Search results for "RB1"

showing 10 items of 81 documents

Early onset of polyglandular failure is associated with HLA-DRB1*03.

2008

ObjectivesPolyglandular failure or autoimmunity (PGA) involves at least two endocrine diseases. Several genes may play a role in its etiology. This study analyzed 1) whether HLA-DRB1, HLA-DQB1, and MHC class I chain-related gene A (MICA) polymorphisms are associated in PGA and 2) whether PGA patients display stronger associations with these immune genes than patients with monoglandular autoimmunity (MGA).DesignAssociation study.MethodsHLA-DRB1, HLA-DQB1, and MICA alleles were analyzed in 73 patients with PGA, 283 with MGA, and 206 healthy controls. The HLA-DRB1 and HLA-DQB1 polymorphisms were determined with PCR-amplified DNA being hybridized with PCR-sequence-specific oligonucleotide probe…

AdultMalemedicine.medical_specialtyAdolescentGenotypeEndocrinology Diabetes and MetabolismBiologymedicine.disease_causePolymerase Chain Reactionlaw.inventionAutoimmunityEndocrinologyGene FrequencylawInternal medicineGermanyHLA-DQ AntigensmedicineHLA-DQ beta-ChainsHumansGenetic Predisposition to DiseaseAlleleAge of OnsetChildPolyendocrinopathies AutoimmuneGeneHLA-DRB1Polymerase chain reactionAllelesPolymorphism GeneticHistocompatibility Antigens Class IGeneral MedicineHLA-DR AntigensMiddle AgedEndocrinologyGenetic markerMicrosatelliteFemaleAge of onsetHLA-DRB1 ChainsEuropean journal of endocrinology
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Evidences of +896 A/G TLR4 Polymorphism as an Indicative of Prevalence of Complications in T2DM Patients

2014

T2DM is today considered as world-wide health problem, with complications responsible of an enhanced mortality and morbidity. Thus, new strategies for its prevention and therapy are necessary. For this reason, the research interest has focused its attention on TLR4 and its polymorphisms, particularly the rs4986790. However, no conclusive findings have been reported until now about the role of this polymorphism in development of T2DM and its complications, even if a recent meta-analysis showed its T2DM association in Caucasians. In this study, we sought to evaluate the weight of rs4986790 polymorphism in the risk of the major T2DM complications, including 367 T2DM patients complicated for th…

AdultMalemedicine.medical_specialtyGenotypeArticle SubjectT2DM TLR4 +896A/G SNP T2DM complicationsImmunologyPolymorphism Single NucleotideLower limbGene FrequencyDiabetes mellitusInternal medicineGenotypelcsh:PathologymedicineHumansSettore MED/05 - Patologia ClinicaGenetic Predisposition to DiseaseAllele frequencyAgedAged 80 and overSettore MED/04 - Patologia Generalebusiness.industryConfoundingTLR4 POLYMORPHISMCell BiologyMiddle Agedmedicine.diseaseSurgeryToll-Like Receptor 4Cumulative riskDiabetes Mellitus Type 2FemaleComplicationbusinessResearch Articlelcsh:RB1-214
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Pulmonary artery sarcoma with angiosarcoma phenotype mimicking pleomorphic malignant fibrous histiocytoma: a case report

2012

Abstract Primary sarcomas of the major blood vessels can be classified based on location in relationship to the wall or by histologic type. Angiosarcomas are malignant neoplasms that arise from the endothelial lining of the blood vessels; those arising in the intimal compartment of pulmonary artery are rare. We report a case of pulmonary artery angiosarcoma in a 36-year old female with pulmonary masses. The patient had no other primary malignant neoplasm, thus excluding a metastatic lesion. Gross examination revealed a thickened right pulmonary artery and a necrotic and hemorrhagic tumor, filling and occluding the vascular lumen. The mass extended distally, within the pulmonary vasculature …

Adultmedicine.medical_specialtyPathologyHistologyBiopsyHemangiosarcomaCase ReportHistiocytoma Malignant FibrousPathology and Forensic MedicinePredictive Value of Testsmedicine.arteryVascular NeoplasmBiomarkers TumorAngiosarcomalcsh:PathologyMedicineHumansAngiosarcomaDiagnostic ErrorsPneumonectomyCell ProliferationPulmonary Artery AngiosarcomaLungbusiness.industrySarcomaGeneral Medicinemedicine.diseaseRight pulmonary arteryImmunohistochemistryVascular NeoplasmsPulmonary arteryHemangiosarcomamedicine.anatomical_structurePhenotypeTreatment OutcomeChemotherapy AdjuvantPulmonary arteryFemaleSarcomaRadiologybusinessTomography X-Ray Computedlcsh:RB1-214Diagnostic Pathology
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New insight into the genetics of age-related macular degeneration in connection with lipid metabolism

