Search results for "Pig"

showing 10 items of 2235 documents

Epigenetic changes underlie the aggressiveness of histologically benign meningiomas that recur

2019

Meningiomas are the most frequent primary brain tumor. Usually, they are curable by surgery, but even after seemingly complete resection, some low-grade lesions recur. Despite recent improvements, signatures having prognostic value in grade I tumors remain poorly characterized. The frequency and delicate location of these tumors suggest that the risk of recurrence might be more accurately predicted. Herein, we show an easy way to evaluate the methylation status of meningiomas and its correlation with the prognosis of the disease. A series of 120 meningiomas, including primary tumors and recurrences, were analyzed histopathologically, and 24 tumor suppressor genes (TSGs) were studied by meth…

AdultMale0301 basic medicineOncologymedicine.medical_specialtyAdolescentBrain tumorDiseaseMLH1Epigenesis GeneticPathology and Forensic MedicineMeningiomaYoung Adult03 medical and health sciences0302 clinical medicineCDKN2BInternal medicineMeningeal NeoplasmsmedicineHumansGenes Tumor SuppressorClinical significanceChildAgedAged 80 and overbusiness.industryDNA MethylationMiddle Agedmedicine.disease030104 developmental biology030220 oncology & carcinogenesisBenign MeningiomaDNA methylationFemaleNeoplasm Recurrence LocalMeningiomabusinessHuman Pathology
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Phenotypical features of two patients diagnosed with PHARC syndrome and carriers of a new homozygous mutation in the ABHD12 gene.

2018

Abstract PHARC (Polyneuropathy, Hearing loss, Ataxia, Retinitis pigmentosa and Cataracts) (MIM# 612674 ) is an autosomal recessive neurodegenerative disease caused by mutations in the ABHD12 gene. We evaluated two Spanish siblings affected with pes cavus, sensorimotor neuropathy, hearing loss, retinitis pigmentosa and juvenile cataracts in whom the genetic test of ABHD12 revealed a novel homozygous frameshift mutation, c.211_223del (p.Arg71Tyrfs*26). The earliest clinical manifestation in these patients was a demyelinating neuropathy manifested with a Charcot-Marie-Tooth phenotype over three decades. Progressive hearing loss, cataracts and retinitis pigmentosa appeared after the age of 30. …

AdultMaleARLID12 genecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyAtaxiagenetic structuresHearing lossUsher syndromeCharcot-Marie-Tooth diseaseCataractFrameshift mutation03 medical and health sciencesPolyneuropathies0302 clinical medicineCataractsRetinitis pigmentosaotorhinolaryngologic diseasesmedicineHumansMuscle SkeletalDeaf-blindnessbusiness.industryPHARCBrainmedicine.diseaseDermatologyMagnetic Resonance Imagingeye diseasesMonoacylglycerol LipasesPedigreePhenotypeNeurologySpainMutation030221 ophthalmology & optometryAtaxiasense organsNeurology (clinical)medicine.symptombusinessUsher syndromePolyneuropathy030217 neurology & neurosurgeryRetinitis PigmentosaRetinopathyJournal of the neurological sciences
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The latent structure of the functional dyspepsia symptom complex: A taxometric analysis

2016

OBJECTIVES: Rome III introduced a subdivision of functional dyspepsia (FD) into postprandial distress syndrome and epigastric pain syndrome, characterized by early satiation/postprandial fullness, and epigastric pain/burning, respectively. However, evidence on their degree of overlap is mixed. We aimed to investigate the latent structure of FD to test whether distinguishable symptom-based subgroups exist. METHODS: Consecutive tertiary care Rome II FD patients completed the dyspepsia symptom severity scale. Confirmatory factor analysis (CFA) was used to compare the fit of a single factor model, a correlated three-factor model based on Rome III subgroups and a bifactor model consisting of a g…

AdultMaleAbdominal painAdolescentPhysiologyEpigastric painYoung Adult03 medical and health sciences0302 clinical medicineSurveys and QuestionnairesmedicineHumansDyspepsiaLatent structureAgedAged 80 and overTertiary HealthcareEndocrine and Autonomic Systemsbusiness.industrySingle factorGastroenterologyNauseaMiddle AgedClassificationPostprandial Periodmedicine.diseaseRome iiiConfirmatory factor analysisAbdominal PainDistress030220 oncology & carcinogenesisFemale030211 gastroenterology & hepatologymedicine.symptombusinessSomatizationClinical psychology
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Translocations Disrupting PHF21A in the Potocki-Shaffer-Syndrome Region Are Associated with Intellectual Disability and Craniofacial Anomalies

