Search results for "Proband"

showing 10 items of 99 documents

Syndrome profiles in alcoholism and panic disorder with or without agoraphobia

2002

It is proposed that alcoholism and panic disorder/agoraphobia demonstrate in part common genetic and environmental origins. Shared subthreshold symptom patterns in the parents' generation could confirm the proposed genetic role in alcoholism and panic disorder/agoraphobia, even if the parents do not fulfil the diagnostic criteria for a primary psychiatric diagnosis. This is the first family study of exploratively analyzing subthreshold symptoms in both disorders. The authors investigated families with panic disorder/agoraphobia and/or alcoholism with the Munich-Composite International Diagnostic Interview (M-CIDI). We documented the diagnoses according to Diagnostic and Statistical Manual o…

PharmacologyProbandmedicine.medical_specialtyAddictionmedia_common.quotation_subjectPanic disorderPanicICD-10medicine.diseasebehavioral disciplines and activitieshumanitiesmental disordersmedicineAnxietymedicine.symptomPsychologyPsychiatryBiological PsychiatryAnxiety disordermedia_commonClinical psychologyAgoraphobiaProgress in Neuro-Psychopharmacology and Biological Psychiatry
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Reaction time paradigms in subjects at risk for schizophrenia.

1994

Abstract Deviant response patterns in experimental reaction time paradigms in schizophrenic probands are well documented. Although simple reaction times are strongly influenced by the current psychopathological status of the proband (e.g. florid psychotic patients versus remitted patients) these influences are less clear for measures obtained from more complex reaction time paradigms. These include the crossover paradigm (reaction time to stimuli presented after constant preparatory intervals in comparison to reaction time to stimuli presented after irregular preparatory intervals) and the modality shift paradigm (reaction time to a stimulus (light or tone) when the modality of the stimulus…

ProbandAdultGenetic MarkersMalePsychosismedicine.medical_specialtyAdolescentCrossoverStimulus (physiology)AudiologyDevelopmental psychologySchizotypal Personality DisorderStimulus modalityRisk FactorsmedicineReaction TimeHumansAttentionBiological PsychiatryPsychiatric Status Rating ScalesCognitionCrossover effectsmedicine.diseasePsychiatry and Mental healthPhenotypeSchizophreniaFemaleSchizophrenic PsychologyPsychologyArousalPsychopathologySchizophrenia research
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Homozygous familial hypobetalipoproteinemia: two novel mutations in the splicing sites of apolipoprotein B gene and review of the literature.

2015

Objective: Familial hypobetalipoproteinemia (FHBL) is autosomal codominant disorder of lipoprotein metabolism characterized by low plasma levels of total cholesterol (TC), low-density lipoproteincholesterol (LDL-C) and apolipoprotein B (apoB) below the 5 th percentile of the distribution in the population. Patients with the clinical diagnosis of homozygous FHBL (Ho-FHBL) are extremely rare and few patients have been characterized at the molecular level. Here we report the medical history and the molecular characterization of one paediatric patient with clinical features of Ho-FHBL. Methods: A one month old infant with failure to thrive, severe hypocholesterolemia and acanthocytosis was clin…

ProbandAdultMaleAcanthocytosiSettore MED/09 - Medicina InternaApolipoprotein BPopulationDNA Mutational AnalysisBiologyHypobetalipoproteinemiasExonHumanseducationGeneGeneticseducation.field_of_studyHomozygoteIntronInfantCholesterol LDLAbetalipoproteinemiaIntronsAlternative SplicingHomozygous familial hypobetalipoproteinemiaCholesterolRNA splicingApolipoprotein B-100Mutationbiology.proteinlipids (amino acids peptides and proteins)FemaleCardiology and Cardiovascular MedicineApolipoprotein BMinigeneAtherosclerosis
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Novel CREB3L3 Nonsense Mutation in a Family With Dominant Hypertriglyceridemia.

