Search results for "Pathogenesis"

showing 10 items of 761 documents

Complement and Atherogenesis

1999

Abstract —Complement activation occurs in temporal correlation with the subendothelial deposition of LDL during early atherogenesis, and complement also plays a pathogenetic role in promoting lesion progression. Two lesion components have been identified that may be responsible for complement activation. First, enzymatic degradation of LDL generates a derivative that can spontaneously activate complement, and enzymatically degraded LDL (E-LDL) has been detected in the lesions. Second, C-reactive protein (CRP) colocalizes with complement C5b-9, as evidenced by immunohistological studies of early atherosclerotic lesions, so the possibility exists that this acute phase protein also fulfills a…

PhosphorylcholineNeuraminidaseComplement Membrane Attack ComplexCoronary Artery DiseaseBiologyPhospholipaseLesionPathogenesismedicineHumansElectrophoresis Gel Two-DimensionalTrypsinComplement Activationchemistry.chemical_classificationPhosphorylcholineC-reactive proteinAcute-phase proteinCholesterol LDLComplement C3Coronary VesselsMolecular biologyComplement systemC-Reactive ProteinEnzymeBiochemistrychemistryType C Phospholipasesbiology.proteinCalciummedicine.symptomCardiology and Cardiovascular MedicineProtein BindingArteriosclerosis, Thrombosis, and Vascular Biology
researchProduct

Activin A and bone metastasis

2010

Activin A, is a multifunctional cytokine of the transforming growth factor-b superfamily of growth factors. This molecule has been shown to be implicated in the regulation of a broad range of important biological functions including bone remodelling. Therefore, a deregulation in the activin signalling pathway may result in disturbances of normal bone metabolism and, eventually, in the onset of severe pathological conditions associated with an altered bone resorption. These observations support the concept that Act A might also be implicated in the pathogenesis of bone metastasis. This review provides insight into the most recent advances in understanding the role of this growth factor in th…

PhysiologyGrowth factormedicine.medical_treatmentClinical BiochemistryBone metastasisBone NeoplasmsCell BiologyBiologymedicine.diseaseHedgehog signaling pathwayBone resorptionBone remodelingActivinsActivin bone metastasisPathogenesisGene Expression Regulation NeoplasticCytokineImmunologymedicineCancer researchAnimalsBone RemodelingActivin type 2 receptorsSignal Transduction
researchProduct

Possible Role of Glymphatic System of the Brain in the Pathogenesis of High-Altitude Cerebral Edema

2018

Simka, Marian, Paweł Latacz, and Joanna Czaja. Possible role of glymphatic system of the brain in the pathogenesis of high-altitude cerebral edema. High Alt Med Biol. 19:394–397, 2018.—In this article, we suggest that the glymphatic system of the brain can play an important role in the pathogenesis of high-altitude cerebral edema (HACE). Water enters the intercellular space of the brain primarily through aquaporin-4 (AQP-4) water channels, the main component of the glymphatic system, whereas acetazolamide, pharmacological agent used in the prevention of HACE, is the blocker of the AQP-4 molecule. In animal experiments, cerebral edema caused by hypobaric hypoxia was associated with an increa…

Physiologyglymphatic systemaquaporin-4Brain EdemaAltitude Sickness030204 cardiovascular system & hematologyCerebral edemaPathogenesis03 medical and health sciences0302 clinical medicinemedicineAnimalsHumansAquaporin 4business.industryAltitudePublic Health Environmental and Occupational HealthastrocytesBrainGeneral MedicineEffects of high altitude on humansmedicine.diseaseSleep in non-human animalsAcetazolamideAquaporin 4Glymphatic systemAcetazolamidebusinessNeuroscience030217 neurology & neurosurgeryHigh-altitude cerebral edemamedicine.drug
researchProduct

Computational Analysis of Clinical and Molecular Markers and New Theranostic Possibilities in Primary Open-Angle Glaucoma

2020

Primary open-angle glaucoma (POAG) is a paramount cause of irreversible visual disability worldwide. We focus on identifying clinical and molecular facts that may help elucidating the pathogenic mechanisms of the disease. By using ophthalmological approaches (biomicroscopy, ocular fundus, optical coherence tomography, and perimetry) and experimental tests (enzyme-linked immunosorbent assay (ELISA), high performance liquid chromatography (HPLC), and Western blot/immunoblotting) directed to evaluate the oxidative stress, inflammation, apoptosis, and neurodegeneration processes, we gather information to build a network of data to perform a computational bioinformatics analysis. Our results sho…

