinNephrosclerosisbiologymedicine.diagnostic_testMalignant nephrosclerosisbusiness.industryInfantPuerperal DisordersCell BiologyAcute Kidney InjuryMiddle Agedmedicine.diseaseMicroscopy Electronmedicine.anatomical_structureChild PreschoolHemolytic-Uremic Syndromebiology.proteinFemaleDifferential diagnosisbusinessPathology - Research and Practice
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