Search results for "Rabbits"

showing 10 items of 501 documents

Nitric oxide, via activation of guanylyl cyclase, suppresses alpha2-adrenoceptor-mediated 5-hydroxytryptamine release from neuroendocrine epithelial …

1998

Isolated tracheae of newborn rabbits were incubated in vitro and the outflow of 5-hydroxytryptamine (5-HT) was determined by HPLC with electrochemical detection. Evidence has previously been provided that this 5-HT outflow derives from neuroendocrine epithelial (NEE) cells of the airway mucosa. Phenylephrine, at a maximally effective concentration of 10 microM, caused a transient increase in 5-HT outflow by about 250%, an effect mediated by alpha2B-adrenoceptors, as previously shown. The phenylephrine-induced 5-HT release remained unchanged in calcium-free medium, but was reduced by 75% when the tracheae were incubated in calcium-free medium which contained 0.5 mM EDTA, a treatment known to…

Malemedicine.medical_specialtySerotoninchemistry.chemical_elementCalciumNitric OxideCalcium in biologyEpitheliumNitric oxidechemistry.chemical_compoundPhenylephrineReceptors Adrenergic alpha-2Internal medicinemedicineAnimalsPhenylephrinePharmacologySnapGeneral MedicineEnzyme ActivationTracheaEndocrinologychemistryAnimals NewbornGuanylate CyclaseBiophysicsLiberationFemaleRabbitsSoluble guanylyl cyclaseIntracellularmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Morphological and biochemical changes in striated muscle after experimental tourniquet ischaemia.

1979

Histological and biochemical changes were studied in the striated muscle following total tourniquet ischaemia between one and four h, the reflow time being 30 min and 24 h. Electronmicroscopy was applied to study the fine structure of the muscle after 24 h reflow. In light microscopy ischaemic changes were not seen even when the tourniquet time was extended to four h. When a four-h ischaemia was followed by a 24-h recovery period, the electron microscopy showed a variety of minor mitochondrial changes such as condensed and slightly dilatated mitochondria. The SDH activities did not vary significantly between the experimental and control samples even after a four-h ischaemia followed by 30 m…

Malemedicine.medical_specialtySuccinic dehydrogenaseTime FactorsIschemiaRecovery periodPneumatic tourniquetIschemiaInternal medicinemedicineAnimalscardiovascular diseasesTourniquetbiologyReflow timeL-Lactate DehydrogenaseChemistrySuccinate dehydrogenaseMusclesGeneral MedicineAnatomyTourniquetsmedicine.diseaseMitochondria MuscleSuccinate DehydrogenaseEndocrinologyTourniquet timebiology.proteinFemaleRabbitsResearch in experimental medicine. Zeitschrift fur die gesamte experimentelle Medizin einschliesslich experimenteller Chirurgie
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Sonography and MRI of experimental muscle injuries

1997

After sonographical examination with a 7.5-MHz linear array scanner, we created an experimental muscle injury of known sitze and location on 28 New Zealand white rabbits by stabbing them with a scalpel in the supraspinatus muscle. The changes in the healing process were followed and documented by sonography and magnetic resonance imaging (MRI) before and 2, 5, 11, 14, 36 and 64 days after injury. The changes in sonography and MRI followed a regular course. Ultrasound revealed an echo-poor area after injury with ever increasing echogenicity from the 14th day. Strong reflexes were found after 2 months. MRI showed few changes, only a slight increase of signal intensity, but a characteristic cu…

Malemedicine.medical_specialtySupraspinatus muscleScarsWounds StabHematomamedicineAnimalsOrthopedics and Sports MedicineMuscle SkeletalUltrasonographyBackWound Healingmedicine.diagnostic_testbusiness.industryUltrasoundEchogenicityMagnetic resonance imagingGeneral MedicineAnatomymedicine.diseaseMagnetic Resonance ImagingOrthopedic surgerySurgeryHistopathologyRabbitsRadiologymedicine.symptombusinessArchives of Orthopaedic and Trauma Surgery
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The effect of dopamine on the overflow of endogenous noradrenaline from the perfused rabbit heart evoked by sympathetic nerve stimulation.

