Search results for "Regulatory"

showing 10 items of 740 documents

Down-regulation of early sea urchin histone H2A gene relies on cis regulative sequences located in the 5' and 3' regions and including the enhancer b…

2004

The tandem repeated sea urchin alpha-histone genes are developmentally regulated by gene-specific promoter elements. Coordinate transcription of the five genes begins after meiotic maturation of the oocyte, continues through cleavage, and reaches its maximum at morula stage, after which these genes are shut off and maintained in a silenced state for the life cycle of the animal. Although cis regulative sequences affecting the timing and the level of expression of these genes have been characterized, much less is known about the mechanism of their repression. Here we report the results of a functional analysis that allowed the identification of the sequence elements needed for the silencing …

Chloramphenicol O-Acetyltransferaseanimal structuresEmbryo NonmammalianMicroinjectionsgenomic insulatorDown-RegulationSettore BIO/11 - Biologia MolecolareBiologyRegulatory Sequences Nucleic AcidDNA-binding proteinHistonesStructural BiologyTranscription (biology)Gene expressionHistone H2Atranscriptional repressionGene silencingAnimalsGene SilencingTransgenesEnhancerPromoter Regions GeneticMolecular BiologyGenePsychological repressionhistone geneRepetitive Sequences Nucleic AcidSequence DeletionGeneticsenhancer blockerGastrulaEnhancer Elements GeneticSea Urchinsembryonic structuresProtein BindingJournal of molecular biology
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Genome-wide association analysis in primary sclerosing cholangitis identifies two non-HLA susceptibility loci

2011

Primary sclerosing cholangitis (PSC) is a chronic bile duct disease affecting 2.4-7.5% of individuals with inflammatory bowel disease. We performed a genome-wide association analysis of 2,466,182 SNPs in 715 individuals with PSC and 2,962 controls, followed by replication in 1,025 PSC cases and 2,174 controls. We detected non-HLA associations at rs3197999 in MST1 and rs6720394 near BCL2L11 (combined P = 1.1 x 10(-16) and P = 4.1 x 10(-8), respectively).

Cholangitis SclerosingPATHOGENESISSingle-nucleotide polymorphismGenome-wide association studyHuman leukocyte antigenBiologyInflammatory bowel diseasePolymorphism Single Nucleotidedigestive systemArticlePrimary sclerosing cholangitisPathogenesisCohort StudiesHLA AntigensProto-Oncogene ProteinsGeneticsmedicineHumansGenetic Predisposition to DiseaseBOWEL-DISEASEGenetic associationBcl-2-Like Protein 11Bile ductHepatocyte Growth FactorGene Expression Profilingdigestive oral and skin physiologyMembrane Proteinsmedicine.diseasedigestive system diseasesmedicine.anatomical_structureGenetic LociImmunologyApoptosis Regulatory ProteinsGenome-Wide Association Study
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Cholera-Like Enterotoxins and Regulatory T cells

2010

Cholera toxin (CT) and the heat-labile enterotoxin of E. coli (LT), as well as their non toxic mutants, are potent mucosal adjuvants of immunization eliciting mucosal and systemic responses against unrelated co-administered antigens in experimental models and in humans (non toxic mutants). These enterotoxins are composed of two subunits, the A subunit, responsible for an ADP-ribosyl transferase activity and the B subunit, responsible for cell binding. Paradoxically, whereas the whole toxins have adjuvant properties, the B subunits of CT (CTB) and of LT (LTB) have been shown to induce antigen specific tolerance when administered mucosally with antigens in experimental models as well as, rece…

Cholera ToxinHealth Toxicology and Mutagenesismedicine.medical_treatmentBacterial Toxinslcsh:MedicineEnterotoxinReviewBiologyToxicologymedicine.disease_causeT-Lymphocytes Regulatoryregulatory T cellsMicrobiologyImmune toleranceAutoimmune DiseasesEnterotoxinsImmune systemAntigenAdjuvants ImmunologicmedicineImmune ToleranceAnimalsHumansAntigen-presenting cellEscherichia coli Proteinslcsh:RCholera toxinCTBIn vitroLTBImmunologyAdjuvantheat-labile enterotoxin of E. colicholera-like enterotoxinsToxins
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cis-Regulatory sequences driving the expression of the Hbox12 homeobox-containing gene in the presumptive aboral ectoderm territory of the Paracentro…

2008

AbstractEmbryonic development is coordinated by networks of evolutionary conserved regulatory genes encoding transcription factors and components of cell signalling pathways. In the sea urchin embryo, a number of genes encoding transcription factors display territorial restricted expression. Among these, the zygotic Hbox12 homeobox gene is transiently transcribed in a limited number of cells of the animal-lateral half of the early Paracentrotus lividus embryo, whose descendants will constitute part of the ectoderm territory. To obtain insights on the regulation of Hbox12 expression, we have explored the cis-regulatory apparatus of the gene. In this paper, we show that the intergenic region …

