Search results for "Polysaccharides"

showing 10 items of 413 documents

Oleanolic acid and hederagenin glycosides from Weigela stelzneri

2015

Four previously undescribed and one known oleanolic acid glycosides were isolated from the roots of Weigela stelzneri, and one previously undescribed and three known hederagenin glycosides were isolated from the leaves. Their structures were elucidated mainly by 2D NMR spectroscopic analysis and mass spectrometry as 3-O-β-D-glucopyranosyl-(1 → 2)-[β-D-xylopyranosyl-(1 → 4)]-β-D-xylopyranosyl-(1 → 4)-β-D-xylopyranosyl-(1 → 3)-α-L-rhamnopyranosyl-(1 → 2)-α-L-arabinopyranosyloleanolic acid, 3-O-β-D-glucopyranosyl-(1 → 2)-[β-D-xylopyranosyl-(1 → 4)]-β-D-xylopyranosyl-(1 → 4)-β-D-xylopyranosyl-(1 → 3)-α-L-rhamnopyranosyl-(1 → 2)-β-D-xylopyranosyloleanolic acid, 3-O-β-D-glucopyranosyl-(1 → 2)-[β-…

LipopolysaccharidesWeigelaStereochemistryInterleukin-1betaStereoisomerismTumor cellsPlant ScienceHorticultureCaprifoliaceaePlant Roots01 natural sciencesBiochemistrychemistry.chemical_compoundGlycosidesOleanolic AcidNuclear Magnetic Resonance BiomolecularMolecular BiologyOleanolic acidchemistry.chemical_classificationDose-Response Relationship DrugLow toxicitybiology010405 organic chemistryGlycosideStereoisomerismGeneral Medicinebiology.organism_classification0104 chemical sciencesPlant Leaves010404 medicinal & biomolecular chemistryHederageninchemistryTwo-dimensional nuclear magnetic resonance spectroscopyPhytochemistry
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Inhibition of leukocyte functions by the alkaloid isaindigotone from Isatis indigotica and some new synthetic derivatives.

2001

The alkaloid isaindigotone (1a) and seven derivatives have been synthesized to study their influence on several leukocyte functions and the generation of inflammatory mediators. Isaindigotone (1a) was found to be a scavenger of superoxide generated either by the hypoxanthine/xanthine oxidase system or stimulated human neutrophils. Isaindigotone (1a) and its acetylated derivative (1b) also inhibited 5-lipoxygenase activity and leukotriene B(4) production in these cells, whereas none of the compounds affected degranulation. In RAW 264.7 macrophages stimulated with lipopolysaccharide, synthetic derivatives exerted higher inhibitory effects on prostaglandin E(2) (PGE(2)) and nitric oxide (NO) g…

LipopolysaccharidesXanthine OxidaseMagnetic Resonance SpectroscopyLeukotriene B4StereochemistryNeutrophilsmedicine.medical_treatmentPharmaceutical ScienceLeukotriene B4DinoprostoneAnalytical ChemistryNitric oxidechemistry.chemical_compoundInhibitory Concentration 50MiceStructure-Activity RelationshipAlkaloidsDrug DiscoverymedicineLeukocytesAnimalsHumansLipoxygenase InhibitorsXanthine oxidaseHypoxanthineCells CulturedPharmacologyInflammationPlants MedicinalbiologyMolecular StructureSuperoxideAlkaloidMacrophagesOrganic ChemistryFree Radical ScavengersComplementary and alternative medicineBiochemistrychemistryArachidonate 5-lipoxygenaseBrassicaceaebiology.proteinQuinazolinesMolecular MedicineChromatography Thin LayerInflammation MediatorsNitric Oxide SynthaseProstaglandin EJournal of natural products
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Structural determination of the lipo-chitin oligosaccharide nodulation signals produced by Rhizobium giardinii bv. giardinii H152

2003

Abstract Rhizobium giardinii bv. giardinii is a microsymbiont of plants of the genus Phaseolus and produces extracellular signal molecules that are able to induce deformation of root hairs and nodule organogenesis. We report here the structures of seven lipochitooligosaccharide (LCO) signal molecules secreted by R. giardinii bv. giardinii H152. Six of them are pentamers of GlcNAc carrying C 16:0 , C 18:0 , C 20:0 and C 18:1 fatty acyl chains on the non-reducing terminal residue. Four are sulfated at C-6 of the reducing terminal residue and one is acetylated in the same position. Six of them are N -methylated on the non-reducing GlcN residue and all the nodulation factors are carbamoylated o…

Lipopolysaccharides[CHIM.POLY] Chemical Sciences/PolymersMagnetic Resonance SpectroscopySTRUCTUREStereochemistrySubstituentRoot hairBiochemistryMass SpectrometryAnalytical ChemistryResidue (chemistry)chemistry.chemical_compoundSulfationGlucosamineCarbohydrate ConformationChromatographyMolecular StructurebiologyFatty AcidsMonosaccharidesOrganic ChemistryGeneral MedicineMethylationbiology.organism_classification[CHIM.POLY]Chemical Sciences/PolymersCarbohydrate SequencechemistryAcetylationPhaseolusRhizobium
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Core oligosaccharide of Escherichia coli B—the structure required for bacteriophage T4 recognition

2015

Abstract The structure of Escherichia coli B strain PCM 1935 core oligosaccharide has been investigated by 1H and 13C NMR spectroscopy, MALDI-TOF MS and ESI MSn. It was concluded that the core oligosaccharide is a pentasaccharide with the following structure: ESI MS/MS analysis revealed that the glycine (a minor component) is linked to the →3,7)- l -α- d -Hepp-(1→ residue.

