Search results for "Reactive"

showing 10 items of 1469 documents

Takotsubo Syndrome: Impact of endothelial dysfunction and oxidative stress

2021

Takotsubo Syndrome (TTS) is characterized by a transient left ventricular dysfunction recovering spontaneously within days or weeks. Although the pathophysiology of TTS remains obscure, there is growing evidence suggesting TTS to be associated with increased production of reactive oxygen species (ROS), which may be involved in causing transient coronary and peripheral endothelial dysfunction leading to a transient impairment of myocardial contraction due to stunning (apical ballooning). Endothelial dysfunction is mainly caused by decreased vascular and myocardial nitric oxide bioavailability in response to increased ROS production. Accordingly, studies in humans and animal models demonstrat…

0301 basic medicinemedicine.medical_specialty1303 Biochemistry610 Medicine & healthSodium hydrosulfidemedicine.disease_causeBiochemistryNitric oxide03 medical and health scienceschemistry.chemical_compound2737 Physiology (medical)0302 clinical medicineSuperoxidesTakotsubo CardiomyopathyPhysiology (medical)Internal medicinemedicineAnimalsHumansEndothelial dysfunctionchemistry.chemical_classificationReactive oxygen speciesNADPH oxidasebiologySuperoxideNADPH OxidasesHydrogen PeroxideMalondialdehydemedicine.diseaseOxidative Stress030104 developmental biologyEndocrinologychemistry10209 Clinic for Cardiologybiology.proteinReactive Oxygen Species030217 neurology & neurosurgeryOxidative stressFree Radical Biology and Medicine
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Impact of Plasma Oxidative Stress Markers on Post-race Recovery in Ultramarathon Runners: A Sex and Age Perspective Overview

2021

Oxidative stress has been widely studied in association to ultra-endurance sports. Although it is clearly demonstrated the increase in reactive oxygen species and free radicals after these extreme endurance exercises, the effects on the antioxidant defenses and the oxidative damage to macromolecules, remain to be fully clarified. Therefore, the aim of this study was to elucidate the impact of an ultramarathon race on the plasma markers of oxidative stress of 32 runners and their post-race recovery, with especial focused on sex and age effect. For this purpose, the antioxidant enzymes glutathione peroxidase (GPx) and glutathione reductase (GR) activity, as well as the lipid peroxidation prod…

0301 basic medicinemedicine.medical_specialtyAntioxidantPhysiologymedicine.medical_treatmentClinical BiochemistryGlutathione reductaseBiologymedicine.disease_causeBiochemistryArticleLipid peroxidation03 medical and health sciencesBasal (phylogenetics)chemistry.chemical_compound0302 clinical medicineultraendurance exerciseInternal medicinemedicineoxidative stressmuscle injuryMolecular Biologychemistry.chemical_classificationReactive oxygen speciesGlutathione peroxidaselcsh:RM1-950030229 sport sciencesCell BiologyMalondialdehydelcsh:Therapeutics. Pharmacologyantioxidants030104 developmental biologyEndocrinologychemistryOxidative stressAntioxidants
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Can aminothiols be distinguished from reactive oxygen species?

2016

Glutathione is considered the major natural antioxidant, protecting cells from oxidative stress. Patel and colleagues used plasma levels of the aminothiols cystine and glutathione to quantify oxidative stress in patients with coronary artery disease, and show that the cystine/glutathione ratio is associated with increased mortality. Is this a new approach for clinical risk stratification?

0301 basic medicinemedicine.medical_specialtyAntioxidantmedicine.medical_treatmentCystine030204 cardiovascular system & hematologymedicine.disease_causeCoronary artery disease03 medical and health scienceschemistry.chemical_compound0302 clinical medicine[SDV.MHEP.CSC]Life Sciences [q-bio]/Human health and pathology/Cardiology and cardiovascular systemInternal medicinemedicineIn patientComputingMilieux_MISCELLANEOUSchemistry.chemical_classificationReactive oxygen speciesbusiness.industryPlasma levelsGlutathionemedicine.disease3. Good health030104 developmental biologyEndocrinologychemistryCardiologyCardiology and Cardiovascular MedicinebusinessOxidative stress
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Exposure to Perfluoroalkyl Substances and Metabolic Outcomes in Pregnant Women: Evidence from the Spanish INMA Birth Cohorts

