Search results for "Retina"

showing 10 items of 864 documents

Upregulation of antibody response to heat shock proteins and tissue antigens in an ocular ischemia model.

2011

PURPOSE. The aim of this study was to characterize the serum antibody reactivities occurring after ocular ischemia reperfusion. The time course of serum antibody responses was examined. METHODS. Wistar rats were exposed to transient ocular ischemia by elevating intraocular pressure to 130 mm Hg for 60 minutes. Axonal damage was evaluated on optic-nerve sections 2 and 4 weeks later. Blood samples collected before and several times after ischemia were used for antibody detection via customized protein microarrays. Different tissue antigens, including heat shock proteins (HSPs) and crystallins, were selected based on previous identification of antibody reactivities in studies on ischemic event…

MalePathologymedicine.medical_specialtyIschemiaHSP27 Heat-Shock ProteinsProtein Array AnalysisVimentinBiologyAutoantigensDownregulation and upregulationAntigenRetinal DiseasesHeat shock proteinGlial Fibrillary Acidic ProteinmedicineAnimalsRats WistarEye ProteinsIntraocular PressureAutoantibodiesGlial fibrillary acidic proteinRetinal VesselsSpectrinMyelin Basic ProteinOptic Nervemedicine.diseaseAxonsRatsUp-RegulationMyelin-Associated GlycoproteinShock (circulatory)Immunoglobulin GReperfusion Injurybiology.proteinMyelin-Oligodendrocyte Glycoproteinmedicine.symptomAntibodyMyelin ProteinsInvestigative ophthalmologyvisual science
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Color Doppler Hemodynamics of Giant Cell Arteritis

1994

Objectives: To determine quantitative and qualitative hemodynamic alterations within the ophthalmic, central retinal, and short posterior ciliary arteries in patients with giant cell arteritis (GCA) proved by biopsy specimen. Design, Patients, and Setting: A consecutive case series of patients with GCA referred to an urban eye hospital who were evaluated with color Doppler imaging that was used to analyze orbital blood flow velocities and vascular resistance in 22 consecutive patients with GCA compared with age and sex-matched controls. Results: Patients with GCA all demonstrated significantly reduced central retinal and short posterior ciliary arterial mean flow velocities as well as signi…

MalePathologymedicine.medical_specialtyRetinal ArteryGiant Cell ArteritisHemodynamicsMethylprednisoloneOptic neuropathyOphthalmic Arterychemistry.chemical_compoundmedicine.arteryInternal medicineLaser-Doppler FlowmetrymedicineHumansAgedAged 80 and overVascular diseasebusiness.industryCiliary BodyRetinalShort posterior ciliary arteriesBlood flowLaser Doppler velocimetryMiddle Agedmedicine.diseaseOphthalmologyGiant cell arteritismedicine.anatomical_structurechemistryRegional Blood FlowOphthalmic arterycardiovascular systemVascular resistanceCardiologyFemaleNeurology (clinical)businessBlood Flow VelocityJournal of Neuro-Ophthalmology
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Ocular Phenotype of Relaxin Gene Knockout (Rln-/-) Mice

2020

Purpose: To test if relaxin deficiency affects ocular structure and function we investigated expression of relaxin (Rln) and RXFP receptors (Rxfp1, Rxfp2), and compared ocular phenotypes in relaxin gene knockout (Rln-/- ) and wild type (Rln+/+ ) mice. Materials and Methods: Rln, Rxfp1 and Rxfp2 mRNA expression was detected in ocular tissues of Rln+/+ mice using RT-PCR. The eyes of 11 Rln-/- and 5 Rln+/+ male mice were investigated. Corneal and retinal thickness was assessed using optical coherence tomography. Intraocular pressure was measured using a rebound tonometer. Retinal, choroidal and sclera morphology and thickness were evaluated histologically. Eyes were collected and fixed for imm…

MalePathologymedicine.medical_specialtygenetic structuresAquaporinsReal-Time Polymerase Chain ReactionRetinaReceptors G-Protein-CoupledCorneaGene Knockout TechniquesMiceTonometry Ocular03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compound0302 clinical medicineCorneamedicineAnimalsRNA MessengerIntraocular PressureGene knockoutMice KnockoutRelaxinExtracellular Matrix ProteinsRetinaChoroidChemistryRelaxinRetinalFluid transporteye diseasesSensory SystemsScleraMice Inbred C57BLOphthalmologyPhenotypemedicine.anatomical_structureGene Expression Regulation030221 ophthalmology & optometryImmunohistochemistryFemalesense organsScleraTomography Optical Coherence030217 neurology & neurosurgeryCurrent Eye Research
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Ultrastructural studies of the retina in infantile neuronal ceroid-lipofuscinosis.

