Search results for "Pose"

showing 10 items of 1189 documents

In vivo triglyceride synthesis in subcutaneous adipose tissue of humans correlates with plasma HDL parameters

2016

Backgrounds and aims: Low concentrations of plasma HDL-C are associated with the development of atherosclerotic cardiovascular diseases and type 2 diabetes. Here we aimed to explore the relationship between the in vivo fractional synthesis of triglycerides (fTG) in subcutaneous (s.q.) abdominal adipose tissue (AT), HDL-C concentrations and HDL particle size composition in non-diabetic humans. Methods: The fTG in s.q. abdominal AT was measured in 16 non-diabetic volunteers (7 women, 9 men; Age: 49 ± 20 years; BMI: 31 ± 5 kg/m; Fasting Plasma Glucose: 90 ± 10 mg/dl) after 2H2O labeling. HDL-C concentration and subclasses, large (L-HDL), intermediate (I-HDL) and small (S-HDL) were measured. Re…

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyAdipocyte; Atheroprotective; Gender-related; HDL-C metabolism; Mass spectrometry; Stable isotope tracer kinetics; Triglycerides; Cardiology and Cardiovascular MedicineAdipose tissueWhite adipose tissueType 2 diabetes030204 cardiovascular system & hematologyTriglycerideArticleAtheroprotective03 medical and health scienceschemistry.chemical_compound0302 clinical medicineInsulin resistanceIn vivoInternal medicineAdipocytemedicineHumansObesityTriglyceridesAgedHDL-C metabolismAdipocyteStable isotope tracer kineticMass spectrometrybusiness.industryLipogenesisCholesterol HDLnutritional and metabolic diseasesMiddle AgedAtherosclerosismedicine.diseaseLipidsObesitySubcutaneous Fat AbdominalGender-related030104 developmental biologyEndocrinologychemistryLipogenesisFemalelipids (amino acids peptides and proteins)Insulin ResistanceCardiology and Cardiovascular MedicinebusinessAtherosclerosis
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Temporal and tissue-specific requirements for T-lymphocyte IL-6 signalling in obesity-associated inflammation and insulin resistance

2017

Low-grade inflammation links obesity to insulin resistance through the activation of tissue-infiltrating immune cells. Interleukin-6 (IL-6) is a crucial regulator of T cells and is increased in obesity. Here we report that classical IL-6 signalling in T cells promotes inflammation and insulin resistance during the first 8 weeks on a high-fat diet (HFD), but becomes dispensable at later stages (after 16 weeks). Mice with T cell-specific deficiency of IL-6 receptor-α (IL-6RαT-KO) exposed to a HFD display improved glucose tolerance, insulin sensitivity and inflammation in liver and EWAT after 8 weeks. However, after 16 weeks, insulin resistance in IL-6RαT-KO epididymal white adipose tissue (EW…

Blood GlucoseMale0301 basic medicinemedicine.medical_specialtyTime FactorsT-LymphocytesT cellScienceGeneral Physics and AstronomyInflammationWhite adipose tissueBiologyDiet High-FatArticleGeneral Biochemistry Genetics and Molecular BiologyMice03 medical and health sciences0302 clinical medicineInsulin resistanceImmune systemInternal medicinemedicineAnimalsHomeostasisObesityReceptorInflammationMice KnockoutMultidisciplinaryInterleukin-6QGeneral ChemistryT lymphocyteLipid Metabolismmedicine.diseaseReceptors Interleukin-6030104 developmental biologymedicine.anatomical_structureEndocrinology030220 oncology & carcinogenesisInsulin Resistancemedicine.symptomHomeostasisSignal TransductionNature Communications
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Chronic peroxisome proliferator-activated receptorβ/δ agonist GW0742 prevents hypertension, vascular inflammatory and oxidative status, and endotheli…

2015

Endothelial dysfunction plays a key role in obesity-induced risk of cardiovascular disease. The aim of the present study was to analyze the effect of chronic peroxisome proliferator-activated receptor (PPAR)β/δ agonist GW0742 treatment on endothelial function in obese mice fed a high-fat diet (HFD).Five-week-old male mice were allocated to one of the following groups: control, control-treated (GW0742, 3 mg/kg per day, by oral gavage), HFD, HFD + GW0742, HFD + GSK0660 (1 mg/kg/day, intraperitoneal) or HFD-GW0742-GSK0660 and followed for 11 or 13 weeks. GW0742 administration to mice fed HFD prevented the gain of body weight, heart and kidney hypertrophy, and fat accumulation. The increase in …