2017

BACKGROUND: Exposure to perfluoroalkyl substances (PFASs) may increase risk for metabolic diseases; however, epidemiologic evidence is lacking at the present time. Pregnancy is a period of enhanced tissue plasticity for the fetus and the mother and may be a critical window of PFAS exposure susceptibility. OBJECTIVE: We evaluated the associations between PFAS exposures and metabolic outcomes in pregnant women. METHODS: We analyzed 1,240 pregnant women from the Spanish INMA [Environment and Childhood Project (INfancia y Medio Ambiente)] birth cohort study (recruitment period: 2003-2008) with measured first pregnancy trimester plasma concentrations of four PFASs (in nanograms/milliliter). We u…

0301 basic medicinemedicine.medical_specialtyC-reactive protein/metabolismHealth Toxicology and Mutagenesis010501 environmental sciences01 natural sciencesCohort Studies03 medical and health sciencesEmbarassadesFluorocarbons/bloodPregnancyEnvironmental healthGlucose IntoleranceHumansmedia_common.cataloged_instanceMedicineEuropean unionAlkanesulfonic acids/bloodEnvironmental pollutants/blood0105 earth and related environmental sciencesmedia_commonFluorocarbonsbusiness.industryObstetricsResearchPregnant womenPublic Health Environmental and Occupational HealthHispanic or LatinoUniversity hospitalMetabolisme3. Good healthDiabetes GestationalC-Reactive Protein030104 developmental biologyMetabolismAlkanesulfonic AcidsCaprylates/bloodEnvironmental PollutantsFemaleMaternal exposureChristian ministryCaprylatesbusinessBirth cohortCohort study
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Relationship between PMN-endothelium interactions, ROS production and Beclin-1 in type 2 diabetes.

2020

Type 2 diabetes is closely related to oxidative stress and cardiovascular diseases. In this study, we hypothesized that polymorphonuclear leukocytes (PMN)-endothelium interactions and autophagy are associated. We evaluated PMN-endothelial interactions, ROS production and autophagy parameters in 47 type 2 diabetic patients and 57 control subjects. PMNs from type 2 diabetic patients exhibited slower rolling velocity (p < 0.001), higher rolling flux (p < 0.001) and adhesion (p < 0.001) in parallel to higher levels of total (p < 0.05) and mitochondrial ROS (p < 0.05). When the protein expression of autophagy markers was analysed, an increase of Beclin-1 (p < 0.05), LC3I (p < 0.05), LC3II (p < 0…

0301 basic medicinemedicine.medical_specialtyEndotheliumNeutrophilsClinical BiochemistryType 2 diabetesMitochondrionmedicine.disease_causeBiochemistry03 medical and health sciences0302 clinical medicineInternal medicineDiabetes mellitusmedicineAutophagyCell AdhesionHumansEndotheliumlcsh:QH301-705.5lcsh:R5-920ChemistryOrganic ChemistryAutophagyRolling velocityType 2 diabetesROSmedicine.diseaseControl subjectsMitochondria030104 developmental biologymedicine.anatomical_structureEndocrinologylcsh:Biology (General)Diabetes Mellitus Type 2Oxidative stressCase-Control StudiesPMN-Endothelium interactionsBeclin-1lcsh:Medicine (General)Reactive Oxygen Species030217 neurology & neurosurgeryOxidative stressResearch PaperRedox biology
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Biotechnological Agents for Patients With Tumor Necrosis Factor Receptor Associated Periodic Syndrome-Therapeutic Outcome and Predictors of Response:…

2021

Objective: To describe the role of biotechnological therapies in patients with tumor necrosis factor receptor associated periodic syndrome (TRAPS) and to identify any predictor of complete response.Methods: Clinical, laboratory, and therapeutic data from 44 Caucasian TRAPS patients treated with biologic agents were retrospectively collected in 16 Italian tertiary Centers.Results: A total of 55 biological courses with anakinra (n = 26), canakinumab (n = 16), anti-TNF-α agents (n = 10), and tocilizumab (n = 3) were analyzed. A complete response was observed in 41 (74.5%) cases, a partial response in 9 (16.4%) cases and a treatment failure in 5 (9.1%) cases. The frequency of TRAPS exacerbation…

