Search results for "Profiling"

showing 10 items of 881 documents

New perspectives on the renal slit diaphragm protein podocin.

2011

Podocin is a critical component of the glomerular filtration barrier, its mutations causing recessive steroid-resistant nephrotic syndrome. A GenBank analysis of the human podocin (NPHS2) gene resulted in the possible existence of a new splice variant of podocin in the kidney, missing the in-frame of exon 5, encoding the prohibitin homology domain. Using RT–polymerase chain reaction and immunoblotting followed by sequence analysis, we are for the first time able to prove the expression of a novel podocin isoform (isoform 2), exclusively and constitutively expressed in human podocytes. Furthermore, we reveal singular extrarenal podocin expression in human and murine testis. Our data show the…

Gene isoformAdultMalePathologymedicine.medical_specialtyendocrine systemkidneySertoli cellsBlotting WesternImmunoblottingMolecular Sequence Datatestisurologic and male genital diseasesReal-Time Polymerase Chain ReactionFilamentous actinPathology and Forensic MedicineSertoli cell-only syndromeMiceYoung AdultmedicineAnimalsHumansProtein IsoformsSertoli cell-only syndromeAmino Acid SequenceProhibitinAgedKidneyMicroscopy ConfocalbiologyBase Sequenceurogenital systemPodocytesGene Expression ProfilingIntracellular Signaling Peptides and ProteinsMembrane ProteinsisoformMiddle Agedmedicine.diseaseSertoli cellfemale genital diseases and pregnancy complicationsWT-1medicine.anatomical_structureSlit diaphragmPodocinbiology.proteinOriginal ArticlepodocinModern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc
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WT1 isoform expression pattern in acute myeloid leukemia.

2013

WT1 plays a dual role in leukemia development, probably due to an imbalance in the expression of the 4 main WT1 isoforms. We quantify their expression and evaluate them in a series of AML patients. Our data showed a predominant expression of isoform D in AML, although in a lower quantity than in normal CD34+ cells. We found a positive correlation between the total WT1 expression and A, B and C isoforms. The overexpression of WT1 in AML might be due to a relative increase in A, B and C isoforms, together with a relative decrease in isoform D expression.

Gene isoformAdultMalecongenital hereditary and neonatal diseases and abnormalitiesCancer ResearchAdolescentCD34HL-60 CellsBiologyurologic and male genital diseasesPositive correlationCohort StudiesYoung AdultDual roleExpression patternhemic and lymphatic diseasesmedicineTumor Cells CulturedHumansProtein IsoformsWT1 ProteinsAgedAged 80 and overurogenital systemGene Expression Regulation LeukemicGene Expression ProfilingMyeloid leukemiaHematologyMiddle Agedmedicine.diseaseMolecular biologyfemale genital diseases and pregnancy complicationsLeukemiaLeukemia Myeloid AcuteOncologyCase-Control StudiesFemaleK562 CellsLeukemia research
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Aberrantly activated claudin 6 and 18.2 as potential therapy targets in non-small-cell lung cancer

2014

Claudins (CLDNs) are central components of tight junctions that regulate epithelial-cell barrier function and polarity. Altered CLDN expression patterns have been demonstrated in numerous cancer types and lineage-specific CLDNs have been proposed as therapy targets. The objective of this study was to assess which fraction of patients with non-small-cell lung cancer (NSCLC) express CLDN6 and CLDN18 isoform 2 (CLDN18.2). Protein expression of CLDN6 and CLDN18.2 was examined by immunohistochemistry on a tissue microarray (n=355) and transcript levels were supportively determined based on gene expression microarray data from fresh-frozen NSCLC tissues (n=196). Both were analyzed with regard to …

Gene isoformCancer ResearchPathologymedicine.medical_specialtyTissue microarrayCancerBiologymedicine.diseaseGene expression profilingOncologymedicineCancer researchAdenocarcinomaImmunohistochemistryClaudinLung cancerInternational Journal of Cancer
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ΔNp73β is oncogenic in hepatocellular carcinoma by blocking apoptosis signaling via death receptors and mitochondria

2010

p73 belongs to the p53 family of transcription factors known to regulate cell cycle and apoptosis. The Trp73 gene has two promoters that drive the expression of two major p73 isoform subfamilies: TA and ΔN. In general, TAp73 isoforms show proapoptotic activities, whereas members of the N-terminally truncated (ΔN) p73 subfamily that lack the transactivation domain show antiapoptotic functions. We found that upregulation of ΔNp73 in hepatocellular carcinoma (HCC) correlated with reduced survival. Here, we investigated the molecular mechanisms accounting for the oncogenic role of ΔNp73 in HCC.ΔNp73β can directly interfere with the transcriptional activation function of the TA (containing the t…