Chromatin ImmunoPrecipitationDNA ComplementaryEmbryo Nonmammaliananimal structuresGreen Fluorescent ProteinsMolecular Sequence DataSettore BIO/11 - Biologia MolecolareEctodermHomeodomainMybBiologyOtxEctoderm specificationHomeobox cis-regulatory elements GFP sea urchinEctodermmedicineAnimalsRegulatory Elements TranscriptionalAboral ectodermSea urchin embryoMolecular BiologyGene transferDNA PrimersRegulator geneCis-regulatory moduleHomeodomain ProteinsGeneticsBase SequenceEmbryogenesisGene Expression Regulation DevelopmentalCell Biologycis-Regulatory moduleGastrulationmedicine.anatomical_structureMutagenesisRegulatory sequenceSea Urchinsembryonic structuresSoxHomeoboxSequence AlignmentDevelopmental BiologyDevelopmental Biology
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Dependence on nuclear factor of activated T-cells (NFAT) levels discriminates conventional T cells from Foxp3 + regulatory T cells

2012

Several lines of evidence suggest nuclear factor of activated T-cells (NFAT) to control regulatory T cells: thymus-derived naturally occurring regulatory T cells (nTreg) depend on calcium signals, the Foxp3 gene harbors several NFAT binding sites, and the Foxp3 (Fork head box P3) protein interacts with NFAT. Therefore, we investigated the impact of NFAT on Foxp3 expression. Indeed, the generation of peripherally induced Treg (iTreg) by TGF-β was highly dependent on NFAT expression because the ability of CD4 + T cells to differentiate into iTreg diminished markedly with the number of NFAT family members missing. It can be concluded that the expression of Foxp3 in TGF-β–induced iTreg depends…

Chromatin ImmunoprecipitationAdoptive cell transferT-LymphocytesImmunoblottingFluorescent Antibody TechniqueLymphocyte ActivationT-Lymphocytes RegulatoryAutoimmune DiseasesProinflammatory cytokineMiceTransforming Growth Factor betaAnimalsHumansHomeodomain ProteinsMultidisciplinaryNFATC Transcription FactorsbiologyFOXP3Forkhead Transcription FactorsNFATTransforming growth factor betaBiological SciencesColitisFlow CytometryNFATC Transcription FactorsAdoptive TransferMolecular biologyCell biologyTransplantationCyclosporinebiology.proteinChromatin immunoprecipitationProceedings of the National Academy of Sciences
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Satb2 Regulates Callosal Projection Neuron Identity in the Developing Cerebral Cortex

2008

SummarySatb2 is a DNA-binding protein that regulates chromatin organization and gene expression. In the developing brain, Satb2 is expressed in cortical neurons that extend axons across the corpus callosum. To assess the role of Satb2 in neurons, we analyzed mice in which the Satb2 locus was disrupted by insertion of a LacZ gene. In mutant mice, β-galactosidase-labeled axons are absent from the corpus callosum and instead descend along the corticospinal tract. Satb2 mutant neurons acquire expression of Ctip2, a transcription factor that is necessary and sufficient for the extension of subcortical projections by cortical neurons. Conversely, ectopic expression of Satb2 in neural stem cells m…

Chromatin ImmunoprecipitationNeuroscience(all)Electrophoretic Mobility Shift AssayMice TransgenicNerve Tissue ProteinsDEVBIOBiologyCorpus callosumMOLNEUROMiceNeural PathwaysmedicineAnimalsCells CulturedCerebral CortexNeuronsRegulation of gene expressionStem CellsGeneral NeuroscienceGene Expression Regulation DevelopmentalMatrix Attachment Region Binding ProteinsDNAEmbryo MammalianNeural stem cellChromatinmedicine.anatomical_structureAnimals NewbornBromodeoxyuridinenervous systemCerebral cortexRegulatory sequenceMutationCorticospinal tractEctopic expressionNeuroscienceTranscription Factors
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RIP-Chip analysis supports different roles for AGO2 and GW182 proteins in recruiting and processing microRNA targets.

2019

Background MicroRNAs (miRNAs) are small non-coding RNA molecules mediating the translational repression and degradation of target mRNAs in the cell. Mature miRNAs are used as a template by the RNA-induced silencing complex (RISC) to recognize the complementary mRNAs to be regulated. To discern further RISC functions, we analyzed the activities of two RISC proteins, AGO2 and GW182, in the MCF-7 human breast cancer cell line. Methods We performed three RIP-Chip experiments using either anti-AGO2 or anti-GW182 antibodies and compiled a data set made up of the miRNA and mRNA expression profiles of three samples for each experiment. Specifically, we analyzed the input sample, the immunoprecipita…