0301 basic medicinemedicine.medical_specialtyMedicine (General)Settore MED/16 - REUMATOLOGIAmedicine.drug_classtumor necrosis factor inhibitorsbiologic therapy interleukin-1 inhibitors personalized medicine tocilizumab tumor necrosis factor inhibitors tumor necrosis factor receptor-associated periodic syndromeinterleukin-1 inhibitorsGastroenterology03 medical and health scienceschemistry.chemical_compoundtocilizumab0302 clinical medicineTocilizumabR5-920Internal medicinemedicinebiologic therapyAdverse effecttumor necrosis factor receptor-associated periodic syndromeOriginal Research030203 arthritis & rheumatologyAnakinraProteinuriabiologymedicine.diagnostic_testbusiness.industryC-reactive proteinGeneral MedicineTumor necrosis factor receptor associated periodic syndromepersonalized medicineCanakinumab030104 developmental biologychemistryErythrocyte sedimentation ratebiology.proteinAutoinflammationCorticosteroidMedicinemedicine.symptombusinessmedicine.drugbiologic therapy; interleukin-1 inhibitors; personalized medicine; tocilizumab; tumor necrosis factor inhibitors; tumor necrosis factor receptor-associated periodic syndrome
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Roles of Vascular Oxidative Stress and Nitric Oxide in the Pathogenesis of Atherosclerosis.

2017

Major reactive oxygen species (ROS)–producing systems in vascular wall include NADPH (reduced form of nicotinamide adenine dinucleotide phosphate) oxidase, xanthine oxidase, the mitochondrial electron transport chain, and uncoupled endothelial nitric oxide (NO) synthase. ROS at moderate concentrations have important signaling roles under physiological conditions. Excessive or sustained ROS production, however, when exceeding the available antioxidant defense systems, leads to oxidative stress. Animal studies have provided compelling evidence demonstrating the roles of vascular oxidative stress and NO in atherosclerosis. All established cardiovascular risk factors such as hypercholesterolemi…

0301 basic medicinemedicine.medical_specialtyNitric Oxide Synthase Type IIIPhysiologyOxidative phosphorylationBiologymedicine.disease_causeNitric OxideNitric oxide03 medical and health scienceschemistry.chemical_compoundSpecies SpecificityInternal medicinemedicineAnimalsHumansXanthine oxidasechemistry.chemical_classificationReactive oxygen speciesNitric Oxide Synthase Type IIIAtherosclerosisEndothelial stem cellOxidative Stress030104 developmental biologyEndocrinologychemistryEndothelium VascularCardiology and Cardiovascular MedicineReactive Oxygen SpeciesOxidative stressNicotinamide adenine dinucleotide phosphateCirculation research
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Gliptins Suppress Inflammatory Macrophage Activation to Mitigate Inflammation, Fibrosis, Oxidative Stress, and Vascular Dysfunction in Models of Nona…

2017

Abstract Aims: Nonalcoholic steatohepatitis (NASH) is characterized by steatosis, panlobular inflammation, liver fibrosis, and increased cardiovascular mortality. Dipeptidyl peptidase-4 inhibitors (gliptins) are indirect glucagon-like peptide 1 agonists with antidiabetic and anti-inflammatory activity, used for the treatment of type 2 diabetes. Their potential and underlying mechanisms to treat metabolic liver inflammation and fibrosis as well as the associated vascular dysfunction remain to be explored. Results: In the methionine/choline-deficient (MCD) diet and Mdr2−/− models of NASH and liver fibrosis, treatment with sitagliptin and linagliptin significantly decreased parameters of steat…

