Search results for "Receptor"

showing 10 items of 6990 documents

Catechol-O-Methyltransferase Gene Polymorphism Is Associated with Skeletal Muscle Properties in Older Women Alone and Together with Physical Activity

2008

BackgroundMuscle strength declines on average by one percent annually from midlife on. In postmenopausal women this decrement coincides with a rapid decline in estrogen production. The genetics underlying the effects of estrogen on skeletal muscle remains unclear. In the present study, we examined whether polymorphisms within COMT and ESR1 are associated with muscle properties and assessed their interaction and their combined effects with physical activity.Methodology/principal findingsA cross-sectional data analysis was conducted with 434 63-76-year-old women from the population-based Finnish Twin Study on Aging. Body anthropometry, muscle cross-sectional area (mCSA), isometric hand grip a…

medicine.medical_specialtymedicine.drug_classScienceeducationPhysical activityWomen's Health/Menopause and Post-Reproductive Women's HealthCatechol O-Methyltransferase03 medical and health sciences0302 clinical medicinePolymorphism (computer science)Internal medicineHand strengthGenetics and Genomics/Population GeneticsMedicineHumansMuscle SkeletalExercise030304 developmental biologyAged0303 health sciencesMultidisciplinaryCatechol-O-methyl transferasePolymorphism Geneticbusiness.industryPhysiology/EndocrinologyQRSkeletal muscleESR1 and Skeletal MuscleMiddle Aged314 Health sciencesTwin studyCOMTEndocrinologymedicine.anatomical_structureEstrogenMedicineESR1 ja luurankolihasFemalePublic Health and Epidemiology/EpidemiologybusinessEstrogen receptor alpha030217 neurology & neurosurgeryResearch ArticlePLoS ONE
researchProduct

Psychotropic Effect of Combined Estrogen-Vit B6 Treatment in Endogenously Depressed Females

1985

Estrogens are among the most commonly prescribed substances in females. Also endogenous estrogen levels change dramatically throughout life and this biological variable has been associated with several psychological signs like premenstrual tension syndrome and depression in older age. Along with clinical practice there is increasing evidence from neuropharmacology suggesting a psychotropic action of estrogens (review: Holsboer, 1982). The most prominent findings are: 1. Reduction of monoamine-oxidase (MAO)-activity by estrogens (McEwen et al., 1978); 2. Competitive inhibition of catechol-o-methyltransferase by 2-hydroxyestrogens, which are major metabolites of estrogens in the CNS (Breuer e…

medicine.medical_specialtymedicine.drug_classbusiness.industryDopaminergicStimulationSerotonergicEndocrinologyEstrogenDopamineInternal medicinemedicineAutoreceptorCholinergicbusinessNeuropharmacologymedicine.drug
researchProduct

Activation of mGlu3 Receptors Stimulates the Production of GDNF in Striatal Neurons

2009

Metabotropic glutamate (mGlu) receptors have been considered potential targets for the therapy of experimental parkinsonism. One hypothetical advantage associated with the use of mGlu receptor ligands is the lack of the adverse effects typically induced by ionotropic glutamate receptor antagonists, such as sedation, ataxia, and severe learning impairment. Low doses of the mGlu2/3 metabotropic glutamate receptor agonist, LY379268 (0.25-3 mg/kg, i.p.) increased glial cell line-derived neurotrophic factor (GDNF) mRNA and protein levels in the mouse brain, as assessed by in situ hybridization, real-time PCR, immunoblotting, and immunohistochemistry. This increase was prominent in the striatum, …

medicine.medical_specialtymedicine.drug_classlcsh:MedicineSubstantia nigraReceptors Metabotropic GlutamateSettore BIO/09 - FisiologiaPolymerase Chain ReactionMiceNeurotrophic factorsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsGlial Cell Line-Derived Neurotrophic FactorRNA MessengerAmino Acidslcsh:ScienceReceptorIn Situ HybridizationNeurological Disorders/Movement DisordersNeuronsMultidisciplinarybiologyNeuroscience/Neuronal and Glial Cell Biologylcsh:RGlutamate receptorBridged Bicyclo Compounds HeterocyclicReceptor antagonistCorpus StriatumEndocrinologyMetabotropic receptornervous systemMetabotropic glutamate receptorSettore BIO/14 - Farmacologiabiology.proteinlcsh:QNeuroscience/Neurobiology of Disease and RegenerationReceptors Metabotropic Glutamate/agonists Glial Cell Line-Derived Neurotrophic FactorResearch Article
researchProduct

Possible Pathogenetic Relevance of Interleukin-1beta in "Destructive" Organ-specific Autoimmune Disease (Hashimoto's Thyroiditis)

