Search results for "Prostaglandin E"

showing 10 items of 98 documents

Leg ulcer and osteomyelitis due to methicillin-susceptible Staphylococcus aureus infection after fracture repair treatment: a case highlighting the p…

2015

Prostaglandins appear to reduce biofilm formation and chronicization of infections, and stimulate a rapid and effective clearance of infecting micro-organisms. We report a case of recovery from methicillin-susceptible Staphylococcus aureus (MSSA) osteomyelitis after multidisciplinary management with antibiotics, anti-thrombotics and prostaglandin E1 (PGE1) vasodilator, in a patient with tibial plateau fracture repaired with internal fixation devices. A 47-year-old HIV-negative male with chronic ulcer on the proximal third of the left leg was admitted to the Orthopaedic Unit of the Orestano Clinic in Palermo, Italy, for suspected osteomyelitis. A biopsy of the skin ulcer and blood cultures w…

MaleStaphylococcus aureusSettore MED/17 - Malattie InfettiveVasodilator AgentsLeg UlcerOsteomyelitisMiddle AgedStaphylococcal InfectionsAnti-Bacterial AgentsTibial FracturesMethicillinTreatment OutcomeFibrinolytic AgentsOrthopaedic Implant-Related Infection MSSA osteomyelitis prostaglandin E1 vasodilatorRisk FactorsHumansDrug Therapy CombinationAlprostadilGentamicinsLe infezioni in medicina
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The specificity of prostaglandin E2 (PGE2) in reducing coronary vascular resistance: A comparison with adenosine.

1978

Experiments were performed on the isolated, electrically driven guinea-pig heart, perfused at constant rate. All animals were pretreated with reserpine. Myocardial contractile force (MCF), coronary perfusion pressure (CPP) and myocardial oxygen consumption (QO2) were monitored continuously. Both adenosine (ADO) and PGE2 produced a concentration-dependent decrease in the CPP. The ED50 (50% of maximum response) was 2.1 +/- 0.6 X 10(-9)M for PGE2 but 40 +/- 7 X 10(-9)M for ADO (P less than 0.01) at 1.8 mM Ca(e). This coronary vasodilation was independent of the external Ca-concentration, which was varied between 0.55-9.0 mM. PGE2 had no effect on MCF or QO2 and the effect of ADO was only sligh…

Malemedicine.medical_specialtyAdenosinePhysiologyGuinea Pigschemistry.chemical_elementProstaglandinBlood PressureOxygenchemistry.chemical_compoundOxygen ConsumptionPhysiology (medical)Internal medicinemedicineAnimalsProstaglandin E2Prostaglandins EReserpineAdenosineCoronary VesselsPerfusionConstant ratemedicine.anatomical_structurechemistryCardiologyCoronary perfusion pressureVascular resistanceFemaleVascular ResistanceCardiology and Cardiovascular Medicinemedicine.drugBasic research in cardiology
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Prostaglandin E2 receptors and COX enxymes in human hepatocellular carcinoma: role in the regulation of cell growth

2008

The aim of this study was to investigate the expression of prostaglandin E 2 receptors (EP 1-4 ), cyclooxygenase-1 (COX-1), and COX-2 in nontumor and tumor human liver tissues, and also to evaluate the antitumor activity of selective EP 1 receptor antagonist used alone or in combination with COX-1 and COX-2 selective inhibitors. Semiquantitative PCR analyses revealed that EP 1-4 , COX-1, and COX-2 mRNA expression was detected in nearly all the tissue samples assayed, although with a high variability between nontumor and tumor tissues. In vitro EP 1 receptor antagonist inhibited anchorage-independent cell growth and reduced the viability of hepatocellular carcinoma (HCC) cells in a dose-depe…

