Search results for "Retrovirus"

showing 10 items of 42 documents

Rapid Detection of the ERV-K(C4) Retroviral Insertion Reveals Further Structural Polymorphism of the Complement C4 Genes in Old World Primates

2001

The fourth component of complement (C4) is coded for by two tandem-duplicated genes located in the class III region of the MHC of humans as well as a number of primates. A C4 gene size polymorphism giving rise to two gene variants of 16 and 22.3 kb length can be attributed to a complete endogenous retroviral insertion of 6.3 kb termed ERV-K(C4) in intron 9 of the long C4 genes. We developed a simple PCR-based screening assay to detect the presence of this insertion, and tested a number of unrelated animals from old world primate species. The presence of the ERV insertion in the orangutan, rhesus macaque and green monkey as well as its absence in gorillas and chimpanzees could be confirmed. …

PrimatesTime FactorsOld WorldVirus IntegrationImmunologyMajor histocompatibility complexPolymerase Chain Reactionbiology.animalGeneticsAnimalsPrimateGeneGenetics (clinical)GeneticsPolymorphism GeneticbiologyEndogenous RetrovirusesIntronComplement C4DNAbiology.organism_classificationIntronsMutagenesis InsertionalRhesus macaqueGreen monkeybiology.proteinBaboonExperimental and Clinical Immunogenetics
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Exceptional Heterogeneity in Viral Evolutionary Dynamics Characterises Chronic Hepatitis C Virus Infection.

2016

The treatment of HCV infection has seen significant progress, particularly since the approval of new direct-acting antiviral drugs. However these clinical achievements have been made despite an incomplete understanding of HCV replication and within-host evolution, especially compared with HIV-1. Here, we undertake a comprehensive analysis of HCV within-host evolution during chronic infection by investigating over 4000 viral sequences sampled longitudinally from 15 HCV-infected patients. We compare our HCV results to those from a well-studied HIV-1 cohort, revealing key differences in the evolutionary behaviour of these two chronic-infecting pathogens. Notably, we find an exceptional level o…

RNA viruses0301 basic medicineMaleHepacivirusHIV InfectionsHepacivirusPathology and Laboratory Medicinemedicine.disease_causeVirus ReplicationHepatitis0302 clinical medicineImmunodeficiency VirusesMedicine and Health Sciences2.2 Factors relating to the physical environmentChronicAetiologylcsh:QH301-705.5Data Managementeducation.field_of_studybiologyHepatitis C virusLiver Diseasevirus diseasesHepatitis C3. Good healthPhylogeneticsInfectious DiseasesMedical MicrobiologyViral PathogensViral evolutionVirusesEvolutionary RateHIV/AIDS030211 gastroenterology & hepatologyFemalePathogensInfectionResearch Articlelcsh:Immunologic diseases. AllergyComputer and Information SciencesEvolutionary ProcessesEvolutionHepatitis C virusPopulationChronic Liver Disease and CirrhosisImmunologyMicrobiologyViral EvolutionVirusEvolution Molecular03 medical and health sciencesHepatitis - CVirologyRetrovirusesGeneticsmedicineHumansEvolutionary SystematicsEvolutionary dynamicseducationMicrobial PathogensMolecular BiologyTaxonomyEvolutionary BiologyFlavivirusesPopulation BiologyLentivirusOrganismsBiology and Life SciencesHIVMolecularHepatitis C Chronicbiology.organism_classificationVirologyHepatitis virusesOrganismal EvolutionViral ReplicationChronic infection030104 developmental biologyEmerging Infectious Diseaseslcsh:Biology (General)Viral replicationMicrobial EvolutionImmunologyHIV-1Parasitologylcsh:RC581-607Digestive DiseasesPopulation GeneticsFollow-Up Studies
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APOBEC4 Enhances the Replication of HIV-1

2016

APOBEC4 (A4) is a member of the AID/APOBEC family of cytidine deaminases. In this study we found a high mRNA expression of A4 in human testis. In contrast, there were only low levels of A4 mRNA detectable in 293T, HeLa, Jurkat or A3.01 cells. Ectopic expression of A4 in HeLa cells resulted in mostly cytoplasmic localization of the protein. To test whether A4 has antiviral activity similar to that of proteins of the APOBEC3 (A3) subfamily, A4 was co-expressed in 293T cells with wild type HIV-1 and HIV-1 luciferase reporter viruses. We found that A4 did not inhibit the replication of HIV-1 but instead enhanced the production of HIV-1 in a dose-dependent manner and seemed to act on the viral L…

