Search results for "Perfusion"

showing 10 items of 714 documents

Riociguat versus sildenafil on hypoxic pulmonary vasoconstriction and ventilation/perfusion matching

2017

Introduction Current treatment with vasodilators for pulmonary hypertension associated with respiratory diseases is limited by their inhibitory effect on hypoxic pulmonary vasoconstriction (HPV) and uncoupling effects on ventilation-perfusion (V'/Q'). Hypoxia is also a well-known modulator of the nitric oxide (NO) pathway, and may therefore differentially affect the responses to phosphodiesterase 5 (PDE5) inhibitors and soluble guanylyl cyclase (sGC) stimulators. So far, the effects of the sGC stimulator riociguat on HPV have been poorly characterized. Materials and methods Contraction was recorded in pulmonary arteries (PA) in a wire myograph. Anesthetized rats were catheterized to record …

MaleAnoxemiaPulmonologyPulmonary FibrosisVasodilator Agentslcsh:MedicineVasodilation030204 cardiovascular system & hematologyPharmacologyVascular Medicinechemistry.chemical_compound0302 clinical medicineSoluble Guanylyl CyclaseHypoxic pulmonary vasoconstrictionPulmonary fibrosisMedicine and Health SciencesPulmonary Arterieslcsh:ScienceHypoxiaMultidisciplinaryVasodilatorsDrugsRespiratory organs diseasesArteriesMiddle AgedChemistryPhysical Sciencescardiovascular systemFemalemedicine.symptomAnatomyMedicamentsmedicine.drugResearch ArticleChemical Elementsinorganic chemicalsSildenafilHypertension PulmonaryChronic Obstructive Pulmonary DiseaseEnzyme ActivatorsIn Vitro TechniquesPulmonary ArteryRiociguatSildenafil CitrateMalalties de l'aparell respiratori03 medical and health sciencesMedical HypoxiamedicineVentilation-Perfusion RatioAnimalsHumansRats WistarAgedPharmacologybusiness.industrylcsh:RAnoxèmiaBiology and Life SciencesHypoxia (medical)Phosphodiesterase 5 Inhibitorsmedicine.diseasePulmonary hypertensionFibrosisRatsOxygenDisease Models AnimalPyrimidines030228 respiratory systemchemistryVasoconstrictionCardiovascular AnatomyPyrazolesBlood Vesselslcsh:QSoluble guanylyl cyclasebusinessDevelopmental Biology
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Carbon Monoxide Protects Against Ischemia-reperfusion Injury in Vitro via Antioxidant Properties

2012

Carbon monoxide (CO) is believed to mediate many of the cytoprotective effects attributed to the activation of heme oxygenase (HO-1), the enzyme responsible for CO production. Recently, the study of CO-releasing molecules (CO-RMs) has provided a new approach for the delivery of CO. In the present study, we examined whether the cardioprotective properties of CO-RM2 in isolated rat hearts subjected to an ischemia-reperfusion (I/R) sequence were associated with the presence of CO. In addition, the antioxidant properties of CO-RM2 were evaluated. In hearts pretreated with CO-RM2, the improvement in contractile function at the end of the reperfusion period after 20 min of global total ischemia w…

MaleAntioxidantCardiotonic AgentsTime FactorsPhysiologymedicine.medical_treatmentIschemiaPharmacologyIn Vitro Techniquesmedicine.disease_causeAntioxidantsVentricular Function Left03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemSuperoxidesEthidiummedicineOrganometallic CompoundsAnimalsRats WistarComputingMilieux_MISCELLANEOUS030304 developmental biologychemistry.chemical_classification0303 health sciencesCarbon MonoxideL-Lactate DehydrogenaseSuperoxideHeartmedicine.diseaseMyocardial ContractionIn vitroRats[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemHeme oxygenaseOxidative StressEnzymechemistryBiochemistry030220 oncology & carcinogenesisReperfusion InjuryReperfusion injuryOxidative stress
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Antioxidant potential of Himanthalia elongata for protection against ischemia-reperfusion injury in the small bowel