1999

Thyroid follicular cells (TFC) abundantly express a variety of immunologically relevant surface molecules in Hashimoto's thyroiditis (HT), for example, MHC antigens and adhesion molecules such as ICAM-1. Cytokines produced by infiltrating type 1 helper and cytotoxic T cells are importantly involved in de novo expression or up-regulation of such molecules. We recently demonstrated that TFC from HT patients almost invariably bear on their surface two additive functional molecules: Fas/Apo1/CD95, an important participant in apoptosis, and B7.1, a member of a family of "co-stimulatory" molecules that are crucial for efficient antigen presentation. To date, 12 out of 14 surgical HT thyroid speci…

medicine.medical_specialtymedicine.medical_treatmentAntigen presentationThyroid Glandmedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyFas ligandAutoimmunityHistory and Philosophy of ScienceInternal medicinemedicineHumansCytotoxic T cellfas ReceptorChemistryGeneral NeuroscienceThyroiditis AutoimmuneInterleukinFas receptorMolecular biologyGraves DiseaseRecombinant ProteinsCytokineEndocrinologyApoptosisB7-1 AntigenCytokinesInterleukin-1Annals of the New York Academy of Sciences
researchProduct

Role of insulin-like growth factors in autocrine growth of human retinoblastoma Y79 cells.

1996

In this study, we have demonstrated that human retinoblastoma Y79 cells produce insulin-like growth factors (IGFs) type I and type II and release them into the medium. We have also ascertained, by means of competitive studies and cross-linking procedure, that Y79 cells contain the type-I IGF receptor (IGF-IR). Furthermore, surface-bound IGF-I is internalised by the receptor, then degraded to amino acids. Insulin, IGF-I and IGF-II caused down-regulation of IGF-IR; the effect is concentration and time dependant. Scatchard analysis demonstrated that incubation with insulin markedly decreased the binding capacity measured for IGF-I while the apparent Kd value calculated for IGF-I binding was no…

medicine.medical_specialtymedicine.medical_treatmentBiologyBiochemistryBinding CompetitiveReceptor IGF Type 1chemistry.chemical_compoundInsulin-Like Growth Factor IIInternal medicineInsulin receptor substratemedicineHumansInsulinInsulin-Like Growth Factor IAutocrine signallingPhosphotyrosineInsulin-like growth factor 1 receptorInsulinRetinoblastomaTyrosine phosphorylationPhosphoproteinsIRS2Insulin receptorautocrine growthEndocrinologychemistrybiology.proteinInsulin Receptor Substrate ProteinsPlatelet-derived growth factor receptorCell DivisionSignal TransductionEuropean journal of biochemistry
researchProduct

Effect of Soluble Interleukin-6 Receptor on Interleukin-6 Synthesis in Human Skin Fibroblasts

1996

Abstract In this study the ability of soluble interleukin-6 receptor (sIL-6R) to stimulate interleukin-6 (IL-6) synthesis in human fibroblasts is described. It was found that sIL-6R, in combination with endogenous or exogenous IL-6, markedly upregulated IL-6 synthesis. These data suggest that increased IL-6 production after stimulation by either interleukin-1 or tumor necrosis factor-α would result in complex formation with sIL-6R, rapid uptake, and further synthesis of this cytokine. Furthermore, it would explain the decrease in sIL-6R plasma levels observed in patients suffering from sepsis.

medicine.medical_specialtymedicine.medical_treatmentBiophysicsGene ExpressionHuman skinStimulationEndogenyBiochemistryDownregulation and upregulationAntigens CDInternal medicinemedicineHumansInterleukin 6ReceptorMolecular BiologyCells CulturedSkinbiologyInterleukin-6Receptors InterleukinCell BiologyFibroblastsReceptors Interleukin-6Recombinant ProteinsKineticsCytokineEndocrinologybiology.proteinTumor necrosis factor alphaBiochemical and Biophysical Research Communications
researchProduct

Role of magnesium in insulin action, diabetes and cardio-metabolic syndrome X

2003

Magnesium (Mg) is one of the most abundant ions present in living cells and its plasma concentration is remarkably constant in healthy subjects. Plasma and intracellular Mg concentrations are tightly regulated by several factors. Among them, insulin seems to be one of the most important. In vitro and in vivo studies have demonstrated that insulin may modulate the shift of Mg from extracellular to intracellular space. Intracellular Mg concentration has also been shown to be effective in modulating insulin action (mainly oxidative glucose metabolism), offset calcium-related excitation-contraction coupling, and decrease smooth cell responsiveness to depolarizing stimuli. A poor intracellular M…

medicine.medical_specialtymedicine.medical_treatmentGlucose uptakeClinical BiochemistryBiologyCarbohydrate metabolismBiochemistryInsulin resistanceDiabetes mellitusInternal medicinemedicineHumansInsulinMagnesiumMolecular BiologyMetabolic SyndromeInsulinGeneral Medicinemedicine.diseaseOxidative StressInsulin receptorEndocrinologyDiabetes Mellitus Type 2Hypertensionbiology.proteinMolecular MedicineInsulin ResistanceVanadatesMetabolic syndromeIntracellularMolecular Aspects of Medicine
researchProduct

