Search results for "RETINAL PIGMENT"

showing 10 items of 68 documents

Human brain organoids assemble functionally integrated bilateral optic vesicles

2021

During embryogenesis, optic vesicles develop from the diencephalon via a multistep process of organogenesis. Using induced pluripotent stem cell (iPSC)-derived human brain organoids, we attempted to simplify the complexities and demonstrate formation of forebrain-associated bilateral optic vesicles, cellular diversity, and functionality. Around day 30, brain organoids attempt to assemble optic vesicles, which develop progressively as visible structures within 60 days. These optic vesicle-containing brain organoids (OVB-organoids) constitute a developing optic vesicle's cellular components, including primitive corneal epithelial and lens-like cells, retinal pigment epithelia, retinal progeni…

OrganogenesisInduced Pluripotent Stem Cellsretinal pigment epitheliumiPSCsEmbryonic DevelopmentBiology03 medical and health sciencesDiencephalonchemistry.chemical_compoundProsencephalon0302 clinical medicineGeneticsOrganoidmedicineHumansInduced pluripotent stem cell030304 developmental biology0303 health sciencesforebrain organoidsRetinal pigment epitheliumbrain organoidsVesicleprimordial eye fieldsOVB-organoidsCell DifferentiationRetinalCell BiologyOptic vesicleHuman brainCell biologyOrganoidsmedicine.anatomical_structurenervous systemchemistryMolecular MedicineFOXG1; OVB-organoids; brain organoids; forebrain organoids; iPSCs; optic vesicles; primary cilium; primordial eye fields; retinal pigment epitheliumoptic vesiclesFOXG1030217 neurology & neurosurgeryprimary ciliumCell Stem Cell
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C21orf2 is mutated in recessive early-onset retinal dystrophy with macular staphyloma and encodes a protein that localises to the photoreceptor prima…

2015

Background/aim We have noted a phenotype of early-onset retinal dystrophy with macular staphyloma but without high myopia. The aim of this study is to report the underlying genetic mutations and the subcellular localisation of the gene product in the retina. Methods Retrospective case series (2012–2015); immunohistochemical analyses of mammalian retina for in situ protein localisation. Results All three probands were first noted to have decreased vision at 3–6 years old which worsened over time. At ages 39, 37 and 12 years old, all had similar retinal findings: dystrophic changes (retinal pigment epithelium mottling, vessel narrowing), macular staphyloma (despite only mild myopia or high hy…

Pathologygenetic structuresSus scrofaPolymerase Chain ReactionPhotoreceptor cellchemistry.chemical_compoundConsanguinityMiceChildFrameshift MutationGeneticsmedicine.diagnostic_testMagnetic Resonance ImagingSensory SystemsTissue DonorsPedigreemedicine.anatomical_structureFemaleRetinal DystrophiesTomography Optical CoherenceDilatation PathologicAdultmedicine.medical_specialtyBlotting WesternMolecular Sequence DataMutation MissenseGenes RecessiveBiologyRetinaCellular and Molecular NeuroscienceRetinal DystrophiesmedicineElectroretinographyAnimalsHumansAmino Acid SequencePhotoreceptor Connecting CiliumRetrospective StudiesRetinaRetinal pigment epitheliumDystrophyProteinsRetinalmedicine.diseaseeye diseasesOphthalmologyCiliopathyCytoskeletal Proteinschemistrysense organsElectroretinographyThe British journal of ophthalmology
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Patterns of cytokeratin and vimentin expression in the human eye

1988

We studied the expression of the various cytokeratin (CK) polypeptides and vimentin in tissues of the human eye by applying immunocytochemical procedures using a panel of monoclonal antibodies as well as by performing biochemical analyses of microdissected tissues. Adult corneal epithelium was found to contain significant amounts of the cornea-specific CKs nos. 3 and 12 as well as CK no. 5, and several additional minor CK components. Among these last CKs, no. 19 was found to exhibit an irregular mosaic-like staining pattern in the peripheral zone of the corneal epithelium, while having a predominantly basal distribution in the limbal epithelium. Both the fetal corneal epithelium and the con…

