Search results for "Real-Time Polymerase Chain Reaction"

showing 10 items of 385 documents

High BCR-ABL Levels At Diagnosis Are Associated with Unfavorable Responses to Imatinib Mesylate.

2012

Abstract Abstract 2790 The approval of second-generation tyrosine kinase inhibitors (TKIs) for the first line treatment of Chronic Myeloid Leukemia (CML) has generated a need for early molecular parameters associated with inadequate responses to Imatinib Mesylate (IM). Recent evidence suggests that CML patients presenting BCR-ABL/ABLIS levels >10% after 3 months of IM or >1% after 6 months of treatment have inferior outcomes in terms of both overall survival (OS) and progression-free survival. We wanted to establish if high BCR-ABL transcripts at diagnosis would also be associated with unfavorable responses to IM. To this end, we correlated quantitative determinations of BCR-ABL measu…

Oncologymedicine.medical_specialtyPathologyABLbusiness.industryImmunologyMyeloid leukemiaCell BiologyHematologymedicine.diseaseBiochemistryLeukemiaImatinib mesylateReal-time polymerase chain reactionhemic and lymphatic diseasesInternal medicinemedicinePopulation studybusinessComplete Hematologic ResponseTyrosine kinase
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High BCR-ABL/GUSIS Levels At Diagnosis Are Associated With Unfavorable Responses To Imatinib

2013

Abstract The approval of three tyrosine kinase inhibitors (TKIs) for the first line treatment of Chronic Myeloid Leukemia (CML) has generated an urgent need for molecular parameters predictive of unfavorable therapeutic outcomes. Recent evidence suggests that failure to achieve early molecular responses (i.e. BCR-ABL/ABLIS levels <10% after 3 months or <1% after 6 months of TKI treatment) results in inferior rates of both overall and progression-free survival. With the current study, we wanted to establish if high BCR-ABL transcripts at diagnosis would be associated with unfavorable responses to Imatinib Mesylate (IM). Thus, we correlated quantitative determinations of BCR-ABL levels …

Oncologymedicine.medical_specialtyPathologyABLbusiness.industryImmunologyMyeloid leukemiaImatinibCell BiologyHematologymedicine.diseaseBiochemistryLeukemiaReal-time polymerase chain reactionImatinib mesylatehemic and lymphatic diseasesInternal medicinemedicinePopulation studybusinessComplete Hematologic Responsemedicine.drug
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Circulating miR-99a-5p Expression in Plasma: A Potential Biomarker for Early Diagnosis of Breast Cancer

2020

MicroRNAs have emerged as new diagnostic and therapeutic biomarkers for breast cancer. Herein, we analysed miR-99a-5p expression levels in primary tumours and plasma of breast cancer patients to evaluate its usefulness as a minimally invasive diagnostic biomarker. MiR-99a-5p expression levels were determined by quantitative real-time PCR in three independent cohorts of patients: (I) Discovery cohort: breast cancer tissues (n = 103) and healthy breast tissues (n = 26)

Oncologymedicine.medical_specialtydiagnosisDown-RegulationBreast NeoplasmsReal-Time Polymerase Chain ReactionArticleCatalysislcsh:ChemistryInorganic Chemistrybreast cancerBreast cancerInternal medicinemicroRNABiomarkers TumorHumansMedicineDiagnostic biomarkerCirculating MicroRNAPhysical and Theoretical Chemistryskin and connective tissue diseaseslcsh:QH301-705.5Molecular BiologyEarly Detection of CancerplasmaSpectroscopyRetrospective StudiesEarly breast cancerPlasma samplesbusiness.industryOrganic ChemistryGeneral MedicineMiddle Agedmedicine.diseaseComputer Science ApplicationsGene Expression Regulation NeoplasticMicroRNAsROC Curvelcsh:Biology (General)lcsh:QD1-999Potential biomarkersCohortbiomarkerBiomarker (medicine)FemalebusinessInternational Journal of Molecular Sciences
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Expression of a mutant HSP110 sensitizes colorectal cancer cells to chemotherapy and improves disease prognosis

2011

Heat shock proteins (HSPs) are necessary for cancer cell survival. We identified a mutant of HSP110 (HSP110ΔE9) in colorectal cancer showing microsatellite instability (MSI CRC), generated from an aberrantly spliced mRNA and lacking the HSP110 substrate-binding domain. This mutant was expressed at variable levels in almost all MSI CRC cell lines and primary tumors tested. HSP110ΔE9 impaired both the normal cellular localization of HSP110 and its interaction with other HSPs, thus abrogating the chaperone activity and antiapoptotic function of HSP110 in a dominant-negative manner. HSP110ΔE9 overexpression caused the sensitization of cells to anticancer agents such as oxaliplatin and 5-fluorou…

