Search results for "Promoter"

showing 10 items of 584 documents

Repression of the nuclear receptor small heterodimer partner by steatotic drugs and in advanced nonalcoholic fatty liver disease.

2015

The small heterodimer partner (SHP) (NR0B2) is an atypical nuclear receptor that lacks a DNA-binding domain. It interacts with and inhibits many transcription factors, affecting key metabolic processes, including bile acid, cholesterol, fatty acid, and drug metabolism. Our aim was to determine the influence of steatotic drugs and nonalcoholic fatty liver disease (NAFLD) on SHP expression and investigate the potential mechanisms. SHP was found to be repressed by steatotic drugs (valproate, doxycycline, tetracycline, and cyclosporin A) in cultured hepatic cells and the livers of different animal models of NAFLD: iatrogenic (tetracycline-treated rats), genetic (glycine N-methyltransferase-defi…

MaleTranscription GeneticThiazepinesResponse elementReceptors Cytoplasmic and NuclearBiologyMiceNon-alcoholic Fatty Liver DiseaseCyclosporin amedicineCCAAT-Enhancer-Binding Protein-alphaAnimalsHumansProtein kinase APromoter Regions GeneticTranscription factorCells CulturedPharmacologyMitogen-Activated Protein Kinase 1KinaseValproic AcidFatty liverTetracyclinemedicine.diseaseFatty LiverDoxycyclineCancer researchSmall heterodimer partnerCyclosporineMolecular MedicineSignal transductionSignal TransductionMolecular pharmacology
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Creating a conditional mutation of Wnt-1 by antisense transgenesis provides evidence that Wnt-1 is not essential for spermatogenesis.

1993

We have used mice transgenic for an antisense construct for Wnt-1 to study the role of this gene in post-meiotic sperm development. The human PGK-2 promoter provided levels of Wnt-1 antisense mRNA in testes in 5 transgenic lines greatly in excess of Wnt-1 mRNA concentrations, and Wnt-1 mRNA levels were greatly decreased in the lines, by 98% in three of them. There was a general correlation between copy number of the insert, levels of antisense RNA, and decreases in mRNA. There was little effect of the antisense transgene on fertility or testicular histology suggesting that normal levels of Wnt-1 transcript are not essential for spermatogenesis.

MaleTransgeneRecombinant Fusion ProteinsMolecular Sequence DataMice Inbred StrainsMice TransgenicWnt1 ProteinBiologyMiceProto-Oncogene ProteinsGene expressionTestisGeneticsAnimalsRNA AntisenseRNA MessengerPromoter Regions GeneticSpermatogenesisRegulation of gene expressionMice KnockoutMessenger RNABase SequenceWnt signaling pathwayRNACell BiologyZebrafish ProteinsMolecular biologyAntisense RNATransgenesisMice Inbred C57BLWnt ProteinsPhosphoglycerate KinaseFertilityGene Expression RegulationOrgan SpecificityDevelopmental BiologyDevelopmental genetics
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Epigenetic upregulation of endogenous VEGF-A reduces myocardial infarct size in mice.

2014

“Epigenetherapy” alters epigenetic status of the targeted chromatin and modifies expression of the endogenous therapeutic gene. In this study we used lentiviral in vivo delivery of small hairpin RNA (shRNA) into hearts in a murine infarction model. shRNA complementary to the promoter of vascular endothelial growth factor (VEGF-A) was able to upregulate endogenous VEGF-A expression. Histological and multiphoton microscope analysis confirmed the therapeutic effect in the transduced hearts. Magnetic resonance imaging (MRI) showed in vivo that the infarct size was significantly reduced in the treatment group 14 days after the epigenetherapy. Importantly, we show that promoter-targeted shRNA upr…

MaleVascular Endothelial Growth Factor ASmall interfering RNAAnatomy and PhysiologyTranscription GeneticMyocardial InfarctionEndogenyCardiovascularCardiovascular SystemEpigenesis GeneticSmall hairpin RNAMiceMolecular cell biologyNucleic AcidsGene expressionProtein IsoformsRNA Small InterferingCyclic AMP Response Element-Binding ProteinPromoter Regions GeneticRegulation of gene expressionMultidisciplinaryChromosome BiologyQRGenomicsGene TherapyChromatinInterventional CardiologyCell biologyUp-RegulationVascular endothelial growth factor AMedicineEpigeneticsDNA modificationHistone modificationResearch ArticleTranscriptional ActivationDrugs and DevicesScienceDNA transcriptionBiologyDownregulation and upregulationGenomic MedicineGeneticsGene silencingAnimalsGene SilencingBiologyBase SequenceInverted Repeat Sequencesta1182Membrane ProteinsDNA MethylationPhosphoproteinsMolecular biologyMice Inbred C57BLRNAGene expressionPloS one
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Vitamin E deficiency induces liver nuclear factor-κB DNA-binding activity and changes in related genes

