Search results for "Protein"

showing 10 items of 21431 documents

Developmental changes and acetylcholinesterase activity in the metamorphosing brain ofTenebrio molitor: Correlation to ecdysteroid titers

1994

The brain of Tenebrio molitor exhibited marked fluctuations in acetylcholinesterase (AChE) activity throughout metamorphosis. This was true AChE activity, since it was inhibited by high substrate concentrations and by 10 μM of the specific AChE inhibitor BW284C51 [(1,5-bis'4-allyldimethylammoniumphenyl)-pentan-3-one dibromide] but not by iso-OMPA (tetraisopropylpyrophosphoramide), a cholinesterase (but not AChE) inhibitor. The histochemical AChE activity was localized in the neuropile and the nuclear envelope of neurons and glial cells. The enzyme extracted from brains with 1% Triton X-100 and 1 M NaCl sedimented as a single peak in a sucrose density gradient, with a sedimentation coefficie…

medicine.medical_specialtyPhysiologyAchémedia_common.quotation_subject20-HydroxyecdysoneBiologyBiochemistrychemistry.chemical_compoundInternal medicinemedicineMetamorphosisCholinesterasemedia_commonchemistry.chemical_classificationEcdysteroidfungiGeneral MedicineAcetylcholinesteraselanguage.human_languageSedimentation coefficientEndocrinologyEnzymechemistryInsect Sciencelanguagebiology.proteinArchives of Insect Biochemistry and Physiology
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Exercise restores decreased physical activity levels and increases markers of autophagy and oxidative capacity in myostatin/activin blocked mdx mice

2013

The importance of adequate levels of muscle size and function and physical activity is widely recognized. Myostatin/activin blocking increases skeletal muscle mass but may decrease muscle oxidative capacity and can thus be hypothesized to affect voluntary physical activity. Soluble activin receptor IIB (sActRIIB-Fc) was produced to block myostatin/activins. Modestly dystrophic mdx mice were injected with sActRIIB-Fc or PBS with or without voluntary wheel running exercise for 7 wk. Healthy mice served as controls. Running for 7 wk attenuated the sActRIIB-Fc-induced increase in body mass by decreasing fat mass. Running also enhanced/restored the markers of muscle oxidative capacity and autoph…

medicine.medical_specialtyPhysiologyActivin Receptors Type IIEndocrinology Diabetes and MetabolismBlotting WesternCitrate (si)-SynthaseMyostatinMotor ActivityHematocritMuscle hypertrophyEatingHemoglobinsMice03 medical and health sciences0302 clinical medicinePhysical Conditioning AnimalPhysiology (medical)Internal medicineAutophagymedicineAnimalsMuscle Skeletalta315Creatine KinaseAdiposity030304 developmental biology0303 health sciencesbiologymedicine.diagnostic_testTumor Necrosis Factor-alphaBody WeightAutophagySkeletal muscleDNAActivin receptorMyostatinActivinsMice Inbred C57BLmedicine.anatomical_structureEndocrinologyHematocritMice Inbred mdxbiology.proteinCreatine kinaseTumor necrosis factor alphaOxidation-Reduction030217 neurology & neurosurgeryAmerican Journal of Physiology-Endocrinology and Metabolism
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Expression of R-3-hydroxybutyrate dehydrogenase, a ketone body converting enzyme in heart and liver mitochondria of ruminant and non-ruminant mammals

1992

1. The properties of rat liver and bovine heart R-3-hydroxybutyrate dehydrogenase (BDH) have been extensively studied in the past 20 years, but little is known concerning the biogenesis and the regulation of this dehydrogenase over different species. 2. In addition, controversial results were often reported concerning the activity, the level and the subcellular location of this enzyme in ruminants. 3. BDH activity found in liver and kidney mitochondria from ruminants (cow and sheep) is low, while it is much higher in rat. 4. However, the enzyme activity is detected in microsomes and in cytosol of liver and of kidney cells from ruminants. These activities are not correlated to ketonaemia lev…

medicine.medical_specialtyPhysiologyBlotting WesternMitochondria LiverDehydrogenaseCross ReactionsBiologyMitochondrionKidneyBiochemistryMitochondria HeartHydroxybutyrate DehydrogenaseInternal medicinemedicineAnimalsHumansMolecular Biologychemistry.chemical_classificationKidneySheepGeneral MedicineEnzyme assayRatsCytosolEnzymemedicine.anatomical_structureEndocrinologyLiverchemistryBiochemistryMicrosomeKetone bodiesbiology.proteinCattleComparative Biochemistry and Physiology Part B: Comparative Biochemistry
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Pharmacological comparison of rat and human melanocortin 3 and 4 receptors in vitro.

