Search results for "Replica"

showing 10 items of 576 documents

Prisoner's dilemma in an RNA virus

1999

The evolution of competitive interactions among viruses1 was studied in the RNA phage φ6 at high and low multiplicities of infection (that is, at high and low ratios of infecting phage to host cells). At high multiplicities, many phage infect and reproduce in the same host cell, whereas at low multiplicities the viruses reproduce mainly as clones. An unexpected result of this study1 was that phage grown at high rates of co-infection increased in fitness initially, but then evolved lowered fitness. Here we show that the fitness of the high-multiplicity phage relative to their ancestors generates a pay-off matrix conforming to the prisoner's dilemma strategy of game theory2,3. In this strateg…

GeneticsMultidisciplinarybiologyvirusesmedia_common.quotation_subjectRNARNA virusPrisoner's dilemmaVirus Replicationbiology.organism_classificationBiological EvolutionModels BiologicalVirusCystovirusBacteriophage phi 6BacteriophageGame TheoryMutationMicrobial cooperationSelfishnessmedia_commonNature
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The cost of replication fidelity in an RNA virus

2005

It is often argued that high mutation rates are advantageous for RNA viruses, because they confer elevated rates of adaptation. However, there is no direct evidence showing a positive correlation between mutation and adaptation rates among RNA viruses. Moreover, theoretical work does not argue in favor of this prediction. We used a series of vesicular stomatitis virus clones harboring single amino acid substitutions in the RNA polymerase to demonstrate that changes inducing enhanced fidelity paid a fitness cost, but that there was no positive correlation between mutation an adaptation rates. We demonstrate that the observed mutation rate in vesicular stomatitis virus can be explained by a t…

GeneticsMutation rateMultidisciplinaryAdaptation BiologicalRNARNA virusDNA-Directed RNA PolymerasesBiological SciencesBiologyVesicular stomatitis Indiana virusVirus Replicationbiology.organism_classificationVirologyVesicular stomatitis Indiana viruschemistry.chemical_compoundAmino Acid SubstitutionchemistryViral replicationVesicular stomatitis virusRNA polymeraseMutation (genetic algorithm)Mutagenesis Site-DirectedSelection GeneticProceedings of the National Academy of Sciences
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Inactivation of folylpolyglutamate synthetase Met7 results in genome instability driven by an increased dUTP/dTTP ratio

2020

AbstractThe accumulation of mutations is frequently associated with alterations in gene function leading to the onset of diseases, including cancer. Aiming to find novel genes that contribute to the stability of the genome, we screened the Saccharomyces cerevisiae deletion collection for increased mutator phenotypes. Among the identified genes, we discovered MET7, which encodes folylpolyglutamate synthetase (FPGS), an enzyme that facilitates several folate-dependent reactions including the synthesis of purines, thymidylate (dTMP) and DNA methylation. Here, we found that Met7-deficient strains show elevated mutation rates, but also increased levels of endogenous DNA damage resulting in gross…

Genome instabilityCell- och molekylärbiologiSaccharomyces cerevisiaeGenome Integrity Repair and ReplicationBiologymedicine.disease_causeGenomic InstabilityFolic AcidGene Expression Regulation FungalGeneticsmedicineThymine NucleotidesPeptide SynthasesDNA FungalUracilGeneCell NucleusRegulation of gene expressionMutationFolylpolyglutamate synthaseFungal geneticsDeoxyguanine NucleotidesMutation AccumulationMolecular biologyMitochondriaMutationDNA methylationGenome FungalDeoxyuracil NucleotidesGene DeletionCell and Molecular BiologyDNA Damage
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Mechanisms of human DNA repair: an update.

2003

The human genome, comprising three billion base pairs coding for 30000-40000 genes, is constantly attacked by endogenous reactive metabolites, therapeutic drugs and a plethora of environmental mutagens that impact its integrity. Thus it is obvious that the stability of the genome must be under continuous surveillance. This is accomplished by DNA repair mechanisms, which have evolved to remove or to tolerate pre-cytotoxic, pre-mutagenic and pre-clastogenic DNA lesions in an error-free, or in some cases, error-prone way. Defects in DNA repair give rise to hypersensitivity to DNA-damaging agents, accumulation of mutations in the genome and finally to the development of cancer and various metab…

