Search results for "Pion"

showing 10 items of 1304 documents

Functional evidence of multidrug resistance transporters (MDR) in rodent olfactory epithelium.

2012

WOS: 000305340700029; International audience; BACKGROUND: P-glycoprotein (Pgp) and multidrug resistance-associated protein (MRP1) are membrane transporter proteins which function as efflux pumps at cell membranes and are considered to exert a protective function against the entry of xenobiotics. While evidence for Pgp and MRP transporter activity is reported for olfactory tissue, their possible interaction and participation in the olfactory response has not been investigated. PRINCIPAL FINDINGS: Functional activity of putative MDR transporters was assessed by means of the fluorometric calcein acetoxymethyl ester (calcein-AM) accumulation assay on acute rat and mouse olfactory tissue slices.…

MaleAnatomy and Physiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionGene Expressionlcsh:MedicineATP-binding cassette transporterPharmacologyMicechemistry.chemical_compoundMolecular Cell Biologypolycyclic compoundslcsh:ScienceMice Inbred BALB CMultidisciplinaryNeuromodulationProbenecidReverse Transcriptase Polymerase Chain ReactionNeurochemistryFluoresceinsSensory SystemsCell biologyElectrophysiologymedicine.anatomical_structureAlimentation et NutritionCyclosporineQuinolinesMedicineFemaleEffluxCellular TypesMultidrug Resistance-Associated Proteinsproduct p-glycoprotein;blood-brain-barrier;receptor neurons;cyclic-nucleotides;tumor-cells;expression;localization;protein;gene;tissuesMultidrug Resistance-Associated ProteinsResearch ArticleATP Binding Cassette Transporter Subfamily BNeurophysiologyBiologyOlfactory Receptor NeuronsOlfactory mucosaPsychologie (Sciences cognitives)Olfactory MucosaPeripheral Nervous SystemmedicineAnimalsFood and NutritionRats WistarBiologyOlfactory SystemOlfactory receptorlcsh:RNeurosciencesEpithelial CellsBiological TransportTransporterRatsCalceinMicroscopy FluorescenceVerapamilchemistryNeurons and Cognitionlcsh:QPropionates[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOlfactory epitheliumNeuroscience
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GAS CHROMATOGRAPHIC ESTIMATION OF ACETYLCHOLINE IN THE RABBIT HEART USING A NITROGEN SELECTIVE DETECTOR

1973

The distribution of ACh in the rabbit heart was investigated by a modified gas chromatographic estimation method. ACh was extracted with perchloric acid, precipitated as reineckate and demethylated with sodium benzenethiolate. The tertiary amines derived from ACh and other choline esters were concentrated by a microdistillation procedure. Gas chromatography was performed using a nitrogen selective detector. In the range of concentrations between 0.4 and 2.5 nmol ACh per tissue sample the coefficient of variation was 5.2 per cent. The recovery of ACh added to heart extracts was 101 per cent. Evidence for the identity of the choline ester isolated from rabbit hearts and authentic ACh was obta…

MaleChromatography GasHeart VentriclesCoefficient of variationSodiumchemistry.chemical_elementBlood PressureBiochemistryCholineCellular and Molecular Neurosciencechemistry.chemical_compoundMethodsmedicineAnimalsBioassayCholineHeart AtriaPerchloric acidButyrylcholineChromatographyChemistryMyocardiumAcetylcholineRatsButyratesBiological AssayFemaleRabbitsGas chromatographyPropionatesAcetylcholinemedicine.drugJournal of Neurochemistry
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The Impact of World and European Football Cups on Stroke in the Population of Dijon, France: A Longitudinal Study from 1986 to 2006

2013

Background: Acute stress may trigger vascular events. We aimed to investigate whether important football competitions involving the French football team increased the occurrence of stroke. Methods: We retrospectively retrieved data of fatal and nonfatal stroke during 4 World Football Cups (1986, 1998, 2002, and 2006) and 4 European Championships (1992, 1996, 2000, and 2004), based on data from the population-based Stroke Registry of Dijon, France. One period of exposure was analyzed: the period of competition extended to 15 days before and after the competitions. The number of strokes was compared between exposed and unexposed corresponding periods of preceding and following years using Poi…