2002

Abstract The melanocortin 3 and 4 receptors are G-protein-coupled receptors found in the hypothalamus with important role in regulation of the energy balance. In this study, we performed pharmacological comparison of the rat and human melancortin (MC) 3 and MC4 receptors. We transiently expressed the genes for these receptors individually in a mammalian cell line and determined the binding affinities to several MSH peptides. The results showed no major difference between the rat and human MC3 receptors while the rat MC4 receptor had higher affinity to several peptides compared with the human MC4 receptor. NDP-, α-, β-, γ-MSH, ACTH(1–24), HS014 and MTII had from 5- to 34-fold higher affinity…

medicine.medical_specialtyPhysiologyClinical BiochemistryHypothalamusClass C GPCRBiologyLigandsBiochemistryBinding CompetitiveCellular and Molecular NeuroscienceChemokine receptorEndocrinologyMelanocortin receptorInternal medicinemedicineCyclic AMPAnimalsHumansACTH receptorReceptor5-HT receptor5-HT2 receptorCell biologyRatsEndocrinologyReceptors Corticotropinalpha-MSHCOS CellsReceptor Melanocortin Type 45-HT1 receptorProtein BindingReceptor Melanocortin Type 3Regulatory peptides
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Increased mRNAs for procollagens and key regulating enzymes in rat skeletal muscle following downhill running.

1999

The purpose of the study was to investigate pre-translational regulation of collagen expression after a single bout of exercise. We analysed steady-state messenger ribonucleic acid (mRNA) levels for collagen types I, III and IV, alpha- and beta-subunits of prolyl 4-hydroxylase and lysyl oxidase (enzymes modifying procollagen chains), and enzyme activity of prolyl 4-hydroxylase from rat soleus muscle (MS) and the red parts of quadriceps femoris muscle (MQF) after 12 h and after 1, 2, 4, 7 and 14 days of downhill (-13.5 degrees ) treadmill running at a speed of 17 m.min-1 for 130 min. Histological and biochemical assays revealed exercise-induced muscle damage in MQF but not MS. Steady-state m…

medicine.medical_specialtyPhysiologyClinical BiochemistryPhysical ExertionProcollagen-Proline DioxygenaseLysyl oxidaseBiologyRunningProtein-Lysine 6-OxidaseHydroxyprolinechemistry.chemical_compoundType IV collagenPhysiology (medical)Internal medicineGene expressionmedicineAnimalsExertionRNA MessengerRats WistarMuscle SkeletalGlucuronidaseSoleus muscleSkeletal muscleBlotting NorthernRatsProcollagen peptidaseEndocrinologymedicine.anatomical_structurechemistryFemaleCollagenProcollagenPflugers Archiv : European journal of physiology
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Is Myeloperoxidase a Key Component in the ROS-Induced Vascular Damage Related to Nephropathy in Type 2 Diabetes?

2013

AbstractIt is still unclear whether microvascular complications of type 2 diabetes correlate with leukocyte-endothelium interactions and/or myeloperoxidase (MPO) levels. In the present study, we found that serum levels of glucose, the rate of ROS and MPO concentration were higher in type 2 diabetic patients. Patients with nephropathy (39.6%) presented higher MPO levels that correlate positively with the albumin/creatinine ratio (r=0.59, p<0.05). In addition, nephropatic patients showed increased leukocyte-endothelium interactions due to an undermining of polymorphonuclear leukocytes (PMN) rolling velocity and increased rolling flux and adhesion, which was accompanied by a rise in levels of …

medicine.medical_specialtyPhysiologyClinical BiochemistryType 2 diabetesmedicine.disease_causeBiochemistryNephropathyProinflammatory cytokinechemistry.chemical_compoundInternal medicinemedicineCell AdhesionHuman Umbilical Vein Endothelial CellsLeukocytesHumansNews & ViewsDiabetic NephropathiesCell adhesionMolecular BiologyCells CulturedGeneral Environmental SciencePeroxidaseCreatininebiologyCell adhesion moleculeCell BiologyMiddle Agedmedicine.diseaseEndocrinologychemistryDiabetes Mellitus Type 2Myeloperoxidasebiology.proteinGeneral Earth and Planetary SciencesCytokinesReactive Oxygen SpeciesCell Adhesion MoleculesOxidative stress
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Response of Bone Metabolism Markers to Ice Swimming in Regular Practitioners

2021

Objective: Both exercise and cold exposure cause physiological stress and they often occur in combination. However, the effects of exercise during severe cold on variation in bone metabolism in humans have remained elusive. The aim of this study was to investigate the variations in circulating bone metabolism markers after ice swimming (IS).Methods: Eighty-seven women and men aged 42–84 years old were recruited to perform regular IS activities. Serum parathyroid hormone (PTH), total calcium (Ca2+), total phosphorus (Pi), total magnesium (Mg2+), N-terminal osteocalcin (N-MID), total propeptide of procollagen 1 (TPINP), and C-terminal telopeptide of type 1 collagen (β-CTX) were measured 30 mi…

medicine.medical_specialtyPhysiologyOsteoporosiscold exposureParathyroid hormoneparathyroid hormone (PTH)bone (re)modeling markersBone remodelingHyperphosphatemiaN-terminal telopeptideInternal medicinePhysiology (medical)medicineQP1-981Femoral neckOriginal ResearchBone mineralbiologyexercisebusiness.industrymedicine.diseaseEndocrinologymedicine.anatomical_structureOsteocalcinbiology.proteinbusinessbone mineral densityFrontiers in Physiology
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Stimuli and sensors that initiate skeletal muscle hypertrophy following resistance exercise

