Search results for "Regulatory"

showing 10 items of 740 documents

A Nullclines Approach to the Study of 2D Artificial Network

2019


 
 The system of two the first order ordinary differential equations arising in the gene regulatory networks theory is studied. The structure of attractors for this system is described for three important behavioral cases: activation, inhibition, mixed activation-inhibition. The geometrical approach combined with the vector field analysis allows treating the problem in full generality. A number of propositions are stated and the proof is geometrical, avoiding complex analytic. Although not all the possible cases are considered, the instructions are given what to do in any particular situation.

GeneralityPhase portraitOrdinary differential equationAttractorStructure (category theory)Gene regulatory networkApplied mathematicsVector fieldGeneral MedicineGeneral ChemistryNullclineMathematicsContemporary Mathematics
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The human fascin gene promoter is highly active in mature dendritic cells due to a stage-specific enhancer.

2003

Abstract Dendritic cells (DC), regarded as the most efficient APCs of the immune system, are capable of activating naive T cells. Thus, DC are primary targets in immunotherapy. However, little is known about gene regulation in DC, and for efficient transcriptional targeting of human DC, a suitable promoter is still missing. Recently, we successfully used the promoter of the murine actin-bundling protein fascin to transcriptionally target DC by DNA vaccination in mice. In this study, we report on isolation of the human fascin promoter and characterization of its regulatory elements. The actively expressed gene was distinguished from a conserved inactive genomic locus and a continuous region …

Genetic MarkersRetroelementsTATA boxImmunologyMolecular Sequence DataCAAT boxRegulatory Sequences Nucleic AcidCell LineTumor Cells CulturedImmunology and AllergyHumansAmino Acid SequenceGene SilencingEnhancerPromoter Regions GeneticGene3' Untranslated RegionsCells CulturedConserved SequenceFascinRegulation of gene expressionbiologyBase SequenceGenome HumanMicrofilament ProteinsPromoterCell DifferentiationDendritic CellsExonsMolecular biologyIntronsEnhancer Elements GeneticGene Expression RegulationRegulatory sequencebiology.proteinCarrier ProteinsPseudogenesJournal of immunology (Baltimore, Md. : 1950)
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Network dynamics of eukaryotic LTR retroelements beyond phylogenetic trees

2009

Abstract Background Sequencing projects have allowed diverse retroviruses and LTR retrotransposons from different eukaryotic organisms to be characterized. It is known that retroviruses and other retro-transcribing viruses evolve from LTR retrotransposons and that this whole system clusters into five families: Ty3/Gypsy, Retroviridae, Ty1/Copia, Bel/Pao and Caulimoviridae. Phylogenetic analyses usually show that these split into multiple distinct lineages but what is yet to be understood is how deep evolution occurred in this system. Results We combined phylogenetic and graph analyses to investigate the history of LTR retroelements both as a tree and as a network. We used 268 non-redundant …

Genetic MarkersRetroelementsvirusesImmunologyGene regulatory networkRetrotransposonCaulimoviridaeBiologyGenomeGeneral Biochemistry Genetics and Molecular BiologyEvolution MolecularPhylogeneticsAnimalsGene Regulatory Networkslcsh:QH301-705.5Ecology Evolution Behavior and SystematicsPhylogenyGeneticsGenomePhylogenetic treeAgricultural and Biological Sciences(all)Biochemistry Genetics and Molecular Biology(all)Applied MathematicsResearchfungiTerminal Repeat Sequencesfood and beveragesEukaryotabiology.organism_classificationLong terminal repeatPhenotypeRetroviridaelcsh:Biology (General)Evolutionary biologyPhylogenetic PatternModeling and SimulationCaulimoviridaeGeneral Agricultural and Biological SciencesBiology Direct
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Founder mutations in BRCA1 and BRCA2 genes

