Search results for "Pulmonary Alveoli"

showing 6 items of 46 documents

Measurements of alveolar pO2 using 19F-MRI in partial liquid ventilation.

2003

Rationale and Objectives: Partial liquid ventilation using Perfluorcarbon (PFC) is an innovative treatment of acute respiratory distress syndrome. However, the underlying mechanisms are not totally clear. The aim was to investigate the distribution of oxygen partial pressure within the PFC-filled lung (ppO 2 ). Methods: Nine pigs underwent partial liquid ventilation, receiving 20 mL PFC/kg bodyweight (bw). Measurements were obtained by a chemical shift selective TurboFLASH sequence at different axial lung levels. ppO 2 was calculated from 1 9 F-MRI by nonlinear curve T 1 -fitting technique after noise correction. Results: Quantification and distribution of ppO 2 was performed successfully. …

Respiratory Distress SyndromeLungLiquid VentilationChemistryPartial PressureDependent lungGeneral MedicinePartial pressureMagnetic Resonance ImagingLimited accessOxygenPulmonary Alveolimedicine.anatomical_structureFluorocarbon PolymersIn vivoAnesthesiamedicineDistribution (pharmacology)AnimalsRadiology Nuclear Medicine and imagingPartial liquid ventilationPulmonary VentilationPerfusionBiomedical engineeringInvestigative radiology
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Post-Transcriptional Regulation of Human Inducible Nitric-Oxide Synthase Expression by the Jun N-terminal Kinase

2007

Human inducible nitric-oxide synthase (iNOS) expression is regulated both at transcriptional and post-transcriptional levels. In the present study, the effect of Jun N-terminal kinase (JNK) on human iNOS expression was investigated. In A549/8 human alveolar epithelial cells, both the inhibition of JNK by a pharmacological inhibitor anthra[1,9-cd]pyrazol-6(2H)-one1,9-pyrazoloanthrone (SP600125) and small interfering RNA (siRNA)-mediated down-regulation of JNK led to a reduction of iNOS mRNA and protein expression. iNOS promoter activity was not affected by these treatments. Hence, JNK seems to regulate iNOS expression through post-transcriptional mechanisms by stabilizing iNOS mRNA. Our labo…

Small interfering RNARNA Stabilityp38 mitogen-activated protein kinasesDown-RegulationNitric Oxide Synthase Type IIRNA-binding proteinNitric Oxidep38 Mitogen-Activated Protein KinasesGene Expression Regulation EnzymologicCell LineTristetraprolinHumansPhosphorylationRNA Small InterferingPromoter Regions GeneticPost-transcriptional regulationAnthracenesPharmacologyRegulation of gene expressionMessenger RNAbiologyChemistryKinaseJNK Mitogen-Activated Protein KinasesEpithelial Cellsrespiratory systemMolecular biologyPulmonary AlveoliNitric oxide synthasebiology.proteinCytokinesMolecular MedicineSignal TransductionMolecular Pharmacology
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Influence of respiratory rate and end-expiratory pressure variation on cyclic alveolar recruitment in an experimental lung injury model

2012

Introduction Cyclic alveolar recruitment/derecruitment (R/D) is an important mechanism of ventilator-associated lung injury. In experimental models this process can be measured with high temporal resolution by detection of respiratory-dependent oscillations of the paO2 (ΔpaO2). A previous study showed that end-expiratory collapse can be prevented by an increased respiratory rate in saline-lavaged rabbits. The current study compares the effects of increased positive end-expiratory pressure (PEEP) versus an individually titrated respiratory rate (RRind) on intra-tidal amplitude of Δ paO2 and on average paO2 in saline-lavaged pigs. Methods Acute lung injury was induced by bronchoalveolar lavag…

Time FactorsRespiratory rateSwinePilot ProjectsLung injuryCritical Care and Intensive Care MedicinePositive-Pressure RespirationRandom AllocationRespiratory Ratevarying shunt fractionsAnimalsMedicineddc:610porcine modelcyclic alveolar recruitmentbusiness.industryResearchLung Injuryrespiratory systemrespiratory- dependent paO 2 oscillationsrespiratory tract diseasesPulmonary AlveoliDisease Models Animalacute lung injuryAnesthesiaHigh temporal resolutionIncreased respiratory ratebusinesscirculatory and respiratory physiologyCritical Care
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Vitamin A deficiency alters rat lung alveolar basement membrane: reversibility by retinoic acid.