Pathologymedicine.medical_specialtyHistologyVimentinEyeCorneaCytokeratinFetusmedicineHumansVimentinPigment Epithelium of EyeMolecular BiologyCorneal epitheliumRetinal pigment epitheliumbiologyCiliary BodyCell BiologyGeneral MedicineImmunohistochemistryeye diseasesEpitheliumStainingMedical Laboratory Technologymedicine.anatomical_structurebiology.proteinKeratinsImmunohistochemistrysense organsAnatomyStem cellGeneral Agricultural and Biological SciencesConjunctivaHistochemistry
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Sodium channels enable fast electrical signaling and regulate phagocytosis in the retinal pigment epithelium

2019

Background Voltage-gated sodium (Nav) channels have traditionally been considered a trademark of excitable cells. However, recent studies have shown the presence of Nav channels in several non-excitable cells, such as astrocytes and macrophages, demonstrating that the roles of these channels are more diverse than was previously thought. Despite the earlier discoveries, the presence of Nav channel-mediated currents in the cells of retinal pigment epithelium (RPE) has been dismissed as a cell culture artifact. We challenge this notion by investigating the presence and possible role of Nav channels in RPE both ex vivo and in vitro. Results Our work demonstrates that several subtypes of Nav cha…

PhotoreceptorsPatch-Clamp TechniquesHuman Embryonic Stem CellsfagosytoosiRetinal Pigment EpitheliumSodium ChannelsRetinaBiokemia solu- ja molekyylibiologia - Biochemistry cell and molecular biologyMicePhagocytosisGenetiikka kehitysbiologia fysiologia - Genetics developmental biology physiologyAnimalsHumans3125 Otorhinolaryngology ophthalmologylcsh:QH301-705.5soluviestintä1184 Genetics developmental biology physiology3112 Neurosciences217 Medical engineeringaistinreseptoritMice Inbred C57BLlcsh:Biology (General)Na-vIon channelsproteiinitRPEPatch clampverkkokalvoNeurotieteet - NeurosciencesNavSignal TransductionResearch Article
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Serous Detachment of the Retina: A Complication of Branch Retinal Vein Stenosis

2005

The author reports a case of serous detachment of the retina, which has developed in an eye with veno-venous collaterals, secondary to stenosis of a branch of the central retinal vein near the optic disc. This complication has not previously been described in this retinal vascular obstructive disease.

RetinaCentral retinal veinRetinal pigment epitheliumRetinal Veingenetic structuresmedicine.diagnostic_testbusiness.industryAnatomymedicine.diseaseFluorescein angiographyeye diseasesOphthalmologySerous fluidmedicine.anatomical_structuremedicineBranch retinal vein occlusionsense organsbusinessOptic discAnnals Of Ophthalmology
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How Does the Eye Breathe?

2003

Visual performance of the vertebrate eye requires large amounts of oxygen, and thus the retina is one of the highest oxygen-consuming tissues of the body. Here we show that neuroglobin, a neuron-specific respiratory protein distantly related to hemoglobin and myoglobin, is present at high amounts in the mouse retina (approximately 100 microm). The estimated concentration of neuroglobin in the retina is thus about 100-fold higher than in the brain and is in the same range as that of myoglobin in the muscle. Neuroglobin is expressed in all neurons of the retina but not in the retinal pigment epithelium. Neuroglobin mRNA was detected in the perikarya of the nuclear and ganglion layers of the n…

RetinaRetinal pigment epitheliumgenetic structuresSkeletal muscleRetinalCell BiologyAnatomyBiologyBiochemistryeye diseasesCell biologyRespiratory proteinchemistry.chemical_compoundmedicine.anatomical_structureMyoglobinchemistryNeuroglobinmedicinesense organsMolecular BiologyPhotoreceptor inner segmentJournal of Biological Chemistry
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A novel murine model of aging of the human retina

2008

Purpose Accumulation of lipids, and especially of cholesteryl esters, under the retinal pigment epithelium and within Bruch’s membrane is a normal feature of aging and has also been observed in human eyes with age-related maculopathy. Our objective was to evaluate the retinal phenotype of apoB100,LDLR-/- mice, a model for lipid metabolism dysfunction and potentially of aging of the retina. Methods ApoB100,LDLR-/- mice were studied at 7 and 14 months of age by standard scotopic and photopic electroretinography by comparison to control animals. Fundus images were obtained with a confocal SLO (Heidelberg Retina Angiograph). The integrity of the vascular system was investigated by means of fluo…