Organoplatinum CompoundsColorectal cancermedicine.medical_treatment[SDV]Life Sciences [q-bio]Blotting WesternFluorescent Antibody TechniqueAntineoplastic AgentsBiologyBioinformaticsReal-Time Polymerase Chain ReactionTransfectionGeneral Biochemistry Genetics and Molecular Biology03 medical and health sciences0302 clinical medicineHeat shock proteinCell Line TumormedicineHumansImmunoprecipitationHSP110 Heat-Shock ProteinsneoplasmsCellular localizationComputingMilieux_MISCELLANEOUS030304 developmental biologyDNA Primers0303 health sciencesChemotherapyMicrosatellite instabilityGeneral MedicineTransfectionmedicine.diseasePrognosisdigestive system diseases3. Good healthOxaliplatinOxaliplatin030220 oncology & carcinogenesisCancer cellMutationCancer researchRegression AnalysisMicrosatellite InstabilityFluorouracilColorectal Neoplasmsmedicine.drugPlasmids
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Gliadin activates arginase pathway in RAW264.7 cells and in human monocytes

2014

AbstractCeliac disease (CD) is an autoimmune enteropathy triggered in susceptible individuals by the ingestion of gliadin-containing grains. Recent studies have demonstrated that macrophages play a key role in the pathogenesis of CD through the release of inflammatory mediators such as cytokines and nitric oxide (NO). Since arginine is the obliged substrate of iNOS (inducible nitric oxide synthase), the enzyme that produces large amount of NO, the aim of this work is to investigate arginine metabolic pathways in RAW264.7 murine macrophages after treatment with PT-gliadin (PTG) in the absence and in the presence of IFNγ. Our results demonstrate that, besides strengthening the IFNγ-dependent …

OrnithineArginineBlotting WesternNitric Oxide Synthase Type IIOrnithine DecarboxylaseReal-Time Polymerase Chain ReactionArginineMonocytesGliadinOrnithine decarboxylaseInterferon-gammaMicechemistry.chemical_compoundmedicineAnimalsHumansCeliac diseaseMacrophageRNA MessengerMolecular BiologyCells CulturedArginasebiologyReverse Transcriptase Polymerase Chain ReactionMacrophagesMonocytenutritional and metabolic diseasesNitric oxideOrnithineMolecular biologyPeptide FragmentsNitric oxide synthaseArginasemedicine.anatomical_structureBiochemistrychemistrybiology.proteinMolecular MedicineInterferon-γGliadinBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
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Induction of a disintegrin and metalloprotease 33 during embryonic lung development and the influence of IL-13 or maternal allergy.

2009

Background Asthma pathogenesis involves gene and environmental interactions. A disintegrin and metalloprotease 33 (ADAM33)/Adam33 is a susceptibility gene for asthma and bronchial hyperresponsiveness in human beings and mice. ADAM33 is almost exclusively expressed in mesenchymal cells, including mesenchymal progenitors in developing lungs. Objective Because maternal allergy is a risk factor for asthma, we hypothesized that an allergic environment affects ADAM33/Adam33 expression during human and mouse lung development. Methods Human embryonic/fetal lung (HEL) tissues were collected from first-trimester terminations of pregnancy. These were processed immediately or used for explant culture ±…

OvalbuminImmunologyADAM33AndrologyMiceOrgan Culture TechniquesGene interactionmedicineHypersensitivityImmunology and AllergyAnimalsHumansProtein IsoformsRNA MessengerLungFetusInterleukin-13medicine.diagnostic_testbiologyGene Expression Regulation Developmentalrespiratory tract diseasesOvalbuminADAM ProteinsBronchoalveolar lavageReal-time polymerase chain reactionInterleukin 13Immunologybiology.proteinLung morphogenesisADAM33 IL-13 Asthma AllergyThe Journal of allergy and clinical immunology
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Identification of miR-187 and miR-182 as biomarkers of early diagnosis and prognosis in patients with prostate cancer treated with radical prostatect…

2014

Purpose: miRNAs are noncoding RNAs that negatively regulate target mRNA gene expression. Aberrant miRNA expression is associated with prostate cancer pathogenesis. We identified miRNAs as potential biomarkers for prostate cancer diagnosis and prognosis. Materials and Methods: Total RNA was obtained from 10 normal prostate and 50 prostate cancer samples, and analyzed using the GeneChip (R) miRNA 2.0 Array. At a median followup of 92 months (range 2 to 189) an independent cohort of 273 paraffin embedded prostate cancer samples was used for validation by quantitative reverse transcriptase-polymerase chain reaction. Another 92 urine samples from patients undergoing prostate biopsy were evaluate…

