Search results for "RATS"

showing 10 items of 3537 documents

Regulation of Anti-Apoptotic SOD2 and BIRC3 in Periodontal Cells and Tissues.

2021

Made available in DSpace on 2021-06-25T10:49:07Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-01-02 Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP) Deutscher Akademischer Austauschdienst Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES) Deutsche Forschungsgemeinschaft The aim of the study was to clarify whether orthodontic forces and periodontitis interact with respect to the anti-apoptotic molecules superoxide dismutase 2 (SOD2) and baculoviral IAP repeat-containing protein 3 (BIRC3). SOD2, BIRC3, and the apoptotic markers caspases 3 (CASP3) and 9 (CASP9) were analyzed in gingiva from periodontally healthy and periodontitis subjects by real-time PCR…

Periodontium0301 basic medicineGingivaApoptosislcsh:ChemistryGingivitis0302 clinical medicineskin and connective tissue diseases610 Medicine &amp; healthperiodontitislcsh:QH301-705.5Cells CulturedSpectroscopybiologyCaspase 3General MedicineGingivitisBaculoviral IAP Repeat-Containing 3 ProteinCaspase 93. Good healthComputer Science Applicationsorthodontic tooth movementHost-Pathogen Interactionscardiovascular systemmedicine.symptomgingivitismedicine.medical_specialtySOD2610 Medicine & healthArticleCatalysisInorganic ChemistrySuperoxide dismutase03 medical and health sciencesstomatognathic systemOrthodontic tooth movementInternal medicinemedicineAnimalsHumansPeriodontal fiberddc:610Physical and Theoretical ChemistryPeriodontitisMolecular BiologyPeriodontitisFusobacterium nucleatumSuperoxide Dismutasebusiness.industry<i>Fusobacterium nucleatum</i>periodontal ligamentOrganic Chemistry030206 dentistryPeriodontiumFibroblastsmedicine.diseasebiology.organism_classificationRatsstomatognathic diseases030104 developmental biologyEndocrinologyGene Expression Regulationlcsh:Biology (General)lcsh:QD1-999Apoptosisbiology.proteinFusobacterium nucleatumbusinessperiodontiumPeriodontal ligament
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Experimental models of orthodontic tooth movement and their effects on periodontal tissues remodelling.

2021

Made available in DSpace on 2022-04-29T08:31:31Z (GMT). No. of bitstreams: 0 Previous issue date: 2021-10-01 Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP) Objectives: The present study aimed to compare two different models of orthodontic tooth movement (OTM) in rats by evaluating tooth movement efficiency and periodontal tissues remodelling. Design: Fifteen animals were randomly distributed into 3 groups: control group (untreated); ligature appliance (LA) as experimental OTM using a closed coil spring fixed around maxillary first molar by steel ligature; occlusal appliance (OA) as experimental OTM using a closed coil spring attached on the occlusal surface of the maxillary f…

PeriodontiumPeriodontal tissueTooth Movement TechniquesPeriodontal Ligamentmedicine.medical_treatmentOsteoclastsBone remodelingBone remodellingOrthodontic tooth movementMaxillary first molarPeriodontal fiberMedicineAnimalsLigatureGeneral DentistryBone mineralOrthodonticsAlveolar bone lossbusiness.industryCell BiologyGeneral MedicineX-Ray MicrotomographyModels TheoreticalRatsOtorhinolaryngologyTooth movementMaxillabusinessArchives of oral biology
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Toxicological evaluation of peroxisome proliferators. Further cellular and molecular aspects.

1996

International audience

Peroxisome proliferatorChemistryGeneral NeuroscienceCell CycleGuinea PigsFibric AcidsPharmacologyMicrobodiesGeneral Biochemistry Genetics and Molecular BiologyRats[SDV.TOX] Life Sciences [q-bio]/ToxicologyClofibric AcidHistory and Philosophy of ScienceLiverSpecies Specificity[SDV.TOX]Life Sciences [q-bio]/ToxicologyAnimalsHumansComputingMilieux_MISCELLANEOUSCells CulturedAnnals of the New York Academy of Sciences
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Phosphorylation of peroxisome proliferator-activated receptor α in rat Fao cells and stimulation by ciprofibrate

1999

The basic mechanism(s) by which peroxisome proliferators activate peroxisome proliferator-activated receptors (PPARs) is (are) not yet fully understood. Given the diversity of peroxisome proliferators, several hypotheses of activation have been proposed. Among them is the notion that peroxisome proliferators could activate PPARs by changing their phosphorylation status. In fact, it is well known that several members of the nuclear hormone receptor superfamily are regulated by phosphorylation. In this report, we show that the rat Fao hepatic-derived cell line, known to respond to peroxisome proliferators, exhibited a high content of PPARalpha. Alkaline phosphatase treatment of Fao cell lysat…

Peroxisome proliferator-activated receptor gammaPhosphataseReceptors Cytoplasmic and NuclearPeroxisome proliferator-activated receptorBiologyMicrobodiesBiochemistryCell LineClofibric AcidmedicineAnimalsEnzyme InhibitorsPhosphorylationPharmacologychemistry.chemical_classificationFibric Acidsfood and beveragesPeroxisomePhosphoric Monoester HydrolasesRatsGene Expression RegulationBiochemistryNuclear receptorchemistryPhosphorylationPeroxisome Proliferatorslipids (amino acids peptides and proteins)Acyl-CoA OxidasePeroxisome proliferator-activated receptor alphaCiprofibrateOxidoreductasesTranscription Factorsmedicine.drugBiochemical Pharmacology
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Functional characterization of a peroxisome proliferator response-element located in the intron 3 of rat peroxisomal thiolase B gene.