RNA virusesMale0301 basic medicineMolecular biologylcsh:MedicineArtificial Gene Amplification and ExtensionCytidinePathology and Laboratory MedicineVirus ReplicationBiochemistryPolymerase Chain ReactionJurkat cellschemistry.chemical_compoundCytidine deaminationImmunodeficiency VirusesTranscription (biology)TestisMedicine and Health Scienceslcsh:SciencePromoter Regions GeneticMultidisciplinaryCytidineTransfectionEnzymesImmunoblot AnalysisMedical MicrobiologyDeaminationViral PathogensViruses293T cellsCell linesPathogensOxidoreductasesBiological culturesLuciferaseResearch ArticleMolecular Probe TechniquesDNA constructionBiologyMicrobiologyCell Line03 medical and health sciencesCytidine DeaminaseRetrovirusesHumansMicrobial PathogensHIV Long Terminal Repeat030102 biochemistry & molecular biologylcsh:RLentivirusHEK 293 cellsOrganismsBiology and Life SciencesHIVProteinsPromoterMolecular biologyResearch and analysis methodsMolecular biology techniques030104 developmental biologychemistryPlasmid ConstructionHIV-1Enzymologylcsh:QEctopic expressionCloningPLOS ONE
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Impact of probiotic Saccharomyces boulardii on the gut microbiome composition in HIV-treated patients: A double-blind, randomised, placebo-controlled…

2017

Dysbalance in gut microbiota has been linked to increased microbial translocation, leading to chronic inflammation in HIV-patients, even under effective HAART. Moreover, microbial translocation is associated with insufficient reconstitution of CD4+T cells, and contributes to the pathogenesis of immunologic non-response. In a double-blind, randomised, placebo-controlled trial, we recently showed that, compared to placebo, 12 weeks treatment with probiotic Saccharomyces boulardii significantly reduced plasma levels of bacterial translocation (Lipopolysaccharide-binding protein or LBP) and systemic inflammation (IL-6) in 44 HIV virologically suppressed patients, half of whom (n = 22) had immun…

RNA virusesMale0301 basic medicinePlacebo-controlled studylcsh:MedicineHIV InfectionsGut floraPathology and Laboratory MedicineSystemic inflammationlaw.inventionPlacebosProbiotic0302 clinical medicineImmunodeficiency ViruseslawMedicine and Health SciencesMedicinelcsh:ScienceImmune ResponseMultidisciplinarybiologyMicrobiotaGenomicsMiddle AgedProbiòticsBacterial PathogensIntestinesSaccharomyces boulardiiMedical MicrobiologyViral PathogensVirusesFemale030211 gastroenterology & hepatologyPathogensmedicine.symptomResearch ArticleSaccharomyces boulardiiAdultImmunologyMicrobial GenomicsMicrobiologySaccharomyces03 medical and health sciencesSigns and SymptomsImmune systemDouble-Blind MethodDiagnostic MedicineRetrovirusesGeneticsVIH (Virus)HumansMicrobiomeMicrobial PathogensInflammationClostridiumBacteriabusiness.industryProbioticsGut BacteriaLentivirusLachnospiraceaelcsh:ROrganismsFungiBiology and Life SciencesHIVbiology.organism_classificationYeast030104 developmental biologyImmunologylcsh:QMicrobiomebusinessPLoS ONE
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Sequential Cross-Sectional Surveys in Orange Farm, a Township of South Africa, Revealed a Constant Low Voluntary Medical Male Circumcision Uptake amo…

2016

Background WHO recommends a male circumcision (MC) prevalence rate higher than 80% to have a substantial impact on the HIV-AIDS epidemic in Eastern and Southern Africa. Orange Farm, a township in South Africa, has a free-for-service voluntary medical male circumcision (VMMC) clinic in operation since 2008. Following an intense campaign from 2008 to 2010, MC prevalence rate increased to 55.4% (ANRS-12126). Ongoing and past VMMC campaigns focused on youths, through school talks, and adults at a community level. The main objective of the study was to assess the change in MC prevalence rate among adults aged 18–19 and 18–49 years in the past 5 years. Methods A cross-sectional survey (ANRS-12285…