PCA3OncologyMalemedicine.medical_specialtyPathologyProstate biopsydiagnosisUrologymedicine.medical_treatmentprostatic neoplasmsProstate cancerProstateInternal medicinemicroRNAmedicineBiomarkers TumorHumansAgedRetrospective StudiesProstatectomymedicine.diagnostic_testbusiness.industryProstatectomyProstatic Neoplasmsmedicine.diseasePrognosismicroRNAsGene Expression Regulation NeoplasticProstate-specific antigenMicroRNAsmedicine.anatomical_structureReal-time polymerase chain reactionEarly Diagnosisprognosisbusinessbiological markersThe Journal of urology
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Molecular Diagnosis of Ewing Sarcoma Family of Tumors

2009

To compare the sensitivity and specificity of fluorescence in situ hybridization (FISH) with reverse transcription polymerase chain reaction (RT-PCR) in the diagnosis of Ewing sarcoma family of tumors (ESFTs) and other small round-cell tumors (SRCTs) in formalin-fixed paraffin-embedded tissue assembled in tissue microarrays (TMAs). The second objective is to confirm the value of molecular methods and immunohistochemical (IHC) assays, to perform a differential diagnosis between ESFTs and SRCTs with similar or overlapping morphology.A total of 560 cases were selected for the present study out the 806 cases collected from the PROgnosis and THerapeutic Targets in the Ewing's Family of TumorS pr…

Pathologymedicine.medical_specialtyBone NeoplasmsSarcoma EwingIn situ hybridizationBiologyTranslocation GeneticPathology and Forensic MedicinePredictive Value of TestsBiomarkers TumormedicineHumansMolecular diagnostic techniquesRNA NeoplasmParaffin embeddingMolecular BiologyIn Situ Hybridization FluorescenceParaffin Embeddingmedicine.diagnostic_testReverse Transcriptase Polymerase Chain ReactionDNA NeoplasmCell Biologymedicine.diseaseReverse transcription polymerase chain reactionReal-time polymerase chain reactionMolecular Diagnostic TechniquesTissue Array AnalysisFish <Actinopterygii>SarcomaFluorescence in situ hybridizationDiagnostic Molecular Pathology
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Contrasting responses of Kupffer cells and inflammatory mononuclear phagocytes to biliary obstruction in a mouse model of cholestatic liver injury.

2012

Background Biliary obstruction and cholestasis are serious complications of many liver diseases. Although resident hepatic macrophages (Kupffer cells) are frequently implicated in disease progression, most studies fail to differentiate the contribution of Kupffer cells and inflammatory mononuclear phagocytes (iMNPs) that infiltrate the liver subsequent to obstruction. Aim This study was undertaken to examine the roles and potential interactions of these two disparate mononuclear phagocyte populations in hepatic injury attending cholestasis. Methods Female, C57Bl/6 mice were injected with magnetic beads on day 3 prior to sham operation or bile duct ligation (BDL) to facilitate subsequent Kup…

Pathologymedicine.medical_specialtyChemokineLiver cytologyKupffer Cellsmedicine.medical_treatmentInflammationCholestasis IntrahepaticBiologyReal-Time Polymerase Chain Reactiondigestive systemMiceCholestasismedicineAnimalsMononuclear Phagocyte SystemLiver injuryHepatologyKupffer cellMononuclear phagocyte systemmedicine.diseaseFlow CytometrySpecific Pathogen-Free OrganismsMice Inbred C57BLmedicine.anatomical_structureCytokineImmunologybiology.proteinFemalemedicine.symptomLiver international : official journal of the International Association for the Study of the Liver
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Real-Time Polymerase Chain Reaction for Detection of Schistosoma DNA in Small-Volume Urine Samples Reflects Focal Distribution of Urogenital Schistos…

2014

Schistosoma haematobium eggs and Schistosoma DNA levels were measured in urine samples from 708 girls recruited from 18 randomly sampled primary schools in South Africa. Microscopic analysis of two 10-mL urine subsamples collected on three consecutive days confirmed high day-to-day variation; 103 (14.5%) girls had positive results at all six examinations, and at least one positive sample was seen in 225 (31.8%) girls. Schistosoma-specific DNA, which was measured in a 200-μL urine subsample by using real-time polymerase chain reaction, was detected in 180 (25.4%) cases, and levels of DNA corresponded significantly with average urine egg excretion. In concordance with microscopic results, pol…

Pathologymedicine.medical_specialtyConcordancePhysiologyUrineBiologyReal-Time Polymerase Chain ReactionSensitivity and Specificitylaw.inventionExcretionSchistosomiasis haematobiaSouth AfricalawVirologyParasite Egg CountmedicineAnimalsHumansChildParasite Egg CountPolymerase chain reactionSchistosomaSchistosoma haematobiumArticlesDNA Helminthbiology.organism_classificationInfectious DiseasesReal-time polymerase chain reactionSchistosoma haematobiumParasitologyFemale
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