Blood GlucoseMaleAgonistmedicine.medical_specialtyNitric Oxide Synthase Type IIIEndotheliumPhysiologymedicine.drug_classCaveolin 1Peroxisome proliferator-activated receptorThiophenesDiet High-FatGW0742MiceInsulin resistanceInternal medicineInternal MedicinemedicineAnimalsObesityPPAR deltaSulfonesEndothelial dysfunctionReceptorPPAR-betaAortachemistry.chemical_classificationInterleukin-6Tumor Necrosis Factor-alphabusiness.industryGlucose Tolerance TestPeroxisomemedicine.diseaseToll-Like Receptor 4VasodilationThiazolesEndocrinologymedicine.anatomical_structureAdipose TissuechemistryHypertensionAdiponectinEndothelium VascularInsulin ResistanceReactive Oxygen SpeciesCardiology and Cardiovascular MedicinebusinessJournal of Hypertension
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Hypothalamic Apelin/Reactive Oxygen Species Signaling Controls Hepatic Glucose Metabolism in the Onset of Diabetes

2014

Aims: We have previously demonstrated that central apelin is implicated in the control of peripheral glycemia, and its action depends on nutritional (fast versus fed) and physiological (normal versus diabetic) states. An intracerebroventricular (icv) injection of a high dose of apelin, similar to that observed in obese/diabetic mice, increase fasted glycemia, suggesting (i) that apelin contributes to the establishment of a diabetic state, and (ii) the existence of a hypothalamic to liver axis. Using pharmacological, genetic, and nutritional approaches, we aim at unraveling this system of regulation by identifying the hypothalamic molecular actors that trigger the apelin effect on liver gluc…

Blood GlucoseMaleSympathetic nervous systemLIVER[SDV.BIO]Life Sciences [q-bio]/BiotechnologyGlycogenolysisPhysiology[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionClinical BiochemistryMice ObeseBiochemistrySYMPATHETIC-NERVE ACTIVITYAPELINBRAINGeneral Environmental ScienceINSULIN-RESISTANCE3. Good healthApelinOriginal Research CommunicationsADIPOSE-TISSUEmedicine.anatomical_structureIntercellular Signaling Peptides and ProteinsSignal TransductionEXPRESSIONmedicine.medical_specialtyGlycogenolysisHypothalamusBiologyCarbohydrate metabolismAutonomic Nervous SystemInsulin resistanceAdipokinesInternal medicineDiabetes mellitusmedicineAnimalsMolecular BiologyGluconeogenesis[ SDV.BIO ] Life Sciences [q-bio]/BiotechnologyCell Biologymedicine.diseaseMice Inbred C57BLMICEGlucoseEndocrinologyDiabetes Mellitus Type 2GluconeogenesisRATGeneral Earth and Planetary SciencesLiver functionReactive Oxygen Species[SDV.AEN]Life Sciences [q-bio]/Food and NutritionSYSTEMAntioxidants & Redox Signaling
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Time course of changes in serum oxidant/antioxidant status in overfed obese rats and their offspring.

2009

The aim of the present study was to determine the time course of changes in oxidant/antioxidant status, as well as serum glucose, insulin, leptin and lipid levels, liver adipose tissue and muscle lipid and protein contents, in cafeteria-diet-fed dams during gestation and lactation, and in their offspring throughout adulthood. Food intake was also evaluated. The cafeteria diet induced a significant increase in maternal body and relative adipose tissue weights, daily energy intake, and plasma glucose, insulin, leptin and lipid levels at parturition (day 0) and at the end of lactation (day 21). Plasma total antioxidant status [ORAC (oxygen radical absorbance capacity)], erythrocyte catalase an…

Blood GlucoseMalemedicine.medical_specialtyErythrocytesOffspringmedicine.medical_treatmentAdipokineAdipose tissueBiologyAntioxidantschemistry.chemical_compoundBlood serumPregnancyInternal medicinemedicineAnimalsInsulinObesityRats WistarPrenatal Nutritional Physiological PhenomenaTriglycerideInsulinLeptinBody WeightProteinsGeneral Medicinemedicine.diseaseOxidantsLipidsRatsPregnancy ComplicationsOxidative StressEndocrinologychemistryPrenatal Exposure Delayed EffectsFemaleMetabolic syndromeEnergy IntakeClinical science (London, England : 1979)
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Implication of corticotropic hormone axis in eating behaviour pattern in obese and type 2 diabetic participants.

