Search results for "Phage"

showing 10 items of 1573 documents

Damping excessive inflammation and tissue damage in Mycobacterium tuberculosis infection by Toll IL-1 receptor 8/single Ig IL-1-related receptor, a n…

2007

Abstract Toll IL-1R 8/single Ig IL-1-related receptor (TIR8/SIGIRR) is a member of the IL-1R family, expressed by epithelial tissues and immature dendritic cells, and is regarded as a negative regulator of TLR/IL-1R signaling. Tir8-deficient mice were rapidly killed by intranasal administration of low doses of Mycobacterium tuberculosis, despite controlling efficiently the number of viable bacilli in different organs. Tir8−/−-infected mice showed an increased number of neutrophils and macrophages in the lungs; however, mycobacteria-specific CD4 and CD8 T cells were similar in Tir8−/− and Tir8+/+ mice. Exaggerated mortality of Tir8−/− mice was due to massive liver necrosis and was accompanie…

TuberculosisNeutrophilsImmunologyInterleukin-1betaInflammationBiologyPeripheral blood mononuclear cellAntibodiesMycobacterium tuberculosisMiceNecrosisCell MovementMacrophages AlveolarmedicineImmunology and AllergyCytotoxic T cellAnimalsReceptorLungTuberculosis PulmonaryInflammationLungTumor Necrosis Factor-alphaToll-Like ReceptorsReceptors Interleukin-1Dendritic CellsMycobacterium tuberculosismedicine.diseasebiology.organism_classificationIn vitroMice Mutant Strainsmedicine.anatomical_structureLiverImmunologyCytokinesmedicine.symptomToll IL-1 Receptor 8/Single Ig IL-1-Related Receptor Inlfammation Mycobacterium tuberculosisInterleukin-1Signal Transduction
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IL-4 depletion enhances host resistance and passive IgA protection against tuberculosis infection in BALB/c mice.

2007

The influence of Th2 cytokines in tuberculosis has been a matter of dispute. Here we report that IL-4 has a profound regulatory effect on the infection of BALB/c mice with Mycobacterium tuberculosis. Depletion of IL-4 with a neutralizing mAb caused only evanescent reduction of lung infection, but when combined with i.n. inoculations of IgA anti-mycobacterial alpha-crystallin mAb and mouse rIFN-gamma, we observed a 40-fold reduction of the bacterial counts in the lungs at 3 wks following i.n. infection (p<0.001). In genetically deficient IL-4-/- BALB/c mice, infection in both lung and spleen was substantially reduced for up to 8 wks without further treatment. Reconstitution of IL-4-/- mice w…

Tuberculosismedicine.medical_treatmentImmunologySpleenNitric OxideBALB/cMicrobiologyProinflammatory cytokineMycobacterium tuberculosisInterferon-gammaMiceImmunitymedicineImmunology and AllergyAnimalsTuberculosis PulmonaryMice KnockoutMice Inbred BALB CbiologyMacrophagesImmunization PassiveImmunotherapyMycobacterium tuberculosisbiology.organism_classificationmedicine.diseaseAntibodies Bacterial infections Cytokines TuberculosisImmunity InnateImmunoglobulin Amedicine.anatomical_structureImmunologybiology.proteinInterleukin-4AntibodyEuropean journal of immunology
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Relevance of the macrophage phenotype in mucosal regeneration in Inflammatory Bowel Disease

2015

La enfermedad Inflamatoria Intestinal (EII) es una enfermedad crónica, recidivante, de carácter sistémico que afecta principalmente al tracto gastrointestinal. Esta patología engloba dos entidades clínicas, la Enfermedad de Crohn (EC) y la Colitis Ulcerosa (CU), que difieren en algunos aspectos pero comparten la disrupción de la barrera epitelial y la respuesta exacerbada del sistema inmunológico a la flora bacteriana. La etiología de la enfermedad es desconocida pero se sabe que factores genéticos (tanto alteraciones a nivel genético como la presencia de miRNAs), ambientales (tales como la dieta, tabaco, actividad física o la calidad del sueño), flora intestinal y sistema inmunológico cont…

UNESCO::CIENCIAS DE LA VIDA::Biología humana ::Fisiología humanawntinflammatory bowel disease:CIENCIAS MÉDICAS ::Patología::Inmunopatología [UNESCO]macrophage phenotypeUNESCO::CIENCIAS MÉDICAS ::Medicina interna::Gastroenterología:CIENCIAS MÉDICAS ::Medicina interna::Gastroenterología [UNESCO]wound healingUNESCO::CIENCIAS MÉDICAS ::Patología::Inmunopatologíamacrophage:CIENCIAS DE LA VIDA::Biología humana ::Fisiología humana [UNESCO]notch
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Hacia la caracterización del epítopo del autoantígeno en el Síndrome de Goodpasture

