Search results for "Progenitor"

showing 10 items of 355 documents

Efficient Reprogramming of Human Fibroblasts and Blood-Derived Endothelial Progenitor Cells Using Nonmodified RNA for Reprogramming and Immune Evasion

2015

mRNA reprogramming results in the generation of genetically stable induced pluripotent stem (iPS) cells while avoiding the risks of genomic integration. Previously published mRNA reprogramming protocols have proven to be inconsistent and time-consuming and mainly restricted to fibroblasts, thereby demonstrating the need for a simple but reproducible protocol applicable to various cell types. So far there have been no published reports using mRNA to reprogram any cell type derived from human blood. Nonmodified synthetic mRNAs are immunogenic and activate cellular defense mechanisms, which can lead to cell death and inhibit mRNA translation upon repetitive transfection. Hence, to overcome RNA…

Homeobox protein NANOGCellular Reprogramming TechniquesInduced Pluripotent Stem CellsVaccinia virusFibroblastsBiologyTransfectionLIN28Molecular biologyCell biologyKruppel-Like Factor 4MicroRNAsSOX2KLF4GeneticsHumansMolecular MedicineCellular Reprogramming TechniquesRNA MessengerProgenitor cellInduced pluripotent stem cellMolecular BiologyReprogrammingEndothelial Progenitor CellsImmune EvasionHuman Gene Therapy
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The role of the human cytomegalovirus UL111A gene in down-regulating CD4+ T-cell recognition of latently infected cells: implications for virus elimi…

2009

AbstractThe capacity of human cytomegalovirus (HCMV) to establish and maintain a latent infection from which it can later reactivate ensures its widespread distribution in the population, but the mechanisms enabling maintenance of latency in the face of a robust immune system are poorly understood. We examined the role of the HCMV UL111A gene, which encodes homologs of the immunosuppressive cytokine interleukin-10 in the context of latent infection of myeloid progenitor cells. A UL111A deletion virus was able to establish, maintain, and reactivate from experimental latency in a manner comparable with parental virus, but major histocompatibility complex class II levels increased significantl…

Human cytomegalovirusCD4-Positive T-LymphocytesIsoantigensMyeloidGenes Viralmedicine.medical_treatmentImmunologyPopulationCytomegalovirusDown-RegulationBiologyIn Vitro Techniquesmedicine.disease_causeBiochemistryAutoantigensHerpesviridaeVirusImmune systemmedicineHumansProgenitor celleducationMyeloid Progenitor Cellseducation.field_of_studyHistocompatibility Antigens Class IICell BiologyHematologymedicine.diseaseVirologyVirus LatencyCytokinemedicine.anatomical_structureImmunologyCytomegalovirus InfectionsHost-Pathogen InteractionsGene DeletionBlood
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The Challenge of Using CB-HSCs As Source for Gene Therapy: Lentiviral Vector Transduction, Phenotypic Characterization and Global Gene Expression Pro…

2015

Abstract Introduction: Genetic modification of autologous hematopoietic stem and progenitor cells (HSPC) is a promising clinical intervention to cure inherited monogenic diseases. Successful gene therapy trials have already been conducted using CD34+ cells from bone marrow and from mobilized peripheral blood. In this regard, cord blood (CB) represents an attractive source of HSCs due to its high concentration of high proliferative HSPC and increased susceptibility to be transduced by lentiviral vectors. Unfortunately, the major disadvantage is the limited number of HSC in the CB collection. Consequently, ex-vivo expansion of CB-HSC is desirable to extend clinical applications. Purposes: To …

ImmunologyCD34Cell BiologyHematologyBiologyCD38BiochemistryMolecular biologyViral vectorGene expression profilingHaematopoiesisSettore BIO/18 - GeneticaCB-HSCs Gene Therapy Gene Expression Profile of Ex-Vivo Expanded CB CD34+ Cells.Cell cultureImmunologyProgenitor cellInterleukin 3
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EPCR/PAR1 Signaling Navigates Long-Term Repopulating Hematopoietic Stem Cell Bone Marrow Homing to Thrombomodulin-Enriched Blood Vessels

2015

Abstract Bone marrow (BM) homing and lodgment of long-term repopulating hematopoietic stem cells (LT-HSCs) is an active and essential first step in clinical stem cell transplantation. EPCR is expressed by murine BM LT-HSCs endowed with the highest repopulation potential and its ligand, activated protein C (aPC), has anticoagulant and anti-sepsis effects in EPCR+/PAR1+ endothelial cells. We recently found that signaling cascades, traditionally viewed as coagulation and inflammation related, also independently control EPCR+ LT-HSC BM retention and recruitment to the blood via distinct PAR1 mediated pathways. EPCR/PAR1 signaling retains LT-HSCs in the BM by restricting nitric oxide (NO) produc…

ImmunologyHematopoietic stem cellCell BiologyHematologyBiologyBiochemistryCell biologyTransplantationEndothelial stem cellHaematopoiesismedicine.anatomical_structureCdc42 GTP-Binding ProteinImmunologymedicineStem cellProgenitor cellHoming (hematopoietic)Blood
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Regulation of hematopoiesis through adhesion receptors

2001

Normal steady-state hematopoiesis takes place in the bone marrow microenvironment. Soluble factors as well as contact interactions between the hematopoietic cells and the marrow microenvironment dictate the fate of hematopoietic stem cells and progenitors. Over the last decade it has become clear that cell-cell and cell-extracellular matrix interactions through adhesion receptors play a major role in the hematopoietic process. They are required for the residence of stem cells and progenitors in the marrow, as well as for homing of stem and progenitor cells to the marrow in the setting of stem cell transplantation. Furthermore, adhesion receptors play an important role in regulation of cell …