2010

Vitamin A is essential for lung development and pulmonary cell differentiation and its deficiency results in alterations of lung structure and function. Basement membranes (BMs) are also involved in those processes, and retinoic acid, the main biologically active form of vitamin A, influences the expression of extracellular matrix macromolecules. Therefore, we have analyzed the ultrastructure and collagen content of lung alveolar BM in growing rats deficient in vitamin A and the recovering effect of all-trans retinoic acid. Male weanling pups were fed a retinol-adequate or -deficient diet until they were 60 days old. A group of vitamin A-deficient pups were recovered by daily intraperitonea…

VitaminCollagen Type IVMalemedicine.medical_specialtymedicine.drug_classEndocrinology Diabetes and MetabolismClinical BiochemistryRetinoic acidTretinoinBiochemistryBasement MembraneCollagen Type ITransforming Growth Factor beta1chemistry.chemical_compoundInternal medicineMalondialdehydemedicineAnimalsRetinoidRNA MessengerRats WistarMolecular BiologyLungPeroxidaseBasement membraneNutrition and DieteticsLungbiologyTumor Necrosis Factor-alphaVitamin A DeficiencyInterleukinsRetinolmedicine.diseaseImmunohistochemistryRatsVitamin A deficiencyPulmonary AlveoliOxidative StressProtein SubunitsEndocrinologymedicine.anatomical_structurechemistryGene Expression RegulationMyeloperoxidasebiology.proteinThe Journal of nutritional biochemistry
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Tachykinin-, calcitonin gene-related peptide-, and protein gene product 9.5-immunoreactive nerve fibers in alveolar walls of mammals.

1991

The presence and distribution of the presumed pan-neural marker protein gene product 9.5 (PGP)- and peptide-immunoreactive (ir) nerve fibers in alveolar walls of various species was investigated by light microscopic single and double staining immunohistochemistry. PGP-, tachykinin (TK)-, and calcitonin gene-related peptide (CRGP)-ir fibers were sparsely distributed in a similar pattern in alveolar walls of all species investigated. No vasoactive intestinal peptide-, peptide histidine isoleucine-, galanin-, and opioid-ir nerve fibers could be detected. PGP-ir fibers outnumbered those staining for TKs and CGRP. There was partial coexistence of PGP and TK as well as of TK and CRGP. PGP-, TK-, …

medicine.medical_specialtyAlveolar EpitheliumCalcitonin Gene-Related PeptideVasoactive intestinal peptideGuinea PigsCalcitonin gene-related peptideBiologyAlveolar cellsDogsNerve FibersSpecies SpecificityInternal medicineCricetinaeTachykininsmedicineAnimalsGalaninMammalsintegumentary systemMesocricetusGeneral NeuroscienceNeuropeptidesrespiratory systemMolecular biologyRatsPulmonary Alveolimedicine.anatomical_structureEndocrinologyCalcitoninPeripheral nervous systemCatsPulmonary alveolusUbiquitin ThiolesteraseBiomarkersNeuroscience letters
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Effect of chest compressions only during experimental basic life support on alveolar collapse and recruitment

2007

Summary Aim The importance of ventilatory support during cardiac arrest and basic life support is controversial. This experimental study used dynamic computed tomography (CT) to assess the effects of chest compressions only during cardiopulmonary resuscitation (CCO-CPR) on alveolar recruitment and haemodynamic parameters in porcine model of ventricular fibrillation. Materials and methods Twelve anaesthetized pigs (26±1kg) were randomly assigned to one of the following groups: (1) intermittent positive pressure ventilation (IPPV) both during basic life support and advanced cardiac life support, or (2) CCO during basic life support and IPPV during advanced cardiac life support. Measurements w…

medicine.medical_specialtyMean arterial pressurePulmonary AtelectasisSwinemedicine.medical_treatmentAtelectasisHeart MassageEmergency NursingReturn of spontaneous circulationInternal medicinemedicineAnimalsCardiopulmonary resuscitationbusiness.industryPulmonary Gas ExchangeAdvanced cardiac life supportHemodynamicsBasic life supportmedicine.diseaseMicrospheresSurgeryAdvanced life supportLife Support CarePulmonary AlveoliDisease Models AnimalRegional Blood FlowVentricular fibrillationVentricular FibrillationEmergency MedicineCardiologyBlood Gas AnalysisCardiology and Cardiovascular MedicinebusinessTomography X-Ray Computed
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