Lipopolysaccharidesanimal structuresStereochemistryElectrospray ionizationMolecular Sequence Datamedicine.disease_causeBiochemistryAnalytical ChemistryBacteriophageResidue (chemistry)13c nmr spectroscopyEscherichia colimedicineBacteriophage T4Escherichia coliChromatographybiologyStrain (chemistry)ChemistryCore oligosaccharideOrganic ChemistryGeneral Medicinebiology.organism_classificationEscherichia coli B; Core oligosaccharide; glycine; NMR; MALDI-TOF; ESI MSCarbohydrate SequenceGlycineCarbohydrate Research
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Structure of a polysaccharide from the lipopolysaccharide of Vibrio vulnificus CECT4602 containing 2-acetamido-2,3,6-trideoxy-3-[(S)- and (R)-3-hydro…

2008

A polysaccharide was isolated by GPC after mild acid treatment of the lipopolysaccharide of Vibrio vulnificus CECT4602 and found to contain L-Rha, D-GlcpNAc and 2-acetamido-2,3,6-trideoxy-3-(3-hydroxybutanoylamino)-L-mannose (L-RhaNAc3NHb). GLC analysis of the trifluoroacetylated (S)-2-octyl esters derived by full acid hydrolysis of the polysaccharide showed that approximately 80% of the 3-hydroxybutanoic acid has the S configuration and approximately 20% the R configuration. The following structure of the polysaccharide was established by (1)H and (13)C NMR spectroscopies, including 2D ROESY and (1)H/(13)C HMBC experiments: [carbohydrate sequence see in text].

Lipopolysaccharideschemistry.chemical_classificationMagnetic Resonance SpectroscopybiologyLipopolysaccharideStereochemistryMolecular Sequence DataPolysaccharides BacterialOrganic ChemistryMannoseGeneral MedicineVibrio vulnificusCarbon-13 NMRbiology.organism_classificationPolysaccharideBiochemistryAnalytical Chemistrychemistry.chemical_compoundCarbohydrate SequencechemistryAcid hydrolysisAcid treatmentMannoseVibrio vulnificusCarbohydrate Research
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CiC3-1a-Mediated Chemotaxis in the Deuterostome Invertebrate Ciona intestinalis (Urochordata)

2003

Abstract Deuterostome invertebrates possess complement genes, and in limited instances complement-mediated functions have been reported in these organisms. However, the organization of the complement pathway(s), as well as the functions exerted by the cloned gene products, are largely unknown. To address the issue of the presence of an inflammatory pathway in ascidians, we expressed in Escherichia coli the fragment of Ciona intestinalis C3-1 corresponding to mammalian complement C3a (rCiC3-1a) and assessed its chemotactic activity on C. intestinalis hemocytes. We found that the migration of C. intestinalis hemocytes toward rCiC3-1a was dose dependent, peaking at 500 nM, and was specific for…

Lipopolysaccharidescomplement system ascidiansHemocytesMolecular Sequence DataIn situ hybridizationPertussis toxinimmunologyHemolymphEscherichia coliAnimalsImmunology and AllergyCiona intestinalisAmino Acid SequencePeptide sequenceinnate immunityInflammationCell-Free SystemChemotactic FactorsbiologyImmune SeraRiboprobeChemotaxisAnatomybiology.organism_classificationRecombinant ProteinsComplement systemCell biologyCiona intestinalisChemotaxis LeukocyteHemocyte migrationPertussis ToxinCell Migration InhibitionComplement C3a
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Inflammatory Characteristics of Monocytes from Pediatric Patients with Tuberous Sclerosis.