1978

1. The effects of dopamine and two dopamine receptor antagonists (pimozide, flupenthixol) on the release of endogenous noradrenaline evoked by electrical stimulation of the postganglionic sympathetic nerves and their influence on cardiac performance were measured in isolated perfused rabbit hearts. 2. Dopamine 0.2μM decreased noradrenaline overflow and ventricular tension development in response to nerve stimulation. 3. Dopamine 2μM increased spontaneous noradrenaline output and tension development. The noradrenaline overflow in response to nerve stimulation was greatly enhanced. This action was only partly reversed by wash out of exogenous dopamine, indicating de novo synthesis and facilit…

Malemedicine.medical_specialtySympathetic Nervous SystemDopamineOxymetazolineStimulationEndogenyFlupenthixolIn Vitro TechniquesNorepinephrinePimozideCocaineDopamineInternal medicinemedicineAnimalsPharmacologyChemistryRabbit heartMyocardiumHeartGeneral MedicineElectric StimulationFlupenthixolEndocrinologyDopamine receptorcardiovascular systemDopamine AntagonistsFemaleRabbitsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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The effects of methacholine and calcium deprivation on the release of the false transmitter, ?-methyladrenaline, from the isolated rabbit heart

1976

1. Anaesthetized rabbits were infused for 20 min with 85 μg · kg−1 · min−1 (±)-α-methyladrenaline. The hearts dissected 15 min after the infusion contained 1.49 μg/g α-methyladrenaline; the endogenous noradrenaline content was correspondingly decreased. 2. Hearts from α-methyladrenaline-infused animals were isolated with the right sympathetic nerves intact and perfused. Ventricular rate, right atrial and right ventricular tensions were recorded using the transverse method. 3. Electrical stimulation (10 Hz, 1 ms, 1 min) of sympathetic nerves, perfusion with the nicotinic drug, p-aminophenethyltrimethylammonium (PAPETA) or perfusion with 54 mM KCl (high K+) solution evoked an output of both α…

Malemedicine.medical_specialtySympathetic Nervous SystemEpinephrineTyraminechemistry.chemical_elementStimulationCalciumPotassium ChlorideNorepinephrinechemistry.chemical_compoundInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsPharmacologyAniline CompoundsChemistryMyocardiumHeartGeneral MedicineTyramineGanglionic StimulantsElectric StimulationQuaternary Ammonium CompoundsAtropineEndocrinologySympatholyticsLiberationCalciumFemaleMethacholineRabbitsPerfusionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Impulse interval-dependent effect of sympathetic nerve stimulation on evoked acetylcholine release from the rabbit perfused atria preparation.

1995

The aim of the present study was to explore possible prejunctional effects mediated by impulse activity of sympathetic terminals on evoked acetylcholine release in an organ innervated by the autonomic ground plexus. Rabbit atria were isolated with the extrinsic right vagus and sympathetic nerves intact and perfused with Tyrode solution. Acetylcholine overflow was determined after labelling of the transmitter stores with [14C]choline and fractionation of the radioactivity on cation exchange columns. The overflow of endogenous noradrenaline was measured by HPLC and electrochemical detection. The vagus nerve was stimulated at 2 Hz for 3 min four times at intervals of 10 min. During the second …

Malemedicine.medical_specialtySympathetic Nervous SystemSympathetic nerveStimulationEndogenyElectrochemical detectionImpulse (physics)In Vitro TechniquesReceptors PresynapticCholinechemistry.chemical_compoundInternal medicinemedicineElectrochemistryCholineAnimalsChromatography High Pressure LiquidPharmacologyPlexusGanglia SympatheticChemistryMyocardiumHeartVagus NerveGeneral MedicineMyocardial ContractionAcetylcholineElectric StimulationPerfusionEndocrinologycardiovascular systemFemaleRabbitsAcetylcholinemedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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The effect of methacholine on noradrenaline release from the rabbit heart perfused with indometacin

1974

The experiments were undertaken in order to study the effect of inhibition of prostaglandin synthesis on the muscarinic inhibition of noradrenaline release evoked by sympathetic nerve stimulation. The right sympathetic nerves of the perfused rabbit heart were stimulated electrically. The noradrenaline output was enhanced after perfusion of the hearts with indometacin 3×10−5 M indicating blockade of the prostaglandin-mediated negative feedback control. Both in the presence and in the absence of indometacin methacholine 4×10−5 M decreased the noradrenaline output by a similar percentage. It is concluded that the muscarinic inhibition of noradrenaline release does not require the functional in…

Malemedicine.medical_specialtySympathetic Nervous SystemTime FactorsHeart VentriclesIndomethacinSympathetic nerveStimulationIn Vitro TechniquesFeedbackNorepinephrineIndometacinHeart RateInternal medicineMuscarinic acetylcholine receptormedicineAnimalsMethacholine CompoundsHeart AtriaPharmacologyChemistryMyocardiumRabbit heartHeartGeneral MedicineElectric StimulationStimulation ChemicalBlockadePerfusionEndocrinologyDepression ChemicalProstaglandinsFemaleMethacholineRabbitsPerfusionmedicine.drugNaunyn-Schmiedeberg's Archives of Pharmacology
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Combining Sirolimus-eluting Stents and External Irradiation in Cholesterol-fed Rabbits Increased Incomplete Stent Apposition and Decreased Re-endothe…

