Search results for "Propidium iodide"

showing 6 items of 56 documents

Swelling and death of neuronal cells by lactic acid.

1993

Lactacidosis occurring in cerebral ischemia or trauma is a major mechanism of cytotoxic brain edema and brain damage. Respective effects of lactacidosis were currently analyzed in vitro by employment of the murine neuronal cell line, Neuro-2A, in order to obtain a better understanding of specific mechanisms underlying cell swelling and cell death in comparison with glial cells. The cells were suspended in a physiological medium in the presence of lactic acid at increasing concentrations. Levels of acidosis reaching from pH 6.8-5.6 were obtained while other parameters, such as osmolarity and electrolyte concentrations, were maintained in the physiological range. Assessment of cell swelling a…

Programmed cell deathPathologymedicine.medical_specialtyBiologyFlow cytometryAndrologychemistry.chemical_compoundMiceNeuroblastomamedicineTumor Cells CulturedAnimalsViability assayPropidium iodideLactic AcidAcidosisNeuronsOsmotic concentrationmedicine.diagnostic_testCell DeathOsmolar ConcentrationHydrogen-Ion ConcentrationFlow CytometryCulture MediaNeurologychemistryCell cultureLactatesNeurology (clinical)medicine.symptomSwellingNeurogliaJournal of the neurological sciences
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Induction of apoptosis in a leukemia cell line by triterpene saponins from Albizia adianthifolia

2004

Abstract Triterpenoid saponins, which are present in plants and some marine animals, exert various important pharmacological effects. The present study examines the effects of adianthifoliosides A, B, and D (AdA, AdB, and AdD) together with two prosapogenins (Pro1 and Pro2) obtained from Albizia adianthifolia (Mimosaceae) on human leukemia T-cells (Jurkat cells) and on splenocytes. AdA, AdB, and AdD were found to exhibit a cytotoxic effect on Jurkat cells, whereas the prosapogenins were found to exert a lymphoproliferative effect on this cell type. Furthermore, all tested compounds were found to exert a synergistic lymphoproliferative activity with concanavalin A (ConA) on splenocytes. The …

T-LymphocytesClinical BiochemistryPharmaceutical ScienceAlbizziaApoptosisHemolysisBiochemistryJurkat cellsCell LineJurkat Cellschemistry.chemical_compoundDrug DiscoveryTumor Cells CulturedHumansCytotoxic T cellPropidium iodideOleanolic AcidCytotoxicityMolecular BiologyPlants MedicinalDose-Response Relationship DrugbiologyChemistryOrganic ChemistryBiological activitySaponinsbiology.organism_classificationAntineoplastic Agents PhytogenicTriterpenesBiochemistryConcanavalin ACell culturebiology.proteinMolecular MedicineAlbizia adianthifoliaBioorganic & Medicinal Chemistry
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In human endothelial cells rapamycin causes mTORC2 inhibition and impairs cell viability and function.

2008

Aim Drug-eluting stents are widely used to prevent restenosis but are associated with late endothelial damage. To understand the basis for this effect, we have studied the consequences of a prolonged incubation with rapamycin on the viability and functions of endothelial cells. Methods and results Human umbilical vein or aorta endothelial cells were exposed to rapamycin in the absence or in the presence of tumour necrosis factor α (TNFα). After a 24 h-incubation, rapamycin (100 nM) caused a significant cell loss associated with the increase of both apoptosis and necrosis, as quantified by propidium iodide staining, caspase 3 activity, and lactate dehydrogenase release. Rapamycin also impair…

Time FactorsPhysiologyApoptosismTORC1Polymerase Chain Reactionchemistry.chemical_compoundCell MovementStress FibersMicroscopy ConfocalCaspase 3TOR Serine-Threonine KinasesNitric Oxide Synthase Type IIIRibosomal Protein S6 Kinases 70-kDaUp-RegulationEndothelial stem cellmedicine.anatomical_structureBiochemistryCardiology and Cardiovascular MedicineE-SelectinEndotheliumNitric Oxide Synthase Type IIICell SurvivalBlotting WesternEnzyme-Linked Immunosorbent AssayBiologyMechanistic Target of Rapamycin Complex 1Nitric OxideTacrolimusNecrosisTheophyllinePhysiology (medical)medicineHumansImmunoprecipitationViability assayPropidium iodideProtein kinase BAdaptor Proteins Signal TransducingSirolimusDose-Response Relationship DrugL-Lactate DehydrogenaseTumor Necrosis Factor-alphaEndothelial CellsProteinsCardiovascular AgentsRegulatory-Associated Protein of mTORMolecular biologyRapamycin-Insensitive Companion of mTOR ProteinchemistryMultiprotein ComplexesTOR Serine-Threonine KinasesCarrier ProteinsProtein KinasesTranscription FactorsCardiovascular research
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Active caspase-3 detection to evaluate apoptosis induced by Verbena officinalis essential oil and citral in chronic lymphocytic leukaemia cells