2012

Contains fulltext : 110038.pdf (Publisher’s version ) (Closed access) Potocki-Shaffer syndrome (PSS) is a contiguous gene disorder due to the interstitial deletion of band p11.2 of chromosome 11 and is characterized by multiple exostoses, parietal foramina, intellectual disability (ID), and craniofacial anomalies (CFAs). Despite the identification of individual genes responsible for multiple exostoses and parietal foramina in PSS, the identity of the gene(s) associated with the ID and CFA phenotypes has remained elusive. Through characterization of independent subjects with balanced translocations and supportive comparative deletion mapping of PSS subjects, we have uncovered evidence that t…

AdultMaleAdolescentGenotypePotocki–Shaffer syndromeChromosome DisordersHaploinsufficiencyBiologyHistone DeacetylasesSodium ChannelsTranslocation GeneticArticleChromatin remodelingCraniofacial Abnormalities03 medical and health sciencesSCN3A0302 clinical medicineIntellectual DisabilityNAV1.3 Voltage-Gated Sodium ChannelmedicineTranscriptional regulationGeneticsAnimalsHumansDeletion mappingGenetics(clinical)CraniofacialZebrafishGenetics (clinical)030304 developmental biologyGenetics0303 health sciencesChromosomes Human Pair 11Infant Newbornmedicine.diseaseGenetics and epigenetic pathways of disease DCN MP - Plasticity and memory [NCMLS 6]Child PreschoolHomeoboxFemaleChromosome DeletionHaploinsufficiencyExostoses Multiple Hereditary030217 neurology & neurosurgeryThe American Journal of Human Genetics
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An evaluation of clinical, radiological and three-dimensional dental tomography findings in ectodermal dysplasia cases

2015

Background:This study aimed to review the results related to head and jaw disorders in cases of ectodermal dysplasia. The evaluation of ectodermal dysplasia cases was made by clincal examination and examination of the jaw and facial areas radiologically and on cone-beam 3-dimensional dental tomography (CBCT) images. Material and Methods: In the 36 cases evaluated in the study, typical clinical findings of pure hypohidrotic ectodermal displasia (HED) were seen, such as missing teeth, dry skin, hair and nail disorders. CBCT images were obtained from 12 of the 36 cases, aged 1.5- 45 years, and orthodontic analyses were made on these images. Results: The clinical and radiological evaluations de…

AdultMaleEctodermal dysplasiaAdolescentSaddle noseTooth resorptionDentistryOdontologíaEctodermal dysplasiaOligodontiathree-dimensional dental tomographyYoung AdultGingivitisImaging Three-Dimensionalstomatognathic systemmedicineHumansChildGeneral DentistryRetrospective StudiesEctodermal dysplasia three-dimensional dental tomographyHypopigmentationbusiness.industryResearchStomatognathic DiseasesInfantCone-Beam Computed TomographyMiddle AgedMedically compromised patients in Dentistry:CIENCIAS MÉDICAS [UNESCO]medicine.diseaseCiencias de la saludstomatognathic diseasesHypodontiamedicine.anatomical_structureOtorhinolaryngologyChild PreschoolUNESCO::CIENCIAS MÉDICASNail (anatomy)FemaleSurgerymedicine.symptombusinessMedicina Oral Patología Oral y Cirugia Bucal
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Leisure-time physical activity and DNA methylation age-a twin study.

2018

Background Epigenetic clocks may increase our understanding on human aging and how genetic and environmental factors regulate an individual aging process. One of the most promising clocks is Horvath’s DNA methylation (DNAm) age. Age acceleration, i.e., discrepancy between DNAm age and chronological age, tells us whether the person is biologically young or old compared to his/her chronological age. Several environmental and lifestyle factors have been shown to affect life span. We investigated genetic and environmental predictors of DNAm age in young and older monozygotic (MZ) and dizygotic (DZ) twins with a focus on leisure time physical activity. Results Quantitative genetic modeling revea…

AdultMaleEpigenetic clockQuantitative geneticsPhysical activityResearchTwin designAge FactorsTwins MonozygoticDNA MethylationMiddle AgedMethylationEpigenesis GeneticCohort StudiesYoung AdultAge DistributionTwins DizygoticHumansFemaleExerciseLife StyleAgedClinical epigenetics
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Two Novel Deletions (Array CGH Findings) in Pigment Dispersion Syndrome

2007

Purpose: We report the first male with pigment dispersion syndrome and a balanced translocation t(10;15)(p11.1;q11.1). Methods: Cytogenetic analyses using Giemsa banding and FISH methods, and array CGH were performed. Results: Array CGH analyses did not show altered DNA sequences in the breakpoints of the translocation, but revealed two novel deletions in 2q22.1 and 18q22.1. Conclusion: We suppose that the coexistence of t(10;15) and pigment dispersion syndrome in our patient is a coincidence. The deletion in 2q22.1, where the gene LRP1B has been located, may play a major role in the dysembryogenesis of the eye and cause the disorder.