Malemedicine.medical_specialtyEP receptorSettore MED/09 - Medicina InternaCarcinoma Hepatocellularmedicine.drug_classProstaglandinmedicine.medical_treatmentGeneral Biochemistry Genetics and Molecular Biologyhepatocellular carcinoma (HCC)History and Philosophy of ScienceInternal medicineCell Line Tumormedicinecell growthHumansReceptors Prostaglandin EProstaglandin E2ReceptorAgedCOX-1ChemistryCell growthGeneral NeuroscienceLiver NeoplasmsCOX-2Middle Agedmedicine.diseaseReceptor antagonistNSAIDIn vitroCyclooxygenaseEndocrinologyProstaglandin-Endoperoxide SynthasesHepatocellular carcinomaSettore BIO/14 - FarmacologiaCancer researchFemaleLiver cancerCell DivisionProstaglandin Emedicine.drug
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Effects of zinc acexamate on blood flow and prostanoid levels in the gastric mucosa of the rat

1989

The effects of the new antiulcer compound zinc acexamate on blood flow and prostanoid levels in the gastric mucosa have been studied. Zinc acexamate (30 and 300 mg/kg) dose-dependently prevents the reduction induced by the perfusion of noradrenaline (3.5 micrograms/kg.min, 30 min) in gastric mucosal blood flow, as measured by 3H-aniline clearance. Zinc acexamate pretreatment also increases the levels of prostaglandin E2 in the gastric mucosa of the rat, both under control conditions and after infusion with noradrenaline. The levels of thromboxane A2 and prostacyclin were not modified by zinc acexamate. These results confirm the importance of microcirculation in pathogenesis and the idea tha…

Malemedicine.medical_specialtyMetabolic Clearance RateClinical BiochemistryProstacyclinBiologyMicrocirculationNorepinephrinechemistry.chemical_compoundThromboxane A2Internal medicinemedicineGastric mucosaAnimalsProstaglandin E2Chromatography High Pressure LiquidAminocaproatesStomachProstanoidRats Inbred StrainsCell BiologyAnti-Ulcer AgentsRatsEndocrinologymedicine.anatomical_structurechemistryGastric MucosaRegional Blood FlowAminocaproic AcidProstaglandinsPerfusionmedicine.drugProstaglandins, Leukotrienes and Essential Fatty Acids
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ACTIONS OF PROSTAGLANDIN E2 ON MYOCARDIAL MECHANICS, CORONARY VASCULAR RESISTANCE AND OXYGEN CONSUMPTION IN THE GUINEA-PIG ISOLATED HEART PREPARATION

1975

Abstract 1 In isolated, electrically driven (3 Hz) hearts of guinea-pigs the action of prostaglandin E2 on left ventricular pressure (LVP), oxygen consumption (Qo2) and coronary vascular resistance (CVR) was studied by establishing cumulative concentration-response curves. The hearts were perfused at a constant flow (10 ml/min) with Tyrode solution (Ca++ 1.8 mM) at 32 degrees C. 2 Under control conditions prostaglandin E2 (2.86 X10(-11) -1.43 X 10(-7) M) decreased LVP, QO2 and CVR in a concentration-dependent manner by maximally 27, 18 and 38%, respectively (P less than 0.05). 3 After reserpine pretreatment there were lower initial values for all parameters measured. The effect of prostagla…

Malemedicine.medical_specialtyReserpineTime Factorsmedicine.medical_treatmentGuinea PigsBlood PressureIn Vitro TechniquesCoronary circulationOxygen ConsumptionHeart RateCoronary CirculationInternal medicineHeart ratemedicineAnimalsProstaglandin E2Pharmacologybusiness.industryProstaglandins EHeartReserpineMyocardial Contractionmedicine.anatomical_structureEndocrinologyBlood pressureVascular resistanceVentricular pressureFemaleVascular ResistancebusinessResearch Articlemedicine.drugProstaglandin EBritish Journal of Pharmacology
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INTERACTIONS OF ISOPRENALINE AND PROSTAGLANDIN E2 WITH RESPECT TO MYOCARDIAL CONTRACTILE FORCE, CORONARY VASCULAR RESISTANCE AND MYOCARDIAL OXYGEN CO…

