Search results for "Predisposition"

showing 10 items of 771 documents

TET2 gene mutation is a frequent and adverse event in chronic myelomonocytic leukemia

2009

Background Acquired somatic deletions and loss-of-function mutations in one or several codons of the TET2 ( Ten-Eleven Translocation-2 ) gene were recently identified in hematopoietic cells from patients with myeloid malignancies, including myeloproliferative disorders and myelodys-plastic syndromes. The present study was designed to determine the prevalence of TET2 gene alterations in chronic myelomonocytic leukemias. Design and Methods Blood and bone marrow cells were collected from 88 patients with chronic phase chronic myelomonocytic leukemia and from 14 with acute transformation of a previously identified disease. Polymerase chain reaction analysis and direct sequencing were used to se…

Malemedicine.medical_specialtyMyeloidDNA Mutational AnalysisChronic myelomonocytic leukemiaSingle-nucleotide polymorphismKaplan-Meier EstimateGene mutationBiologymedicine.disease_causeDioxygenasesGene FrequencyMonocytosisInternal medicinehemic and lymphatic diseasesProto-Oncogene ProteinsmedicineHumansGenetic Predisposition to DiseaseLetters to the EditorAgedProportional Hazards ModelsAged 80 and overComparative Genomic HybridizationMutationHematologyLeukemia Myelomonocytic ChronicHematologyMiddle Agedmedicine.diseaseMyelodysplastic-Myeloproliferative DiseasesDNA-Binding ProteinsLeukemiamedicine.anatomical_structureImmunologyMutationFemaleOriginal Article
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Association between AT C573T polymorphism and cardiovascular risk factors in myocardial infarction.

2011

Abstract Background Polymorphisms in the AT1 gene have been associated with various parameters related to the pathogenesis of cardiovascular diseases and to myocardial infarction. This study analyzed the relationship between two polymorphisms of the angiotensin II AT-1 receptor gene (AT1_1166 and AT1_573) and the risk of ischemic heart disease by studying their association with several cardiovascular risk factors. Methods The sample population comprised 356 subjects: 174 patients who had survived myocardial infarction (61.01±8.15 years), and 182 age- and gender-matched controls (mean age of 60.25±9.43). The polymorphisms of the angiotensin II AT1-receptor gene (C573T and A1166C) were studie…

Malemedicine.medical_specialtyMyocardial InfarctionDiseaseGastroenterologyPolymorphism Single NucleotideRisk AssessmentReceptor Angiotensin Type 1Pathology and Forensic Medicinelaw.inventionPathogenesisGene FrequencylawRisk FactorsInternal medicineGenotypemedicineOdds RatioHumansGenetic Predisposition to DiseaseMyocardial infarctionPolymerase chain reactionAgedRetrospective StudiesAnalysis of VarianceAngiotensin II receptor type 1Chi-Square Distributionbusiness.industryGeneral MedicineAnthropometryMiddle Agedmedicine.diseaseAngiotensin IILogistic ModelsPhenotypeHaplotypesSpainCase-Control StudiesCardiologyFemaleCardiology and Cardiovascular MedicinebusinessCardiovascular pathology : the official journal of the Society for Cardiovascular Pathology
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The parkin gene is not a major susceptibility locus for typical late-onset Parkinson's disease

2001

We investigated the parkin gene in 118 patients with typical Parkinson's disease (PD), i. e. in patients who had an onset of PD after the age of 45 years. The study group included 95 subjects with sporadic PD and 23 subjects from 18 families with autosomal recessive PD. No pathogenetic mutations in the parkin gene were detected either in familial or in sporadic patients. Our findings indicate that the parkin gene is not involved in the pathogenesis of classic late-onset PD.

Malemedicine.medical_specialtyNeurologyParkinson's diseaseUbiquitin-Protein LigasesDNA Mutational AnalysisMolecular Sequence DataLate onsetGenes RecessiveDermatologyDiseaseParkinPathogenesisLigasesParkinsonian DisordersmedicineHumansPoint MutationGenetic Predisposition to DiseaseGenetic TestingAge of OnsetAgedGeneticsbusiness.industryGeneral MedicineExonsParkin geneMiddle Agedmedicine.diseasenervous system diseasesPsychiatry and Mental healthSusceptibility locusChromosomes Human Pair 6FemaleNeurology (clinical)business
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Involvement of -308 TNF-alpha and 1267 Hsp70-2 polymorphisms and zinc status in the susceptibility of coronary artery disease (CAD) in old patients.

