Search results for "Postsynaptic potential"

showing 10 items of 371 documents

Effects of Substantia Nigra Stimulation on Electrical and Mechanical Activities of the Duodeno-Jejunal Loop, in the Cat

1985

The effect of substantia nigra stimulation on duodeno-jejunal intraluminal pressure and electrical activity was analysed in the cat. An inhibitory influence was noted to occur on both the intestinal pressure and the electrical response activity (ERA): the effect seemed to be in correlation with the stimulus parameters employed. After bilateral vagotomy, the inhibition remained substantially unchanged. The possibility of the existence of a sympathetic pathway mediating the effect is emphasized.

Malemedicine.medical_specialtyDuodenumPhysiologymedicine.medical_treatmentStimulationSubstantia nigraInhibitory postsynaptic potentialBiochemistryFunctional LateralityMidbrainIleumInternal medicinemedicineCarnivoraAnimalsbiologyFissipediaElectric Conductivitybiology.organism_classificationVagotomyElectric StimulationSubstantia NigraEndocrinologyCatsFemalemedicine.symptomMuscle contractionArchives Internationales de Physiologie et de Biochimie
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Pre- and postjunctional effects of diadenosine polyphosphates in the guinea-pig vas deferens.

1995

Abstract The pre- and postjunctional activities of a number of diadenosine polyphosphates were examined in the guinea-pig isolated vas deferens at the level of the membrane-potential, using a modified sucrose-gap technique. P1,P3-Di(adenosine 5′)triphosphate (Ap3A), P1,P4-di(adenosine 5′)tetraphosphate (Ap4A) and P1,P5-di(adenosine 5′)pentaphosphate (Ap5 A) all caused concentration-dependent depolarization of the smooth muscle membrane. The potency order was: Ap5A > Ap4A. Ap3A. P1, P2-Di(adenosine 5′)pyrophosphate (Ap2A) did not evoke depolarization even at the highest concentration tested (1 mM). All the dinucleotides caused a reduction in the amplitude of evoked excitatory junction…

Malemedicine.medical_specialtyGuinea PigsPharmaceutical ScienceIn Vitro TechniquesMembrane PotentialsVas DeferensAdenine nucleotideInternal medicinemedicineAnimalsPharmacologyDose-Response Relationship DrugChemistryPurinergic receptorAntagonistVas deferensReceptors PurinergicDepolarizationAdenosine receptorAdenosinemedicine.anatomical_structureEndocrinologyExcitatory postsynaptic potentialDinucleoside Phosphatesmedicine.drugThe Journal of pharmacy and pharmacology
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Nitric oxide and glutamate interaction in the control of cortical and hippocampal excitability.

1999

Summary: Purpose: We investigated the role of nitric oxide (NO) as a new neurotransmitter in the control of excitability of the hippocampus and the cerebral cortex, as well as the possible functional interaction between NO and the glutamate systems. Methods: The experiments were performed on anesthetized rats. The bioelectrical activities of the somatosensory cortex and the CA1 region of the hippocampus of these rats were recorded. Pharmacologic inhibition of NO synthase (NOS) through the nonselective and brain-selective inhibitors, N-nitro-L-arginine methyl ester (l-NAME) and 7-nitroindazole (7-NI), was performed. Results: The treatments caused the appearance of an interictal discharge act…

Malemedicine.medical_specialtyHippocampusGlutamic AcidHippocampal formationNeurotransmissionBiologyNitric OxideHippocampusReceptors N-Methyl-D-Aspartatechemistry.chemical_compoundInternal medicinemedicineAnimalsRats WistarNeurotransmitterCerebral CortexEpilepsyGlutamate receptorSomatosensory CortexRatsEndocrinologyNeurologychemistryCNQXExcitatory postsynaptic potentialNMDA receptorNeurology (clinical)Epilepsia
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Inhibition of nitric oxide synthase influences the activity of striatal neurons in the rat

2002

The activity of single units in the striatum of urethane-anesthetized rats was recorded before and after the systemic administration of 7-nitro-indazole (7-NI; 50 mg/kg intraperitoneally), a selective inhibitor of neuronal nitric oxide (NO) synthase. Two neuronal types were clearly distinguishable electrophysiologically, on the basis of either discharge frequency pattern or features of the individual spike waveform (spike duration, negative phase/total duration ratio, and negative phase/total amplitude ratio). Only sporadically discharging neurons (basal firing rate, <0.1 spikes/s) were influenced by 7-NI, which caused a statistically significant increase in their firing rate. In contrast, …