Genome instabilityGeneticsDNA ReplicationDNA RepairBase pairDNA repairDNA damageBase Pair MismatchDNA replicationDNABiologyToxicologyDNA Repair ProteinAnimalsHumansHuman genomePoly(ADP-ribose) PolymerasesGeneDNA DamageToxicology
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Molecular and physiological consequences of faulty eukaryotic ribonucleotide excision repair

2019

Abstract The duplication of the eukaryotic genome is an intricate process that has to be tightly safe‐guarded. One of the most frequently occurring errors during DNA synthesis is the mis‐insertion of a ribonucleotide instead of a deoxyribonucleotide. Ribonucleotide excision repair (RER) is initiated by RNase H2 and results in error‐free removal of such mis‐incorporated ribonucleotides. If left unrepaired, DNA‐embedded ribonucleotides result in a variety of alterations within chromosomal DNA, which ultimately lead to genome instability. Here, we review how genomic ribonucleotides lead to chromosomal aberrations and discuss how the tight regulation of RER timing may be important for preventin…

Genome instabilityRibonucleotideDNA RepairDNA repairDNA damageRibonucleotide excision repairRibonuclease HContext (language use)ReviewBiologyGenomic InstabilityGeneral Biochemistry Genetics and Molecular Biology570 Life sciences03 medical and health scienceschemistry.chemical_compound0302 clinical medicineAnimalsHumansMolecular Biology030304 developmental biology0303 health sciencesGeneral Immunology and MicrobiologyGeneral NeuroscienceRNA–DNA hybridDNA Replication Repair & RecombinationEukaryotaDNAtopoisomerase 1ChromatinChromatinCell biologychemistryribonucleotide excision repairGenetic FitnessRNase H2030217 neurology & neurosurgeryDNA570 BiowissenschaftenThe EMBO Journal
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The sf32 unique gene of Spodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV) is a non-essential gene that could be involved in nucleocapsid o…

2013

A recombinant virus lacking the sf32 gene (Sf32null), unique to the Spodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV), was generated by homologous recombination from a bacmid comprising the complete viral genome (Sfbac). Transcriptional analysis revealed that sf32 is an early gene. Occlusion bodies (OBs) of Sf32null contained 62% more genomic DNA than viruses containing the sf32 gene, Sfbac and Sf32null-repair, although Sf32null DNA was three-fold less infective when injected in vivo. Sf32null OBs were 18% larger in diameter and contained 17% more nucleocapsids within ODVs than those of Sfbac. No significant differences were detected in OB pathogenicity (50% lethal concentration)…

GenotypevirusesScienceGenome ViralSpodopteraSpodopteraVirus ReplicationOcclusion-derived virionsRecombinant virusHomology (biology)VirusViral Proteins03 medical and health sciencesAnimalsNucleocapsidSpodoptera frugiperda multiple nucleopolyhedrovirus (SfMNPV)Gene030304 developmental biology0303 health sciencesGenes Essential[SDV.BA.MVSA]Life Sciences [q-bio]/Animal biology/Veterinary medicine and animal HealthMultidisciplinaryNucleocapsid organizationbiology030306 microbiologyfungiQVirionRbiology.organism_classificationVirologyNucleopolyhedroviruses3. Good healthViral replicationEssential geneLarvaDNA Viral[SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/VirologyMedicinesf32Homologous recombinationResearch ArticlePLoS ONE
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Photochemical sample treatment: a greener approach to chlorobenzene determination in sediments.

2014

Abstract Due to worker׳s exposure, solvent and stationary phases׳ consumption, sample purification is one of the most polluting steps in analytical procedures for determination of organic pollutants in real samples. The use of photochemical sample treatment represents a valid alternative methodology for extracts clean up allowing for a reduction of the used amount of organic solvents. In this paper we report the first application on the photolytic destruction of organic substances to eliminate some of the interferences in the analysis of Chlorobenzenes in sediment samples. The method׳s efficiency and robustness were compared with classic silica column purification process currently used in …

Geologic SedimentsPhotochemistryPhotochemistryChlorobenzenesSettore CHIM/12 - Chimica Dell'Ambiente E Dei Beni CulturaliGas Chromatography-Mass SpectrometryAnalytical ChemistrySpecimen Handlingchemistry.chemical_compoundLimit of DetectionSediment analysiClean upSoil PollutantsSettore CHIM/01 - Chimica Analiticasample treatmentPollutantDetection limitReproducibilityChromatographyChromatographySediment AnalysisPhotochemicalUV irradiationTemperatureReproducibility of ResultsWaterGreen Chemistry TechnologySettore CHIM/06 - Chimica OrganicaReplicateClean-upchemistryChlorobenzeneEnvironmental chemistrySolventsSediment analysisAnalytical proceduresChlorobenzeneTalanta
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12-month follow-up analysis of a multicenter, randomized, prospective trial in de novo liver transplant recipients (LIS2T) comparing cyclosporine mic…