1976

Left ventricular pressure (LVP), left ventricular pressure derivative (LV dp/dtmax), coronary vascular resistance (CVR) and myocardial oxygen consumption (Qo2) were measured simultaneously in isolated, electrically driven hearts of guinea-pigs at constant perfusion rate. 2 LVP, LV dp/dtmax, CVR and Qo2 were greatly decreased by either the addition of prostaglandin E2 (50 ng/ml) to the perfusion fluid or pretreatment of the animals with reserpine. 3 Isoprenaline (0.5 nM to 100 nM) induced increases in LVP, LV dp/dtmax and Qo2. In the presence of prostaglandin E2, there was a parallel shift of the isoprenaline concentration-response curve for LVP and LV dp/dtmax. This effect was not seen, aft…

Malemedicine.medical_specialtyReserpinemedicine.medical_treatmentGuinea PigsIndomethacinProstaglandinBlood PressureNorepinephrinechemistry.chemical_compoundOxygen ConsumptionInternal medicineIsoprenalinemedicineAnimalsDrug InteractionsProstaglandin E2PharmacologyDose-Response Relationship Drugbusiness.industryMyocardiumProstaglandins EIsoproterenolReserpineCoronary VesselsMyocardial ContractionBlood pressuremedicine.anatomical_structurechemistryCardiologyVentricular pressureVascular resistanceFemaleVascular ResistancebusinessResearch Articlemedicine.drugProstaglandin EBritish Journal of Pharmacology
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Involvement of secretory phospholipase A2 activity in the zymosan rat air pouch model of inflammation.

1996

1. In the zymosan rat air pouch model of inflammation we have assessed the time dependence of phospholipase A2 (PLA2) accumulation in the inflammatory exudates as well as cell migration, myeloperoxidase activity, prostaglandin E2 (PGE2) and leukotriene B4 (LTB4) levels. 2. A significant increase in PLA2 activity was detected in 1,200 g supernatants of exudates 8 h after injection of zymosan into rat air pouch. This event coincided with peaks in cell accumulation (mainly neutrophils) and myeloperoxidase activity in exudates and was preceded by a rise in eicosanoid levels. 3. This enzyme (without further purification) behaved as a secretory type II PLA2 with an optimum pH at 7-8 units, lack o…

Malemedicine.medical_specialtySesterterpenesLeukotriene B4NeutrophilsAnti-Inflammatory AgentsLeukotriene B4DinoprostonePhospholipases AManoalidechemistry.chemical_compoundPhospholipase A2Internal medicinemedicineAnimalsProstaglandin E2Rats WistarPeroxidasePharmacologyInflammationAnalysis of VariancebiologyTerpenesZymosanZymosanRatsPhospholipases A2EndocrinologyEicosanoidBiochemistrychemistryMyeloperoxidasebiology.proteinHomosteroidslipids (amino acids peptides and proteins)PouchColchicinemedicine.drugResearch ArticleBritish journal of pharmacology
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Protection against 2,4,6-trinitrobenzenesulphonic acid-induced colonic inflammation in mice by the marine products bolinaquinone and petrosaspongioli…

2005

Proinflammatory mediators, namely eicosanoids, reactive oxygen and nitrogen species and cytokines, are clearly involved in the pathogenesis of intestinal bowel disease. bolinaquinone (BQ) and petrosaspongiolide M (PT), two marine products with potent anti-inflammatory action, have been shown to control the production of mediators in acute and chronic inflammatory processes. Hence, we have tested here the hypothesis that BQ and PT could ameliorate inflammation and oxidative stress parameters in 2,4,6-trinitrobenzenesulphonic acid (TNBS)-induced colitis in Balb/c mice. BQ and PT were given orally in doses of 10 or 20mg/kg/day. Treatment of the animals with BQ or PT at the highest dose signifi…