2003

Expression of the rat peroxisomal 3-ketoacyl-CoA thiolase gene B is induced by peroxisome proliferators. Although a sequence element like a peroxisome proliferator-activated receptor (PPAR)-binding site is located in the promoter region of this gene, we previously found that this element is competent for the activation by hepatocyte nuclear factor-4, but not functional with PPARalpha. We describe here a new peroxisome proliferator-response element located in the intron 3 (+1422/+1434) that binds in vitro the PPARalpha/retinoid X receptor alpha heterodimer and confers the induction by PPARalpha in transfection assays. We propose a model of regulation of the rat thiolase B gene involving thos…

Peroxisome proliferator-activated receptor gammaResponse elementBiophysicsPeroxisome proliferator-activated receptorReceptors Cytoplasmic and NuclearRetinoid X receptorBiochemistryGene Expression Regulation EnzymologicStructure-Activity RelationshipPeroxisomesAnimalsAcetyl-CoA C-AcetyltransferaseMolecular BiologyCells Culturedchemistry.chemical_classificationThiolaseChemistryCell BiologyPhosphoproteinsMolecular biologyIntronsRatsDNA-Binding ProteinsBiochemistryHepatocyte Nuclear Factor 4LiverPeroxisome proliferator-activated receptor deltaPeroxisome ProliferatorsPeroxisome proliferator-activated receptor alphaPPARGC1BTranscription FactorsBiochemical and biophysical research communications
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Nonlinearities in amoxycillin pharmacokinetics. II. Absorption studies in the rat.

1992

Most factors influencing amoxycillin oral absorption are, even today, unknown. Since many dosage schedules have been shown to lead to incomplete absorption, it would be desirable to find a suitable animal model where these factors could be studied in depth. In this paper, it is shown that, in the rat, plasma level curves obtained after oral doses of 7 and 28 mg kg-1 are poorly fitted using first-order absorption kinetics and that the best fit is obtained through the use of an input equation combining zero and first-order kinetics. In contrast, plasma level curves found after intraduodenal administration of amoxycillin solutions (7 mg kg-1) are well fitted by first-order input kinetics. It w…

PharmacologyAbsorption (pharmacology)MaleChemistryKineticsPharmaceutical ScienceAmoxicillinBiological AvailabilityRats Inbred StrainsGeneral MedicinePharmacologyAmoxicillinModels BiologicalIntestinal absorptionBioavailabilityRatsPharmacokineticsIntestinal AbsorptionOral administrationCurve fittingmedicineAnimalsPharmacology (medical)medicine.drugBiopharmaceuticsdrug disposition
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Behavioural studies on rats injected with l-cycloserine and other compounds

1964

l-cycloserine, a cyclic derivative of O-substituted hydroxylamine, causes the suppression of the conditioned avoidance reaction in the rat. The subsequent injection of equimolar amounts of pyridoxine hydrochloride is responsible for a complete reversal of this suppression. The suppression of the conditioned response is not observed with the d-isomer of cycloserine. Isoniazid is a strong antagonist of l-cycoserine. Changes in the brain level of GABA under several conditions are described. These changes suggest though do not establish a relationship between the block of the conditioned response and the elevation of the aminoacid above its physiological concentration.

PharmacologyAminobutyratesResearchCycloserineAntagonistBrainPyridoxineMetabolismPharmacologyPyridoxineAminobutyric acidRatschemistry.chemical_compoundMetabolismHydroxylamineBiochemistrychemistryCycloserineConditioning PsychologicalAvoidance LearningIsoniazidmedicineAvoidance reactionPyridoxine Hydrochloridemedicine.drugPsychopharmacologia
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Changes of the renin-angiotensin and of the kallikrein-kinin system after administration of saline, christalloid and colloid solution in the rat. Eff…

1988

PharmacologyBlood VolumeCaptoprilChemistrymedicine.medical_treatmentCaptoprilKininsKininPharmacologySodium ChlorideRatsRenin-Angiotensin SystemSolutionsColloidGlucoseFurosemideRenin–angiotensin systemmedicineAnimalsKallikreinsColloidsSalinemedicine.drugPharmacological research communications
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The effect of cannabinoid receptor agonist WIN 55,212-2 on anxiety-like behavior and locomotion in a genetic model of absence seizures in the elevate…

2022

GAERS and NEC rats were treated with cannabinoid 1/2 receptor agonist WIN 55,212-2 2 mg/kg and tested on the Elevated Plus-Maze

PharmacologyCannabinoid Receptor AgonistsModels GeneticMorpholinesNECWIN 55212-2 2AnxietyNaphthalenesSettore BIO/09 - FisiologiaBenzoxazinesRatsElevated Plus Maze TestPsychiatry and Mental healthEpilepsy AbsenceSeizuresPhysiology (medical)GAERSAnimalsPharmacology (medical)Elevated Plus-MazeRats WistarLocomotionCNS neurosciencetherapeutics
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Isolated myocardiocytes and DNA synthesis: a possible experimental model to predict the in vivo cardiotoxicity

1989

PharmacologyCardiotoxicityHeart DiseasesDNA synthesisBiological modelingbusiness.industryExperimental modelMyocardiumDNAPharmacologyModels BiologicalRatsMiceDoxorubicinIn vivoToxicityAnimalsMedicineMitoxantronebusinessPharmacological Research
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