Search results for "REACTIVE OXYGEN SPECIES"

showing 10 items of 879 documents

Wine Consumption and Oral Cavity Cancer: Friend or Foe, Two Faces of Janus

2020

The health benefits of moderate wine consumption have been extensively studied during the last few decades. Some studies have demonstrated protective associations between moderate drinking and several diseases including oral cavity cancer (OCC). However, due to the various adverse effects related to ethanol content, the recommendation of moderate wine consumption has been controversial. The polyphenolic components of wine contribute to its beneficial effects with different biological pathways, including antioxidant, lipid regulating and anti-inflammatory effects. On the other hand, in the oral cavity, ethanol is oxidized to form acetaldehyde, a metabolite with genotoxic properties. This rev…

Antioxidantmedicine.medical_treatmentAnti-Inflammatory AgentsPharmaceutical ScienceReviewresveratrolResveratrolOral cavityAntioxidantsAnalytical Chemistrychemistry.chemical_compound0302 clinical medicineRisk FactorsDrug DiscoveryFood science0303 health sciencesfood and beveragesLipidsReactive Nitrogen SpeciesChemistry (miscellaneous)030220 oncology & carcinogenesisMolecular MedicineMouth NeoplasmscarcinogenesisAlcohol Drinkinglcsh:QD241-44103 medical and health scienceslcsh:Organic chemistrymedicineAnimalsHumanswinePhysical and Theoretical Chemistry030304 developmental biologyConsumption (economics)Winebusiness.industryOrganic Chemistryoral cavity cancerAcetaldehydePolyphenolsCancerDNAmedicine.diseasechemistryEthanol contentethanolReactive Oxygen SpeciesbusinessMutagensacetaldehydeMolecules
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Direct and indirect antioxidant properties of α-lipoic acid and therapeutic potential.

2012

International audience; Diabetes has emerged as a major threat to worldwide health. The exact mechanisms underlying the disease are unknown; however, there is growing evidence that the excess generation of reactive oxygen species (ROS) associated with hyperglycemia, causes oxidative stress in a variety of tissues. In this context, various natural compounds with pleiotropic actions like lipoic acid (LA) are of interest, especially in metabolic diseases such as diabetes. LA, either as a dietary supplement or a therapeutic agent, modulates redox potential because of its ability to match the redox status between different subcellular compartments as well as extracellularly. Both the oxidized (d…

Antioxidantmedicine.medical_treatmentContext (language use)InflammationBiologymedicine.disease_causeAntioxidants03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemDiabetes mellitusmedicineDiabetes MellitusAnimalsHumans030304 developmental biologyChelating Agentschemistry.chemical_classificationInflammation0303 health sciencesReactive oxygen speciesThioctic AcidEndothelial CellsMetabolismmedicine.disease3. Good health[SDV.MHEP.CSC] Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemMitochondriaLipoic acidOxidative StresschemistryBiochemistryCardiovascular Diseases030220 oncology & carcinogenesisHyperglycemiaDietary Supplementsmedicine.symptomReactive Oxygen SpeciesOxidation-ReductionOxidative stressFood ScienceBiotechnologyMolecular nutritionfood research
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Ursolic acid ameliorates stress and reactive oxygen species in C. elegans knockout mutants by the dopamine Dop1 and Dop3 receptors.

2020

Abstract Background Depression and stress-related disorders are leading causes of death worldwide. Standard treatments elevating serotonin or noradrenaline levels are not sufficiently effective and cause adverse side effects. A connection between dopamine pathways and stress-related disorders has been suggested. Compounds derived from herbal medicine could be a promising alternative. We examined the neuroprotective effects of ursolic acid (UA) by focusing on dopamine signalling. Methods Trolox equivalent capacity assay was used to determine the antioxidant activities of UA in vitro. C. elegans N2 wildtype and dopamine receptor-knockout mutants (dop-1-deficient RB665 and dop-3-deficient LX70…

Antioxidantmedicine.medical_treatmentDopamineLongevityPharmaceutical SciencePharmacologyNeuroprotectionAntioxidants03 medical and health scienceschemistry.chemical_compoundGene Knockout Techniques0302 clinical medicineDopamineStress PhysiologicalDrug DiscoverymedicineAnimalsHumansReceptorCaenorhabditis elegansCaenorhabditis elegans Proteins030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesChemistryReceptors Dopamine D2Receptors Dopamine D1Receptors Dopamine D3TriterpenesMolecular Docking SimulationComplementary and alternative medicineDopamine receptor030220 oncology & carcinogenesisMutationMolecular MedicineSerotoninTroloxReactive Oxygen Speciesmedicine.drugSignal TransductionPhytomedicine : international journal of phytotherapy and phytopharmacology
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Melatonin protects against ischemia-reperfusion injury and inhibits apoptosis in isolated working rat heart.