Gene isoformCarcinoma HepatocellularMolecular Sequence DataApoptosisBiologyModels BiologicalTransactivationDownregulation and upregulationCell Line TumorHumansProtein IsoformsMolecular BiologyTranscription factorGenes DominantOligonucleotide Array Sequence Analysisbcl-2-Associated X ProteinRegulation of gene expressionBase SequenceSettore BIO/11Gene Expression ProfilingTumor Suppressor ProteinsLiver NeoplasmsNuclear ProteinsTumor Protein p73PromoterReceptors Death DomainCell BiologyCell cyclePrognosisMitochondriaCell biologyDNA-Binding ProteinsEnzyme ActivationGene Expression Regulation NeoplasticDrug Resistance NeoplasmCaspasesCancer researchTumor Suppressor Protein p53Signal transductionPrecancerous ConditionsSignal TransductionDevelopmental BiologyCell Cycle
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Expression of multiple epigenetically regulated cancer/germline genes in nonsmall cell lung cancer.

2005

Cancer/germline (CG) antigens represent promising targets for widely applicable mono- and multiantigen cancer vaccines for nonsmall cell lung cancer (NSCLC). Since little is known about their composite expression in this tumor type, we analyzed 7 CG genes (MAGE-A3, NY-ESO-1, LAGE-1, BRDT, HOM-TES-85, TPX-1 and LDHC) in 102 human NSCLC specimens. About 81% of NSCLC express at least 1 and half of the specimen at least 2 CG genes. Activation of most of these genes occurs more frequently in squamous cell cancer than in adenocarcinomas. Even though we found all genes but one to be regulated by genomic methylation, not all of them are co-expressed. In particular, combining CG genes not localized …

Genetic MarkersCancer ResearchLung Neoplasms/geneticsLung NeoplasmsBiologyGermlineEpigenesis GeneticGermline mutationAntigens NeoplasmCarcinoma Non-Small-Cell Lung/geneticsCarcinoma Non-Small-Cell LungmedicineTumor Cells CulturedHumansGeneGerm-Line MutationGene Expression ProfilingCancerMethylationDNA Methylationmedicine.diseaseGene expression profilingOncologyDNA methylationImmunologyCancer researchCancer/testis antigensInternational journal of cancer
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MGFM: a novel tool for detection of tissue and cell specific marker genes from microarray gene expression data

2015

Background Identification of marker genes associated with a specific tissue/cell type is a fundamental challenge in genetic and cell research. Marker genes are of great importance for determining cell identity, and for understanding tissue specific gene function and the molecular mechanisms underlying complex diseases. Results We have developed a new bioinformatics tool called MGFM (Marker Gene Finder in Microarray data) to predict marker genes from microarray gene expression data. Marker genes are identified through the grouping of samples of the same type with similar marker gene expression levels. We verified our approach using two microarray data sets from the NCBI’s Gene Expression Omn…

Genetic MarkersCancer ResearchMicroarraysBiologyMarker genesWeb BrowserProteomicsMarker geneBioconductorGeneticsGeneGenetic Association StudiesGeneticsMicroarray analysis techniquesMethodology ArticleGene Expression ProfilingComputational BiologyReproducibility of Results3. Good healthGene expression profilingSamplesGene OntologyGenetic markerOrgan SpecificityDNA microarrayBiotechnologyBMC Genomics
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Fine mapping of the 2p11 dyslexia locus and exclusion of TACR1 as a candidate gene.

2003

Developmental dyslexia, or reading disability, is a multigenic complex disease for which at least five loci, i.e. DYX1-3 and DYX5-6, have been clearly identified from the human genome. To date, DYX1C1 is the only dyslexia candidate gene cloned. We have previously reported linkage to 2p11 and 7q32 in 11 Finnish pedigrees. Here, we report the fine mapping of the approximately 40-cM linked region from chromosome 2 as we increased marker density to one per 1.8 cM. Linkage was supported with the highest NPL score of 3.0 (P=0.001) for marker D2S2216. Association analysis using the six pedigrees showing linkage pointed to marker D2S286/rs3220265 (P value0.001) in the near vicinity of D2S2216. We w…