ImmunologyMembrane ProteinsBone Marrow CellsStem cell factorCell BiologyBiologyHematopoietic Stem CellsHematopoietic ProcessHematopoiesisCell biologyEndothelial stem cellmedicine.anatomical_structureCell AdhesionmedicineAnimalsHumansImmunology and AllergyBone marrowStem cellProgenitor cellCell adhesionHoming (hematopoietic)
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IL-4-mediated drug resistance in colon cancer stem cells

2008

Cancer stem cells are defined as cells able to both extensively self-renew and differentiate into progenitors. Cancer stem cells are thus likely to be responsible for maintaining or spreading a cancer, and may be the most relevant targets for cancer therapy. The CD133 glycoprotein was recently described as a reliable cancer stem-like cell marker in colon carcinoma. CD133+ cells are both necessary and sufficient to initiate tumour growth in animal models. The CD133+ cell population and spheroid cultures contain cells expressing the stem cell marker Musashi-1 which is involved in maintenance of stem cell fate in several tissues and importantly, this expression is maintained in stem-like cells…

Induced stem cellsCancerStem cell factorAntineoplastic AgentsCell BiologyBiologymedicine.diseaseStem cell markercolon carcinoma cancer stem cells (CSCs) CD133 musashi-1 (Msi-1) interleukin-4 (IL-4) apoptosis tumor chemoresistanceCancer stem cellDrug Resistance NeoplasmImmunologyColonic NeoplasmsmedicineCancer researchNeoplastic Stem CellsAnimalsHumansInterleukin-4Stem cellProgenitor cellSettore MED/46 - Scienze Tecniche Di Medicina Di LaboratorioMolecular BiologyDevelopmental BiologyAdult stem cellCell cycle (Georgetown, Tex.)
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Complicaciones infecciosas en el trasplante de sangre de cordón umbilical de donante no emparentado en adultos con enfermedades hematológicas malignas

2019

El trasplante de sangre de cordón umbilical (TSCU) es una opción terapéutica con potencial curativo para pacientes con enfermedades hematológicas malignas u otras enfermedades con grave afectación de la hematopoyesis que carecen de un donante familiar idéntico y precisan ser trasplantados en un breve plazo. Sin embargo, una de las principales limitaciones del TSCU es la importante morbilidad y mortalidad infecciosa que se observa con este procedimiento. Un detallado conocimiento del perfil clínico y microbiológico de las infecciones que desarrollan los pacientes sometidos a un TSCU es una condición necesaria para tratar de disminuir su impacto en la morbilidad y mortalidad. Así, el análisis…

InfeccionesUNESCO::CIENCIAS MÉDICASEnfermedades hematológicas malignasTrasplante de sangre de cordón umbilicalTrasplante alogénico de progenitores hematopoyéticos:CIENCIAS MÉDICAS [UNESCO]Neoplasias hematológicas
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ID: 156

2015

Innate lymphoid cells (ILCs) are a recently discovered family of innate lymphocytes that are substantially represented at mucosal surfaces and have been implicated in the protection of epithelial barriers. Various types of ILCs can be discriminated based on the expression of distinct transcription factors controlling the expression of a distinct set of cytokine genes endowing the various ILC subsets with a specific range of effector functions. Currently, three groups of ILCs are being recognized. Group 1 ILCs (ILC1s) are a diverse group of ILCs comprised of natural killer (NK) cells and other, poorly defined subsets of ILCs. It is believed that the ILC1 fate decision is controlled by the T-…

Innate immune systemEffectorImmunologyInnate lymphoid cellHematologyT helper cellBiologyBiochemistrybody regionsmedicine.anatomical_structureImmunologyGene expressionTranscriptional regulationmedicineImmunology and Allergyskin and connective tissue diseasesMolecular BiologyTranscription factorProgenitorCytokine
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Candida albicansstimulatesin vivodifferentiation of haematopoietic stem and progenitor cells towards macrophages by a TLR2-dependent signalling

2013

Toll-like receptors (TLRs) are expressed by haematopoietic stem and progenitor cells (HSPCs), and may play a role in haematopoiesis in response to pathogens during infection. We have previously demonstrated that (i) inactivated yeasts of Candida albicans induce in vitro differentiation of HSPCs towards the myeloid lineage, and (ii) soluble TLR agonists induce in vivo their differentiation towards macrophages. In this work, using an in vivo model of HSPCs transplantation, we report for the first time that HSPCs sense C. albicans in vivo and subsequently are directed to produce macrophages by a TLR2-dependent signalling. Purified lineage-negative cells (Lin(-)) from bone marrow of C57BL/6 mic…

Innate immune systemMyeloidCellular differentiationImmunologyBiologyMicrobiologyCell biologyTransplantationHaematopoiesisTLR2medicine.anatomical_structureVirologyImmunologymedicineBone marrowProgenitor cellCellular Microbiology
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TLRs control hematopoiesis during infection

2013

Recent research has shown that (i) Toll-like receptor (TLR) agonists drive hematopoietic stem and progenitor cells (HSPCs) to proliferate and differentiate along the myeloid lineage in vitro, and (ii) direct TLR-mediated stimulation of HSPCs also promotes macrophage differentiation in vivo following infection. These new insights demonstrate that TLR signaling in HSPCs, in addition to other TLR-dependent mechanisms, can contribute to HSPC expansion and myeloid differentiation after infection. Evidence is, therefore, mounting that direct TLR-induced programming of hematopoiesis plays a key role in host defense by rapidly replenishing the innate immune system with the cells needed to deal with…

Innate immune systemMyeloidCellular differentiationImmunologyStem cell factorBiologyCell biologyHaematopoiesismedicine.anatomical_structureImmunologymedicineImmunology and AllergyProgenitor cellSignal transductionReceptorEuropean Journal of Immunology
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