2018

One of the most striking adaptations to exercise is the skeletal muscle hypertrophy that occurs in response to resistance exercise. A large body of work shows that a mammalian target of rapamycin complex 1 (mTORC1)-mediated increase of muscle protein synthesis is the key, but not sole, mechanism by which resistance exercise causes muscle hypertrophy. While much of the hypertrophy signaling cascade has been identified, the initiating, resistance exercise-induced and hypertrophy-stimulating stimuli have remained elusive. For the purpose of this review, we define an initiating, resistance exercise-induced and hypertrophy-stimulating signal as “hypertrophy stimulus,” and the sensor of such a s…

medicine.medical_specialtyPhysiologySkeletal muscle hypertrophylihaksetmuscle protein030204 cardiovascular system & hematologyMechanotransduction CellularMuscle hypertrophyWeight-Bearing03 medical and health sciencesskeletal muscle hypertrophy0302 clinical medicineStress PhysiologicalPhysiology (medical)Internal medicinemedicineAnimalsHumansMechanotransductionta315Muscle Skeletalbusiness.industryResistance trainingSkeletal muscleResistance Training030229 sport sciencesHypertrophyhypertrophy sensorEndocrinologymedicine.anatomical_structurelihasmassahypertrophy stimulusvoimaharjoitteluproteiinitSignal transductionbusiness
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Restoration of cerebral and systemic microvascular architecture in APP/PS1 transgenic mice following treatment with Liraglutide™.

2015

OBJECTIVE: Cerebral microvascular impairments occurring in AD may reduce Aβ peptide clearance and impact upon circulatory ultrastructure and function. We hypothesized that microvascular pathologies occur in organs responsible for systemic Aβ peptide clearance in a model of AD and that Liraglutide (Victoza(®)) improves vessel architecture. METHODS: Seven-month-old APP/PS1 and age-matched wild-type mice received once-daily intraperitoneal injections of either Liraglutide or saline (n = 4 per group) for eight weeks. Casts of cerebral, splenic, hepatic, and renal microanatomy were analyzed using SEM. RESULTS: Casts from wild-type mice showed regularly spaced microvasculature with smooth lumenal…

medicine.medical_specialtyPhysiologySpleenMice TransgenicKidneyMicrocirculationAmyloid beta-Protein PrecursorMiceAlzheimer DiseaseGlucagon-Like Peptide 1Physiology (medical)Internal medicinemedicinePresenilin-1AnimalsHumansHypoglycemic AgentsMolecular BiologyKidneybusiness.industryLiraglutideMicrocirculationBrainLiraglutideGlucagon-like peptide-1Extravasationmedicine.anatomical_structureEndocrinologyCerebrovascular CirculationCirculatory systemMicrovesselsSystemic administrationCardiology and Cardiovascular MedicinebusinessSpleenmedicine.drugMicrocirculation (New York, N.Y. : 1994)
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C-Reactive Protein Is Elevated Only in High Creatine Kinase Responders to Muscle Damaging Exercise.

2019

The purpose of this study was to investigate if exertional rhabdomyolysis induced by an acute bout of plyometric exercise in untrained individuals was associated with histological characteristics of skeletal muscle, creatine kinase (CK) polymorphism or secondary damage. Twenty-six healthy male untrained individuals completed a bout of plyometric exercise (10 sets of 10 maximal squat jumps, with each standardized to achieve at least 95% of individual maximal jump height). Blood samples were taken, and perceived pain was scored immediately before the exercise intervention and 6 h, 1, 2, and 3 days post-intervention. Muscle biopsies were collected 9 or 4 days before (baseline) and 3 days after…

medicine.medical_specialtyPhysiologySquatlcsh:Physiology03 medical and health sciences0302 clinical medicinesingle nucleotide polymorphismPhysiology (medical)Internal medicineWhite blood cellmedicinePlyometricsEccentricOriginal Researchlcsh:QP1-981biologybusiness.industrycreatine kinaseC-reactive proteinSkeletal muscle030229 sport sciencesmedicine.diseaseeccentric exercise and muscle damagemyeloperoxidasemedicine.anatomical_structuremyoglobinExertional rhabdomyolysisbiology.proteinCardiologyCreatine kinasebusiness030217 neurology & neurosurgeryFrontiers in physiology
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