2015

Objective— Cyclic AMP responsive element–binding protein 3–like 3 ( CREB3L3 ) is a novel candidate gene for dominant hypertriglyceridemia. To date, only 4 kindred with dominant hypertriglyceridemia have been found to be carriers of 2 nonsense mutations in CREB3L3 gene (245fs and W46X). We investigated a family in which hypertriglyceridemia displayed an autosomal dominant pattern of inheritance. Approach and Results— The proband was a 49-year-old woman with high plasma triglycerides (≤1300 mg/dL; 14.68 mmol/L). Her father had a history of moderate hypertriglyceridemia, and her 51-year-old brother had triglycerides levels as high as 1600 mg/dL (18.06 mmol/L). To identify the causal mutation …

ProbandAdultMaleCandidate geneHeterozygoteSettore MED/09 - Medicina InternaHeredityAdolescentNonsense mutationDNA Mutational AnalysisPenetranceBiologymedicine.disease_causeSeverity of Illness IndexFrameshift mutationExonYoung AdultmedicineHumansGenetic Predisposition to DiseasetriglycerideCyclic AMP Response Element-Binding ProteinTriglyceridesAgedGeneticsAged 80 and overHypertriglyceridemiaMutationHypertriglyceridemiaMiddle Agedmedicine.diseasePenetrancePedigreePhenotypeCodon NonsenseFemalemutationCardiology and Cardiovascular MedicineBiomarkersArteriosclerosis, thrombosis, and vascular biology
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Germline correction of an epimutation related to Silver-Russell syndrome.

2015

Like genetic mutations, DNA methylation anomalies or epimutations can disrupt gene expression and lead to human diseases. However, unlike genetic mutations, epimutations can in theory be reverted through developmental epigenetic reprograming, which should limit their transmission across generations. Following the request for a parental project of a patient diagnosed with Silver-Russell syndrome (SRS), and the availability of both somatic and spermatozoa DNA from the proband and his father, we had the exceptional opportunity to evaluate the question of inheritance of an epimutation. We provide here for the first time evidence for efficient reversion of a constitutive epimutation in the sperm…

ProbandAdultMaleGenetic counselingRussell-Silver SyndromeBiologymedicine.disease_causeGermlineEpigenesis GeneticGenomic ImprintingGene OrderGeneticsmedicineHumansExomeEpigeneticsPromoter Regions GeneticMolecular BiologyGenetics (clinical)GeneticsMutationSilver–Russell syndromeHigh-Throughput Nucleotide SequencingGeneral MedicineDNA Methylationmedicine.diseaseSilver-Russell SyndromeGerm CellsPhenotypeGene Expression RegulationGenetic LociDNA methylationCpG IslandsFemaleRNA Long NoncodingHuman molecular genetics
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The relationship between bipolar disorder and alcoholism: a controlled family study.

1995

SYNOPSISBipolar disorder and alcoholism are familial disorders. The familial–genetic relationship between both is controversial and has received insufficient study. This study explores whether bipolar disorder and alcoholism share familial risk factors, and whether the co-occurrence of lifetime diagnosis of bipolar disorder and alcoholism is familial. We report on first-degree relatives of 146 consecutively admitted patients with either bipolar disorder or/and alcoholism; relatives of the patients (in total 728 relatives directly interviewed) were compared with first-degree relatives of 109 general population probands (320 relatives directly interviewed). Overlap between the familial compon…

ProbandAdultMaleRiskmedicine.medical_specialtyBipolar DisorderAdolescentPopulationComorbidityGermanymental disordersmedicineHumansLife TablesBipolar disorderPsychiatryeducationApplied PsychologyAgededucation.field_of_studyDepressive DisorderModels GeneticFamilial riskMiddle Agedmedicine.diseaseComorbidityPsychiatry and Mental healthAlcoholismPhenotypeFemalePsychologyClinical psychologyPsychological medicine
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The impact of gender and age at onset on the familial aggregation of schizophrenia.

1993

Some recent family studies have shown that the familial risk for schizophrenia is higher in female than in male schizophrenics. It is debated whether the risks for the other disorders, such as schizotypal personality disorder or affective disorders in families of schizophrenics are similarly influenced by the proband's gender. Also, the reason for the effect of proband's gender on the recurrence risk for schizophrenia has not been clarified. This family study (159 probands, 589 first degree relatives) confirms that schizophrenia, but also schizophrenia spectrum disorders were more frequent in families of female compared with male schizophrenics. Neither age at onset in probands nor the inte…

ProbandAdultMalecongenital hereditary and neonatal diseases and abnormalitiesmedicine.medical_specialtyPsychosisComorbidityPersonality DisordersSex Factorsmental disordersmedicineHumansPharmacology (medical)Spectrum disorderFamilyRisk factorFirst-degree relativesPsychiatryBiological PsychiatryAgedPsychiatric Status Rating ScalesMood DisordersAge FactorsFamily aggregationGeneral MedicineMiddle Agedmedicine.diseaseSchizotypal personality disorderPsychiatry and Mental healthSchizophreniaSchizophreniaFemalePsychologyEuropean archives of psychiatry and clinical neuroscience
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Continuity and discontinuity of affective disorders and schizophrenia. Results of a controlled family study.