Retinamedicine.medical_specialtyRetinal pigment epitheliumgenetic structuresLipid metabolismRetinalGeneral MedicineAnatomyFundus (eye)Biologymedicine.diseaseeye diseasesOphthalmologychemistry.chemical_compoundmedicine.anatomical_structurechemistryOphthalmologymedicineMaculopathylipids (amino acids peptides and proteins)sense organsScotopic visionPhotopic visionActa Ophthalmologica
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OPTICAL COHERENCE TOMOGRAPHIC FINDINGS IN BIETTI’S CRYSTALLINE TAPETORETINAL DYSTROPHY

2014

Purpose: To report the optical coherence tomography (OCT) findings of Bietti crystalline tapetoretinal dystrophy. Design: Observational case report. Methods: A subject with Bietti crystalline tapetoretinal dystrophy was evaluated with ophthalmoscopy, fluorescein angiography, and OCT (Stratus). Results: OCT showed thinning and hyporeflectivity of the outer nuclear layer in the macula due to photoreceptor degeneration. The retinal areas showing atrophy of the retinal pigment epithelium displayed greater penetration of the optical beam into the choroid and visualization of residual choroidal vessels. The hyperreflective band normally seen under the neurosensory retina was extremely wide when a…

Retinamedicine.medical_specialtyRetinal pigment epitheliumgenetic structuresmedicine.diagnostic_testbusiness.industryRetinalGeneral MedicineFluorescein angiographyeye diseasesOphthalmoscopyOphthalmologychemistry.chemical_compoundmedicine.anatomical_structureOptical coherence tomographychemistryOphthalmologymedicinesense organsChoroidbusinessOuter nuclear layerRETINAL Cases & Brief Reports
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Disruption of the retinitis pigmentosa 28 gene Fam161a in mice affects photoreceptor ciliary structure and leads to progressive retinal degeneration.

2014

Mutations in the FAM161A gene were previously identified as the cause for autosomal-recessive retinitis pigmentosa 28. To study the effects of Fam161a dysfunction in vivo, we generated gene-trapped Fam161a(GT/GT) mice with a disruption of its C-terminal domain essential for protein-protein interactions. We confirmed the absence of the full-length Fam161a protein in the retina of Fam161a(GT/GT) mice using western blots and showed weak expression of a truncated Fam161a protein by immunohistochemistry. Histological analyses demonstrated that photoreceptor segments were disorganized in young Fam161a(GT/GT) mice and that the outer retina was completely lost at 6 months of age. Reactive microglia…

Retinal degenerationMaleOpsinGenotypeVision DisordersAction PotentialsGene ExpressionMice TransgenicRetinal Pigment EpitheliumBiologyRetinaMiceRetinitis pigmentosaGeneticsmedicineAnimalsHumansPhotoreceptor CellsPeripherin 2Eye ProteinsMolecular BiologyGenetics (clinical)Retinal regenerationRetinaGene therapy of the human retinaCiliumRetinal DegenerationGeneral Medicinemedicine.diseaseeye diseasesCell biologyProtein Transportmedicine.anatomical_structureGenetic LociGene TargetingMutationFemalesense organsMicrogliaCarrier ProteinsProtein BindingHuman molecular genetics
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PRCD is concentrated at the base of photoreceptor outer segments and is involved in outer segment disc formation.

2019

Abstract Mutations of the PRCD gene are associated with rod-cone degeneration in both dogs and humans. Prcd is expressed in the mouse eye as early as embryonic day 14. In the adult mouse retina PRCD is expressed in the outer segments of both rod and cone photoreceptors. Immunoelectron microscopy revealed that PRCD is located at the outer segment rim, and that it is highly concentrated at the base of the outer segment. Prcd-knockout mice present with progressive retinal degeneration, starting at 20 weeks of age and onwards. This process is reflected by a significant and progressive reduction of both scotopic and photopic electroretinographic responses, and by thinning of the retina, and spec…

Retinal degenerationMalegenetic structuresImmunoelectron microscopyRetinal Pigment EpitheliumBiologyRetinachemistry.chemical_compoundMicePhagocytosisGeneticsmedicineAnimalsScotopic visionOuter nuclear layerEye ProteinsMolecular BiologyGenetics (clinical)Mice KnockoutRetinaRetinal DegenerationMembrane ProteinsRetinalGeneral Medicinemedicine.diseaseRod Cell Outer SegmentPhotoreceptor outer segmenteye diseasesCell biologyMice Inbred C57BLmedicine.anatomical_structurechemistryRetinal Cone Photoreceptor CellsFemalesense organsCone-Rod DystrophiesRetinitis PigmentosaPhotopic visionSignal TransductionHuman molecular genetics
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