Search results for "Progression"

showing 10 items of 1251 documents

Global Initiative for Asthma (GINA) Strategy 2021 - Executive summary and rationale for key changes

2021

Funder: Global Initiative for Asthma

ExacerbationRespiratory SystemINHALED CORTICOSTEROIDSCritical Care and Intensive Care MedicineAsthma managementSINGLE-INHALERDOUBLE-BLINDRisk Factorsimmune system diseasesAdrenal Cortex HormonesRELIEVER THERAPYFormoterol FumarateMedicine and Health SciencesImmunology and AllergyAnti-Asthmatic AgentsChild11 Medical and Health SciencesLung functionMILD PERSISTENT ASTHMAExecutive summaryBiologic therapiesCombined Modality TherapyLUNG-FUNCTIONChild PreschoolPractice Guidelines as TopicPerspectiveDisease ProgressionDrug Therapy Combinationmedicine.drugPulmonary and Respiratory MedicineAdultmedicine.medical_specialtyBUDESONIDE/FORMOTEROL MAINTENANCEAdolescentMild asthmaMEDLINEInhaled corticosteroidsCONTROLLED-TRIALSkills trainingGINA Strategy 2021: Executive SummaryBUDESONIDE-FORMOTEROLAdministration InhalationmedicineHumansasthma preventionIntensive care medicineSEVERE EXACERBATIONSAsthmaasthma diagnosisbusiness.industryPatient AcuityInfantasthmamedicine.diseaseAsthmarespiratory tract diseasesSelf Carewf_140Formoterolwf_100businessasthma management
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Recommendations for eye care during the alarm state by the coronavirus disease pandemic COVID-19.

2020

Abstract Objective Minimize exposure to the SARS-CoV-2, reduce the chances of cross-transmission between patients and healthcare personnel, and prevent the development of postoperative complications from the management of patients with eye diseases during the 2019 coronavirus disease pandemic (COVID-19). Methods COVID-19 literature review and consensus establishment between different Spanish ophthalmology societies in order to provide guidelines and recommendations of maximum resources primarily conditioned by the state of alert, confinement and social distancing that occurs in Spain since March 16, 2020. Results The recommendations will promote the adoption of action and protection measure…

Eye DiseasesAtención oftalmológicaOphthalmologic Surgical ProceduresDiseaseRecommendationsPostoperative Complications0302 clinical medicineRisk FactorsHealth carePandemicOutpatient clinicMedicine030212 general & internal medicineSocieties Medicaleducation.field_of_studySocial distanceChloroquineGeneral MedicineEye careDisease ProgressionOftalmologíaMedical emergencySymptom AssessmentCoronavirus InfectionsAtención oftalmológica COVID-19 Eye care Oftalmología Ophthalmology Pandemia Pandemic Recomendaciones Recommendations SARS-CoV-2HydroxychloroquineInfectious Disease Transmission Patient-to-ProfessionalContact LensesBlood SafetyPneumonia ViralPopulationArticleAntimalarialsBetacoronavirus03 medical and health sciencesHumanseducationPandemicsPersonal protective equipmentPandemiaGovernmentPandemicSARS-CoV-2business.industryCOVID-19medicine.diseaseOphthalmologyWithholding TreatmentSpainAsymptomatic DiseasesRecomendaciones030221 ophthalmology & optometrybusiness
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Defective apoptosis and tumorigenesis: role of p53 mutation and Fas/FasL system dysregulation

2010

The transcription factor p53 and the cytokine receptor FasL are two of the most famous regulators of cell life, and their alterations can cause a large number of pathologies, including cancer. In this review, we focused on how they can determine defective apoptosis, one of the causes of tumorigenesis and tumor progression. The importance of this knowledge lies in the new perspectives that gene therapy can offer to cure cancer.