2016

Seaweed has been associated with the prevention and/or treatment of various diseases related to oxidative stress because of its antioxidant activity. We investigated the protective potential of extract of Himanthalia elongata against ischemia-reperfusion (I/R) injury in the intestine of rats.Seventy-two (72) male Wistar albino rats were randomly assigned into 12 groups as follows: sham, I/R only, I/R plus vehicle at 3 time points, and I/R plus extract at 3 time points. The degree of intestinal injury was determined by oxidative stress using lipid peroxidation, superoxide dismutase, catalase, and glutathione peroxidase after mesenteric ischemia-reperfusion. A histological study was also perf…

MaleAntioxidantmedicine.medical_treatmentPharmacologymedicine.disease_causeSensitivity and SpecificityAntioxidantsLipid peroxidationSuperoxide dismutaseRandom Allocation03 medical and health scienceschemistry.chemical_compound0302 clinical medicineIntestine SmallHimanthalia elongatamedicineAnimalsIntestinal MucosaRats Wistarchemistry.chemical_classificationbiologyPlant ExtractsGlutathione peroxidaseBiopsy NeedleSeaweedmedicine.diseasebiology.organism_classificationImmunohistochemistryRatsDisease Models AnimalTreatment OutcomechemistryBiochemistryCatalaseReperfusion Injury030220 oncology & carcinogenesisbiology.protein030211 gastroenterology & hepatologySurgeryLipid PeroxidationReperfusion injuryOxidative stressPhytotherapySurgery
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GABA receptors are involved in the modulation of the release of 5-hydroxytryptamine from the vascularly perfused small intestine of the guinea-pig

1989

Isolated small intestinal segments of the guinea-pig were perfused arterially and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent was determined by HPLC with electrochemical detection. Test substances were applied intraarterially. Muscimol (1 microM) time dependently first increased then decreased the release of 5-HT and 5-HIAA. The stimulatory effect was prevented by tetrodotoxin (TTx) or scopolamine, indicating that it was mediated by the release of acetylcholine. Bicuculline concentration dependently decreased (1 microM) or increased (10, 50 microM) the release of 5-HT and 5-HIAA, indicating that endogenous GABA also activ…

MaleBaclofenSerotoninmedicine.medical_specialtyGuinea PigsTetrodotoxinIn Vitro TechniquesBiologyGABAB receptorBicucullineInhibitory postsynaptic potential5-Hydroxytryptophanchemistry.chemical_compoundInternal medicineIntestine SmallmedicineAnimalsReceptorPharmacologyMuscimolGABAA receptorOxotremorineMuscle SmoothHydroxyindoleacetic AcidBicucullineReceptors GABA-APerfusionEndocrinologynervous systemMuscimolchemistryFemaleSerotoninAcetylcholinemedicine.drugEuropean Journal of Pharmacology
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Nifedipine improves blood flow and oxygen supply, but not steady-state oxygenation of tumours in perfusion pressure-controlled isolated limb perfusio…

2002

Isolated limb perfusion allows the direct application of therapeutic agents to a tumour-bearing extremity. The present study investigated whether the dihydropyridine-type Ca2+-channel blocker nifedipine could improve blood flow and oxygenation status of experimental tumours during isolated limb perfusion. Perfusion was performed by cannulation of the femoral artery and vein in rats bearing DS-sarcoma on the hind foot dorsum. Perfusion rate was adjusted to maintain a perfusion pressure of 100–140 mmHg throughout the experiment. Following equilibration, nifedipine was continuously infused for 30 min (8.3 μg min−1 kg−1 BW). During constant-pressure isolated limb perfusion, nifedipine can signi…

MaleCancer ResearchNifedipinecalcium channel blockerCell RespirationHemodynamicsFemoral arteryMicrocirculationRats Sprague-DawleyNifedipinemedicine.arterymedicineLaser-Doppler FlowmetryAnimalsExperimental TherapeuticsInfusions Intravenoustumour vascular resistancebusiness.industrytumour perfusionBlood flowOxygenationHypoxia (medical)Calcium Channel BlockersRatsOxygenOncologyRegional Blood FlowAnesthesiaChemotherapy Cancer Regional PerfusionSarcoma Experimentalmedicine.symptombusinessPerfusiontumour oxygenationmedicine.drugisolated limb perfusionBritish journal of cancer
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Betulinic acid protects against cerebral ischemia–reperfusion injury in mice by reducing oxidative and nitrosative stress

