Search results for "RATS"

showing 10 items of 3537 documents

Toxicity of the antimalarial artemisinin and its dervatives.

2010

As long as no effective malaria vaccine is available, chemotherapy belongs to the most important weapons fighting malaria. One of the most promising new drug developments is the sesquiterpene artemisinin (ARS) and its derivatives, e.g., artemether, arteether, and sodium artesunate. Large clinical studies and meta-analyses did not show serious side effects, although proper monitoring of adverse effects in developing countries might not be a trivial task. There is a paucity of large-scale clinical trials suitable to detect rare but significant toxicity. Therefore, a final and definitive statement on the safety of artemisinins still cannot be made. In contrast, animal experiments show consider…

ArtemisininsDrug-Related Side Effects and Adverse ReactionsArtesunatePharmacologyToxicologychemistry.chemical_compoundAntimalarialsDogsparasitic diseasesMedicineAnimalsHumansArtemetherArtemisininAdverse effectDeveloping CountriesClinical Trials as Topicbusiness.industryMalaria vaccineDrug Administration Routesmedicine.diseaseArtemisininsMalariaRatschemistryArtesunateToxicityArtemetherRabbitsbusinessSesquiterpenesMalariamedicine.drugCritical reviews in toxicology
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Pharmacological blockade of the fatty acid amide hydrolase (FAAH) alters neural proliferation, apoptosis and gliosis in the rat hippocampus, hypothal…

2015

Endocannabinoids participate in the control of neurogenesis, neural cell death and gliosis. The pharmacological effect of the fatty acid amide hydrolase (FAAH) inhibitor URB597, which limits the endocannabinoid degradation, was investigated in the present study. Cell proliferation (phospho-H3(+) or BrdU(+) cells) of the main adult neurogenic zones as well as apoptosis (cleaved caspase-3(+)), astroglia (GFAP(+)), and microglia (Iba1(+) cells) were analyzed in the hippocampus, hypothalamus and striatum of rats intraperitoneally treated with URB597 (0.3 mg/kg/day) at one dose/4-days resting or 5 doses (1 dose/day). Repeated URB597 treatment increased the plasma levels of the N-acylethanolamine…

AstrocitosNeurobiologia del desenvolupamentAmidohidrolasasCannabinoid receptorCarbamatos:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Intracellular Signaling Peptides and Proteins::Apoptosis Regulatory Proteins::Caspases [Medical Subject Headings]:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Differentiation::Neurogenesis [Medical Subject Headings]medicine.medical_treatment:Chemicals and Drugs::Carbohydrates::Monosaccharides::Hexoses::Glucose [Medical Subject Headings]Apoptosis:Phenomena and Processes::Physiological Phenomena::Body Constitution::Body Weights and Measures::Body Size::Body Weight [Medical Subject Headings]chemistry.chemical_compound:Chemicals and Drugs::Amino Acids Peptides and Proteins::Proteins::Membrane Proteins::Receptors Cell Surface::Receptors G-Protein-Coupled::Receptors Cannabinoid::Receptor Cannabinoid CB1 [Medical Subject Headings]0302 clinical medicine:Chemicals and Drugs::Organic Chemicals::Carboxylic Acids::Acids Acyclic::Carbamates [Medical Subject Headings]Fatty acid amide hydrolaseReceptor cannabinoide CB1:Organisms::Eukaryota::Animals [Medical Subject Headings]FAAHGliosishealth care economics and organizations:Chemicals and Drugs::Nucleic Acids Nucleotides and Nucleosides::Nucleosides::Deoxyribonucleosides::Deoxyuridine::Bromodeoxyuridine [Medical Subject Headings]:Chemicals and Drugs::Lipids::Glycerides::Triglycerides [Medical Subject Headings]Original Research0303 health sciencesNeurogenesisBenzamidas:Chemicals and Drugs::Polycyclic Compounds::Steroids::Cholestanes::Cholestenes::Cholesterol [Medical Subject Headings]Endocannabinoid systemEtanolaminas3. Good healthEndocannabinoides:Chemicals and Drugs::Lipids::Fatty Acids::Fatty Acids Unsaturated::Fatty Acids Monounsaturated::Oleic Acids [Medical Subject Headings]CannabinoidesMicroglíalipids (amino acids peptides and proteins)medicine.symptomColesterol:Chemicals and Drugs::Organic Chemicals::Hydrocarbons::Terpenes::Cannabinoids [Medical Subject Headings]:Chemicals and Drugs::Lipids::Fatty Acids::Palmitic Acids [Medical Subject Headings]psychological phenomena and processesProliferación celularmedicine.medical_specialtyCerebroNeurogenesiseducationBiologyBromodesoxiuridina:Anatomy::Nervous System::Neuroglia::Microglia [Medical Subject Headings]Triglicéridoslcsh:RC321-571Ácidos oléicosRatas03 medical and health sciencesCellular and Molecular NeuroscienceInternal medicineHipocampomedicineCaspasa 3:Anatomy::Nervous System::Central Nervous System::Brain::Limbic System::Hippocampus [Medical Subject Headings]:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Growth Processes::Cell Proliferation [Medical Subject Headings]lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry030304 developmental biologyPalmitoylethanolamide:Chemicals and Drugs::Chemical Actions and Uses::Pharmacologic Actions::Molecular Mechanisms of Pharmacological Action::Neurotransmitter Agents::Endocannabinoids [Medical Subject Headings]:Chemicals and Drugs::Enzymes and Coenzymes::Enzymes::Hydrolases::Amidohydrolases [Medical Subject Headings]Cannabinoids:Anatomy::Cells::Neuroglia::Astrocytes [Medical Subject Headings]Peso corporalEnergy metabolism:Anatomy::Nervous System::Central Nervous System::Brain [Medical Subject Headings]:Anatomy::Nervous System::Central Nervous System::Brain::Limbic System::Hypothalamus [Medical Subject Headings]URB597:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Death [Medical Subject Headings]:Diseases::Pathological Conditions Signs and Symptoms::Pathologic Processes::Gliosis [Medical Subject Headings]:Chemicals and Drugs::Organic Chemicals::Amines::Amino Alcohols::Ethanolamines [Medical Subject Headings]Muerte celular:Phenomena and Processes::Cell Physiological Phenomena::Cell Physiological Processes::Cell Death::Apoptosis [Medical Subject Headings]:Organisms::Eukaryota::Animals::Chordata::Vertebrates::Mammals::Rodentia::Muridae::Murinae::Rats [Medical Subject Headings]EndocrinologyURB597chemistryGliosisnervous systemGlucosaCannabinoidEnergy Metabolism:Chemicals and Drugs::Organic Chemicals::Amides::Benzamides [Medical Subject Headings]HipotálamoÁcidos palmíticos030217 neurology & neurosurgeryNeuroscienceFrontiers in Cellular Neuroscience
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Evidence that adenosine is not involved in the non-adrenergic non-cholinergic relaxation in the rat duodenum.