RNA virusesMaleCross-sectional studyEpidemiologyPrevalenceSocial Scienceslcsh:MedicineHIV InfectionsOrange (colour)Pathology and Laboratory MedicineGeographical locationsCultural AnthropologySouth Africa0302 clinical medicineImmunodeficiency VirusesSociologyEnvironmental protectionCircumcisionMedicine and Health SciencesPrevalenceMedicine030212 general & internal medicineYoung adultReproductive System Procedureslcsh:ScienceMultidisciplinarySchoolsMiddle Aged3. Good healthReligionMale circumcisionMedical MicrobiologyHIV epidemiologyViral PathogensVirusesInfectious diseasesPathogens0305 other medical scienceDemand creationResearch ArticleAdultAdolescentSurgical and Invasive Medical ProceduresViral diseasesMicrobiologyEducation03 medical and health sciencesYoung AdultRetrovirusesAdultsHumansSex organMicrobial Pathogens030505 public healthbusiness.industryLentiviruslcsh:ROrganismsBiology and Life SciencesHIVPatient Acceptance of Health CareCross-Sectional StudiesCircumcision MaleTurnoverAge GroupsAnthropologyPeople and PlacesAfricaPopulation Groupingslcsh:Qbusiness[SDV.MHEP]Life Sciences [q-bio]/Human health and pathologyDemography
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Home-based HIV testing: Using different strategies among transgender women in Argentina.

2019

Background In Argentina, HIV prevalence among transgender women (TGW) has been reported at 34%. The stigma is one of the most important factors limiting their access to healthcare services. The aims of this study were to compare different HIV testing methodologies, to determine the factors associated with HIV diagnosis and to determine the feasibility of a home-based HIV testing service for TGW. Methods A multidisciplinary team performed home-based HIV testing interventions in four cities of Argentina. Participants self-identified as TGW, older than 14 years and with a negative or unknown HIV status. Blood samples were screened by two rapid tests (RT), one based on antibodies (Determine™ HI…

RNA virusesMultivariate analysisEpidemiologyPsychological interventionSocial SciencesHIV InfectionsLogistic regressionPathology and Laboratory MedicineTransgender womenGeographical locations0302 clinical medicineImmunodeficiency VirusesSociologyMedicine and Health Sciences030212 general & internal medicineYoung adultEnzyme-Linked Immunoassayseducation.field_of_studyMultidisciplinarySchoolsQRvirus diseasesHIV diagnosis and managementMedical MicrobiologyHIV epidemiologyViral PathogensVirusesMedicineInfectious diseasesFemalePathogens0305 other medical scienceViral loadResearch ArticleAdultmedicine.medical_specialtySciencePopulationArgentinaSexually Transmitted DiseasesHiv testingViral diseasesResearch and Analysis MethodsTransgender PersonsMicrobiologyEducation03 medical and health sciencesYoung AdultInternal medicineRetrovirusesmedicineHumanseducationImmunoassaysMicrobial Pathogens030505 public healthbusiness.industryLentivirusOrganismsBiology and Life SciencesHIVSouth AmericaDiagnostic medicineLogistic ModelsMultivariate AnalysisImmunologic TechniquesFeasibility StudiesReagent Kits DiagnosticPeople and placesbusinessPloS one
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Evolutionary plasticity of SH3 domain binding by Nef proteins of the HIV-1/SIVcpz lentiviral lineage

2021

The accessory protein Nef of human and simian immunodeficiency viruses (HIV and SIV) is an important pathogenicity factor known to interact with cellular protein kinases and other signaling proteins. A canonical SH3 domain binding motif in Nef is required for most of these interactions. For example, HIV-1 Nef activates the tyrosine kinase Hck by tightly binding to its SH3 domain. An archetypal contact between a negatively charged SH3 residue and a highly conserved arginine in Nef (Arg77) plays a key role here. Combining structural analyses with functional assays, we here show that Nef proteins have also developed a distinct structural strategy—termed the "R-clamp”—that favors the formation …

RNA virusesviruksetvirusesSimian Acquired Immunodeficiency SyndromeHIV InfectionsPathology and Laboratory MedicineSH3 domainWhite Blood CellsImmunodeficiency VirusesAnimal CellsMedicine and Health SciencesBiology (General)MammalsGenetics11832 Microbiology and virology0303 health sciencesKinase030302 biochemistry & molecular biologyEukaryotavirus diseasesTransfection3. Good healthSIVMedical MicrobiologyViral PathogensViral evolutionVirusesVertebratesProto-Oncogene Proteins c-hckApesSimian Immunodeficiency VirusPathogensCellular TypesTyrosine kinaseResearch ArticlePrimateskinaasitEvolutionary ImmunologyLineage (genetic)QH301-705.5Immune CellsImmunologyevoluutioBiologyTransfectionResearch and Analysis MethodsHIV-tartuntaMicrobiologyViral EvolutionEvolution Molecularsrc Homology Domains03 medical and health sciencesVirologyRetrovirusesGeneticsAnimalsHumansLuciferaseAmino Acid Sequencenef Gene Products Human Immunodeficiency VirusChimpanzeesMolecular Biology TechniquesMicrobial PathogensMolecular Biology030304 developmental biologyEvolutionary BiologyBlood CellsSequence Homology Amino AcidMacrophagesLentivirusOrganismsBiology and Life SciencesHIVCell BiologyRC581-607Organismal Evolution3121 General medicine internal medicine and other clinical medicineMicrobial EvolutionAmniotesHIV-1ParasitologySalt bridgeproteiinitImmunologic diseases. AllergyZoology
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Comparative cytogenetics of human chromosome 3q21.3 reveals a hot spot for ectopic recombination in hominoid evolution