Posterior Eye Segmentmedicine.medical_specialtytheranosticsprimary open-angle glaucomaOpen angle glaucomagenetic structuresGlaucomalcsh:MedicineVisual disabilityDiseaseArticlePathogenesis03 medical and health sciences0302 clinical medicineOphthalmologymedicineoxidative stressComputational analysisbusiness.industrypathogenesisNeurodegenerationlcsh:RapoptosisneurodegenerationGeneral Medicinemedicine.diseaseeye diseasesinflammation030221 ophthalmology & optometrysense organsbusiness030217 neurology & neurosurgeryJournal of Clinical Medicine
researchProduct

Granulozytenfunktion bei Frühgeborenen ab der 34. Schwangerschaftswoche und bei reifen Neugeborenen

1999

BACKGROUND Preterm and term neonates have an increased risk to develop severe bacterial infections. Impairment of neutrophil function may be responsible for this increased risk. Other diseases related to prematurity like retinopathia of prematurity (ROP) or broncho-pulmonary dysplasia (BPD) on the other hand may be due to poorly controlled O2-radical production. PATIENTS AND METHODS Blood samples of 112 premature (34 weeks of gestation and older) and term neonates were analysed. Blood samples of 23 healthy adults (18 to 50 years old) served as controls. O2-radical production and phagocytosis of neutrophils were determined by flow cytometry, using a commercial test system. RESULTS Under the …

Pregnancybusiness.industryRespiratory diseaseStimulationGranulocytemedicine.diseasePathogenesismedicine.anatomical_structureBronchopulmonary dysplasiaDysplasiaPediatrics Perinatology and Child HealthImmunologymedicineGestationbusinessKlinische Pädiatrie
researchProduct

Oxidative stress and mitochondrial dysfunction in Kindler syndrome

2014

This is an Open Access article distributed under the terms of the Creative Commons Attribution License.-- et al.

Premature agingMaleKeratinocytesAdolescentComputingMilieux_LEGALASPECTSOFCOMPUTINGMitochondrionmedicine.disease_causePathogenesisKindler syndrome03 medical and health scienceschemistry.chemical_compound0302 clinical medicineBlistermedicineHumansGenetics(clinical)Pharmacology (medical)Photosensitivity DisordersGenodermatosisChildGenetics (clinical)Cells CulturedPeriodontal Diseases030304 developmental biologyAged 80 and overMedicine(all)0303 health sciencesintegumentary systemResearchGeneral MedicineGlutathioneMiddle Agedmedicine.diseaseMalondialdehydeMolecular biology3. Good healthMitochondriaOxidative StresschemistryOxidative stress030220 oncology & carcinogenesisChild PreschoolFemaleSkin cancerEpidermolysis BullosaKindlin1Oxidative stressOrphanet Journal of Rare Diseases
researchProduct

Human parvovirus B19 induced apoptotic bodies contain altered self-antigens that are phagocytosed by antigen presenting cells.

2013

Human parvovirus B19 (B19V) from the erythrovirus genus is known to be a pathogenic virus in humans. Prevalence of B19V infection has been reported worldwide in all seasons, with a high incidence in the spring. B19V is responsible for erythema infectiosum (fifth disease) commonly seen in children. Its other clinical presentations include arthralgia, arthritis, transient aplastic crisis, chronic anemia, congenital anemia, and hydrops fetalis. In addition, B19V infection has been reported to trigger autoimmune diseases such as systemic lupus erythematosus and rheumatoid arthritis. However, the mechanisms of B19V participation in autoimmunity are not fully understood. B19V induced chronic dise…

Programmed cell deathScienceAntigen-Presenting CellsArthritisApoptosisAutoimmunitySpodopteraViral Nonstructural ProteinsBiologymedicine.disease_causeAutoantigensVirusautoimmuniteettiImmune toleranceAutoimmunityParvoviridae InfectionsPathogenesis03 medical and health sciences0302 clinical medicineImmune systemPhagocytosisImmune ToleranceParvovirus B19 HumanSf9 CellsHuman Parvovirus B19medicineta319AnimalsHumansAntigen-presenting cellself-antigens030304 developmental biology0303 health sciencesMultidisciplinaryQta1182RHep G2 CellsFlow Cytometrymedicine.diseaseVirology3. Good healthImmunologyMicroscopy Electron ScanningMedicineResearch Article030215 immunologyPLoS ONE
researchProduct