2005

The biological functions of vitamin E have been classically attributed to its property as a potent inhibitor of lipid peroxidation in cellular membranes. However, in 1991, Azzi's group first described that alpha-tocopherol inhibits smooth muscle cell proliferation in a protein kinase C (PKC)-dependent way, demonstrating a non-antioxidant cell signalling function for vitamin E. More recently, the capacity of alpha-tocopherol to modulate gene expression with the implication of different transcription factors, beyond its antioxidant properties, has also been established. This study was to determine the effect of vitamin E-deficiency on liver nuclear factor-kappa B (NF-kappaB) DNA-binding activ…

MaleVitaminChromatin ImmunoprecipitationGlutamate-Cysteine Ligasemedicine.medical_treatmentBlotting WesternBiologyBiochemistrychemistry.chemical_compoundCyclin D1CyclinsMalondialdehydemedicineAnimalsVitamin EVitamin E DeficiencyRNA MessengerRats WistarTranscription factorVitamin EBody WeightNF-kappa BPromoterDNAGeneral MedicineCell cycleGlutathioneRatsCell biologyGene Expression RegulationLiverBiochemistrychemistryVitamin E deficiencyChromatin immunoprecipitationFree Radical Research
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The Gpr1/Zdbf2 locus provides new paradigms for transient and dynamic genomic imprinting in mammals

2014

Many loci maintain parent-of-origin DNA methylation only briefly after fertilization during mammalian development: Whether this form of transient genomic imprinting can impact the early embryonic transcriptome or even have life-long consequences on genome regulation and possibly phenotypes is currently unknown. Here, we report a maternal germline differentially methylated region (DMR) at the mouse Gpr1/Zdbf2 (DBF-type zinc finger-containing protein 2) locus, which controls the paternal-specific expression of long isoforms of Zdbf2 (Liz) in the early embryo. This DMR loses parental specificity by gain of DNA methylation at implantation in the embryo but is maintained in extraembryonic tissue…

Male[SDV]Life Sciences [q-bio]Locus (genetics)Receptors G-Protein-CoupledEvolution MolecularHistonesGenomic ImprintingMice03 medical and health sciences0302 clinical medicineGeneticsAnimalsHumansEpigeneticsImprinting (psychology)Promoter Regions GeneticSpermatogenesisEmbryonic Stem Cells030304 developmental biologyMammalsGenetics0303 health sciencesbiologyGene Expression Regulation DevelopmentalDNA MethylationEmbryonic stem cellHistoneDNA methylationbiology.proteinFemaleGenomic imprintingReprogramming030217 neurology & neurosurgeryResearch PaperDevelopmental BiologyGenes & Development
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Liver-specific methionine adenosyltransferase MAT1A gene expression is associated with a specific pattern of promoter methylation and histone acetyla…

2000

Methionine adenosyltransferase (MAT) is the enzyme that catalyzes the synthesis of S-adenosylmethionine (AdoMet), the main donor of methyl groups in the cell. In mammals MAT is the product of two genes, MAT1A and MAT2A. MAT1A is expressed only in the mature liver whereas fetal hepatocytes, extrahepatic tissues and liver cancer cells express MAT2A. The mechanisms behind the tissue and differentiation state specific MAT1A expression are not known. In the present work we examined MAT1A promoter methylation status by means of methylation sensitive restriction enzyme analysis. Our data indicate that MAT1A promoter is hypomethylated in liver and hypermethylated in kidney and fetal rat hepatocytes…

Malemedicine.drug_classBiologyBiochemistryGene Expression Regulation EnzymologicHistonesGeneticsmedicineAnimalsGene SilencingRats WistarPromoter Regions GeneticMolecular BiologyRegulation of gene expressionHistone deacetylase inhibitorNucleic Acid HybridizationAcetylationMethylationMethionine AdenosyltransferaseDNA MethylationMolecular biologyChromatinRatsHistoneLiverAcetylationHistone methyltransferaseDNA methylationCancer researchbiology.proteinBiotechnologyFASEB journal : official publication of the Federation of American Societies for Experimental Biology
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Dietary fat interacts with the -514CT polymorphism in the hepatic lipase gene promoter on plasma lipid profiles in a multiethnic Asian population: th…

2003

We have previously reported an interaction between -514CT polymorphism at the hepatic lipase (HL) gene and dietary fat on high-density lipoprotein-cholesterol (HDL-C) metabolism in a representative sample of white subjects participating in the Framingham Heart Study. Replication of these findings in other populations will provide proof for the relevance and consistency of this marker as a tool for risk assessment and more personalized cardiovascular disease prevention. Therefore, we examined this gene-nutrient interaction in a representative sample of Singaporeans (1324 Chinese, 471 Malays and 375 Asian Indians) whose dietary fat intake was recorded by a validated questionnaire. When no str…