2006

Coronary artery disease (CAD) is characterized by an inflammatory status and it represents the major cause of death in elderly. Zinc deficiency and inflammatory genes within major histocompatibility complex (MHC) region are implicated in ischaemic heart diseases. TNF-alpha is present in coronary artery plaques and may provoke plaque instability. Hsp70 plays instead a pro-atherogenic role, via proinflammatory cytokine production, in atherosclerotic lesions contributing to plaque rupture. Contradictory data report the association between -308 TNF-alpha polymorphism and CAD, while no investigations exist on Hsp70-2 gene in CAD. In the current study, we analysed -308 TNF-alpha and 1267 Hsp70-2 …

Malemedicine.medical_specialtyPathologyAgingCoronary Artery DiseaseMajor histocompatibility complexGastroenterologyRisk AssessmentProinflammatory cytokineCoronary artery diseaseGene FrequencyRisk FactorsInternal medicineGenotypemedicineOdds RatioHumansCarotid StenosisGenetic Predisposition to DiseaseHSP70 Heat-Shock ProteinsAlleleCause of deathAgedPolymorphism Geneticbiologybusiness.industryInterleukin-6Tumor Necrosis Factor-alphaMiddle Agedmedicine.diseaseZincmedicine.anatomical_structureHaplotypesItalyCase-Control Studiesbiology.proteinTumor necrosis factor alphaFemaleGeriatrics and GerontologybusinessGerontologyBiomarkersArteryBiogerontology
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Boy with pseudohypoparathyroidism type 1a caused byGNASgene mutation (deltaN377), Crouzon-like craniosynostosis, and severe trauma-induced bleeding

2009

We report on a 6-month-old boy with craniosynostosis, pseudohypoparathyroidism type 1a (PHP1A), and a GNAS gene mutation. He had synostoses of the coronal, frontal, and sagittal sutures, brachyturricephaly, and hydrocephaly. He also had congenital hypothyroidism, round face, full cheeks, shortness of limbs, mild developmental delay, and muscular hypotonia. Because of progressive hydrocephaly, the synostosis was corrected surgically but circulatory decompensation led to disseminated intravascular coagulation and cerebral infarctions. Our patient died 8 days later. Postmortem molecular studies of GNAS, the gene for guanine nucleotide-binding protein, alpha-stimulating activity polypeptide (ge…

Malemedicine.medical_specialtyPathologyCraniosynostosisFatal OutcomeInternal medicineChromograninsCongenital HypothyroidismGTP-Binding Protein alpha Subunits GsGeneticsmedicineGNAS complex locusHumansGenetic Predisposition to DiseaseGenetics (clinical)PseudohypoparathyroidismDisseminated intravascular coagulationbiologyMuscular hypotoniabusiness.industryCraniofacial DysostosisInfantDysostosisSynostosismedicine.diseaseCongenital hypothyroidismEndocrinologyBrain InjuriesPseudohypoparathyroidismMutationbiology.proteinbusinessIntracranial HemorrhagesHydrocephalusAmerican Journal of Medical Genetics Part A
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Genetic, clinical and radiographic signs in knee osteoarthritis susceptibility

2014

Abstract INTRODUCTION: Osteoarthritis (OA) is considered to be a multifactorial and polygenic disease and diagnosis is mainly clinical and radiological. Correlation between radiographic data and clinical status has been reported. However, very few studies, especially in Caucasian people, describe the association between the Kellgren and Lawrence OA grading scale (KL) and genetic alterations to better understand OA etiopathogenesis and susceptibility. In order to update the knee OA grading, in this study we assessed the associations between KL grade, clinical features such as American Knee Society Score (AKSS), age, and polymorphisms in the principal osteoarthritis susceptibility (OS) genes …

Malemedicine.medical_specialtyPathologydbSNPGenotypeSingle Nucleotide PolymorphismsImmunologySingle-nucleotide polymorphismOsteoarthritisPolymorphism Single NucleotideRadiographicRheumatologyInternal medicineSettore MED/33 - Malattie Apparato LocomotoreOMIM : Online Mendelian Inheritance in ManHumansImmunology and AllergyMedicineGenetic Predisposition to DiseaseGrading (tumors)AgedAged 80 and overReverse Transcriptase Polymerase Chain Reactionbusiness.industryMiddle AgedOsteoarthritis Kneemedicine.diseaseRheumatologyRadiographyOrthopedic surgeryCohortFemaleOsteoarthritibusinessResearch ArticleArthritis Research & Therapy
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West syndrome followed by juvenile myoclonic epilepsy: a coincidental occurrence?

2013

Background: West syndrome is an age-dependent epilepsy with onset peak in the first year of life whose aetiology may be symptomatic or cryptogenic. Long-term cognitive and neurological prognosis is usually poor and seizure outcome is also variable. Over the past two decades a few patients with favourable cognitive outcome and with total recovery from seizures were identified among the cryptogenic group suggesting an idiopathic aetiology. Recent research has described two children with idiopathic WS who later developed a childhood absence epilepsy. Case presentation: We reviewed the medical records of patients with West syndrome admitted to the our Child Neuropsychiatry Unit in the last 15 y…