T cell-independent joint destruction

1998

Rheumatoid Arthritis (RA) is a chronic systemic disorder of unknown etiology. Although, early and late stages of the disease may be driven by different processes, affected joints are characterized by inflammation, synovial hyperplasia, and abnormal immune responses [1]. The abundance of T cells within the rheumatoid synovium as well as the association of certain major histocompatibility complex (MHC) class II molecules with RA [2] implied a central role for T cells in the pathophysiology of the disease. However, recent advances in molecular biology have fostered new concepts for the pathogenesis of RA. Specifically, the investigation of early stages of disease, the development of novel anim…

Property (philosophy)media_common.quotation_subjectT cellInflammationDiseaseBiologyMajor histocompatibility complexExperiential learningExistentialismPathogenesisMode (music)Immune systemPerceptionSynovitismedicineRelation (history of concept)media_commonTime perceptionmedicine.diseasemedicine.anatomical_structureRheumatoid arthritisImmunologySpitebiology.proteinmedicine.symptomPsychologyCognitive psychology
researchProduct

Dominant role of paraoxonases in inactivation of the Pseudomonas aeruginosa quorum-sensing signal N-(3-oxododecanoyl)-L-homoserine lactone.

2008

Pseudomonas aeruginosa is an opportunistic bacterium which causes serious infections in immunocompromised and cystic fibrosis patients (10). As with many gram-negative bacteria, P. aeruginosa produces acyl-homoserine lactone (AHL) quorum-sensing (QS) signaling molecules termed autoinducers which allow the single-celled organisms to coordinate their actions (36). N-(3-Oxododecanoyl)-l-homoserine lactone (3OC12-HSL) is a key autoinducer synthesized by P. aeruginosa which regulates the expression of extracellular virulence factors and biofilm formation (5, 36). Rats and mice experimentally infected with P. aeruginosa mutants deficient in the ability to produce or respond to 3OC12-HSL exhibited…

ProteasesCell signalingImmunologyHomoserineBiologymedicine.disease_causeMicrobiologyMicrobiologychemistry.chemical_compoundMice4-ButyrolactonemedicineHomoserineLeukocyte proliferationAnimalsHumansLungEdetic AcidMice Inbred ICRPseudomonas aeruginosaAryldialkylphosphataseHydrolysisBiofilmEsterasesfood and beveragesQuorum SensingGene Expression Regulation BacterialMolecular PathogenesisQuorum sensingInfectious DiseasesBiochemistrychemistryLiverMetalsPseudomonas aeruginosaParasitologyAutoinducerInfection and immunity
researchProduct

A proteomic approach to studying plant response to crenate broomrape (Orobanche crenata) in pea (Pisum sativum)

2004

Abstract Crenate broomrape ( Orobanche crenata ) is a parasitic plant that threatens legume production in Mediterranean areas. Pea ( Pisum sativum ) is severely affected, and only moderate levels of genetic resistance have so far been identified. In the present work we selected the most resistant accession available (Ps 624) and compared it with a susceptible (Messire) cultivar. Experiments were performed by using pot and Petri dish bioassays, showing little differences in the percentage of broomrape seed germination induced by both genotypes, but a significant hamper in the number of successfully installed tubercles and their developmental stage in the Ps 624 compared to Messire. The prote…

Proteomics0106 biological sciencesSilver StainingGenotypeParasitic plantNitrogen assimilationGene ExpressionPlant ScienceHorticultureOrobanche crenataPeptide MappingPlant Roots01 natural sciencesBiochemistryFructokinasePisum03 medical and health sciencesSativumGlutamine synthetaseElectrophoresis Gel Two-DimensionalDatabases ProteinMolecular Biology[SDV.BV.PEP] Life Sciences [q-bio]/Vegetal Biology/Phytopathology and phytopharmacyComputingMilieux_MISCELLANEOUSPlant Proteins030304 developmental biologyPathogenesis-related protein2. Zero hunger0303 health sciencesbiologyOrobanchePeasGeneral Medicinebiology.organism_classification[SDV.BV.PEP]Life Sciences [q-bio]/Vegetal Biology/Phytopathology and phytopharmacyBiochemistrySpectrometry Mass Matrix-Assisted Laser Desorption-IonizationElectrophoresis Polyacrylamide Gel010606 plant biology & botany
researchProduct