2003

INTRODUCTION: Melatonin (MEL), a pineal hormone, is well known as a potent antioxidant in a variety of ischemia-reperfusion models. Recent studies have assumed a pivotal role of reactive oxygen species (ROS) in the development of apoptosis. There are few pieces of information concerning a possible protective role of MEL against apoptosis in ischemia-reperfusion injury of myocardium. METHODS: We conducted an in vitro experiment: (1) to study the effect of MEL in the model of isolated and perfused working rat heart; (2) to evaluate the antioxidant capacity of MEL by a simple fluorescence test; and (3) to analyze the extent of apoptosis inhibition by MEL. Four groups of male Wistar rat were us…

Antioxidantmedicine.medical_treatmentIschemiaPharmacologymedicine.disease_causePathology and Forensic MedicineMelatonin03 medical and health sciences0302 clinical medicinehemic and lymphatic diseasesPhysiology (medical)medicineneoplasms030304 developmental biologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesTUNEL assaymedicine.disease3. Good healthchemistryApoptosisAnesthesiaReperfusion injury030217 neurology & neurosurgeryOxidative stressmedicine.drugPathophysiology : the official journal of the International Society for Pathophysiology
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The triterpenoid ursolic acid ameliorates stress in Caenorhabditis elegans by affecting the depression-associated genes skn-1 and prdx2.

2021

Abstract Introduction Depression is one of the leading causes of death worldwide. Lower antioxidant concentrations and increased oxidative stress levels contribute to the development of depression. Effective and tolerable medications are urgently needed. Nrf2 and PRDX2 are promising targets in the treatment of oxidative stress and, therefore, promising for the development of novel antidepressants. Ursolic acid (UA), a natural triterpenoid found in various plants is known to exert neuroprotective and antioxidant effects. Skn-1 (which corresponds to human Nrf2) and prdx2 deficient mutants of the nematode Caenorhabditis elegans are suitable models to study the effect of UA on these targets. Ad…

Antioxidantmedicine.medical_treatmentPharmaceutical SciencePharmacologymedicine.disease_causeProtective AgentsNeuroprotectionAntioxidants03 medical and health scienceschemistry.chemical_compound0302 clinical medicineUrsolic acidStress PhysiologicalDrug DiscoveryAdaptogenmedicineAnimalsCaenorhabditis elegansCaenorhabditis elegans ProteinsCaenorhabditis elegans030304 developmental biologyPharmacologychemistry.chemical_classification0303 health sciencesReactive oxygen speciesbiologyDepressionPeroxiredoxinsbiology.organism_classificationAntidepressive AgentsTriterpenesDNA-Binding ProteinsMolecular Docking SimulationOxidative StressComplementary and alternative medicinechemistryGene Expression Regulation030220 oncology & carcinogenesisMutationMolecular MedicineReactive Oxygen SpeciesJugloneOxidative stressTranscription FactorsPhytomedicine : international journal of phytotherapy and phytopharmacology
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Proteomic Analysis of Saccharomyces cerevisiae Response to Oxidative Stress Mediated by Cocoa Polyphenols Extract

2020

The present study addressed the protective effects against oxidative stress (OS) of a cocoa powder extract (CPEX) on the protein expression profile of S. cerevisiae. A proteomic analysis was performed after culture preincubation with CPEX either without stress (&minus

Antioxidantmedicine.medical_treatmentSaccharomyces cerevisiaePharmaceutical Scienceantioxidant activitySaccharomyces cerevisiaeamino acid metabolismmedicine.disease_causeAmino acid metabolismAnalytical Chemistrycocoa polyphenolslcsh:QD241-441<i>Saccharomyces cerevisiae</i>03 medical and health sciencesHistone H3chemistry.chemical_compoundBiosynthesisAntioxidant activitylcsh:Organic chemistryprotein identificationDrug DiscoverymedicineDeletion mutantsoxidative stressPhysical and Theoretical ChemistryReactive oxygen species metabolic process030304 developmental biologychemistry.chemical_classification0303 health sciencesbiologyCocoa polyphenolsChemistry030302 biochemistry & molecular biologyOrganic Chemistrybiology.organism_classificationYeastAmino acidBiochemistryChemistry (miscellaneous)Oxidative stressMolecular MedicineProtein identificationdeletion mutantsOxidative stressMolecules
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Impact of Hydrogen Peroxide on Protein Synthesis in Yeast.

2021

This article belongs to the Special Issue Thiol-Based Redox Regulation of Cellular and Organismal Function.