1993

Background: It is widely acknowledged that the genetic diatheses for schizophrenia and affective disorders are independent. However, there are increasing doubts about this classic view, and empirical evidence for a dichotomy of these two prototypes of functional psychoses is limited. A controlled family study of consecutive admissions was conducted to determine whether familial risks for schizophrenic (SCZ) and affective disorders were independent or overlapping. Methods: Index probands met Research Diagnostic Criteria for SCZ (n=146), schizoaffective (SA [n=115]), bipolar (BP [n=80]), or unipolar major depressive (UP [n=184])disorder. Comparison probands met Research Diagnostic Criteria fo…

ProbandAdultMalemedicine.medical_specialtyBipolar DisorderPopulationResearch Diagnostic CriteriaComorbiditySex FactorsArts and Humanities (miscellaneous)Risk FactorsmedicineHistory of depressionPrevalenceHumansFamilyBipolar disordereducationPsychiatryDepression (differential diagnoses)education.field_of_studyDepressive DisorderModels GeneticAge FactorsMiddle Agedmedicine.diseaseComorbidityPsychiatry and Mental healthPhenotypePsychotic DisordersSchizophreniaSchizophreniaFemalePsychologyClinical psychologyArchives of general psychiatry
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The risk of minor depression in families of probands with major depression: sex differences and familiality.

1992

Currently it is not clear whether minor forms of unipolar depression not matching the criteria of “major depression” should be considered as a separate diagnostic category. A controlled family study examined the familial aggregation of minor depression among probands with unipolar major depression. In the families of these probands the relative risk for minor depression was elevated by a similar magnitude to the risk for major depression. Threrefore, the diagnostic category “minor depression” would not increase diagnostic sensitivity at the expense of diagnostic specificity as far as familiality is the criterion. In agreement with recent epidemiological studies, minor depression did not rev…

ProbandAdultMalemedicine.medical_specialtyMinor (academic)Sex FactorsRisk FactorsEpidemiologymedicinePrevalenceHumansPharmacology (medical)Risk factorPsychiatryBiological PsychiatryDepression (differential diagnoses)Family HealthPsychiatric Status Rating ScalesDepressive DisorderFamily aggregationGeneral MedicinePsychiatry and Mental healthPsychotic DisordersRelative riskSchizophreniaFemalePsychologySex ratioEuropean archives of psychiatry and clinical neuroscience
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A family with various symptomatology suggestive of Anderson-Fabry disease and a genetic polymorphism of alpha galactosidase A gene.

2014

Background: Anderson/Fabry disease expresses a wide range of clinical variability in patients that it is possible to explain referring to a genetic variability with numerous mutations described in the literature (more than 600). Methods: We report some clinical cases of some members of a Sicilian family to express phenotypical variability of this disease in subjects with the same genetic mutation. Results: The first case was a 59-year-old female. Brain MRI revealed right frontal periventricular white matter of likely vascular-degenerative origin. The proband's alpha galactosidase A activity was 3.7. nmol/mL/h. Molecular genetics revealed a polymorphism: - 10 C. >. T; IVS 2-76_80del5; IVS…

ProbandAdultMalemedicine.medical_specialtyPathologySettore MED/09 - Medicina InternaAdolescentClinical BiochemistryMolecular Sequence DataBiologyAnderson-Fabry diseaseNucleic Acid DenaturationGastroenterologyPolymorphism (computer science)Internal medicineMolecular geneticsmedicineHaplotypeHumansFamilyGenetic Predisposition to DiseaseGenetic variabilitySymptomatologyChildPolymorphism GeneticBase SequenceHaplotypeHeterozygote advantageGeneral MedicineMiddle Agedmedicine.diseaseFabry diseaseMagnetic Resonance ImagingPedigreealpha-GalactosidaseFabry DiseaseMicroalbuminuriaFemaleHumanClinical biochemistry
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