2011

Increased production of reactive oxygen and nitrogen species following cerebral ischemia-reperfusion is a major cause for neuronal injury. In hypercholesterolemic apolipoprotein E knockout (ApoE-KO) mice, 2h of middle cerebral artery (MCA) occlusion followed by 22h of reperfusion led to an enhanced expression of NADPH oxidase subunits (NOX2, NOX4 and p22phox) and isoforms of nitric oxide synthase (neuronal nNOS and inducible iNOS) in the ischemic hemisphere compared with the non-ischemic contralateral hemisphere. This was associated with elevated levels of 3-nitrotyrosine, an indicator of peroxynitrite-mediated oxidative protein modification. Pre-treatment with betulinic acid (50mg/kg/day f…

MaleCancer ResearchPhysiologyClinical BiochemistryIschemiaPharmacologymedicine.disease_causeBiochemistryBrain IschemiaMicechemistry.chemical_compoundStress PhysiologicalEnosBetulinic acidmedicineAnimalsRNA MessengerBetulinic AcidMice KnockoutNADPH oxidasebiologyChemistryBrainNADPH Oxidasesbiology.organism_classificationmedicine.diseaseReactive Nitrogen SpeciesTriterpenesNitric oxide synthaseOxidative StressBiochemistryReperfusion Injurycardiovascular systembiology.proteinTyrosineP22phoxNitric Oxide SynthasePentacyclic TriterpenesReperfusion injuryOxidative stressNitric Oxide
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Tumor blood flow: The principal modulator of oxidative and glycolytic metabolism, and of the metabolic micromilieu of human tumor xenograftsin vivo

1989

We have investigated therapeutically relevant pathophysiological parameters of human breast and lung cancer xenografts in nude rats. All lung cancers and one breast cancer exhibited rapid growth and high blood flow values paralleled by high metabolic rates. The tissue of these tumors was well oxygenated up to very advanced growth stages. Xenografts from other breast cancer cell lines grew much more slowly, were poorly perfused, and exhibited low metabolic rates. Here, tumor hypoxia and tissue acidosis were evident. These results indicate that significant differences in the metabolic micromilieu can be detected in human tumors; these are due to varying perfusion rates and may be partly respo…

MaleCancer Researchmedicine.medical_specialtyLung NeoplasmsTransplantation HeterologousBiologyOxygen ConsumptionBreast cancerIn vivoInternal medicinemedicineAnimalsHumansGlycolysisLung cancerLungTumor hypoxiaMammary Neoplasms ExperimentalHydrogen-Ion Concentrationmedicine.diseaseRatsTransplantationEndocrinologymedicine.anatomical_structureOncologyCancer researchFemaleGlycolysisOxidation-ReductionPerfusionNeoplasm TransplantationInternational Journal of Cancer
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Localized hypothermia: impact on oxygenation, microregional perfusion, metabolic and bioenergetic status of subcutaneous rat tumours.

1998

The effect of localized hypothermia on microcirculatory and metabolic parameters in s.c. DS sarcomas on the hind foot dorsum of Sprague-Dawley rats was investigated. Tumours were cooled by superfusion of the tumour surface with cooled saline solution to 25 degrees C or 15 degrees C. Control tumours remained at 35 degrees C. These temperatures were maintained for 30 min. In tumour oxygenation measurements, hypothermia at 25 degrees C and 15 degrees C caused progressive decreases in the size of the fraction of pO2 measurements between 0 and 2.5 mmHg together with a reduction in pO2 variability. No significant changes in median or mean pO2 or in the fraction of pO2 measurements between 0 and 5…

MaleCancer Researchmedicine.medical_specialtyPathologySkin Neoplasmsmedicine.medical_treatmentBlood viscosityMicrocirculationRats Sprague-DawleyOxygen ConsumptionHypothermia InducedInternal medicinemedicineLaser-Doppler FlowmetryAnimalsSalinebusiness.industryMicrocirculationOxygenationLaser Doppler velocimetryHypothermiaBlood ViscosityRatsRed blood cellEndocrinologymedicine.anatomical_structureOncologySarcoma Experimentalmedicine.symptombusinessPerfusionResearch ArticleBritish Journal of Cancer
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Regional perfusion and oxygenation of tumors upon methylxanthine derivative administration