2010

Evaluation of: Zerbib J, Seddon JM, Richard F et al. rs5888 variant of SCARB1 gene is a possible susceptibility factor for age-related macular degeneration. PLoS ONE 4(10), e7341 (2009).Age-related macular degeneration (AMD) is the leading cause of visual loss in elderly populations of Western countries. According to demographic forecasts, and owing to the improvement in life expectancy, the number of people suffering from this pathology is expected to dramatically increase in the near future, thereby becoming a significant socioeconomic burden. There has been great progress in defining risk factors for AMD over the last few decades. Beyond advanced age, environmental and genetic factors ha…

Candidate genegenetic structures[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionBiomedical EngineeringPolymorphism (computer science)[SDV.IDA]Life Sciences [q-bio]/Food engineeringmedicine[SPI.GPROC]Engineering Sciences [physics]/Chemical and Process EngineeringLIPIDRisk factorGeneGeneticsbusiness.industryLipid metabolismAGE-RELATED MACULAR DEGENERATIONMacular degenerationmedicine.diseaseGENESCARB1eye diseasesPOLYMORPHISM3. Good healthOphthalmology[SDV.AEN] Life Sciences [q-bio]/Food and NutritionLife expectancyNUTRITIONsense organsbusiness[SDV.AEN]Life Sciences [q-bio]/Food and NutritionOptometry
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A Case of Follicular Tumor of Uncertain Malignant Potential (FT-UMP) with Glomeruloid Features Showing Capsular Mucinous Degeneration

2021

The most recent revision of the World Health Organization (WHO) Classification of Tumours of Endocrine Organs introduced a new variant of follicular thyroid carcinoma (FTC). It is characterized by a “glomeruloid” architectural pattern of growth. We present a case of follicular tumor with glomeruloid features, with Alcian Blue positive mucinous stromal degeneration in foci of questionable capsular microinvasion. At our knowledge, this the second case of glomeruloid follicular tumor in the literature and the first case in which Alcian Blue staining was used to investigate capsular invasion. Moreover, RAS mutation further supports that this is a variant of follicular tumor with uncertain malig…

Capsular InvasionPathologymedicine.medical_specialtyendocrine systemStromal cell030209 endocrinology & metabolismCase ReportSettore MED/08 - Anatomia PatologicaMucinous degenerationWorld healthThyroid carcinomaGlomeruloid Features03 medical and health sciences0302 clinical medicineFollicular phasemedicinePathologyEndocrine systemRB1-214Follicular Tumor of Uncertain Malignant Potential (FT-UMP)business.industryGeneral MedicineStainingSettore MED/18 - Chirurgia Generale030220 oncology & carcinogenesisbusinessCase Reports in Pathology
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The human gene for mannan-binding lectin-associated serine protease-2 (MASP-2), the effector component of the lectin route of complement activation, …

2001

The proteases of the lectin pathway of complement activation, MASP-1 and MASP-2, are encoded by two separate genes. The MASP1 gene is located on chromosome 3q27, the MASP2 gene on chromosome 1p36.23-31. The genes for the classical complement activation pathway proteases, C1r and C1s, are linked on chromosome 12p13. We have shown that the MASP2 gene encodes two gene products, the 76 kDa MASP-2 serine protease and a plasma protein of 19 kDa, termed MAp19 or sMAP. Both gene products are components of the lectin pathway activation complex. We present the complete primary structure of the human MASP2 gene and the tight cluster that this locus forms with non-complement genes. A comparison of the …

Chromosomes Artificial BacterialTranscription GeneticGenetic LinkageRNA SplicingImmunologyMolecular Sequence DataBiologyGeneticsHumansPromoter Regions GeneticComplement ActivationGenetics (clinical)Mannan-binding lectinGeneticsComplement component 2Base SequenceCD69Serine EndopeptidasesC4AChromosome MappingCollectinsKLRB1Chromosomes Human Pair 1Lectin pathwayMannose-Binding Protein-Associated Serine ProteasesMultigene Familybiology.proteinCarrier ProteinsMASP2MASP1
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A novel tunicate (Botryllus schlosseri) putative C-type lectin features an immunoglobulin domain.