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical BiochemistryAnti-Inflammatory AgentsGene ExpressionInflammationType 2 diabetes030204 cardiovascular system & hematologymedicine.disease_causeBiochemistryAntioxidantsProinflammatory cytokineMice03 medical and health sciences0302 clinical medicineNon-alcoholic Fatty Liver DiseaseFibrosisInternal medicinemedicineAnimalsMyeloid CellsMolecular BiologyDipeptidyl peptidase-4General Environmental ScienceInflammationMice KnockoutDipeptidyl-Peptidase IV Inhibitorsbusiness.industryMacrophagesCell BiologyMacrophage Activationmedicine.diseaseFibrosisDietDisease Models AnimalOxidative Stress030104 developmental biologyEndocrinologyLiverNADPH Oxidase 2General Earth and Planetary SciencesTumor necrosis factor alphaSteatosismedicine.symptomReactive Oxygen SpeciesbusinessBiomarkersOxidative stressAntioxidants &amp; Redox Signaling
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The Adverse Effects of Environmental Noise Exposure on Oxidative Stress and Cardiovascular Risk

2018

Abstract Epidemiological studies have provided evidence that traffic noise exposure is linked to cardiovascular diseases such as arterial hypertension, myocardial infarction, and stroke. Noise is a nonspecific stressor that activates the autonomous nervous system and endocrine signaling. According to the noise reaction model introduced by Babisch and colleagues, chronic low levels of noise can cause so-called nonauditory effects, such as disturbances of activity, sleep, and communication, which can trigger a number of emotional responses, including annoyance and subsequent stress. Chronic stress in turn is associated with cardiovascular risk factors, comprising increased blood pressure and …

0301 basic medicinemedicine.medical_specialtyPhysiologyClinical Biochemistrytraffic noise exposure030204 cardiovascular system & hematologyenvironmental risk factorsmedicine.disease_causeBiochemistryendothelial dysfunctionCoronary artery disease03 medical and health sciences0302 clinical medicinestress hormonesComprehensive Invited ReviewRisk FactorsInternal medicineDiabetes mellitusmedicineHumansChronic stressMyocardial infarctionEndothelial dysfunctionMolecular BiologyStrokeGeneral Environmental Sciencebusiness.industryaircraft noise exposureEnvironmental ExposureCell Biologymedicine.diseaseStrokeOxidative Stress030104 developmental biologyBlood pressureCardiovascular DiseasesHypertensionCardiologyGeneral Earth and Planetary SciencesEnvironmental PollutantsNoiseReactive Oxygen SpeciesbusinessOxidative stressAntioxidants &amp; Redox Signaling
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The Mitochondria-Targeted Antioxidant MitoQ Modulates Mitochondrial Function and Endoplasmic Reticulum Stress in Pancreatic β Cells Exposed to Hyperg…

2019

Background/aims Mitochondria-targeted antioxidants such as mitoquinone (MitoQ) have demonstrated protective effects against oxidative damage in several diseases. The increase in reactive oxygen species (ROS) production during glucose metabolism in β cells can be exacerbated under hyperglycaemic conditions such as type 2 diabetes (T2D), thus contributing to β cell function impairment. In the present work, we aimed to evaluate the effect of MitoQ on insulin secretion, oxidative stress, endoplasmic reticulum (ER) stress and nuclear factor kappa B (NFκB) signalling in a pancreatic β cell line under normoglycaemic (NG, 11.1 mM glucose), hyperglycaemic (HG, 25 mM glucose) and lipidic (palmitic ac…

0301 basic medicinemedicine.medical_specialtyPhysiologyUbiquinoneCarbohydrate metabolismmedicine.disease_causeMitoQlcsh:PhysiologyPancreatic β cellsAntioxidantsProinflammatory cytokinelcsh:Biochemistry03 medical and health scienceschemistry.chemical_compound0302 clinical medicineOrganophosphorus CompoundsInternal medicineCell Line TumorInsulin-Secreting CellsmedicineAnimalslcsh:QD415-436chemistry.chemical_classificationReactive oxygen speciesMitoQlcsh:QP1-981Endoplasmic reticulumGlutathioneEndoplasmic Reticulum StressType 2 DiabetesMitochondriaRatsOxidative Stress030104 developmental biologyEndocrinologyGlucosechemistry030220 oncology & carcinogenesisHyperglycemiaUnfolded protein responseER stressMitochondrial dysfunctionReactive Oxygen SpeciesOxidative stressSignal Transduction
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