2011

Verbena officinalis L., Verbenaceae, commonly known as vervain, is a plant widely used in medicine. Despite of its widespread use in different traditional practices, the mechanisms of pharmacological actions of the plant and its volatile oil are still unclear. We evaluated the pro-apoptotic activity of V. officinalis essential oil and of its main component, citral, on lymphocytes collected from ten patients with chronic lymphocytic leukaemia (CLL), a disease in which a faulty apoptotic mechanism is still retained one of the primary pathogenic events, by adding to treated mononuclear cells, annexin-V, propidium iodide, and CD19. Apoptosis was also evaluated using anti-active-caspase-3 monocl…

caspase-3lcsh:RS1-441Caspase 3PharmacologyCitralCD19law.inventionlcsh:Pharmacy and materia medicachemistry.chemical_compoundlawhemic and lymphatic diseasesPropidium iodideGeneral Pharmacology Toxicology and PharmaceuticscitralEssential oilbiologyflow cytometryVerbena officinalisapoptosisbiology.organism_classificationVerbena officinalisBiochemistrychemistryApoptosisOfficinalisbiology.proteinchronic lymphocytic leukaemiaRevista Brasileira de Farmacognosia
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Prevention of 7-Ketocholesterol-Induced Overproduction of Reactive Oxygen Species, Mitochondrial Dysfunction and Cell Death with Major Nutrients (Pol…

2020

The brain, which is a cholesterol-rich organ, can be subject to oxidative stress in a variety of pathophysiological conditions, age-related diseases and some rare pathologies. This can lead to the formation of 7-ketocholesterol (7KC), a toxic derivative of cholesterol mainly produced by auto-oxidation. So, preventing the neuronal toxicity of 7KC is an important issue to avoid brain damage. As there are numerous data in favor of the prevention of neurodegeneration by the Mediterranean diet, this study aimed to evaluate the potential of a series of polyphenols (resveratrol, RSV

eicosapentaenoic acidPharmaceutical ScienceResveratrolDiet Mediterraneanmedicine.disease_causequercetinAnalytical ChemistryMicechemistry.chemical_compound0302 clinical medicineDrug Discoveryoxidative stressKetocholesterolsNeuronschemistry.chemical_classification0303 health sciencesCell Deathfood and beveragesdocosahexaenoic acidEicosapentaenoic acidMitochondriaBiochemistryChemistry (miscellaneous)Docosahexaenoic acid030220 oncology & carcinogenesisMolecular Medicineα-linolenic acidN2a cellsArticlelcsh:QD241-44103 medical and health sciencesresveratrol.lcsh:Organic chemistryCell Line TumorMediterranean dietFatty Acids Omega-3medicineAnimalsPropidium iodidePhysical and Theoretical Chemistry7-ketocholesterol030304 developmental biologyapigeninReactive oxygen speciesOrganic ChemistryNeurotoxicityPolyphenolsmedicine.diseaseOleic acidoleic acidchemistryReactive Oxygen SpeciesOxidative stressMolecules
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Treatment of B-CLL Cells with Bortezomib and Rituximab Reduces Cell Viability In Vitro..

2004

Introduction : In B-CLL, somatic mutation of IgVH genes defines a group of patients with favorable prognosis, whereas the absence of IgVH mutations is correlated with a worse outcome. Our previous data suggested that BCL-6 mutations identify a subgroup of patients with high risk of progression despite the presence of mutated IgVH gene, but the clinical significance of this molecular alteration remains uncertain. New approaches are now being tested for the treatment of B-CLL. Proteasome inhibitor, Bortezomib (Btz), and monoclonal antibodies specific for surface antigens, Rituximab (Rtx), represent potential therapeutic strategy. Objetives : To study the effects of Btz and Rtx on viability of…

medicine.diagnostic_testBortezomibLymphocyteImmunologyCell BiologyHematologyBiochemistryMolecular biologyFlow cytometrychemistry.chemical_compoundmedicine.anatomical_structurechemistryAntigenImmunologymedicineProteasome inhibitorDoubling timePropidium iodideViability assaymedicine.drugBlood
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