2007

BRCA1 and BRCA2 germline mutations contribute to a significant number of familial and hereditary breast and/or ovarian cancers. The proportion of high-risk families with breast and/or ovarian cancer cases due to mutations in these tumor suppressor genes varies widely among populations. In some population, a wide spectrum of different mutations in both genes are present, whereas in other groups specific mutations in BRCA1 and BRCA2 have been reported with high frequency. Most of these mutations are prevalent in restricted populations as consequence of a founder effect. The comparison of haplotypes between families with the same mutation can distinguish whether high-frequency alleles derive f…

Genetic counselingPopulationBiologymedicine.disease_causeGermline mutationEthnicitymedicineHumansGenetic TestingeducationGenetic testingBRCA2 ProteinGeneticseducation.field_of_studyMutationmedicine.diagnostic_testBRCA1 ProteinHaplotypeHematologyPenetranceFounder EffectOncologyMutationApoptosis Regulatory ProteinsBRCA1 BRCA2 founder mutationFounder effect
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Cutting Edge: TGF-β Signaling Is Required for the In Vivo Expansion and Immunosuppressive Capacity of Regulatory CD4+CD25+ T Cells

2004

Abstract Data regarding the role of TGF-β for the in vivo function of regulatory CD4+CD25+ T cells (Treg) are controversial. A transgenic mouse model with impaired TGF-β signaling specifically in T cells was used to assess the role of endogenous TGF-β for the in vivo function of CD4+CD25+ Treg in a murine model of colitis induced by dextran sulfate. Transfer of wild-type, but not transgenic CD4+CD25+ Treg was found to suppress colitis in wild-type mice. In addition, by transferring CFSE-labeled CD4+CD25+ Treg we could demonstrate that endogenous TGF-β promotes the expansion of CD4+CD25+ Treg in vivo. Transgenic mice themselves developed reduced numbers of peripheral CD4+CD25+ Treg and were …

Genetically modified mouseAdoptive cell transferTransgeneImmunologychemical and pharmacologic phenomenaEndogenyBiologyT-Lymphocytes RegulatoryMiceTransforming Growth Factor betaIn vivomedicineAnimalsHumansImmunology and AllergyGenetic Predisposition to DiseaseLymphocyte CountIL-2 receptorColitisReceptorCell DifferentiationReceptors Interleukin-2hemic and immune systemsColitismedicine.diseaseAdoptive TransferCell biologyImmunologySignal TransductionThe Journal of Immunology
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TGF-beta1 in liver fibrosis: an inducible transgenic mouse model to study liver fibrogenesis.

1999

Transforming growth factor-beta1 (TGF-beta1) is a powerful stimulus for collagen formation in vitro. To determine the in vivo effects of TGF-beta1 on liver fibrogenesis, we generated transgenic mice overexpressing a fusion gene [C-reactive protein (CRP)/TGF-beta1] consisting of the cDNA coding for an activated form of TGF-beta1 under the control of the regulatory elements of the inducible human CRP gene promoter. Two transgenic lines were generated with liver-specific overexpression of mature TGF-beta1. After induction of the acute phase response (15 h) with lipopolysaccharide (100 microgram ip), plasma TGF-beta1 levels reached600 ng/ml in transgenic animals, which is100 times above normal …

Genetically modified mouseLipopolysaccharidesmedicine.medical_specialtyTranscription GeneticPhysiologyTransgeneRecombinant Fusion ProteinsMice TransgenicBiologyRegulatory Sequences Nucleic AcidLiver Cirrhosis ExperimentalMiceDownregulation and upregulationFibrosisIn vivoTransforming Growth Factor betaPhysiology (medical)Internal medicinemedicineAnimalsHumansRNA MessengerPromoter Regions GeneticRegulation of gene expressionHepatologyGastroenterologymedicine.diseaseMolecular biologyImmunohistochemistryEndocrinologymedicine.anatomical_structureC-Reactive ProteinGene Expression RegulationLiverHepatocyteHepatic stellate cellCollagenProcollagen
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A key pathogenic role for the STAT1/T-bet signaling pathway in T-cell-mediated liver inflammation.