2014

El síndrome de Goodpasture (GP) es un desorden autoinmune que cursa con glomerulonefritis rápidamente progresiva y hemorragia pulmonar. El ataque inmunológico se lleva a cabo mediante (auto)-anticuerpos (anticuerpos GP) dirigidos contra el dominio C terminal, no colagenoso (NC1), de la cadena α3 del colágeno IV, presente en la membrana basal glomerular (GBM), así como en la membrana basal alveolar. Tres cadenas α se asocian para construir una molécula de colágeno IV, que recibe el nombre de protómero, dos protómeros de colágeno IV se unen a través de sus extremos NC1 para formar una estructura cuaternaria de naturaleza hexamérica (hexámero) en la red de colágeno IV. Estudios cristalográfico…

UNESCO::CIENCIAS DE LA VIDA::Biología molecularUNESCO::CIENCIAS DE LA VIDA::Inmunología::Reacción antígeno-anticuerpo:CIENCIAS MÉDICAS::Patología::Inmunopatología [UNESCO]GoodpastureAntígeno-anticuerpoepítopophage display:CIENCIAS DE LA VIDA::Inmunología::Reacción antígeno-anticuerpo [UNESCO]Autoinmune:CIENCIAS DE LA VIDA::Biología molecular [UNESCO]UNESCO::CIENCIAS MÉDICAS::Patología::Inmunopatología
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Association of AUUUA-binding Protein with A + U-rich mRNA during nucleo-cytoplasmic transport

1992

Resealed nuclear envelope (NE) vesicles from rat liver containing entrapped exogenous RNA were used to study the effect of adenosine+uridine binding factor (AUBF), present in cytosolic cell extracts, on ATP-dependent transport of A+U-rich RNA (AU+RNA) and A+U-free RNA (AU-RNA) across the NE. This factor specifically binds to A+U-rich sequences present in the 3' untranslated regions of lymphokine and cytokine mRNAs, containing overlapping AUUUA boxes (granulocyte-macrophage colony stimulating factor, interleukin-3). Addition of AUBF to the extravesicular compartment markedly increased the efflux of the in vitro transcribed, capped and polyadenylated AU+ RNAs. Export of entrapped AU- control …

Untranslated regionCytoplasmAdenosineTranscription GeneticPolyadenylationNuclear EnvelopeMolecular Sequence DataRNA-binding proteinBiologyCell LineStructural BiologyTranscription (biology)EndoribonucleasesAnimalsHumansNuclear MatrixRNA MessengerBinding siteNuclear export signalUridineMolecular BiologyCell NucleusMessenger RNABinding SitesBase SequenceGranulocyte-Macrophage Colony-Stimulating FactorInterferon-alphaRNA-Binding ProteinsRNAMolecular biologyRatsKineticsLiverRibonucleoproteinsInterleukin-3Carrier ProteinsPlasmidsPolyribonucleotidesProtein BindingJournal of Molecular Biology
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In Vivo Modulation of Angiogenesis and Immune Response on a Collagen Matrix via Extracorporeal Shockwaves

2020

The effective management of tissue integration and immunological responses to transplants decisively co-determines the success of soft and hard tissue reconstruction. The aim of this in vivo study was to evaluate the eligibility of extracorporeal shock wave therapy (ESWT) with respect to its ability to modulate angiogenesis and immune response to a collagen matrix (CM) for tissue engineering in the chorioallantoic membrane (CAM) assay, which is performed with fertilized chicken eggs. CM were placed on the CAM on embryonic development day (EDD) 7

Vascular Endothelial Growth Factor A0301 basic medicineAngiogenesismedicine.medical_treatmentNitric Oxide Synthase Type IIChick EmbryoChorioallantoic Membranelcsh:ChemistryNeovascularizationangiogenesischemistry.chemical_compoundmacrophage response0302 clinical medicineTissue engineeringlcsh:QH301-705.5Spectroscopyoral inflammationTissue Scaffoldsvascular endothelial growth factorGeneral MedicineComputer Science ApplicationsVascular endothelial growth factorChorioallantoic membraneExtracorporeal shockwave therapyCollagenmedicine.symptomchorioallantoic membrane assayNeovascularization PhysiologicArticleCatalysisAvian ProteinsInorganic ChemistryAndrology03 medical and health sciencesImmune systemIn vivomatrix metalloproteasesmucoderm®medicineAnimalsddc:610Physical and Theoretical Chemistrymucoderm<sup>®</sup>Molecular BiologyTissue Engineeringbusiness.industryOrganic Chemistrycollagen matrix030206 dentistryextracorporeal shockwave therapyHypoxia-Inducible Factor 1 alpha SubunitMatrix Metalloproteinases030104 developmental biologylcsh:Biology (General)lcsh:QD1-999chemistrybusiness
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High levels of HIF-2α highlight an immature neural crest-like neuroblastoma cell cohort located in a perivascular niche