1990

In rat isolated duodenal segments, adenosine induced, in the presence of atropine and guanethidine, a dose-dependent, long-lasting (about 20 s), tetrodotoxin (TTX)-resistant relaxation both in endoluminal pressure and in isometric tension. Electrical field stimulation (EFS) induced, in the presence of atropine and guanethidine, a TTX-sensitive short-lasting (about 6 s) relaxation followed by a sustained rebound contraction. Theophylline, a P1 receptor antagonist, at the concentration of 100 microM caused a marked inhibition of the adenosine-induced relaxation, while the EFS-induced relaxation was not modified. Our results suggest that adenosine induces relaxation of the rat duodenal smooth …

AtropineGuanethidinemedicine.medical_specialtyAdenosinePhysiologyDuodenumMuscle RelaxationTetrodotoxinIn Vitro TechniquesBiochemistrychemistry.chemical_compoundTheophyllineInternal medicineIsometric ContractionmedicinePressureAnimalsTheophyllineNeurotransmitterReceptorGuanethidineReceptors PurinergicMuscle SmoothAdenosineAdenosine receptorElectric StimulationRatsAtropineEndocrinologychemistryTetrodotoxinmedicine.drugArchives internationales de physiologie et de biochimie
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Endotoxin inhibits gastric emptying in rats via a capsaicin-sensitive afferent pathway.

2001

The effects of endotoxin on gastric emptying of a solid nutrient meal and the neural mechanisms involved in such a response were investigated in conscious rats. The intraperitoneal (i.p.) administration of E. coli endotoxin (40 mug/kg) significantly reduced the 4-h rate of gastric emptying of a standard solid nutrient meal. Ablation of primary afferent neurons by systemic administration of high doses of capsaicin (20+30+50 mg/kg s.c.) to adult rats did not modify the rate of gastric emptying in control animals but prevented the delay in gastric transit induced by endotoxin. Local application of capsaicin to the vagus nerve rather than application of capsaicin to the celiac ganglion signific…