2003

TH1 cytokines have been suggested to contribute to the pathogenesis of T-cell-mediated liver injury and inflammation. However, the molecular signaling pathways involved in such injury are still poorly understood. In the present study, we investigated the role of the STAT1/T-bet signaling pathway in a murine model of T-cell-mediated liver inflammation induced by the application of concanavalin A (Con A) using newly created STAT1 transgenic mice as well as STAT1- and T-bet-deficient mice. Liver injury induced by Con A was associated with an increase of both pSTAT1 and T-bet levels in the liver. Furthermore, functional studies suggested a pathogenic role for STAT1 in Con A-induced liver injury…

Genetically modified mouseT cellTransgeneT-LymphocytesInflammationMice TransgenicBiologyHepatitisInterferon-gammaMicemedicineConcanavalin AAnimalsInterferon gammaLiver injuryHepatologymedicine.diseasePhosphoproteinsDNA-Binding ProteinsMice Inbred C57BLIRF1medicine.anatomical_structureSTAT1 Transcription FactorLiverImmunologyTrans-ActivatorsSignal transductionmedicine.symptomT-Box Domain Proteinsmedicine.drugInterferon Regulatory Factor-1Signal TransductionTranscription FactorsHepatology (Baltimore, Md.)
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Recent advances in the expression, evolution, and dynamics of prokaryotic genomes

2007

Work on the molecular and cellular biology of prokaryotic microorganisms and their phage continues to be at the cutting edge in many areas of fundamental research in the life sciences. An important stimulus has been the large number of complete bacterial and archaeal genome sequences that have appeared over the past decade and that now are being produced at a rate of several hundred per year. This information is the foundation for all of the “-omics” leading to a global evaluation of the state of the cell under various conditions of growth or stress. An overview of the sequenced prokaryotic genomes has also greatly influenced our thinking about the evolution of life on earth. The EMBO Confe…

Genetics0303 health sciences030306 microbiologyLibrary scienceBiological evolutionBiologyPathogenicityMicrobiologyGenome[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/BacteriologyMeeting ReviewRegulatory rna03 medical and health sciencesMolecular BiologyComputingMilieux_MISCELLANEOUS030304 developmental biology
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The cis-regulatory sequences required for expression of the Drosophila melanogaster adult cuticle gene ACP65A.

2009

Post-embryonic development in insects requires successive molts. Molts are triggered by ecdysteroids, and the nature of the molt (larval, pupal or adult) is determined by juvenile hormones. The genes encoding cuticle proteins are targets of both classes of hormones, and therefore are interesting models to study hormone action at the molecular level. The Drosophila ACP65A cuticle gene is expressed exclusively during the synthesis of the adult exoskeleton, in epidermal domains synthesising flexible cuticle. We have examined the cis -regulatory sequences of ACP65A using phylogenetic comparisons and functional analysis, and find that only about 180 bp are essential, including an 81 bp intron. T…

GeneticsBase SequenceCuticlefungiMolecular Sequence DataIntronBiologyRegulatory Sequences Nucleic Acidbiology.organism_classificationDrosophila melanogasterGene Expression RegulationRegulatory sequenceInsect ScienceJuvenile hormoneGene expressionGeneticsAnimalsInsect ProteinsRegulatory Elements TranscriptionalDrosophila melanogasterMolecular BiologyGeneFunctional genomicsPhylogenyInsect molecular biology
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Gene Duplication Models and Reconstruction of Gene Regulatory Network Evolution from Network Structure

2016

The work was supported by Latvian Council of Science grant 258/2012 and Latvian State Research programme project NexIT (2014-2017).

GeneticsGeneral Computer ScienceGene duplicationGene regulatory networklanguageLatvianNetwork structureComputational biologyGraph algorithmsBiologylanguage.human_languageBaltic Journal of Modern Computing
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