2009

Restenosis after the implantation of a drug-eluting stent or after vascular irradiation therapy shares similar physiopathological mechanisms. No experimental data are currently available on vascular wall behavior after external irradiation on arteries stented with sirolimus-eluting stents (SES). Ten New Zealand white rabbits received a 0.5% cholesterol-enriched chow for 1 month. Bilateral iliac artery stent implantation was then performed with an SES (Cypher; Cordis Corp). The animals were randomized into either an irradiated group (I, 2 Gy external x-ray irradiation, n = 5) or a control group (C, n = 5). The cholesterol-enriched chow was continued for 1 additional month after stent implant…

Malemedicine.medical_specialtyTime FactorsEndotheliummedicine.medical_treatmentUrologyCholesterol Dietarychemistry.chemical_compoundRestenosismedicineAnimalsSirolimusPharmacologyCholesterolbusiness.industryX-RaysStentDose-Response Relationship RadiationDrug-Eluting StentsIncomplete stent appositionequipment and suppliesmedicine.diseaseImmunohistochemistryPathophysiologySurgeryStenosisCholesterolmedicine.anatomical_structurechemistrySirolimusEndothelium VascularRabbitsTunica IntimaTunica MediaCardiology and Cardiovascular MedicinebusinessImmunosuppressive Agentsmedicine.drugJournal of Cardiovascular Pharmacology
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Effects of a nitrate-free interval on tolerance, vasoconstrictor sensitivity and vascular superoxide production

2000

Abstract OBJECTIVES In the present study, we tested whether a nitrate-free interval is able to prevent increases in vascular superoxide (O2•−) and the development of hypersensitivity to vasoconstrictors and whether this may result in restoration of vascular nitroglycerin (NTG) sensitivity. BACKGROUND Intermittent NTG-patch treatment (12 h patch on/patch off) has been shown to increase ischemic periods in patients with stable coronary arteries, suggesting a rebound-like situation during the patch-off period. Recently, we demonstrated that long-term treatment with NTG induces tolerance, which was in part related to increases in vascular O2•− and increased vasoconstrictor sensitivity. METHODS …

Malemedicine.medical_specialtyTime Factorsgenetic structuresVasodilator AgentsBlotting WesternVasodilationIn Vitro TechniquesSuperoxide dismutaseNitroglycerinchemistry.chemical_compoundDrug toleranceInternal medicinemedicineAnimalsEndothelial dysfunctionPhenylephrinebiologySuperoxide DismutaseSuperoxidebusiness.industryDrug Tolerancemedicine.diseaseAngiotensin IIAcetylcholineeye diseasesOxidative StressEndocrinologychemistryVasoconstrictionbiology.proteinFemaleEndothelium VascularRabbitsmedicine.symptombusinessCardiology and Cardiovascular MedicineVasoconstrictionmedicine.drugJournal of the American College of Cardiology
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Aspirin treatment improves bladder function after outlet obstruction in rabbits.

2001

Abstract Objectives. To examine whether bladder smooth muscle dysfunction after outlet obstruction could be altered by treatment with aspirin. Long-term outlet obstruction causes contractile and metabolic dysfunction of the bladder in vivo and in vitro. The evidence is growing that a decrease in bladder perfusion is an important cause of this phenomenon. The platelet aggregation inhibitor, acetylsalicylic acid (aspirin), has been used to improve perfusion of the heart for decades. Methods. Ten male New Zealand white rabbits were obstructed for 4 weeks. Five rabbits received no further treatment (Obs), and 5 rabbits received 2 mg/kg/day aspirin (Obs+aspirin), administered by an osmotic pump …

Malemedicine.medical_specialtyUrologyInjections SubcutaneousUrinary BladderUrologyConnective tissueStimulationurologic and male genital diseasesBladder outlet obstructionBleeding timeInternal medicinemedicineAnimalsDrug ImplantsAspirinmedicine.diagnostic_testAspirinbusiness.industryMuscle SmoothOrgan SizeUrinary Bladder Neck ObstructionNeck of urinary bladderEndocrinologymedicine.anatomical_structurePlatelet aggregation inhibitorRabbitsbusinessPerfusionmedicine.drugUrology
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