2006

The LIS2T study was an open-label, multicenter study in which recipients of a primary liver transplant were randomized to cyclosporine microemulsion (CsA-ME) (Neoral) (n = 250) (monitoring of blood concentration at 2 hours postdose) C2 or tacrolimus (n = 245) (monitoring of trough drug blood level [predose]) C0 to compare efficacy and safety at 3 and 6 months and to evaluate patient status at 12 months. All patients received steroids with or without azathioprine. At 12 months, 85% of CsA-ME patients and 86% of tacrolimus patients survived with a functioning graft (P not significant). Efficacy was similar in deceased- and living-donor recipients. Significantly fewer hepatitis C–positive pati…

Graft RejectionMaleTime Factorsmedicine.medical_treatmentTACROLIMUSAzathioprineHepacivirusHEPATITIS-CLiver transplantationmedicine.disease_causeGastroenterologychemistry.chemical_compoundLiving DonorsLongitudinal StudiesC-2IMMUNOSUPPRESSIONHEPATITIS-C DIABETES-MELLITUS C-2 REPLICATION RECURRENCE SURVIVALGraft SurvivalHepatitis CTreatment Outcomesurgical procedures operativeCreatinineSURVIVALEmulsionsFemaleSteroidsImmunosuppressive Agentsmedicine.drugmedicine.medical_specialtyHepatitis C virusRenal functionRANDOMIZED STUDYAge DistributionInternal medicinemedicineDiabetes MellitusHumansHypoglycemic AgentsRECURRENCEMonitoring PhysiologicHepatitisTransplantationCreatinineHepatologybusiness.industryLIVER TRANSPLANTATIONDIABETES-MELLITUSmedicine.diseaseSurvival AnalysisTacrolimusSurgerychemistryREPLICATIONCYCLOSPORINESurgerybusinessFollow-Up Studies
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Modulation of base excision repair of 8-oxoguanine by the nucleotide sequence.

2013

8-Oxoguanine (8-oxoG) is a major product of oxidative DNA damage, which induces replication errors and interferes with transcription. By varying the position of single 8-oxoG in a functional gene and manipulating the nucleotide sequence surrounding the lesion, we found that the degree of transcriptional inhibition is independent of the distance from the transcription start or the localization within the transcribed or the non-transcribed DNA strand. However, it is strongly dependent on the sequence context and also proportional to cellular expression of 8-oxoguanine DNA glycosylase (OGG1)-demonstrating that transcriptional arrest does not take place at unrepaired 8-oxoG and proving a causal…

GuanineBase SequenceDNA RepairTranscription GeneticNucleotidesDNA-binding domainBase excision repairDNABiologyGenome Integrity Repair and ReplicationMolecular biologyDNA GlycosylasesDNA glycosylaseGenes ReporterCoding strandGeneticsDNA supercoilHumansAP siteheterocyclic compoundsNucleotide excision repairTranscription bubbleHeLa CellsNucleic acids research
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Widespread transcriptional gene inactivation initiated by a repair intermediate of 8-oxoguanine.

2016

DNA damage can significantly modulate expression of the affected genes either by direct structural interference with transcription components or as a collateral outcome of cellular repair attempts. Thus, DNA glycosylases of the base excision repair (BER) pathway have been implicated in negative transcriptional response to several spontaneously generated DNA base modifications, including a common oxidative DNA base modification 8-oxoguanine (8-oxoG). Here, we report that single 8-oxoG situated in the non-transcribed DNA strand of a reporter gene has a pronounced negative effect on transcription, driven by promoters of various strength and with different structural properties, including viral…

GuanineDNA RepairTranscription GeneticDNAGenome Integrity Repair and ReplicationHydroxamic AcidsResponse ElementsDNA GlycosylasesDNA-(Apurinic or Apyrimidinic Site) LyaseHumansGene SilencingPromoter Regions GeneticHeLa CellsPlasmidsSequence DeletionNucleic acids research
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