MaleCompetitive BehaviorLongitudinal studyTime FactorsPopulationFootballRisk Assessmentsymbols.namesakeRisk FactorsSoccerOdds RatioHumansMedicineLongitudinal StudiesRegistriesPoisson regressionChampionshipeducationStrokeAgedRetrospective Studieseducation.field_of_studyChi-Square Distributionbusiness.industryRehabilitationEuphoriaMiddle Agedmedicine.diseaseConfidence intervalStrokeRelative riskMultivariate AnalysissymbolsFemaleTelevisionSurgeryFranceNeurology (clinical)Cardiology and Cardiovascular MedicinebusinessStress PsychologicalDemographyJournal of Stroke and Cerebrovascular Diseases
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Effects of bupropion, alone or coadministered with nicotine, on social behavior in mice

2008

Bupropion, administered alone or combined with nicotine, is presently used to treat nicotine dependence. Despite experimental evidence of the complex behavioral actions of this drug, there have been little data reported about its effects on social behavior. Our main aim was to investigate the effects of acute administration of bupropion, alone or plus nicotine, on social interaction in mice. OF1 group-housed male mice were confronted in a neutral cage with an anosmic opponent during a 10 minutes encounter. Time allocated to body care and digging was reduced by administration of bupropion (40 mg/kg) both when administered alone and with nicotine (1 and 0.5 mg/kg). The lowest dose of bupropio…

MaleDrugNicotinemedicine.drug_classmedia_common.quotation_subjectMedicine (miscellaneous)Male miceAnxietyPharmacologyAnxiolyticDrug Administration ScheduleNicotineMiceDopamine Uptake Inhibitorsmental disordersmedicineAnimalsSocial BehaviorNicotine dependenceBupropionmedia_commonPharmacologyBupropionBehavior AnimalLow doseTobacco Use Disordermedicine.diseaseGanglionic StimulantsAggressionPsychiatry and Mental healthExploratory BehaviorPsychologymedicine.drugAddiction Biology
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Effects of co-administration of bupropion and nicotinic agonists on the elevated plus-maze test in mice

2005

There is evidence that the cholinergic nicotinic system is involved in the modulation of anxiety. Anxiolytic and anxiogenic effects of nicotine agonists have been reported in mice. Bupropion is an antidepressant drug which may alleviate some symptoms of nicotine withdrawal, although its effects on anxiety are not clear. It has been suggested that the interaction between bupropion and nicotinic mechanisms could be complex. The aim of the present study was to investigate acute effects of co-administration of bupropion and nicotinic agonists on the elevated plus-maze test in NMRI mice. Effects of nicotine, lobeline, and cytisine (0.35 and 0.175 mg/kg), administered alone or combined with bupro…

MaleElevated plus mazemedicine.drug_classPharmacologyAnxiolyticNicotineMicechemistry.chemical_compoundDopamine Uptake Inhibitorsmental disordersmedicineAnimalsLobelineNicotinic AgonistsMaze LearningBupropionBiological PsychiatryPharmacologyBupropionAnalysis of VarianceBehavior AnimalDose-Response Relationship Drugbusiness.industrymedicine.diseaseDrug CombinationsNicotinic agonistNicotine withdrawalchemistryAnxiogenicbusinesspsychological phenomena and processesmedicine.drugProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Endothelium- and nitric oxide-dependent vasorelaxing activities of gamma-butyrobetaine esters: possible link to the antiischemic activities of mildro…

2004

Mildronate [3-(2,2,2-trimethylhydrazine) propionate (THP)] is an antiischemic drug acting mainly via inhibition of fatty acid beta-oxidation. Some effects of the drug cannot be explained by the latter mechanism. We tested the eventual nitric oxide (NO) dependence of the mildronate action. Mildronate, gamma-butyrobetaine (GBB) and GBB methyl ester induced transient increases in nitric oxide (NO) concentrations in rat blood and myocardium. In vitro, these compounds neither modified the activities of purified neuronal and endothelial recombinant nitric oxide synthases (NOSs) nor were able to interact with their active site. GBB induced vasodilatation at high concentrations only (EC50 = 5 x 10(…