2015

In Algeria, eating behaviour has been increasingly deviated from its traditional Mediterranean diet to modern fast food style. The present study examines the interactions between eating behaviour pattern (EBP), corticotropic hormone axis and the metabolic syndrome. Our Algerian population cohort comprised of 410 participants (130 obese, 170 type 2 diabetics and 110 healthy participants). The EBP was evaluated by the Three-Factor Eating Questionnaire test. The anthropometric and metabolic parameters (glucose, TAG, HDL, LDL and cholesterol) and the concentrations of hormones (insulin, adrenocorticotropin hormone (ACTH), cortisol and growth hormone) were determined by biometrics, spectrophotom…

Blood GlucoseMalemedicine.medical_specialtyHydrocortisonemedicine.medical_treatmentRadioimmunoassayMedicine (miscellaneous)Adipose tissueType 2 diabetesDiet MediterraneanCohort StudiesInsulin resistanceAdrenocorticotropic HormoneInternal medicineDiabetes mellitusSurveys and QuestionnairesmedicineHumansInsulinObesityGlycated HemoglobinMetabolic SyndromeNutrition and DieteticsAnthropometrybusiness.industryInsulinCholesterol HDLCholesterol LDLFeeding Behaviormedicine.diseaseObesityDietEndocrinologyDiabetes Mellitus Type 2AlgeriaGrowth HormoneMultivariate AnalysisFemaleMetabolic syndromebusinessHormoneThe British journal of nutrition
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Time course of changes in serum glucose, insulin, lipids and tissue lipase activities in macrosomic offspring of rats with streptozotocin-induced dia…

1999

The aim of this investigation was to determine the time course of changes in serum glucose, insulin and lipid levels, as well as lipid and protein content and lipolytic activities in insulin target organs (liver, adipose tissue and muscle), in macrosomic offspring of streptozotocin-induced mildly hyperglycaemic rats. Food intake and nutritional efficiency were also evaluated. Mild hyperglycaemia in pregnant rats was induced by intraperitoneal injection of streptozotocin (40 mg/kg body weight) on day 5 of gestation. Control pregnant rats were injected with citrate buffer. At birth, macrosomic pups (birth weight > 1.7 S.D. greater than the mean value for the control pups) had higher serum …

Blood GlucoseMalemedicine.medical_specialtyTime Factorsmedicine.medical_treatmentLipolysisAdipose tissueBiologyDiabetes Mellitus ExperimentalFetal Macrosomiachemistry.chemical_compoundEatingInsulin resistanceSex FactorsDiabetes mellitusAdipocyteInternal medicinemedicineFetal macrosomiaAnimalsInsulinObesityRats WistarMuscle SkeletalInsulinLipid metabolismGeneral MedicineLipasemedicine.diseaseStreptozotocinLipidsRatsEndocrinologychemistryAdipose TissueLiverFemalemedicine.drugClinical science (London, England : 1979)
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Eicosapentaenoic acid modulates fatty acid metabolism and inflammation in Psammomys obesus.

2015

Abstract The desert gerbil, Psammomys obesus, is a unique polygenic animal model of metabolic syndrome (insulin resistance, obesity and type 2 diabetes), and these pathological conditions resemble to those in human beings. In this study, the animals were fed ad libitum either a natural diet (ND) which contained desertic halophile plants or a standard laboratory diet (STD) or a diet which contained eicosapentaenoic acid (EPA), hence, termed as EPA diet (EPAD). In EPAD, 50% of total lipid content was replaced by EPA oil. By employing real-time PCR, we assessed liver expression of key genes involved in fatty acid metabolism such as PPAR-α, SREBP-1c, LXR-α and CHREBP. We also studied the expres…