AtropineLipopolysaccharidesMaleendotoxinmedicine.medical_specialtyCalcitonin Gene-Related PeptidePharmacology toxicologyMuscarinic AntagonistscapsaicinRats Sprague-Dawleychemistry.chemical_compoundgastric emptyingtransitNeurons EfferentCalcitonin Gene-Related Peptide Receptor AntagonistsInternal medicinemedicineAnimalsDrug InteractionsCGRPNeurons AfferenttachykininsPhentolamineAfferent PathwayAdrenergic alpha-AntagonistsPharmacologyMealAfferent PathwaysGastric emptyingdigestive oral and skin physiologyGeneral MedicinePeptide FragmentsRatsEndocrinologychemistryGastric EmptyingCapsaicinCapsaicinNaunyn-Schmiedeberg's archives of pharmacology
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PACAP induces bradycardia in guinea-pig heart by stimulation of atrial cholinergic neurones

1996

Based on previous studies which indicated that pituitary adenylate cyclase activating peptide (PACAP) acts as a positive inotropic and chronotropic substance in different species via the cAMP signal transduction pathway, the objective of the present work was to investigate cAMP-regulated myocardial key proteins in response to PACAP in isolated ventricular cells of the guinea pig. Surprisingly, the two molecular forms of PACAP, PACAP(1-27) and PACAP(1-38), showed no effect on intracellular cAMP-levels, L-type Ca2+ channel current or phosphorylation of troponin inhibitor (TnI) and phospholamban (PLB). Additionally, inotropy of isolated guinea-pig ventricular strips was not affected by the neu…

AtropineMaleChronotropicendocrine systemmedicine.medical_specialtyGuinea PigsReceptors Pituitary Adenylate Cyclase-Activating PolypeptideStimulationIn Vitro TechniquesBiologyInternal medicineBradycardiaCyclic AMPmedicineAnimalsHeart AtriaReceptors Pituitary HormonePatch clampNeuronsPharmacologyNeurotransmitter AgentsMyocardiumCalcium-Binding ProteinsNeuropeptidesGeneral MedicineAcetylcholineRatsPhospholambanElectrophysiologyAtropinePituitary adenylate cyclase-activating peptideEndocrinologycardiovascular systemPituitary Adenylate Cyclase-Activating PolypeptideCholinergicFemalehormones hormone substitutes and hormone antagonistsAcetylcholineReceptors Pituitary Adenylate Cyclase-Activating Polypeptide Type ISignal Transductionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Evidence for the presence of functional protease activated receptor 4 (PAR4) in the rat colon

2004

Background and aims: Protease activated receptors (PARs) have been postulated to play a role during intestinal inflammation. The presence and role played by PAR4 in gastrointestinal functions have not been fully clarified. The aims of this study were: (i) to examine expression of PAR4 in rat proximal colon; (ii) to determine the mechanical effects induced by PAR4 activation in longitudinal muscle; and (iii) to characterise the underlying mechanisms. Methods: PAR4 expression was determined by reverse transcription-polymerase chain reaction (RT-PCR) and immunohistochemistry. Mechanical activity was recorded as changes in isometric tension. Results: A PCR product corresponding to the predicted…

AtropineMaleQuinuclidinesmedicine.medical_specialtyColonMotilityInflammationTetrodotoxinPROTEASE-ACTIVATED RECEPTORSBiologyIntestine InflammationSettore BIO/09 - Fisiologiachemistry.chemical_compoundNeurokinin-1 Receptor AntagonistsPiperidinesInternal medicinemedicineAnimalsRNA MessengerRats WistarReceptorSettore MED/12 - GastroenterologiaDose-Response Relationship DrugReverse Transcriptase Polymerase Chain ReactionGastroenterologyMuscle SmoothReceptors Neurokinin-2ColitisImmunohistochemistryRatsEndocrinologyMechanism of actionchemistryCapsaicinCROSS-REACTIVITYBenzamidesGASTRIC SMOOTH-MUSCLETetrodotoxinReceptors ThrombinCapsaicinmedicine.symptomGastrointestinal MotilityOligopeptidesAcetylcholineMuscle Contractionmedicine.drugMuscle contractionGut
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Effects of cimetidine, atropine and prostaglandin E2 on rat mucosal erosions produced by intragastric distension

1980

Abstract The effects of three typical antisecretory agents: cimetidine, atrophine and prostaglandin E2 were compared on an acute rat gastric ulcer model which consisted of perfusing the stomach continuously, at a high intraluminal pressure (120 mm H2O), with a simulated gastric juice (0.1 M HCl plus 600 mg pepsin/1). As the acid and pepsin are given exogenously the inhibitory action of the antisecretory drugs is obviated in this model. Cimetidine and atropine failed to reduce gastric erosions, whereas prostaglandin E2 markedly reduced the severity of the mucosal lesions with respect to control values. Long-term treatment with cimetidine also failed to increase the resistance of the gastric …