MaleEndotheliumNitric Oxide Synthase Type IIIStereochemistryDrug Evaluation PreclinicalMyocardial IschemiaVasodilationAorta ThoracicNitric OxideMuscle Smooth VascularNitric oxidechemistry.chemical_compoundCarnitinemedicineAnimalsEndotheliumRats WistarPharmacologychemistry.chemical_classificationbiologyElectron Spin Resonance SpectroscopyActive siteFatty acidDrug SynergismRatsNitric oxide synthaseBetaineVasodilationDrug Combinationsmedicine.anatomical_structureEnzymeNG-Nitroarginine Methyl Esterchemistrybiology.proteinPropionateNitric Oxide SynthaseDitiocarbMethylhydrazinesEuropean journal of pharmacology
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Clinical and Biological Heterogeneity in Children with Moderate Asthma

2003

To evaluate the relationship between inflammatory markers and severity of asthma in children, the amount of interleukin-8 (IL-8) and granulocyte/macrophage colony-stimulating factor (GM-CSF) released by peripheral blood mononuclear cells, exhaled nitric oxide (FE NO) levels, p65 nuclear factor-kappaB subunit, and phosphorylated IkBalpha expression by peripheral blood mononuclear cells were assessed in six control subjects, 12 steroid-naives subjects with intermittent asthma, and 17 children with moderate asthma. To investigate their predictive value, biomarker levels were correlated with the number of exacerbations during a 18-month follow-up period. We found that GM-CSF release was higher …

MaleExacerbationAnti-Inflammatory AgentsCritical Care and Intensive Care MedicineSynaptotagminsMedicineChildSalmeterol XinafoateCalcium-Binding ProteinMembrane GlycoproteinsRespiratory diseaseNF-kappa Binflammatory markersBronchodilator AgentsAnti-Inflammatory AgentSynaptotagmin IBiomarker (medicine)FemaleMembrane GlycoproteinAndrostadienes; Anti-Inflammatory Agents; NF-kappa B; Leukocytes Mononuclear; Membrane Glycoproteins; Granulocyte-Macrophage Colony-Stimulating Factor; Humans; Synaptotagmins; Albuterol; Asthma; Child; Receptors Cell Surface; Nerve Tissue Proteins; Nitric Oxide; Synaptotagmin I; Calcium-Binding Proteins; Interleukin-8; Adolescent; Bronchodilator Agents; Male; Biological Markers; Femalemedicine.symptomHumanmedicine.drugPulmonary and Respiratory MedicineAdolescentNerve Tissue ProteinsReceptors Cell SurfaceInflammationNitric OxidePeripheral blood mononuclear cellFluticasone propionateHumansAlbuterolBronchodilator AgentAsthmaAndrostadienefluticasone propionatebusiness.industryCalcium-Binding ProteinsInterleukin-8Granulocyte-Macrophage Colony-Stimulating Factormedicine.diseaseSynaptotagminAsthmaAndrostadienesasthma; inflammatory markers; fluticasone propionateNerve Tissue ProteinBiological MarkerExhaled nitric oxideImmunologyLeukocytes MononuclearFluticasonebusinessBiomarkersAmerican Journal of Respiratory and Critical Care Medicine
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Long-term efficacy of botulinum toxin A for treatment of blepharospasm,hemifacial spasm, and spastic entropion: a multicentre study using two drug-do…

2009

PURPOSE: To investigate the long-term effectiveness and safety of botulinum neurotoxin A (BoNT-A) treatment in patients with blepharospasm (BEB), hemifacial spasm (HFS), and entropion (EN) and to use for the first time two modified indexes, 'botulin toxin escalation index-U' (BEI-U) and 'botulin toxin escalation index percentage' (BEI-%), in the dose-escalation evaluation. METHODS: All patients in this multicentre study were followed for at least 10 years and main outcomes were clinical efficacy, duration of relief, BEI-U and BEI-%, and frequency of adverse events. RESULTS: BEB, HFS, and EN patients received a mean BoNT-A dose with a significant inter-group difference (P<0.0005, respectivel…