Blood GlucoseMalemedicine.medical_specialtymedicine.medical_treatmentInterleukin-1betaAdipose tissueGene ExpressionBiologyBiochemistrychemistry.chemical_compoundInsulin resistanceInternal medicinemedicineAnimalsInsulinPPAR alphaRNA MessengerTriglyceridesLiver X Receptorschemistry.chemical_classificationFatty acid metabolismReverse Transcriptase Polymerase Chain ReactionTumor Necrosis Factor-alphaInsulinBody WeightFatty AcidsGeneral Medicinebiology.organism_classificationmedicine.diseaseLipid MetabolismOrphan Nuclear ReceptorsEicosapentaenoic acidEndocrinologychemistryAdipose TissueEicosapentaenoic AcidLiverlipids (amino acids peptides and proteins)PsammomysMetabolic syndromeInflammation MediatorsGerbillinaeSterol Regulatory Element Binding Protein 1Polyunsaturated fatty acidBiochimie
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Effects of Two Months of Very Low Carbohydrate Ketogenic Diet on Body Composition, Muscle Strength, Muscle Area, and Blood Parameters in Competitive …

2021

Background: Ketogenic diet (KD) is a nutritional approach that restricts daily carbohydrates, replacing most of the reduced energy with fat, while maintaining an adequate quantity of protein. Despite the widespread use of KD in weight loss in athletes, there are still many concerns about its use in sports requiring muscle mass accrual. Thus, the present study sought to investigate the influence of a KD in competitive natural body builders. Methods: Nineteen volunteers (27.4 &plusmn

Blood GlucoseMalemedicine.medical_treatmentDiet Carbohydrate-Restricted0302 clinical medicineNeurotrophic factorsWeight lossInsulinTestosteroneHealth Ketogenic diet Resistance training StrengthInsulin-Like Growth Factor ITestosteroneNutrition and DieteticsMuscleshealthAdipose Tissueketogenic dietBody CompositionCytokinesmedicine.symptomDiet Ketogenicstrengthlcsh:Nutrition. Foods and food supplySettore M-EDF/01 - Metodi E Didattiche Delle Attivita' MotorieAdultmedicine.medical_specialtyAdolescentCarbohydrates030209 endocrinology & metabolismlcsh:TX341-641ArticleProinflammatory cytokineHealth; Ketogenic diet; Resistance training; Strength03 medical and health sciencesYoung AdultInternal medicineWeight LossmedicineHumansMuscle StrengthTriglyceridesbusiness.industryInsulinBrain-Derived Neurotrophic Factor030229 sport sciencesEndocrinologyGlucoseAthletesDiet WesternBasal metabolic rateLean body massBasal Metabolismresistance trainingbusinessFood ScienceKetogenic dietNutrients
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Altered adipose tissue metabolism in offspring of dietary obese rat dams.

2011

To investigate further the mechanisms of developmental programming, we analysed the effects of maternal overnutrition and of postnatal high-fat feeding on adipose tissue metabolism in the offspring. Postnatal changes in serum adiponectin, leptin and TAG [triacylglycerol (triglyceride)] levels, adipose tissue TAGs, fatty acids and enzyme activities were determined in offspring of cafeteria-diet-fed dams during gestation and lactation, weaned on to standard chow or on to cafeteria diet. Obese rats showed higher adiposity (+35% to 85%) as well as a significant increase in serum glucose, insulin, leptin, adiponectin and TAG levels (P<0.01) and adipose tissue LPL (lipoprotein lipase) and …

Blood Glucosemedicine.medical_specialtyOffspringAdipokineAdipose tissueBiologychemistry.chemical_compoundOvernutritionPregnancyInternal medicinemedicineAdipocytesAnimalsObesityRats WistarPrenatal Nutritional Physiological PhenomenaTriglycerideschemistry.chemical_classificationTriglycerideAdiponectinLeptinBody WeightFatty AcidsGeneral MedicineFeeding BehaviorOrgan Sizemedicine.diseaseHormonesRatsPregnancy ComplicationsEndocrinologychemistryAdipose TissuePrenatal Exposure Delayed EffectsFemalePolyunsaturated fatty acidClinical science (London, England : 1979)
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