1988

A 9-year-old boy who had died of infantile neuronal ceroid-lipofuscinosis had experienced retina-derived visual failure. Ophthalmologically and morphologically, his retina was severely atrophic and scarred by a dense fibrillary gliosis while photoreceptor cells had completely disappeared, cells of the bipolar layer had decreased in number and had become atrophic beyond cytologic recognition. Retinal pigment epithelial cells had undergone either atrophy or proliferation. Disease-specific granular lipopigments had accumulated in perikarya and processes of remaining cells and were infrequently associated with melanin within huge melanolipofuscin bodies and RPE cells of sessile and migrating na…

MalePathologymedicine.medical_specialtygenetic structuresInfantile neuronal ceroid lipofuscinosisCytoplasmic GranulesRetinaLipofuscinMelaninchemistry.chemical_compoundAtrophyNeuronal Ceroid-LipofuscinosesCytologymedicineHumansChildMelaninsRetinaMembranesbusiness.industryRetinalGeneral MedicinePigments Biologicalmedicine.diseaseLipidseye diseasesOphthalmologymedicine.anatomical_structurechemistryUltrastructuresense organsbusinessRetinopathyRetina (Philadelphia, Pa.)
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GE-25-like immunoreactivity in the rat eye.

2012

Abstract This study aimed to investigate the presence and distribution of the chromogranin A-derived peptide GE-25 in the rat eye. The molecular form detected by the GE-25 antiserum was evaluated in the rat trigeminal ganglion, retina and remaining tissues of the rat eye by means of Western blots and the distribution pattern of GE-25-like immunoreactivity was studied in the rat eye and rat trigeminal ganglion by immunofluorescence. One single band of approximately 70 kDa was stained in the trigeminal ganglion and retina which represents the uncleaved intact chromogranin A indicating that the proteolytic processing of chromogranin A to GE-25 is limited in these tissues. Sparse GE-25-like imm…

MalePathologymedicine.medical_specialtygenetic structuresPhysiologyBlotting WesternFluorescent Antibody TechniqueIn Vitro TechniquesEyeBiochemistryCellular and Molecular NeuroscienceCiliary processesTrigeminal ganglionEndocrinologyCiliary bodymedicineAnimalsIris (anatomy)RetinabiologyChromogranin AAnatomyeye diseasesPeptide FragmentsGanglionRatsmedicine.anatomical_structureTrigeminal Ganglionbiology.proteinChromogranin Asense organsChoroidPeptides
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Branch Retinal Vein Occlusion Followed by Central Retinal Artery Occlusion in Churg-Strauss Syndrome: Unusual Ocular Manifestations in Allergic Granu…

2009

Purpose To describe a rare branch retinal vein occlusion (BRVO) followed by central retinal artery occlusion (CRAO) in a patient with Churg-Strauss syndrome (CSS). Methods A 55-year-old man with a not yet diagnosed CSS developed a BRVO in the left eye and 1 year later a CRAO with painless and acute vision loss in the same eye. Medical history included bronchial asthma, history of allergy, eosinophilic pneumonia, bilateral pleuric and pericardial effusion, hypereosinophilia, and purpuric vasculitis. Results CRAO in the left eye was diagnosed by retinal whitening and a cherry red spot with coexisting old BRVO evidenced by previous laser photocoagulation. Corticosteroids and cyclophosphamide t…

MalePathologymedicine.medical_specialtygenetic structuresRetinal Artery OcclusionChurg-strauss syndromeHypereosinophiliaChurg-Strauss SyndromeBlindness03 medical and health sciences0302 clinical medicineRetinal Vein OcclusionmedicineHumansFluorescein AngiographyAllergic granulomatous angiitisbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseeye diseasesOphthalmology030221 ophthalmology & optometryBranch retinal vein occlusionCentral retinal artery occlusionmedicine.symptombusiness030217 neurology & neurosurgerySystemic vasculitisEuropean Journal of Ophthalmology
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Further delineation of eye manifestations in homozygous 15q13.3 microdeletions including TRPM1: a differential diagnosis of ceroid lipofuscinosis.

2014

The 15q13.3 heterozygous microdeletion is a fairly common microdeletion syndrome with marked clinical variability and incomplete penetrance. The average size of the deletion, which comprises six genes including CHRNA7, is 1.5 Mb. CHRNA7 has been identified as the gene responsible for the neurological phenotype in this microdeletion syndrome. Only seven patients with a homozygous microdeletion that includes at least CHRNA7, and is inherited from both parents have been described in the literature. The aim of this study was to further describe the distinctive eye manifestations from the analysis in the three French patients diagnosed with the classical 1.5 Mb homozygous microdeletion. Patients…