2007

High HIF-2alpha protein levels in the sympathetic nervous system-derived childhood tumour neuroblastoma as well as immature phenotype correlate to unfavourable outcome. Here we show that a small subset of perivascularly located, strongly HIF-2alpha-positive tumour cells (MYCN amplified) lacks expression of differentiation markers, but expresses neural crest and early sympathetic progenitor marker genes such as Notch-1, HES-1, c-Kit, dHAND, and vimentin. HIF-2alpha- and CD68-positive tumour-associated macrophages were frequently found close to the immature and HIF-2alpha-positive neuroblastoma cells and as VEGF levels are high in the perivascular niche, we hypothesize that neuroblastoma neur…

Vascular Endothelial Growth Factor APathologymedicine.medical_specialtySympathetic Nervous SystemAngiogenesisVimentinPathology and Forensic MedicineNeuroblastomaNeuroblastomaBasic Helix-Loop-Helix Transcription FactorsTumor Cells CulturedmedicineHumansMacrophageProgenitorOncogene ProteinsN-Myc Proto-Oncogene ProteinNeovascularization PathologicbiologyMacrophagesNuclear ProteinsNeural crestmedicine.diseasePhenotypeCell HypoxiaNeoplasm ProteinsNeural CrestNeoplastic Stem Cellsbiology.proteinCancer researchStem cellThe Journal of Pathology
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On the pathogenesis of atherosclerosis: enzymatic transformation of human low density lipoprotein to an atherogenic moiety.

1995

Combined treatment with trypsin, cholesterol esterase, and neuraminidase transforms LDL, but not HDL or VLDL, to particles with properties akin to those of lipid extracted from atherosclerotic lesions. Single or double enzyme modifications, or treatment with phospholipase C, or simple vortexing are ineffective. Triple enzyme treatment disrupts the ordered and uniform structure of LDL particles, and gives rise to the formation of inhomogeneous lipid droplets 10-200 nm in diameter with a pronounced net negative charge, but lacking significant amounts of oxidized lipid. Enzymatically modified LDL (E-LDL), but not oxidatively modified LDL (ox-LDL), is endowed with potent complement-activating c…

Very low-density lipoproteinArteriosclerosisImmunologyNeuraminidaseComplement Membrane Attack Complexchemistry.chemical_compoundLipid dropletmedicineExtracellularImmunology and AllergyHumansTrypsinReceptors ImmunologicComplement ActivationGlycoproteinsReceptors Lipoproteinchemistry.chemical_classificationReceptors ScavengerPhospholipase CCholesterolMacrophagesMembrane ProteinsComplement C3Complement System ProteinsArticlesScavenger Receptors Class BSterol EsteraseTrypsinLipid MetabolismLipoproteins LDLEnzymechemistryBiochemistryLow-density lipoproteinlipids (amino acids peptides and proteins)medicine.drugFoam CellsThe Journal of experimental medicine
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July 2003: 62-year-old female with progressive muscular weakness

2004

The July 2003 Case of the Month (COM). A 62-year-old female patient experienced progressive muscular weakness over the last ten years, involving shoulder and pelvic girdle muscles, paraspinal and facial muscles. A biopsy was taken from the left deltoid muscle where hepatitis vaccination had taken place 4 weeks previously. The specimen revealed macrophagic myofasciitis due to the injection of aluminium-bound vaccines. The finding can be reproduced experimentally by injecting vaccines in rats. The pathomechanism is supposed to involve immune stimulation due to long term persistence of the adjuvant. Macrophagic myofasciitis has been suggested to occasionally cause myopathy but is supposed to b…

Viral Hepatitis Vaccinesmedicine.medical_specialtyAluminum HydroxideMass SpectrometryCases of the Month: July to September 2003Pathology and Forensic MedicineDiagnosis DifferentialBiopsymedicineHumansMuscle SkeletalMyopathyInclusion BodiesHepatitisMuscle WeaknessPelvic girdlemedicine.diagnostic_testbusiness.industryMacrophagesGeneral NeuroscienceMacrophagic myofasciitisMiddle Agedmedicine.diseaseMuscular Dystrophy FacioscapulohumeralSurgeryVaccinationMicroscopy ElectronFacial musclesmedicine.anatomical_structureFemaleNeurology (clinical)medicine.symptombusinessProgressive muscular weaknessBrain Pathology
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Optimization of the detection of bacteriophages induced from Listeria sp.

1997

It is necessary to isolate new phages in order to improve the rate of typeability of Listeria monocytogenes strains. We propose a method which increases the detection of induced phages in the presence of inhibitory substances synthesized or liberated by the cells during phage production. Of the 29 phages isolated, 11 (38%) were detected by the spot-on-the-lawn technique and 18 (62%) were revealed by the soft-agar technique. To increase the rate of phage detection, both techniques appear useful. Listeria cultures were subjected to phage typing procedures utilizing these newly isolated phages and the French International set of phages. It appears that the newly isolated phages are good tools …

Viral Plaque AssayListeriavirusesViral Plaque AssayBiologybiology.organism_classificationmedicine.disease_causeListeria monocytogenesApplied Microbiology and BiotechnologyVirologyVirusMicrobiologyBacteriophageListeria monocytogenesListeriamedicineBacteriophagesTypingBacteriophage TypingBacteriaPhage typingLetters in Applied Microbiology
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