Fas Ligand ProteinHistologyFree RadicalsCellBiophysicsApoptosisBiologymedicine.disease_causeFas ligandNeoplasmsmedicineAnimalsHumansfas ReceptorTranscription factorlcsh:QH301-705.5Membrane GlycoproteinsfungiCancerfood and beveragesGenetic TherapyCell BiologyGenes p53medicine.diseaseGenes bcl-2medicine.anatomical_structurelcsh:Biology (General)ApoptosisTumor progressionImmunologyCarcinogenesisCytokine receptorEuropean Journal of Histochemistry
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Staging systems in bipolar disorder: an International Society for Bipolar Disorders Task Force Report

2014

Objective: We discuss the rationale behind staging systems described specifically for bipolar disorders. Current applications, future directions and research gaps in clinical staging models for bipolar disorders are outlined. Method: We reviewed the literature pertaining to bipolar disorders, focusing on the first episode onwards. We systematically searched data on staging models for bipolar disorders and allied studies that could inform the concept of staging. Results: We report on several dimensions that are relevant to staging concepts in bipolar disorder. We consider whether staging offers a refinement to current diagnoses by reviewing clinical studies of treatment and functioning and t…

First episodeBipolar DisorderTask forceStandard treatmentDisease progressionAdvisory Committeesmedicine.diseaseSeverity of Illness IndexPsychiatry and Mental healthNeuroimagingmedicineDisease ProgressionHumansBipolar disorderMedical diagnosisPsychologyNeurocognitiveBiomarkersSocieties MedicalClinical psychology
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The Influence of Infective Dose on the Virulence of a Generalist Pathogen in Rainbow Trout (Oncorhynchus mykiss) and Zebra Fish (Danio rerio)

2015

Pathogen density and genetic diversity fluctuate in the outside-host environment during and between epidemics, affecting disease emergence and the severity and probability of infections. Although the importance of these factors for pathogen virulence and infection probability has been acknowledged, their interactive effects are not well understood. We studied how an infective dose in an environmentally transmitted opportunistic fish pathogen, Flavobacterium columnare, affects its virulence both in rainbow trout, which are frequently infected at fish farms, and in zebra fish, a host that is not naturally infected by F. columnare. We used previously isolated strains of confirmed high and low …

Fish farmingScienceVirulenceBiologyFlavobacteriumMicrobiologyFish DiseaseskirjolohiFlavobacteriaceae InfectionsmedicineAnimals14. Life underwaterseeprakalaPathogenZebrafishInfectivityMultidisciplinaryVirulenceCoinfectionQRbiology.organism_classificationmedicine.diseaseTroutDisease Models AnimalOncorhynchus mykissFlavobacterium columnareCoinfectionDisease ProgressionMedicineRainbow troutpathogenbiologiaResearch ArticlePLoS ONE
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Hepatobiliary phase hypointensity predicts progression to hepatocellular carcinoma for intermediate-high risk observations, but not time to progressi…

2020

Purpose: To determine whether hepatobiliary phase hypointensity, enhancing “capsule” and size provide prognostic information regarding the risk of progression to hepatocellular carcinoma (HCC), as well as the time to progression, of intermediate to high risk observations ≥ 10 mm with arterial phase hyperenhancement (APHE). Method: This retrospective dual-institution study included 160 LR-3 and 26 LR-4 observations measuring more than 10 mm and having APHE in 136 patients (mean age [SD], 57 [11] years old). A composite reference standard of pathologic analysis and imaging follow-up was used. The prognostic performance of hepatobiliary phase hypointensity, enhancing “capsule” and size (cut-of…

Gadolinium DTPAMalemedicine.medical_specialtyGadoxetic acidCarcinoma HepatocellularContrast MediaIndependent predictorGastroenterologyTime030218 nuclear medicine & medical imagingCohort Studies03 medical and health sciences0302 clinical medicineInternal medicineImage Interpretation Computer-AssistedmedicineHumansRadiology Nuclear Medicine and imagingReference standardsRetrospective StudiesObserver Variationbusiness.industryTime to progressionLiver NeoplasmsMean ageGeneral MedicineMiddle AgedImage EnhancementPrognosismedicine.diseaseMagnetic Resonance ImagingLiverGadoxetic acid LI-RADS Hepatobiliary phase Hepatocellular carcinoma030220 oncology & carcinogenesisHepatocellular carcinomaDisease ProgressionHepatobiliary phaseFemalebusinessFollow-Up StudiesArterial phasemedicine.drug
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The “Janus” Role of C/EBPs Family Members in Cancer Progression

2020

CCAAT/enhancer-binding proteins (C/EBPs) constitute a family of transcription factors composed of six members that are critical for normal cellular differentiation in a variety of tissues. They promote the expression of genes through interaction with their promoters. Moreover, they have a key role in regulating cellular proliferation through interaction with cell cycle proteins. C/EBPs are considered to be tumor suppressor factors due to their ability to arrest cell growth (contributing to the terminal differentiation of several cell types) and for their role in cellular response to DNA damage, nutrient deprivation, hypoxia, and genotoxic agents. However, C/EBPs can elicit completely opposi…