MalePathologymedicine.medical_specialtygenetic structuresalpha7 Nicotinic Acetylcholine ReceptorEncephalopathyTRPM Cation ChannelsChromosome DisordersBiologyBlindnessEyePupilNeuronal Ceroid-LipofuscinosesNight BlindnessSeizuresIntellectual DisabilityRetinal DystrophiesGeneticsmedicineElectroretinographyMyopiaHumansEye AbnormalitiesChildGenetics (clinical)TRPM1Genetic Association StudiesCongenital stationary night blindnessGeneticsChromosomes Human Pair 15DystrophyEye Diseases HereditaryGenetic Diseases X-LinkedOptic NerveMicrodeletion syndromemedicine.diseasePenetranceChild PreschoolFemalesense organsDifferential diagnosisChromosome DeletionAmerican journal of medical genetics. Part A
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LATE-ONSET OCULAR HYPERTENSION AFTER VITRECTOMY: A Multicenter Study of 6,048 Eyes

2018

Purpose: To determine the incidence and risk factors for late-onset ocular hypertension (LOH) after vitrectomy. Methods: From the electronic medical records of consecutive patients who underwent primary vitrectomy, from January 2010 to December 2015, at 5 tertiary vitreoretinal centers in Italy, patient demographics, systemic, ophthalmic, operative, and postoperative data were drawn. Main outcome measure was the presence of LOH, defined as intraocular pressure >21 mmHg detected more than 2 months after vitrectomy on at least 2 consecutive visits. Results: Among 6,048 patients, LOH was found in 294 (4.9%) vitrectomized eyes and in 87 (1.4%) fellow eyes, (chi square; P < 0.001). Multiva…

MalePediatricsmedicine.medical_specialtyTime Factorsmedicine.medical_treatmentMEDLINEOcular hypertensionVitrectomyLate onset01 natural sciences03 medical and health sciences0302 clinical medicineRetinal DiseasesRisk FactorsVitrectomyHumansMedicineIntraocular PressureAgedRetrospective Studiesbusiness.industryIncidenceMedical recordIncidence (epidemiology)Retrospective cohort studyGeneral Medicinemedicine.disease0104 chemical sciences010404 medicinal & biomolecular chemistryOphthalmologyItalyMulticenter study030221 ophthalmology & optometryFemaleOcular Hypertensionbusinesslate-onset ocular hypertensionFollow-Up Studies
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Comparative study of serine-plasmalogens in human retina and optic nerve: identification of atypical species with odd carbon chains

2012

International audience; The objective of this work was to detect and identify phosphatidylserine plasmalogen species in human ocular neurons represented by the retina and the optic nerve. Plasmalogens (vinyl-ether bearing phospholipids) are commonly found in the forms of phosphatidylcholine and phosphatidylethanolamine in numerous mammalian cell types, including the retina. While their biological functions are still unclear, the alteration of cellular plasmalogen content has been associated with several human disorders such as Rhizomelic Chondrodysplasia Punctata Type 2 and Primary Open-Angle Glaucoma.By using liquid-chromatography coupled to high resolution and tandem mass spectrometry, we…

MalePlasmalogenPlasmalogensQD415-436PhosphatidylserinesBiologyTandem mass spectrometryBiochemistryRetinaSerine03 medical and health scienceschemistry.chemical_compound0302 clinical medicineEndocrinologySpecies SpecificityTandem Mass Spectrometryphospholipids/phosphatidylserinePhosphatidylcholineSerinemedicineFood and NutritionHumansphospholipidsmass spectrometryAged030304 developmental biologyAged 80 and overchemistry.chemical_classificationPhosphatidylethanolamine0303 health sciencesRhizomelic chondrodysplasia punctataFatty acidOptic NerveCell BiologyPhosphatidylserineEye/retinamedicine.diseaseCarbonchemistryBiochemistryAlimentation et NutritionFemalePatient-Oriented and Epidemiological Research[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryChromatography LiquidJournal of Lipid Research
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Synaptic vesicle alterations in rod photoreceptors of synaptophysin-deficient mice.

2001

Abstract The abundance of the integral membrane protein synaptophysin in synaptic vesicles and its multiple possible functional contributions to transmitter exocytosis and synaptic vesicle formation stand in sharp contrast to the observed lack of defects in synaptophysin knockout mice. Assuming that deficiencies are compensated by the often coexpressed synaptophysin isoform synaptoporin, we now show that retinal rod photoreceptors, which do not synthesize synaptoporin either in wild-type or in knockout mice, are affected by the loss of synaptophysin. Multiple pale-appearing photoreceptors, as seen by electron microscopy, possess reduced cytoplasmic electron density, swollen mitochondria, an…

MalePresynaptic TerminalsSynaptophysinAction PotentialsFluorescent Antibody TechniqueDark AdaptationBiologyRibbon synapseSynaptic vesicleSynaptic TransmissionExocytosisExocytosisMiceRetinal Rod Photoreceptor CellsElectroretinographySynaptic vesicle recyclingAnimalsMice KnockoutSex CharacteristicsGeneral NeuroscienceVesicleMembrane ProteinsClathrin-Coated VesiclesSynaptoporinCell biologyMice Inbred C57BLMicroscopy ElectronProtein TransportKnockout mouseSynaptophysinbiology.proteinFemaleSynaptic VesiclesNeurosciencePhotic StimulationNeuroscience
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