Genetic MarkersCandidate geneLocus (genetics)Quantitative trait locusBiologyPolymorphism Single NucleotideDyslexia03 medical and health sciences0302 clinical medicineGene mappingGenetic linkageGeneticsmedicineHumansGenetics (clinical)FinlandReceptors Tachykinin030304 developmental biologyGenetics0303 health sciencesGene Expression ProfilingHaplotypeDyslexiaChromosome Mappingmedicine.diseaseBlotting NorthernPedigreeGenetic markerChromosomes Human Pair 2030217 neurology & neurosurgeryMicrosatellite RepeatsHuman genetics
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Novel circulating microRNA signature as a potential non-invasive multi-marker test in ER-positive early-stage breast cancer:A case control study

2014

Introduction There are currently no highly sensitive and specific minimally invasive biomarkers for detection of early‐stage breast cancer. MicroRNAs (miRNAs) are present in the circulation and may be unique biomarkers for early diagnosis of human cancers. The aim of this study was to investigate the differential expression of miRNAs in the serum of breast cancer patients and healthy controls. Methods Global miRNA analysis was performed on serum from 48 patients with ER‐positive early‐stage breast cancer obtained at diagnosis (24 lymph node‐positive and 24 lymph node‐negative) and 24 age‐matched healthy controls using LNA‐based quantitative real‐time PCR (qRT‐PCR). A signature of miRNAs was…

Genetic MarkersOncologyCancer Researchmedicine.medical_specialtyEstrogen receptorBreast NeoplasmsBiologyReal-Time Polymerase Chain ReactionBioinformaticsSerum markersBreast cancerBreast cancerInternal medicinemicroRNABiomarkers TumorGeneticsmedicineHumansBreastStage (cooking)Lymph nodeResearch ArticlesAgedmiRNAReceiver operating characteristicGene Expression ProfilingCase-control studyGeneral MedicineMiddle Agedmedicine.diseaseGene Expression Regulation NeoplasticMicroRNAsCirculating MicroRNAmedicine.anatomical_structureReceptors EstrogenOncologyCase-Control StudiesMolecular MedicineFemaleMiRNAResearch Article
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D-2-hydroxyglutarate produced by mutant IDH2 causes cardiomyopathy and neurodegeneration in mice.

2014

Mutations in isocitrate dehydrogenase 1 and 2 (IDH1/2) have been discovered in several cancer types and cause the neurometabolic syndrome D2-hydroxyglutaric aciduria (D2HGA). The mutant enzymes exhibit neomorphic activity resulting in production of D2-hydroxyglutaric acid (D-2HG). To study the pathophysiological consequences of the accumulation of D-2HG, we generated transgenic mice with conditionally activated IDH2R140Q and IDH2R172K alleles. Global induction of mutant IDH2 expression in adults resulted in dilated cardiomyopathy, white matter abnormalities throughout the central nervous system (CNS), and muscular dystrophy. Embryonic activation of mutant IDH2 resulted in more pronounced ph…

Genetically modified mouseTransgeneMutantCardiomyopathyMice NudeBiologyIDH2Cell LineGlutarateschemistry.chemical_compoundMiceGeneticsmedicineAnimalsHumansMuscular dystrophyMice Inbred BALB CGlycogenGene Expression ProfilingGene Expression Regulation DevelopmentalHeartNeurodegenerative Diseasesmedicine.diseaseMolecular biologyIsocitrate DehydrogenaseIsocitrate dehydrogenasechemistryMutationCardiomyopathiesDevelopmental BiologyResearch PaperGenesdevelopment
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Fingerprinting and diversity of bacterial copA genes in response to soil types, soil organic status and copper contamination

2007

A molecular fingerprinting assay was developed to assess the diversity of copA genes, one of the genetic determinants involved in bacterial resistance to copper. Consensus primers of the copA genes were deduced from an alignment of sequences from proteobacterial strains. A PCR detection procedure was optimized for bacterial strains and allowed the description of a novel copA genetic determinant in Pseudomonas fluorescens. The copA DNA fingerprinting procedure was optimized for DNA directly extracted from soils differing in their physico-chemical characteristics and in their organic status (SOS). Particular copA genetic structures were obtained for each studied soil and a coinertia analysis …

Genetics0303 health sciencesGenetic diversityEcologybiology030306 microbiologySoil organic matterPseudomonas fluorescensSoil classificationbiology.organism_classificationApplied Microbiology and BiotechnologyMicrobiology03 medical and health sciencesDNA profilingGenetic variationGene clusterSoil microbiology030304 developmental biologyFEMS Microbiology Ecology
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