Malemedicine.medical_specialtyChinaGenotypeMedicine (miscellaneous)IndiaBiologyPolymerase Chain ReactionPolymorphism Single Nucleotidechemistry.chemical_compoundFramingham Heart StudyAsian PeopleInternal medicineGenotypeBlood plasmamedicineEthnicityHumansPromoter Regions GeneticTriglyceridesDNA PrimersNutrition and Dieteticsmedicine.diagnostic_testTriglycerideBase SequenceCholesterolConfoundingMalaysiaLipaseDietary FatsEndocrinologychemistryLiverFemaleHepatic lipaseLipid profileThe Journal of nutrition
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Interleukin-6 gene promoter –174G/C polymorphism and insulin resistance: a pilot study

2007

Background: The aim of this pilot study was to evaluate the relationship between interleukin-6 promoter -174G/C (IL-6 -174G/C) polymorphism and insulin resistance (IR) in obese patients with coronary heart disease (CHD). Methods: Twenty obese male patients with CHD were selected from a larger database of patients (n=606). IL-6 -174G/C genotype was previously analysed and only homozygotes with the CC genotype (n=10) or GG genotype (n=10) were selected. IR was measured using the homeostasis model assessment for IR (HOMA-IR) method. Results: Differences in age, body mass index, total cholesterol, low-density lipoprotein cholesterol, high-density lipoprotein cholesterol (HDL-C), triglyceride (T…

Malemedicine.medical_specialtyGenotypemedicine.medical_treatmentClinical BiochemistryCoronary DiseasePilot ProjectsBiologychemistry.chemical_compoundInsulin resistanceRisk FactorsPolymorphism (computer science)Internal medicineGenotypemedicineHumansObesityPromoter Regions GeneticPancreatic hormoneAgedPolymorphism GeneticTriglycerideInterleukin-6InsulinCholesterol HDLBiochemistry (medical)Age Factorsnutritional and metabolic diseasesGeneral MedicineMiddle Agedmedicine.diseaseObesityEndocrinologychemistryCardiovascular DiseasesFemaleInsulin ResistanceBody mass indexClinical Chemical Laboratory Medicine
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Interleukin-6 gene polymorphism is an age-dependent risk factor for myocardial infarction in men.

2005

Summary Several studies show that inflammatory components may contribute to atherosclerosis and increase the risk for myocardial infarction (MI). Interleukin-6 (IL-6) is a key pro-inflammatory and immune-modulatory cytokine of relevance for cardiovascular diseases. In this case-control study, 200 patients with MI and 257 healthy controls were genotyped for the polymorphism present in −174 promoter region of the IL-6 gene. Plasma concentrations of IL-6 and C-reactive protein (CRP) in a group of patients and controls were measured. The −174 C allele was associated with an increased risk of developing MI (OR = 2.886, c.i. = 1.801–4.624, P = 0.0001) in older patients, while no association was f…

Malemedicine.medical_specialtyGenotypemedicine.medical_treatmentImmunologyMyocardial InfarctionGastroenterologyPolymorphism Single NucleotidePolymorphism (computer science)Risk FactorsInternal medicineGenotypeGeneticsmedicineHumansGenetic Predisposition to DiseaseMyocardial infarctionRisk factorAllelePromoter Regions GeneticMolecular BiologyGeneGenetics (clinical)AllelesAgedbusiness.industryInterleukin-6Age FactorsPromoterGeneral MedicineMiddle Agedmedicine.diseaseC reactive protein DNA interleukin 6CytokineC-Reactive ProteinCase-Control StudiesImmunologybusiness
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Down-regulation of the expression of endothelial NO synthase is likely to contribute to glucocorticoid-mediated hypertension.

1999

Hypertension is a side effect of systemically administered glucocorticoids, but the underlying molecular mechanism remains poorly understood. Ingestion of dexamethasone by rats telemetrically instrumented increased blood pressure progressively over 7 days. Plasma concentrations of Na + and K + and urinary Na + and K + excretion remained constant, excluding a mineralocorticoid-mediated mechanism. Plasma NO 2 − /NO 3 − (the oxidation products of NO) decreased to 40%, and the expression of endothelial NO synthase (NOS III) was found down-regulated in the aorta and several other tissues of glucocorticoid-treated rats. The vasodilator response of resistance arterioles was tested by intravital m…

Malemedicine.medical_specialtyNitric Oxide Synthase Type IIIDown-RegulationVasodilationBiologyEndothelial NOSRats Inbred WKYUmbilical veinDexamethasonechemistry.chemical_compoundInternal medicinemedicineAnimalsRNA MessengerPromoter Regions GeneticAortaCells CulturedNitritesDNA PrimersMultidisciplinaryNitratesBase SequenceAntiglucocorticoidNitric Oxide Synthase Type IIIBiological SciencesRatsNitric oxide synthaseVasodilationEndocrinologychemistryHypertensionbiology.proteinEndothelium VascularNitric Oxide SynthaseGlucocorticoidIntravital microscopymedicine.drugTranscription FactorsProceedings of the National Academy of Sciences of the United States of America
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