Chromatin ImmunoprecipitationSupport Vector MachineRIP-Chip data analysisMiRNA bindingComputational biologyBiologylcsh:Computer applications to medicine. Medical informaticsBiochemistryAutoantigens03 medical and health sciencesOpen Reading Frames0302 clinical medicineStructural BiologymicroRNARIP-Chip data analysiCoding regionGene silencingHumansRNA MessengerMolecular BiologyGenelcsh:QH301-705.5030304 developmental biology0303 health sciencesBinding SitesApplied MathematicsGene Expression ProfilingResearchRNARNA-Binding ProteinsmicroRNA target predictionRISC proteins AGO2 and GW182Computer Science ApplicationsSettore BIO/18 - GeneticaMicroRNAslcsh:Biology (General)Gene Expression Regulation030220 oncology & carcinogenesismicroRNA regulatory activityArgonaute ProteinsMCF-7 Cellslcsh:R858-859.7DNA microarrayRIP-ChipBMC bioinformatics
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Topological structure analysis of chromatin interaction networks.

2019

Abstract Background Current Hi-C technologies for chromosome conformation capture allow to understand a broad spectrum of functional interactions between genome elements. Although significant progress has been made into analysis of Hi-C data to identify biologically significant features, many questions still remain open, in particular regarding potential biological significance of various topological features that are characteristic for chromatin interaction networks. Results It has been previously observed that promoter capture Hi-C (PCHi-C) interaction networks tend to separate easily into well-defined connected components that can be related to certain biological functionality, however, …

Chromatin interaction networksFunctionally related modulesComputer scienceCellStructure (category theory)Topologylcsh:Computer applications to medicine. Medical informaticsBiochemistryGenomeChromosome conformation capture03 medical and health sciences0302 clinical medicineGraph topologyStructural BiologyComponent (UML)medicineHumansGene Regulatory NetworksCell type specificityPromoter Regions GeneticMolecular Biologylcsh:QH301-705.5030304 developmental biologyConnected component0303 health sciencesApplied MathematicsResearchChromatinComputer Science ApplicationsChromatinHematopoiesisIdentification (information)medicine.anatomical_structurelcsh:Biology (General)Gene Expression RegulationTopological graph theorylcsh:R858-859.7DNA microarray030217 neurology & neurosurgeryAlgorithmsBMC bioinformatics
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Reverse engineering a mouse embryonic stem cell-specific transcriptional network reveals a new modulator of neuronal differentiation

2012

Gene expression profiles can be used to infer previously unknown transcriptional regulatory interaction among thousands of genes, via systems biology 'reverse engineering' approaches. We 'reverse engineered' an embryonic stem (ES)-specific transcriptional network from 171 gene expression profiles, measured in ES cells, to identify master regulators of gene expression ('hubs'). We discovered that E130012A19Rik (E13), highly expressed in mouse ES cells as compared with differentiated cells, was a central 'hub' of the network. We demonstrated that E13 is a protein-coding gene implicated in regulating the commitment towards the different neuronal subtypes and glia cells. The overexpression and …

Chromosomal Proteins Non-HistoneCellular differentiationNeurogenesisNerve Tissue ProteinsBiologyCell LineMiceGene expressionProtein Interaction MappingGeneticsTranscriptional regulationmedicineAnimalsGene Regulatory NetworksTransgenesEmbryonic Stem CellsGene Expression ProfilingSystems BiologyNeurogenesisBrainComputational BiologyEmbryonic stem cellCell biologyGene expression profilingmedicine.anatomical_structurenervous systemNeuron differentiationNeurogliaTranscriptome
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Maternal Oct-4 is a potential key regulator of the developmental competence of mouse oocytes

2008

Abstract Background The maternal contribution of transcripts and proteins supplied to the zygote is crucial for the progression from a gametic to an embryonic control of preimplantation development. Here we compared the transcriptional profiles of two types of mouse MII oocytes, one which is developmentally competent (MIISN oocyte), the other that ceases development at the 2-cell stage (MIINSN oocyte), with the aim of identifying genes and gene expression networks whose misregulated expression would contribute to a reduced developmental competence. Results We report that: 1) the transcription factor Oct-4 is absent in MIINSN oocytes, accounting for 2) the down-regulation of Stella, a matern…

Chromosomal Proteins Non-HistoneCleavage Stage OvumRegulatorEmbryonic DevelopmentBiologyOct-4MicemedicineAnimalsCluster AnalysisGene Regulatory Networkslcsh:QH301-705.5MetaphaseOligonucleotide Array Sequence AnalysisRegulation of gene expressionGeneticsZygoteGene Expression ProfilingGene Expression Regulation DevelopmentalOocyteEmbryonic stem cellCell biologyGene expression profilingMice Inbred C57BLRepressor ProteinsRNA Messenger Storedmedicine.anatomical_structurelcsh:Biology (General)OocytesFemaleDevelopmental biologyOctamer Transcription Factor-3Developmental BiologyResearch ArticleBMC Developmental Biology
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