Malemedicine.medical_specialtyIndazolesAction PotentialsNerve Tissue ProteinsNitric Oxide Synthase Type IStriatumNeurotransmissionBiologyNitric OxideInhibitory postsynaptic potentialTonic (physiology)Nitric oxidechemistry.chemical_compoundInternal medicineBasal gangliamedicineAnimalsEnzyme InhibitorsRats WistarGeneral NeuroscienceCorpus StriatumRatsElectrophysiologyNitric oxide synthaseElectrophysiologyEndocrinologynervous systemchemistrybiology.proteinNitric Oxide SynthaseNeuroscienceNeuroscience Letters
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Streptozotocin diabetic mice display depressive-like behavior and alterations in the structure, neurotransmission and plasticity of medial prefrontal…

2015

Diabetes mellitus patients are at increased risk of developing depression, although the neurobiological bases of this comorbidity are not yet fully understood. These patients show CNS alterations, similar to those found in major depression, including changes in the structure and neurotransmission of excitatory neurons. However, although depressive patients and animal models also display alterations in inhibitory networks, little is known about the effects of diabetes on interneurons. Our main objective was to study the impact of diabetes on interneurons of the medial prefrontal cortex (mPFC), one of the regions most affected by major depression. For this purpose we have induced diabetes wit…

Malemedicine.medical_specialtyInterneuronGlutamate decarboxylaseGreen Fluorescent ProteinsSynaptophysinPrefrontal CortexMice TransgenicNeural Cell Adhesion Molecule L1NeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionDiabetes Mellitus ExperimentalInterneuronsInternal medicinemedicineAnimalsPrefrontal cortexDepressive DisorderNeuronal PlasticitybiologyGlutamate Decarboxylasemusculoskeletal neural and ocular physiologyGeneral NeuroscienceDendritesTail suspension testEndocrinologymedicine.anatomical_structurenervous systemExcitatory postsynaptic potentialSynaptophysinbiology.proteinSialic AcidsPsychologyNeuroscienceBrain research bulletin
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Presynaptic nicotine receptors mediating a positive feed-back on transmitter release from the rat phrenic nerve.

1986

The effects of 1,1-dimethyl-4-phenylpiperazinium (DMPP) and of nicotine receptor antagonists on [3H]acetylcholine release from the rat phrenic nerve preincubated with [3H]choline were investigated in the absence and presence of cholinesterase inhibitors (presynaptic effects). Additionally, the effects of hexamethonium and tubocurarine on the muscle contraction of the indirectly stimulated diaphragm were examined (postsynaptic effects). DMPP (1-30 microM) increased (76-92%), whereas hexamethonium (0.001-1 mM) and tubocurarine (1-10 microM) decreased (52-60%) the release of [3H]acetylcholine following a train of 100 pulses at 5 Hz. The release caused by a longer train (750 pulses at 5 Hz) was…

Malemedicine.medical_specialtyMotor nerveTubocurarineHexamethonium CompoundsIn Vitro TechniquesReceptors NicotinicNeuromuscular junctionFeedbackchemistry.chemical_compoundPostsynaptic potentialInternal medicinemedicineAnimalsCholinesterasePhrenic nervePharmacologyNeurotransmitter AgentsbiologyRats Inbred StrainsGeneral Medicinemusculoskeletal systemElectric StimulationRatsPhrenic NerveEndocrinologymedicine.anatomical_structurechemistrybiology.proteinHexamethoniummedicine.symptomDimethylphenylpiperazinium IodideAcetylcholineMuscle contractionmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Effects of excitatory amino acids and neuropeptide Y on the discharge activity of suprachiasmatic neurons in rat brain slices

1997

Effects of L-glutamate, AMPA, NMDA and NPY on the discharge activity of neurons located in the ventral subdivision of the suprachiasmatic nucleus were examined in submerged coronal slices of the rat hypothalamus. All substances were bath applied. Application of L-glutamate (14 neurons examined) induced an excitatory response in 8 suprachiasmatic neurons (+248.9 +/- 122.24%, mean +/- S.E.M.; P0.001). A biphasic response, i.e. an initial transient excitation (+54.3 +/- 8.21%; P0.001) succeeded by an inhibition (-66.2 +/- 9.31%; P0.001), was observed in 6 neurons. Application of AMPA (36 neurons examined) resulted in an excitation of 31 neurons (+209.2 +/- 58.58%; P0.0001). Application of NMDA…

Malemedicine.medical_specialtyN-MethylaspartateGlutamic AcidAMPA receptorInhibitory postsynaptic potentialRats Sprague-Dawleychemistry.chemical_compoundOrgan Culture TechniquesInternal medicineExcitatory Amino Acid AgonistsmedicineAnimalsNeuropeptide Yalpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic AcidMolecular BiologyGlycine receptorNeuronsDose-Response Relationship DrugChemistrySuprachiasmatic nucleusGeneral NeuroscienceGlycine AgentsStrychnineStrychninehumanitiesCircadian RhythmRatsElectrophysiologyEndocrinologynervous systemHypothalamusExcitatory postsynaptic potentialNMDA receptorSuprachiasmatic NucleusNeurology (clinical)NeuroscienceDevelopmental BiologyBrain Research
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Motor intracortical inhibition in PD: L-DOPA modulation of high-frequency rTMS effects