MaleEye diseaseEcchymosisBlepharospasmBlepharospasmBlepharospasm Hemifacial spasmPtosismedicineHumansHemifacial SpasmLongitudinal StudiesBotulinum Toxins Type AAdverse effectAgedDiplopiaAged 80 and overDose-Response Relationship Drugbusiness.industrySettore MED/30 - Malattie Apparato VisivoEntropionMiddle Agedmedicine.diseasebotulinum toxin A; Blepharospasm Hemifacial spasm; entropion; drug-dose escalation indexdrug-dose escalation indexEntropionOphthalmologyDrug CombinationsNeuromuscular AgentsMuscle SpasticityAnesthesiaFemalemedicine.symptombotulinum toxin AbusinessHemifacial spasmFollow-Up Studies
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Tolerance-Inducing Dose of 3-Nitropropionic Acid Modulates bcl-2 and bax Balance in the Rat Brain: A Potential Mechanism of Chemical Preconditioning

2000

Many studies have reported ischemia protection using various preconditioning techniques, including single dose 3-nitropropionic acid (3-NPA), a mitochondrial toxin. However, the cellular signal transduction cascades resulting in ischemic tolerance and the mechanisms involved in neuronal survival in the tolerant state still remain unclear. The current study investigated the mRNA and protein expression of the antiapoptotic bcl-2 and the proapoptotic bax, two antagonistic members of the bcl-2 gene family, in response to a single dose of 3-NPA, to global cerebral ischemia–reperfusion, and to the combination of both 3-NPA-pretreatment and subsequent global cerebral ischemia–reperfusion. Brain h…

MalePathologymedicine.medical_specialtyIschemiaPharmacologyBiology030218 nuclear medicine & medical imaging03 medical and health sciences0302 clinical medicineBcl-2-associated X proteinDrug toleranceProto-Oncogene ProteinsGene expressionmedicineAnimalsRNA MessengerRats WistarIschemic Preconditioningbcl-2-Associated X ProteinMessenger RNABrainDrug ToleranceNitro Compoundsmedicine.diseasePathophysiologyRatsReal-time polymerase chain reactionProto-Oncogene Proteins c-bcl-2NeurologyIschemic Attack TransientApoptosisbiology.proteinNeurology (clinical)PropionatesCardiology and Cardiovascular Medicine030217 neurology & neurosurgeryJournal of Cerebral Blood Flow &amp; Metabolism
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Increased Hypoxic Tolerance by Chemical Inhibition of Oxidative Phosphorylation: “Chemical Preconditioning”

1997

A short ischemic episode preceding sustained ischemia is known to increase tolerance against ischemic cell death. We report early-onset long-lasting neuroprotection against in vitro hypoxia by preceding selective chemical inhibition of oxidative phosphorylation: “chemical preconditioning.” The amplitude of CA1population spikes (psap) in hippocampal slices prepared from control animals (control slices) was 31 ± 27% (mean ± SD) upon 45-min recovery from 15-min in vitro hypoxia. In slices prepared from animals treated in vivo with 20 mg/kg 3-nitropropionate (3-np) 1–24 h prior to slice preparation (preconditioned slices), psap improved to 90 ± 15% (p &lt; 0.01). Posthypoxic oxygen free radical…

MalePotassium ChannelsFree RadicalsPopulationIschemiaNerve Tissue ProteinsBiologyPharmacologyHippocampusNeuroprotectionOxidative PhosphorylationBrain Ischemia030218 nuclear medicine & medical imagingGlibenclamide03 medical and health sciencesAdenosine Triphosphate0302 clinical medicineSlice preparationIn vivoGlyburidemedicineAnimalsEnzyme InhibitorsRats WistarHypoxia BraineducationNeuronseducation.field_of_studyAntagonistHypoxia (medical)NADNitro Compoundsmedicine.diseaseCell HypoxiaRatsSuccinate DehydrogenaseNeuroprotective AgentsNeurologyAnesthesiaNeurology (clinical)Propionatesmedicine.symptomReactive Oxygen SpeciesCardiology and Cardiovascular Medicine030217 neurology & neurosurgerymedicine.drugJournal of Cerebral Blood Flow &amp; Metabolism
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