1997

We have cloned a putative C-type lectin of Botryllus schlosseri [Ascidiacea], whose deduced protein of 333 amino acids features three building blocks: (i) a Greek-key motif signature at the amino-terminus, (ii) a C-type lectin domain signature, and (iii) an immunoglobulin (Ig) domain at the carboxyl terminus. This C-type lectin was termed BSCLT. Similarity searches revealed that the Ig domain in BSCLT, which is evidently not polymorphic, is best classified as an Intermediate-type Ig domain. Rabbit antibodies, raised against recombinant BSCLT, cross-reacted in a Western blot with a 38-kD polypeptide in tunicate crude extract. Presumably, this bimodal tunicate protein is the first description…

DNA ComplementaryMolecular Sequence DataImmunoglobulinsBotryllus schlosseriImmunoglobulin domainC-type lectinLectinsGeneticsAnimalsLectins C-TypeAmino Acid SequenceUrochordataMolecular Biologychemistry.chemical_classificationbiologyBase SequenceSequence Homology Amino AcidCD69LectinCell BiologyGeneral Medicinebiology.organism_classificationMolecular biologyAmino acidTunicateKLRB1chemistrybiology.proteinDNA and cell biology
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Class II HLA interactions modulate genetic risk for multiple sclerosis

2015

Association studies have greatly refined the understanding of how variation within the human leukocyte antigen (HLA) genes influences risk of multiple sclerosis. However, the extent to which major effects are modulated by interactions is poorly characterized. We analyzed high-density SNP data on 17,465 cases and 30,385 controls from 11 cohorts of European ancestry, in combination with imputation of classical HLA alleles, to build a high-resolution map of HLA genetic risk and assess the evidence for interactions involving classical HLA alleles. Among new and previously identified class II risk alleles (HLA-DRB1*15:01, HLA-DRB1*13:03, HLA-DRB1*03:01, HLA-DRB1*08:01 and HLA-DQB1*03:02) and cla…

Geneticsmusculoskeletal diseasesMultiple SclerosisHistocompatibility Antigens Class IISingle-nucleotide polymorphismGenome-wide association studyEpistasis GeneticHuman leukocyte antigenBiologyPolymorphism Single NucleotideArticleHistocompatibilityGenetic variationGeneticsHumansGenetic Predisposition to DiseaseAllele10. No inequalityHLA-DRB1AllelesGenetic association
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MHC class II genes influence the susceptibility to chronic active hepatitis C

1997

Chronic hepatitis C develops in more than 70% of hepatitis C virus infected subjects. Viral factors influence the disease course, but little is known about the importance of host factors.Frequencies of major histocompatibility complex (MHC) class I and class II antigens were analyzed in two groups of patients with chronic hepatitis C virus infection and in control subjects. MHC class I typing was done by standard microlymphocytotoxicity assays. DRB1 and DQA1 genotyping was done by PCR based typing methods.DRB1*0301 was found in 26 of 75 patients with chronic hepatitis C virus infection (34.7%) and in 12 of 101 control subjects (11.9%) (relative risk 3.9; p0.001). Homozygosity for this allel…

GenotypeHepatitis C virusGenes MHC Class IIBiologymedicine.disease_causePolymerase Chain ReactionHLA-DQ alpha-ChainsVirusMHC Class II GeneReference ValuesHLA-DQ AntigensMHC class ImedicineHumansGenetic Predisposition to DiseaseAllelesAntilymphocyte SerumHepatitis ChronicHepatitisMHC class IIHepatologyHistocompatibility Antigens Class IHomozygoteHistocompatibility Antigens Class IIHLA-DR AntigensHepatitis Cmedicine.diseaseHepatitis CVirologyHistocompatibilityImmunologyDisease Progressionbiology.proteinDisease SusceptibilityHLA-DRB1 ChainsJournal of Hepatology
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Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis.

2011

Multiple sclerosis is a common disease of the central nervous system in which the interplay between inflammatory and neurodegenerative processes typically results in intermittent neurological disturbance followed by progressive accumulation of disability. Epidemiological studies have shown that genetic factors are primarily responsible for the substantially increased frequency of the disease seen in the relatives of affected individuals, and systematic attempts to identify linkage in multiplex families have confirmed that variation within the major histocompatibility complex (MHC) exerts the greatest individual effect on risk. Modestly powered genome-wide association studies (GWAS) have ena…

Immunity Cellular/geneticsCellular immunityMultiple SclerosisGenome-wide association studyCLEC16ABiologyPolymorphism Single NucleotideCell Differentiation/immunologyEurope/ethnologyMajor Histocompatibility Complex/geneticsMajor Histocompatibility Complex03 medical and health sciences0302 clinical medicinemedicineGenetic predispositionHumansGenetic Predisposition to DiseaseHLA-A Antigens/geneticsAlleles030304 developmental biologyGenetic associationGenetics0303 health sciencesImmunity CellularMultidisciplinaryHLA-A AntigensGenome HumanMultiple sclerosisGenetic Predisposition to Disease/geneticsHLA-DR Antigens/geneticsLymphocyte differentiationCell DifferentiationHLA-DR AntigensT-Lymphocytes Helper-InducerRC346medicine.diseasePolymorphism Single Nucleotide/geneticsGenetic architecture3. Good healthEuropeSample SizeImmunologyGenome Human/geneticsMultiple Sclerosis/genetics030217 neurology & neurosurgeryT-Lymphocytes Helper-Inducer/cytologyGenome-Wide Association StudyHLA-DRB1 Chains
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