AtropineMalemedicine.medical_treatmentPharmacologyGuanidinesPepsinmedicineGastric mucosaAnimalsStomach UlcerProstaglandin E2CimetidinePharmacologyGastric Juicebiologybusiness.industryProstaglandins EGastric distensionStomachdigestive oral and skin physiologyRatsDisease Models AnimalAtropinemedicine.anatomical_structureAnesthesiabiology.proteinmedicine.symptomCimetidinebusinessProstaglandin Emedicine.drugEuropean Journal of Pharmacology
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Mode and mechanism of neurotensin action in rat proximal colon

1997

Abstract This study examined the mechanism of action of neurotensin on intraluminal pressure in rat proximal colon. The direct and indirect contractile response to neurotensin (100 nM) was abolished in Ca 2+ -free solution, and was antagonized by nifedipine (1–5–10 nM) and potentiated by Bay K 8644 (methyl 1,4-dihydro-2,6-dimethyl-3-nitro-4-(2-trifluoromethylphenyl)-pyridine-5-carboxylate) (10–100–1000 nM). Neurotensin, in the presence of nifedipine (10 nM) and atropine (1 μM), induced a tetrodotoxin-insensitive inhibitory effect, which was antagonized by SR 48692 (2[(1-(7-chloro-4-quinolinyl)-5-(2,6-dimethoxy-phenyl)pyrazol-3-yl) carbonyl amino]tricyclo (3.3.1.1. 3.7 ) decan-2-carboxylic a…

Atropinemedicine.medical_specialtyNifedipineColonchemistry.chemical_elementCholinergic AgonistsIn Vitro TechniquesCalciumInhibitory postsynaptic potentialApaminCholinergic Antagonistschemistry.chemical_compoundNifedipineInternal medicinemedicineAnimalsReceptors NeurotensinRats WistarNeurotensinPharmacologyChemistryMuscle Smooth3-Pyridinecarboxylic acid 14-dihydro-26-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)- Methyl esterBethanecholCalcium Channel BlockersRatsCalcium Channel AgonistsEndocrinologyApaminMechanism of actionQuinolinesExcitatory postsynaptic potentialBiophysicsPyrazolesCalciummedicine.symptomMuscle Contractionmedicine.drugMuscle contractionNeurotensinEuropean Journal of Pharmacology
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Absence of muscarinic modulation of vasopressin release from the isolated rat neurohypophysis

1975

1. Isolated rat neurohypophyses were incubated in Locke solution at 37°C and the vasopressin output into the medium determined by bioassay. 2. Potassium chloride 60 mM caused a 9-fold increase in the rate of vasopressin release that was abolished when calcium chloride was omitted from the Locke solution. 3. Acetylcholine 5.5×10−4 M neither alone nor in the presence of atropine 2.9×10−6 M changed the “resting” release of vasopressin. 4. Neither acetylcholine 5.5×10−4 M nor oxotremorine 10−4 and 3×10−4 M altered the vasopressin release evoked by potassium chloride 60 mM. 5. In contrast to the peripheral adrenergic nerve fibres, the secretory terminal fibres of the neurohypophysis do not appea…

Atropinemedicine.medical_specialtyVasopressinVasopressinschemistry.chemical_elementIn Vitro TechniquesCalciumInhibitory postsynaptic potentialPotassium ChloridePituitary Gland PosteriorInternal medicineMuscarinic acetylcholine receptormedicineOxotremorineAnimalsPharmacologyNeurosecretionChemistryOxotremorineGeneral MedicineAcetylcholineRatsAtropineEndocrinologyNicotinic agonistParasympathomimeticsCalciumFemaleAcetylcholinemedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Auditory cortical event-related potentials to pitch deviances in rats

1998

Abstract We recorded epidural event-related potentials (ERPs) from the auditory cortex in anesthetized rats when pitch-deviant tones were presented in a homogenous series of standard tones (oddball condition). Additionally, deviant tones were presented without standard tones (deviant-alone condition). ERPs to deviant tones in the oddball condition differed significantly from ERPs to standard tones at the latency range of 63–243 ms. On the other hand, ERPs to deviant tones in the deviant-alone condition did not differ from ERPs to standard tones until 196 ms from stimulus onset. The results suggest that oddball stimuli can be neurophysiologically discriminated in anesthetized rats. Furthermo…

Auditory Cortexmedicine.medical_specialtygenetic structuresGeneral NeuroscienceeducationMismatch negativityStimulus (physiology)AudiologyAuditory cortexbehavioral disciplines and activitiesRatsPitch DiscriminationElectrophysiologyEvent-related potentialmental disordersEvoked Potentials AuditorymedicineAnimalsRats WistarPsychologypsychological phenomena and processesNeuroscience Letters
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