2015

Objective  Therapeutic options for the tuberous sclerosis complex (TSC) syndrome showed varying outcomes. Malfunctional tsc1 / tsc2 genes leave mTOR uninhibited, a positive downstream modulator of the innate proinflammatory immune system, which has not yet been described in pediatric patients with TSC. Methods  Using polymerase chain reaction (PCR) gene expression levels of monocytes after cultivation with lipopolysaccharide (LPS) or with LPS + mTOR inhibitor rapamycin, patients with TSC ( n  = 16) were compared with healthy subjects ( n  = 20). Results  Compared with monocytes from healthy controls, LPS showed a more prominent gene expression pattern in patients with TSC (CCL24, CXCL10, IL…

Lipopolysaccharidescongenital hereditary and neonatal diseases and abnormalitiesLipopolysaccharideGene ExpressionMonocytesProinflammatory cytokinechemistry.chemical_compoundTuberous sclerosisTuberous SclerosisGene expressionmedicineCXCL10HumansChildInflammationSirolimusbusiness.industryTOR Serine-Threonine KinasesInfant NewbornInfantGeneral Medicinemedicine.diseasemedicine.anatomical_structureCross-Sectional StudieschemistryChild PreschoolPediatrics Perinatology and Child HealthImmunologyCytokinesNeurology (clinical)TSC1TSC2Inflammation MediatorsbusinessCCL24Immunosuppressive AgentsNeuropediatrics
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The lipopolysaccharide O side chain of Vibrio vulnificus serogroup E is a virulence determinant for eels

1997

Vibrio vulnificus is a gram-negative bacterium capable of producing septicemic infections in eels and immunocompromised humans. Two biotypes are classically recognized, with the virulence for eels being specific to strains belonging to biotype 2, which constitutes a homogeneous lipopolysaccharide (LPS)-based O serogroup (which we have designated serogroup E). In the present study we demonstrated that the O side chain of this LPS determines the selective virulence of biotype 2 for eels: (i) biotype 1 strains (which do not belong to serogroup E) are destroyed by the bactericidal action of nonimmune eel serum (NIS) through activation of the alternative pathway of complement, (ii) biotype 2 str…

Lipopolysaccharidesendocrine systemanimal structuresLipopolysaccharideComplement Pathway AlternativeImmunologyMutantVirulenceVibrio vulnificusBiologyMicrobiologyMicrobiologychemistry.chemical_compoundPhagocytosisVibrionaceaeAnimalsVibrioEelsVirulenceO Antigensbiology.organism_classificationVirologyVibrioInfectious DiseaseschemistryAlternative complement pathwayImmunizationParasitologyBacteriaResearch Article
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Tiratricol neutralizes bacterial endotoxins and reduces lipopolysaccharide-induced TNF-alpha production in the cell.

2008

Contains fulltext : 70610.pdf (Publisher’s version ) (Closed access) The screening of a commercially available library of compounds has proved a successful strategy for the identification of a lead compound in a drug discovery programme. Here, we analysed 880 off-patent drugs, which initially comprised the Prestwick Chemical library, as sources of bacterial endotoxin neutralizers. We identified 3,3',5-triiodo-thyroacetic acid (tiratricol) as a non-antibacterial compound that neutralizes the toxic lipopolysaccharide.

LipopolysaccharidesendotoxinLipopolysaccharideCelllipopolysaccharide-antagonistsBiology:Enginyeria dels materials [Àrees temàtiques de la UPC]BiochemistryCell LineChemical libraryMicrobiologyLipid ASepsissepsisMiceStructure-Activity Relationshipchemistry.chemical_compoundtumour necrosis factor-alphaDrug DiscoveryEscherichia colimedicineAnimalsDrugs--Designlipid APharmacologyTriiodothyroacetic acidMedicaments -- DissenyTumor Necrosis Factor-alphaDrug discoveryOrganic Chemistrylipopolysaccharidetumour necrosis factor-amedicine.diseaseAnti-Bacterial AgentsEndotoxinsmedicine.anatomical_structurechemistryTriiodothyronineMolecular Medicineseptic shockLead compoundImmunity infection and tissue repair [NCMLS 1]
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Corynebacterium parvum (Propionibacterium acnes): an inducer of tumor necrosis factor-alpha in human peripheral blood mononuclear cells and monocytes…

1990

The present study investigates the potential capacity of the immunostimulant Corynebacterium parvum (C.p.) to induce tumor necrosis factor-alpha (TNF-alpha) in human peripheral blood mononuclear cells (PBMC) and blood monocytes (BMo) in vitro. Both at the mRNA and protein level, stimulation of PBMC and BMo upon C.p. induces TNF-alpha. Compared to the hitherto used TNF-alpha inducers in vitro such as Sendai virus, phytohemagglutinin or lipopolysaccharide the C.p. stimulus displayed a threefold stronger induction of TNF-alpha production (p less than 0.001). Using C.p. as an inducer it was possible to demonstrate that TNF-alpha production is regulated by prostaglandin E2; preincubation of the …

Lipopolysaccharidesmedicine.drug_classLymphocyteImmunologyEnzyme-Linked Immunosorbent AssayBiologyIn Vitro TechniquesPeripheral blood mononuclear cellImmunostimulantDinoprostoneMonocytesInterferon-gammamedicineImmunology and AllergyHumansInterferon gammaInducerPropionibacterium acnesProstaglandin E2Cells CulturedDose-Response Relationship DrugTumor Necrosis Factor-alphaMonocyteBlotting NorthernMolecular biologymedicine.anatomical_structureImmunologyLeukocytes MononuclearRNATumor necrosis factor alphaImmunizationDNA Probesmedicine.drugEuropean journal of immunology
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