AdultMaleEye DiseasesLRP1BG bandingChromosomal translocationBiologyTranslocation GeneticDNA sequencingmedicineHumansPigment Epithelium of EyeGeneIn Situ Hybridization FluorescenceGenetics (clinical)Sequence DeletionGeneticsChromosomes Human Pair 15Chromosomes Human Pair 10BreakpointNucleic Acid Hybridizationmedicine.diseaseMolecular biologyOphthalmologyPediatrics Perinatology and Child HealthPigment dispersion syndromeFish <Actinopterygii>Retinal PigmentsOphthalmic Genetics
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Mutations in the gene encoding the basal body protein RPGRIP1L, a nephrocystin-4 interactor, cause Joubert syndrome.

2007

Peters, T.A./0000-0001-8443-5500; van Beersum, Sylvia E.C./0000-0002-4552-2908; Cremers, Frans/0000-0002-4954-5592; Roepman, Ronald/0000-0002-5178-8163 WOS: 000247619800019 PubMed: 17558407 Protein- protein interaction analyses have uncovered a ciliary and basal body protein network that, when disrupted, can result in nephronophthisis ( NPHP), Leber congenital amaurosis, Senior- Loken syndrome ( SLSN) or Joubert syndrome ( JBTS)(1-6). However, details of the molecular mechanisms underlying these disorders remain poorly understood. RPGRIP1- like protein ( RPGRIP1L) is a homolog of RPGRIP1 ( RPGR-interacting protein 1), a ciliary protein defective in Leber congenital amaurosis(7,8). We show t…

AdultMaleHealth aging / healthy living [IGMD 5]Eye DiseasesGenetics and epigenetic pathways of disease [NCMLS 6]TMEM67Molecular Sequence DataMembrane transport and intracellular motility [NCMLS 5]Biologymedicine.disease_causeJoubert syndromeCell LineGenomic disorders and inherited multi-system disorders [IGMD 3]NephronophthisisCerebellar DiseasesGeneticsmedicinePerception and Action [DCN 1]Basal bodyAnimalsHumansNeurosensory disorders [UMCN 3.3]CiliaAdaptor Proteins Signal TransducingRenal disorder [IGMD 9]GeneticsMutationCiliumCiliary transition zoneProteinsSyndromemedicine.diseasePedigreeRatsCytoskeletal ProteinsGenetic defects of metabolism [UMCN 5.1]RPGRIP1LFemaleKidney DiseasesFunctional Neurogenomics [DCN 2]Ciliary Motility Disorders
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Cadmium, Smoking, and Human Blood DNA Methylation Profiles in Adults from the Strong Heart Study

2020

The epigenetic effects of individual environmental toxicants in tobacco remain largely unexplored. Cadmium (Cd) has been associated with smoking-related health effects, and its concentration in tobacco smoke is higher in comparison with other metals. We studied the association of Cd and smoking exposures with human blood DNA methylation (DNAm) profiles. We also evaluated the implication of findings to relevant methylation pathways and the potential contribution of Cd exposure from smoking to explain the association between smoking and site-specific DNAm. We conducted an epigenome-wide association study of urine Cd and self-reported smoking (current and former vs. never, and cumulative smoki…

AdultMaleHealth Toxicology and Mutagenesischemistry.chemical_elementPhysiology010501 environmental sciencesBiology01 natural sciencesEpigenesis Genetic03 medical and health sciences0302 clinical medicineHumans030212 general & internal medicineEpigenetics0105 earth and related environmental sciencesAgedCadmiumHuman bloodResearchSmokingPublic Health Environmental and Occupational HealthEnvironmental ExposureDNA MethylationMiddle AgedchemistryDNA methylationFemaleCadmiumGenome-Wide Association StudyEnvironmental Health Perspectives
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Dimensions of the laryngeal framework in adults

1994

The increasing application of sophisticated electrophysiological, radiological and surgical methods to the diagnosis and treatment of laryngeal disorders requires a profound knowledge of the size and proportions of the human larynx and it's cartilaginous components. Only inadequate data regarding this subject have so far been accessible. The larynges of 53 patients (28 male and 25 female, age 25-88 years, in the means 59 years) were removed during routine autopsy 12-48 h post mortem and immediately submitted to morphometric investigation. None of the patients had histories or visible signs of laryngeal disease. Anatomical preparations were performed with customary surgical tools and morphom…

AdultMaleLarynxmedicine.medical_specialtyLaryngeal CartilagesAutopsyEpiglottic CartilagePathology and Forensic MedicineCricoid cartilagemedicineHumansRadiology Nuclear Medicine and imagingAgedAged 80 and overAnthropometrybusiness.industryThyroidArytenoid cartilageAnatomyMiddle AgedLaryngeal Disordermedicine.anatomical_structureOrthopedic surgeryFemaleSurgeryLarynxAnatomybusinessSurgical and Radiologic Anatomy
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