Malemedicine.medical_specialtyPediatricsNeurologyLevetiracetamAdolescentHairy elbows syndromeMyoclonic JerkClinical NeurologyCase ReportEpilepsyChildhood absence epilepsyJuvenile myoclonic epilepsySettore M-PSI/08 - Psicologia ClinicamedicineHumansEpilepsy evolutionPsychiatrySettore M-PSI/02 - Psicobiologia E Psicologia Fisiologicabusiness.industryGenetic predispositionMyoclonic Epilepsy JuvenileBrainInfantWest SyndromeGeneral MedicineWest syndromemedicine.diseaseMagnetic Resonance ImagingPiracetamSettore MED/39 - Neuropsichiatria InfantileWest syndrome Juvenile myoclonic epilepsy Epilepsy evolution Genetic predisposition Hairy elbows syndromeDisease ProgressionMyoclonic epilepsyNeurology (clinical)LevetiracetamJuvenile myoclonic epilepsybusinessSpasms Infantilemedicine.drugBMC neurology
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Association of a Mineralocorticoid Receptor Gene Polymorphism With Hypertension in a Spanish Population

2009

BACKGROUND To assess the association of polymorphisms and haplotypes of the mineralocorticoid receptor (MR) (NR3C2) gene to the risk of essential hypertension (HTN) in a Spanish population. METHODS This is a population-based study which included 1,502 subjects (748 women) >18 years old. Twenty-four polymorphisms of NR3C2 gene were analyzed by using SNPlex (Genotyping System based on OLA/PCR technology). RESULTS Alleles of the single-nucleotide polymorphism (SNP) rs5522 were significantly associated with the risk of HTN, both in the recessive and codominant models adjusted by age, gender, and body mass index (BMI). Genotype GG of the rs5522 showed to be protective against HTN odds ratio (OR)…

Malemedicine.medical_specialtyPopulationSingle-nucleotide polymorphismEssential hypertensionPolymerase Chain ReactionPolymorphism (computer science)Internal medicineGenotypePrevalenceInternal MedicineHumansMedicineGenetic Predisposition to Diseasecardiovascular diseaseseducationAllelesAgededucation.field_of_studyPolymorphism Geneticbusiness.industryHaplotypeDNAOdds ratioMiddle Agedmedicine.diseaseReceptors MineralocorticoidEndocrinologyHaplotypesSpainHypertensionFemaleGene polymorphismbusinessAmerican Journal of Hypertension
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Association of cathepsin B gene polymorphisms with tropical calcific pancreatitis

2006

Background and aims: Tropical calcific pancreatitis (TCP) is a type of chronic pancreatitis unique to countries in the tropics. Mutations in pancreatic secretory trypsin inhibitor (SPINK1) rather than cationic trypsinogen (PRSS1) explain the disease in only 50% of TCP patients. As cathepsin B (CTSB) is known to activate cationic trypsinogen, we attempted to understand the role of CTSB mutations in TCP. Evidence of epistatic interaction was investigated with the previously associated N34S SPINK1 allele, a variant considered to be a modifier rather than a true susceptibility allele. Subjects and methods: We sequenced the coding region of CTSB gene in 51 TCP patients and 25 controls and furthe…

Malemedicine.medical_specialtyTrypsinogenSingle-nucleotide polymorphismBiologyPolymerase Chain ReactionGastroenterologyCathepsin BCathepsin Bchemistry.chemical_compoundGene FrequencyPancreatitis ChronicInternal medicinemedicineHumansGenetic Predisposition to DiseaseTrypsinogen activationAlleleAllele frequencyGeneticsPolymorphism GeneticHaplotypeGastroenterologyCase-control studyCalcinosisHaplotypesPancreatitischemistryCase-Control StudiesMutationAcute DiseaseFemaleGut
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The association between the 4G/5G polymorphism in the promoter of the plasminogen activator inhibitor-1 gene and extension of postsurgical calf vein …

2013

The objective of this study was to evaluate whether the presence of a plasminogen activator inhibitor type 1 (PAI-1) promoter polymorphism 4G/5G could significantly influence the proximal extension of vein thrombosis in spite of anticoagulant treatment in patients with calf vein thrombosis (CVT) following orthopaedic, urological and abdominal surgery. We studied 168 patients with CVT, who had undergone orthopaedic, urological and abdominal surgery, subdivided as follows: first, 50 patients with thrombosis progression; second, 118 patients without thrombosis progression. The 4G/5G polymorphism of the plasminogen activator inhibitor 1 was evaluated in all patients and in 70 healthy matched co…

Malemedicine.medical_specialtymedicine.medical_treatmentGastroenterologychemistry.chemical_compoundPostoperative ComplicationsGene FrequencyRisk FactorsInternal medicineFibrinolysisPlasminogen Activator Inhibitor 1Factor V LeidenmedicineOdds RatioHumansGenetic Predisposition to Disease4G/5G genotype PAI-1 thrombotic lesionsPromoter Regions GeneticAllelesAgedVenous ThrombosisPolymorphism Geneticbusiness.industryHematologyGeneral MedicineOdds ratioMiddle Agedmedicine.diseaseNadroparin calciumThrombosisSurgerychemistryPlasminogen activator inhibitor-1Case-Control StudiesFemalebusinessPlasminogen activatorAbdominal surgerymedicine.drugBlood coagulationfibrinolysis : an international journal in haemostasis and thrombosis
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