2004

Fluorescence in situ hybridization mapping of fully integrated human BAC clones to primate chromosomes, combined with precise breakpoint localization by PCR analysis of flow-sorted chromosomes, was used to analyze the evolutionary rearrangements of the human 3q21.3-syntenic region in orangutan, siamang gibbon, and silvered-leaf monkey. Three independent evolutionary breakpoints were localized within a 230-kb segment contained in BACs RP11-93K22 and RP11-77P16. Approximately 200 kb of the human 3q21.3 sequence was not present on the homologous orangutan, siamang, and Old World monkey chromosomes, suggesting a genomic DNA insertion into the breakpoint region in the lineage leading to humans a…

Recombination GeneticGeneticsGenome evolutionModels GeneticGenome HumanEndogenous RetrovirusesBreakpointChromosome MappingChromosome BreakageHominidaeBiologyGenomeEvolution MolecularMolecular evolutionCytogenetic AnalysisGeneticsAnimalsHumansEctopic recombinationHuman genomeChromosomes Human Pair 3Segmental duplicationSyntenyGenomics
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Gypsy endogenous retrovirus maintains potential infectivity in several species of Drosophilids.

2008

Abstract Background Sequences homologous to the gypsy retroelement from Drosophila melanogaster are widely distributed among drosophilids. The structure of gypsy includes an open reading frame resembling the retroviral gene env, which is responsible for the infectious properties of retroviruses. Results In this study we report molecular and phylogeny analysis of the complete env gene from ten species of the obscura group of the genus Drosophila and one species from the genus Scaptomyza. Conclusion The results indicate that in most cases env sequences could produce a functional Env protein and therefore maintain the infectious capability of gypsy in these species.

RetroelementsEvolutionvirusesGenome InsectEndogenous retrovirusSequence alignmentGenes InsectGenes envEvolution MolecularOpen Reading FramesViral Envelope ProteinsPhylogeneticsDrosophilidaeQH359-425AnimalsDrosophilidaeRNA MessengerDrosophila (subgenus)Cloning MolecularGeneEcology Evolution Behavior and SystematicsPhylogenyGeneticsLikelihood FunctionsbiologyModels GeneticReverse Transcriptase Polymerase Chain ReactionEndogenous RetrovirusesDNASequence Analysis DNAbiology.organism_classificationOpen reading frameProtein BiosynthesisDrosophila melanogasterSequence AlignmentResearch ArticleBMC evolutionary biology
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Regulation of the tumor marker Fascin by the viral oncoprotein Tax of human T-cell leukemia virus type 1 (HTLV-1) depends on promoter activation and …

2015

AbstractAdult T-cell leukemia/lymphoma is a highly infiltrative neoplasia of CD4+ T-lymphocytes that occurs in about 5% of carriers infected with the deltaretrovirus human T-cell leukemia virus type 1 (HTLV-1). The viral oncoprotein Tax perturbs cellular signaling pathways leading to upregulation of host cell factors, amongst them the actin-bundling protein Fascin, an invasion marker of several types of cancer. However, transcriptional regulation of Fascin by Tax is poorly understood. In this study, we identified a triple mode of transcriptional induction of Fascin by Tax, which requires (1) NF-κB-dependent promoter activation, (2) a Tax-responsive region in the Fascin promoter, and (3) a p…

Transcriptional ActivationT-LymphocytesTaxmacromolecular substancesBiologyModels BiologicalFascinDownregulation and upregulationVirologyTranscriptional regulationmedicineHumansPromoter Regions GeneticProtein Kinase InhibitorsOncogeneFascinRegulation of gene expressionHuman T-lymphotropic virus 1NF‐kappa B (NF‐KB)Microfilament ProteinsNF-kappa BPromoterTumor virusTranscription regulationGene Products taxmedicine.diseasebiology.organism_classificationCell Transformation ViralPP2DeltaretrovirusLeukemiasrc-Family KinasesGene Expression RegulationHTLV-1ATLHuman T-lymphotropic virus 1Cancer researchbiology.proteinSignal transductionCarrier ProteinsSignal TransductionVirology
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