1998

Abstract Purpose: The use of methylxanthine derivatives has been postulated as a means of increasing tumor perfusion and thus ameliorating tumor hypoxia. The aim of this study was to quantify and compare the effects of three methylxanthine derivatives: pentoxifylline (PX), torbafylline (TB), and HWA 138 (HW) on tumor perfusion and oxygenation. Methods and Materials: Anesthetized Sprague Dawley rats with DS-sarcomas implanted subcutaneously onto the hind foot dorsum were used in this study. Mean arterial blood pressure (MABP) was measured throughout experiments. Regional red blood cell (RBC) flux was monitored using a multichannel laser Doppler device and tumor oxygenation on a more global l…

MaleCancer Researchmedicine.medical_specialtyVasodilator AgentsPentoxifyllineRats Sprague-DawleyOxygen ConsumptionPharmacokineticsInternal medicinemedicineAnimalsRadiology Nuclear Medicine and imagingPentoxifyllineRadiationTumor hypoxiabusiness.industryNeoplasms ExperimentalBlood flowOxygenationTumor OxygenationRatsSurgeryBlood pressureEndocrinologyOncologyRegional Blood FlowSarcoma ExperimentalbusinessPerfusionmedicine.drugInternational Journal of Radiation Oncology*Biology*Physics
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Sex differences in anthracycline-induced cardiotoxicity: the benefits of estrogens

2019

Anthracyclines are the cornerstone for many oncologic treatments, but their cardiotoxicity has been recognized for several decades. Female subjects, especially before puberty and adolescence, or after menopause, seem to be more at increased risk, with the prognostic impact of this sex issue being less consistent compared to other cardiovascular risk factors. Several studies imply that sex differences could depend on the lack of the protective effect of sex hormones against the anthracycline-initiated damage in cardiac cells, or on differential mitochondria-related oxidative gene expression. This is also reflected by the results obtained with different diagnostic methods, such as cardiovascu…

MaleCardiac & Cardiovascular SystemsMagnetic Resonance Spectroscopyand protection from anthracycline cardiotoxicitymedicine.disease_causeBioinformaticsRisk FactorsAnthracycline cardiotoxicityGender differenceGender differencesAnthracyclinesGonadal Steroid Hormones1102 Cardiorespiratory Medicine and HaematologyAMERICAN SOCIETYCardioprotectionSex CharacteristicsHeartPrognosisMitochondriaMenopauseEchocardiographyReperfusion InjuryHEART-FAILUREAnthracycline cardiotoxicity; Gender differences; Pathophysiology monitoring and protection from anthracycline cardiotoxicity; Anthracyclines; Biomarkers; Cardiotonic Agents; Cardiotoxicity; Echocardiography; Female; Gonadal Steroid Hormones; Heart; Heart Failure; Humans; Magnetic Resonance Spectroscopy; Male; Mitochondria; Nuclear Medicine; Oxidative Stress; Prognosis; Reperfusion Injury; Risk Factors; Sex CharacteristicsFemaleCardiology and Cardiovascular MedicineLife Sciences & BiomedicinePOSITION PAPERCARDIAC DYSFUNCTIONCardiotonic AgentsAnthracyclineSPECKLE-TRACKINGIschemiaDRUG CARDIOTOXICITYPathophysiologymedicineHumansCHILDHOOD-CANCER SURVIVORSBREAST-CANCERPathophysiology monitoring and protection from anthracycline cardiotoxicityHeart FailureCardiotoxicityScience & Technologybusiness.industryWORKING GROUPmedicine.diseaseCardiotoxicityOxidative StressmonitoringCardiovascular System & HematologyHeart failureCardiovascular System & CardiologyRISK-FACTORSNuclear MedicinebusinessOxidative stressAnthracycline cardiotoxicity; Gender differences; Pathophysiology monitoring and protection from anthracycline cardiotoxicityBiomarkersHormone
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