Gene isoformCell typeDNA damagetumor suppressorCellular differentiationReviewBiologyCatalysisInorganic Chemistrylcsh:ChemistryStructure-Activity RelationshipSettore BIO/13 - Biologia ApplicataNeoplasmsAnimalsHumansProtein IsoformscancerPhysical and Theoretical ChemistryCell Cycle ProteinMolecular BiologyTranscription factorlcsh:QH301-705.5SpectroscopyCell growthOrganic Chemistrytumor promoterPromoterGeneral MedicineC/EBPComputer Science ApplicationsCell biologyGene Expression Regulation Neoplasticlcsh:Biology (General)lcsh:QD1-999Multigene FamilyCCAAT-Enhancer-Binding ProteinsDisease ProgressionDisease SusceptibilityProtein BindingSignal TransductionInternational Journal of Molecular Sciences
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ALS monocyte-derived microglia-like cells reveal cytoplasmic TDP-43 accumulation, DNA damage, and cell-specific impairment of phagocytosis associated…

2022

Abstract Background Amyotrophic lateral sclerosis (ALS) is a multifactorial neurodegenerative disease characterised by the loss of upper and lower motor neurons. Increasing evidence indicates that neuroinflammation mediated by microglia contributes to ALS pathogenesis. This microglial activation is evident in post-mortem brain tissues and neuroimaging data from patients with ALS. However, the role of microglia in the pathogenesis and progression of amyotrophic lateral sclerosis remains unclear, partly due to the lack of a model system that is able to faithfully recapitulate the clinical pathology of ALS. To address this shortcoming, we describe an approach that generates monocyte-derived mi…

General NeuroscienceAmyotrophic Lateral SclerosisImmunologyNeurodegenerative DiseasesMonocytesInflammasomeDNA-Binding ProteinsCellular and Molecular NeurosciencePhagocytosisNeurologyDisease ProgressionHumansSettore MED/26 - NeurologiaMicrogliaTDP-43 inclusionsAmyotrophic lateral sclerosiDNA DamageJournal of Neuroinflammation
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T Cell-Specific Overexpression of TGFß1 Fails to Influence Atherosclerosis in ApoE-Deficient Mice

2013

Clinical data have indicated a negative correlation between plasma TGFß1 concentrations and the extent of atherosclerosis and have thus led to the hypothesis that the pleiotropic cytokine may have anti-atherogenic properties. T-cells are currently discussed to significantly participate in atherogenesis, but the precise role of adaptive immunity in atherogenesis remains to be elucidated. TGFß1 is known to strongly modulate the function of T-cells, however, inhibition of TGFß1 signalling in T-cells of atherosclerosis-prone knock-out mice failed to unequivocally clarify the role of the cytokine for the development of atherosclerosis. In the present study, we thus tried to specify the role of T…

Genetically modified mouseApolipoprotein ELipoproteinsT-LymphocytesScienceCD3medicine.medical_treatmentT cellTransgeneMutantGene ExpressionMice TransgenicBiologyTransforming Growth Factor beta1MiceApolipoproteins EmedicineAnimalsHumansMultidisciplinaryQRAtherosclerosisAcquired immune systemCytokinemedicine.anatomical_structureImmunologyDisease Progressionbiology.proteinMedicineFemaleResearch ArticlePLoS ONE
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IL-6 Signaling Promotes Tumor Growth in Colorectal Cancer

2004

Recent investigations support an important role for TGF-beta in the development of colorectal cancer. However, the molecular consequences of TGF-beta signaling in the colon remains incompletely understood. In a recent study in Immunity, we analyzed the role of TGF-beta in a murine model of colon cancer. Using transgenic mice overexpressing TGF-beta or a dominant negative TGF-beta receptor II under control of the CD2 minigene, we show that TGF-beta signaling in tumor infiltrating T lymphocytes regulates the growth of dysplastic colon epithelial cells, as determined by histology and a novel system for high resolution chromoendoscopy in vivo. At the molecular level, TGF-beta signaling in T cel…

Genetically modified mouseCell growthColorectal cancerCell BiologyBiologyEndoglinmedicine.diseaseTumor progressionIn vivoImmunologyCancer researchbiology.proteinmedicineADAM17 ProteinInterleukin 6Molecular BiologyDevelopmental BiologyCell Cycle
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