Anesthetics and Cerebral Protection in Patients Undergoing Carotid Endarterectomy

2015

EREBRAL ISCHEMIA/HYPOXIA may occur in a vari-ety of perioperative circumstances. The main pathophy-siologic aspects involved in cerebral ischemia/reperfusion arecaused by adenosine triphosphate (ATP) consumption, theexcitotoxic actions of glutamate, changes in ionic homeostasis,and formation of free radicals (Fig 1). Outcomes from suchevents range from subclinical neurocognitive deficits to cata-strophic neurologic morbidity or death.

medicine.medical_specialtymedicine.medical_treatmentIschemiaCarotid endarterectomyAnesthesia GeneralNeuroprotectionSevofluraneBrain Ischemia03 medical and health sciences0302 clinical medicine030202 anesthesiologyInternal medicinemedicineAnimalsHumansIschemic PreconditioningSubclinical infectionEndarterectomy Carotidbusiness.industryGlutamate receptorPerioperativeHypoxia (medical)medicine.disease3. Good healthNeuroprotective AgentsAnesthesiology and Pain MedicineAnesthesiaAnesthetics InhalationCardiologymedicine.symptomCardiology and Cardiovascular Medicinebusiness030217 neurology & neurosurgerymedicine.drugJournal of Cardiothoracic and Vascular Anesthesia
researchProduct

Cholesterol as stabilizer of the oxytocin receptor

2002

AbstractThe function of the oxytocin receptor system is strongly dependent on steroids as demonstrated by several physiological studies. One key element of this dependence on steroids may be the interaction of cholesterol and the oxytocin receptor. In this study, we show that cholesterol stabilizes the solubilized human oxytocin receptor against thermal inactivation and proteolytic degradation. In the absence of additional cholesterol, the soluble receptor inactivates within minutes. Maximal stabilization of the oxytocin receptor requires a continuous supply with cholesterol from a cholesterol-rich environment. A structure–activity analysis of various cholesterol analogues and their effect …

medicine.medical_specialtymedicine.medical_treatmentProteolysisGreen Fluorescent ProteinsBiophysicsTransfectionBiochemistrySteroidCell Linechemistry.chemical_compoundInternal medicineEndopeptidasesmedicineHumansDenaturation (biochemistry)ReceptorOxytocin receptormedicine.diagnostic_testCholesterolTemperatureTransfectionCell BiologyHydrogen-Ion ConcentrationOxytocin receptorDenaturationLuminescent ProteinsEndocrinologyCholesterolchemistrySolubilityCell cultureReceptors OxytocinProteolysislipids (amino acids peptides and proteins)hormones hormone substitutes and hormone antagonistsBiochimica et Biophysica Acta (BBA) - Biomembranes
researchProduct

Inflammation et immunité : implications dans l’obésité et le diabète de type 2

2006

The evidences have been increasingly accumulated on the implication of inflammatory mediators like tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) in the pathological states related to insulin resistance like obesity, type 2 diabetes and atherosclerosis. There seems a link between insulin resistance and these pro-inflammatory agents, secreted by macrophages and adipocytes. Th (helper) cells are differentiated into either Th1 or Th2 phenotypes. It is generally considered that Th1 phenotype is pro-inflammatory whereas Th2 phenotype exerts anti-inflammatory (protective) effects. The upregulation of Th1 phenotype may aggravate these pathologies. One of the adipokines, i.e., adiponectin…

medicine.medical_specialtyobesitymedicine.medical_treatmentPeroxisome proliferator-activated receptorInflammationlcsh:TP670-699BiologyBiochemistryPPARfatty acidsInsulin resistanceDownregulation and upregulationInternal medicinemedicinechemistry.chemical_classificationAdiponectinInsulinmedicine.diseaseInsulin receptorimmune systemEndocrinologyCytokinechemistryinflammationImmunologybiology.proteinmedicine.symptomlcsh:Oils fats and waxesFood ScienceOléagineux, Corps gras, Lipides
researchProduct