2007

Dopaminergic drugs and deep brain stimulation restore cortical inhibition in Parkinson disease (PD) patients. High-frequency rTMS was also found to increase cortical inhibition in PD but its therapeutic effect is still controversial. Here we hypothesize that, if dopaminergic drugs reverse to normal cortical excitability in M1, the effect of high-frequency (hf)-rTMS in PD patients could depend on whether they are in a medicated or unmedicated state. The present study aims to explore the lasting effects of sub-threshold hf rTMS trains over M1 on cortical inhibition in patients with "on" and without "off" L-DOPA treatment. Fourteen PD patients were examined twice while "on" and "off" medicatio…

Malemedicine.medical_specialtyNeurologyDeep brain stimulationCORTICAL SILENT PERIODmedicine.medical_treatmentStimulationMotor ActivityInhibitory postsynaptic potentialPAIRED-PULSE INHIBITIONAntiparkinson AgentsLevodopaDegenerative diseaseEVOKED-POTENTIALSmedicineHumansCORTEX EXCITABILITYAgedAged 80 and overSUBTHALAMIC NUCLEUSPARKINSONS-DISEASE PATIENTSGeneral NeuroscienceDopaminergicMotor CortexNeural InhibitionParkinson DiseaseMiddle Agedmedicine.diseaseFACILITATIONCombined Modality TherapyTranscranial Magnetic StimulationTranscranial magnetic stimulationBRAIN-STIMULATIONmedicine.anatomical_structureSettore MED/26 - NeurologiaFemaleREPETITIVE TMSPsychologyNeuroscienceMotor cortexExperimental Brain Research
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Inhibition of mechanical activity by neurotensin in rat proximal colon: involvement of nitric oxide.

1997

The aim of the present study was to define the nature of inhibitory action of neurotensin in rat proximal colon. Mechanical activity was detected as changes of intraluminal pressure. Neurotensin (10(-10) to 10(-7) M), in the presence of atropine (10(-6) M), guanethidine (10(-6) M), and nifedipine (10(-8) M), induced a tetrodotoxin-insensitive inhibitory effect characterized by the complete disappearance of the spontaneous phasic contractions. The inhibitory effect of neurotensin (10(-7) M) was abolished by scorpion venom (Leiurus quinquestriatus hebraeus) (10(-6) g/ml) or high K+ (40 mM KCl), whereas it persisted in the presence of omega-conotoxin GVIA, (10(-7) M). N omega-nitro-L-arginine…

Malemedicine.medical_specialtyPhysiologyColonNeuropeptideScorpion VenomsTetrodotoxinIn Vitro TechniquesInhibitory postsynaptic potentialNitric Oxidecomplex mixturesNitric oxidechemistry.chemical_compoundomega-Conotoxin GVIAPhysiology (medical)Internal medicinemedicineAnimalsOmega-Conotoxin GVIAEnzyme InhibitorsRats WistarGuanethidineNeurotensinHepatologybiologyGastroenterologyRatsNitric oxide synthaseEndocrinologyNG-Nitroarginine Methyl EsterMechanism of actionchemistrybiology.proteinPotassiumFemalemedicine.symptomGastrointestinal MotilityPeptidesmedicine.drugNeurotensinThe American journal of physiology
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Impaired neuromuscular transmission during partial inhibition of acetycholinest-erase: The of stimulus-induced antiromic backfiring in the generation…

1992

Neuromuscular transmission was studied in the rat phrenic nerve-hemidiaphragm preparation with acetylcholinesterase (AChE) partially inactivated. Enzyme inhibition resulted in (1) increased single-twitch tension of the diaphragm; (2) compound muscle action potential (CMAP) containing repetitive discharges; (3) stimulus-induced antidromic backfiring (SIAB) seen in the phrenic nerve; and (4) repetitive nerve stimulation (RNS) eliciting a decrement-increment (D-I) phenomenon (i.e., amplitude reduction maximal with the second CMAP). Using a high-calcium and low-magnesium solution, SIAB and the decrement of the second CMAP during RNS were intensified, whereas closely spaced trains and (+)-tubocu…

Malemedicine.medical_specialtyPhysiologyDiaphragmNeuromuscular JunctionNeuromuscular transmissionAction PotentialsReceptors NicotinicSynaptic TransmissionRats Sprague-DawleyCellular and Molecular NeurosciencePostsynaptic potentialPhysiology (medical)Internal medicinemedicineAnimalsRepetitive nerve stimulationEvoked PotentialsPhrenic nerveChemistrymusculoskeletal systemElectric StimulationNeostigmineRatsAntidromicCompound muscle action potentialPhrenic NerveEndocrinologymedicine.anatomical_structurePeripheral nervous systemAcetylcholinesteraseNeurology (clinical)NeuroscienceAcetylcholinemedicine.drugMuscle &amp; Nerve
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