Antioxidantprotein synthesisPhysiologymedicine.medical_treatmentClinical Biochemistryhydrogen peroxideReviewRM1-950Mitochondrionmedicine.disease_causeBiochemistryCysteine thiolscysteine thiolschemistry.chemical_compoundmedicineProtein biosynthesisHydrogen peroxideMolecular Biologychemistry.chemical_classificationReactive oxygen speciesTranslation (biology)Cell BiologyHydrogen peroxideSignalingCell biologychemistryTherapeutics. PharmacologyProtein synthesissignalingOxidative stressIntracellularAntioxidants (Basel, Switzerland)
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Cytoprotective effect of resveratrol diastereomers in CHO-K1 cells exposed to beauvericin

2015

Beauvericin (BEA) causes cytotoxicity, lipid peroxidation and reactive oxygen species in CHO-K1 cells. Resveratrol (RSV) is a polyphenol with multiple biological properties, including antioxidant effects. RSV has two forms: trans and cis. The aims of this study were to determine the cytoprotective effect of trans-RSV and diastereomers mixtures (50:50 trans/cis-RSV and 70:30 trans/cis-RSV) incubated alone and in combination with BEA in ovarian (CHO-K1) cells. The results demonstrated that cell viability increases (from 9% to 77%) when they were exposed to low concentration of RSV. Moreover, when the cells were pre-treated with RSV and then exposed to BEA, a cytoprotective effect (from 25% to…

Antioxidantvirusesmedicine.medical_treatmentCHO CellsResveratrolToxicologymedicine.disease_causeLipid peroxidationchemistry.chemical_compoundCricetulusCricetinaeDepsipeptidesStilbenesmedicineAnimalsViability assayCytotoxicitychemistry.chemical_classificationReactive oxygen speciesChemistryvirus diseasesGeneral Medicinerespiratory systemMolecular biologyBeauvericinBiochemistryCytoprotectionResveratrolLipid PeroxidationReactive Oxygen SpeciesOxidative stressFood ScienceFood and Chemical Toxicology
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Deficiency of glutathione peroxidase-1 accelerates the progression of atherosclerosis in apolipoprotein E-deficient mice.

2007

Background— We have recently demonstrated that activity of red blood cell glutathione peroxidase-1 is inversely associated with the risk of cardiovascular events in patients with coronary artery disease. The present study analyzed the effect of glutathione peroxidase-1 deficiency on atherogenesis in the apolipoprotein E-deficient mouse. Methods and Results— Female apolipoprotein E-deficient mice with and without glutathione peroxidase-1 deficiency were placed on a Western-type diet for another 6, 12, or 24 weeks. After 24 weeks on Western-type diet, double-knockout mice (GPx-1 −/− ApoE −/− ) developed significantly more atherosclerosis than control apolipoprotein E-deficient mice. Moreover…

Apolipoprotein Emedicine.medical_specialtyGPX1AntioxidantApolipoprotein Bmedicine.medical_treatmentLipoproteinsApoptosisBlood Pressuremedicine.disease_causeNitric OxideMitochondria HeartMonocyteschemistry.chemical_compoundMiceApolipoproteins EGlutathione Peroxidase GPX1SuperoxidesInternal medicinePeroxynitrous AcidmedicineAnimalsAortaCell Proliferationchemistry.chemical_classificationMice KnockoutReactive oxygen speciesGlutathione PeroxidaseMembranesbiologyGlutathione peroxidaseGlutathioneAtherosclerosisEndocrinologyPhenotypechemistryImmunologybiology.proteinDisease ProgressionFemaleCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidation-ReductionOxidative stressArteriosclerosis, thrombosis, and vascular biology
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Superoxide Flux in Endothelial Cells via the Chloride Channel-3 Mediates Intracellular Signaling

2007

Reactive oxygen species (ROS) have been implicated in both cell signaling and pathology. A major source of ROS in endothelial cells is NADPH oxidase, which generates superoxide (O2.−) on the extracellular side of the plasma membrane but can result in intracellular signaling. To study possible transmembrane flux of O2.−, pulmonary microvascular endothelial cells were preloaded with the O2.−-sensitive fluorophore hydroethidine (HE). Application of an extracellular bolus of O2.−resulted in rapid and concentration-dependent transient HE oxidation that was followed by a progressive and nonreversible increase in nuclear HE fluorescence. These fluorescence changes were inhibited by superoxide dism…

ApoptosisMembrane PotentialsSuperoxide dismutasechemistry.chemical_compoundChloride ChannelsSuperoxidesExtracellularAnimalsHumansEnzyme InhibitorsRNA Small InterferingMolecular BiologyLungCells CulturedFluorescent Dyeschemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxideAngiotensin IIThrombinAcetophenonesEndothelial CellsNADPH OxidasesCell BiologyArticlesCell biologyMitochondriaPhenanthridinesOxygenchemistryDIDSbiology.proteinCalciumSignal transductionOxidation-ReductionIntracellularSignal Transduction
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