Malemedicine.medical_treatmentAnti-Inflammatory AgentsNitric Oxide Synthase Type IIInflammationNerve Tissue ProteinsPharmacologymedicine.disease_causeBiochemistryProinflammatory cytokinechemistry.chemical_compoundMiceSynaptotagminsDysideamedicineAnimalsOleanolic AcidPharmacologyMice Inbred BALB CMembrane GlycoproteinsbiologySuperoxideNitrotyrosineCalcium-Binding ProteinsInterleukinMembrane ProteinsColitisInflammatory Bowel DiseasesImmunohistochemistryNitric oxide synthasechemistryBiochemistryTrinitrobenzenesulfonic AcidCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesSynaptotagmin IHeme Oxygenase (Decyclizing)biology.proteinmedicine.symptomNitric Oxide SynthaseSesquiterpenesOxidative stressHeme Oxygenase-1Prostaglandin EInterleukin-1Biochemical pharmacology
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Toll-like receptor 2 mediates prostaglandin E2 production in murine peritoneal macrophages and splenocytes in response to Candida albicans

2004

The involvement of Toll-like receptor 2 (TLR2) and TLR4 in triggering signal transduction pathways leading to prostaglandin E(2) (PGE(2)) production in response to Candida albicans has been studied in cells from wild-type, TLR2-/- and TLR4-/- knockout mice. In vitro PGE(2) production by macrophages challenged with zymosan, yeast or hypha cells was strongly inhibited in TLR2-deficient cells, but not in TLR4-/- cells, as compared to macrophages from wild-type mice. PGE(2) production was dependent on de novo cyclooxygenase-2 (Cox2) synthesis, since unchallenged cells failed to produce PGE(2) and specific Cox2 inhibition during challenge totally blocked PGE(2) production. Similar results were o…

Malemedicine.medical_treatmentReceptors Cell SurfaceBiologyMicrobiologyDinoprostoneMicechemistry.chemical_compoundCandida albicansmedicineAnimalsProstaglandin E2Candida albicansMolecular BiologyCells CulturedMice KnockoutToll-like receptorZymosanGeneral Medicinebiology.organism_classificationMolecular biologyToll-Like Receptor 2Corpus albicansToll-Like Receptor 4TLR2chemistryCyclooxygenase 2Prostaglandin-Endoperoxide SynthasesImmunologyMacrophages PeritonealTLR4Femalelipids (amino acids peptides and proteins)Signal Transductionmedicine.drugProstaglandin EResearch in Microbiology
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Cytotoxicity of Root Canal Filling Materials to Three Different Human Cell Lines

2001

The aim of this study was to investigate the biological compatibility of five root canal sealers (Sealapex, Endion, Super-EBA, Ketac-Endo, and AH Plus) and regular and calcium hydroxide-based gutta-percha in three different human cell lines. Cultures without root canal sealers were used as controls. Cell growth, cell morphology, cell viability, protein content of the cells, and prostaglandin E 2 (PGE 2 ) release were used as parameters to determine the cytotoxicity of the materials. The protein content of the three cell lines—nasal fibroblasts, gingival fibroblasts, and epithelial tumor cells—was significantly reduced (p ≤ 0.001) by all materials tested. Determinations of PGE 2 release show…

Materials scienceCell SurvivalRoot canalmedicine.medical_treatmentStatistics as TopicCellGingivaDentistryBiocompatible MaterialsCell morphologyDinoprostoneCell LineCalcium HydroxideRoot Canal Filling Materialschemistry.chemical_compoundTumor Cells CulturedmedicineHumansNeoplasms Glandular and EpithelialViability assayCytotoxicityGeneral DentistryCalcium hydroxideEpoxy ResinsCell growthbusiness.industryProteinsFibroblastsMolecular biologySalicylatesNasal Mucosamedicine.anatomical_structurechemistryGlass Ionomer CementsDentin-Bonding AgentsGutta-PerchabusinessBiomarkersCell DivisionProstaglandin EJournal of Endodontics
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