Search results for "Reuptake"

showing 10 items of 96 documents

Diagnostic techniques and multidisciplinary approach in idiopathic granulomatous mastitis: A revision of the literature

2019

Idiopathic granulomatous mastitis (IGM) is a chronic benign inflammatory disease of the breast that may mimic breast cancer. It is most common in parous young fertile women, although it can occur in nulliparous women and in men. IGM is an idiopathic disease due to the influence of some environmental factors in genetically predisposed subjects. Several pathogenic hypothesis have been proposed in the last years (autoimmune, hormonal, infective genesis). IGM presents as a painful palpable mass located in one of the two udders. The skin is usually normal but could present signs of inflammation with or without lymph nodes involvement. Ultrasonography, mammography, magnetic resonance can be diagn…

Settore MED/18 - Chirurgia GeneraleBreast cancerIdiopatic mastitiHumansidiopatic mastitisFemaleReviewGranulomatous MastitisChronic benign inflammatory diseaseSelective Serotonin Reuptake Inhibitors
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Effects of oxotremorine and physostigmine on the inhibitory avoidance impairment produced by amitriptyline in male and female mice.

2009

We have previously observed that amitriptyline and other antidepressants produce impairing effects on inhibitory avoidance (also called passive avoidance) in mice of both sexes. In the present study we investigated the involvement of the cholinergic system in the inhibitory avoidance impairment produced by acute amitriptyline in male and female CD1 mice. For this purpose, the effects on said task of acute i.p. administration of several doses of amitriptyline, either alone or in combination with the cholinergic agonists oxotremorine and physostigmine, were evaluated. Pre-training administration of 5, 7.5, 10 or 15 mg/kg of amitriptyline produced a significant impairment of inhibitory avoidan…

Malemedicine.medical_specialtyPhysostigmineTime FactorsAmitriptylinePhysostigmineMice Inbred StrainsPharmacologyAntidepressive Agents TricyclicCholinergic AgonistsBehavioral Neurosciencechemistry.chemical_compoundMiceRandom AllocationInternal medicineOxotremorineAvoidance LearningMedicineAnimalsAmitriptylineNeurotransmitterCholinesteraseSex Characteristicsbiologybusiness.industryLearning DisabilitiesOxotremorineEndocrinologychemistrybiology.proteinAntidepressantCholinergicFemalebusinessReuptake inhibitormedicine.drugBehavioural brain research
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Clinical responses to antidepressants among 1036 acutely depressed patients with bipolar or unipolar major affective disorders.

2012

Whether responses to antidepressants differ in bipolar and unipolar depression remains unresolved.We analyzed patient characteristics and outcomes of antidepressant treatment of 1036 depressed patients with bipolar-I or bipolar-II disorder, or unipolar major depression, using bivariate and multivariate methods and survival analysis, testing the hypothesis that responses would be superior in unipolar depression.Antidepressants were given to 84.8% (878/1036) of depressed patients: 58.9% of 93 bipolar-I, 80.1% of 117 bipolar-II, and 91.3% of 668 unipolar disorder cases. The 158 not given antidepressants had more manias/year, spent more months in mania and depression, and were far more likely t…

AdultMalemedicine.medical_specialtyBipolar DisorderMonoamine Oxidase InhibitorsAntidepressive Agents Tricyclicbehavioral disciplines and activitiesInternal medicinemental disordersmedicineHumansBipolar disorderPsychiatrySurvival analysisDepression (differential diagnoses)Depressive Disorder MajorManic MoodMiddle Agedmedicine.diseaseAntidepressive AgentsPsychiatry and Mental healthMoodTreatment OutcomeMajor depressive disorderAntidepressantFemalemedicine.symptomPsychologyManiaSelective Serotonin Reuptake InhibitorsActa psychiatrica Scandinavica
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Pharmacokinetics of selective serotonin reuptake inhibitors

2000

The five selective serotonin reuptake inhibitors (SSRIs), fluoxetine, fluvoxamine, paroxetine, sertraline, and citalopram, have similar antidepressant efficacy and a similar side effect profile. They differ, however, in their pharmacokinetic properties. Under steady-state concentrations, their half-lives range between 1 and 4 days for fluoxetine (7 and 15 days for norfluoxetine) and between 21 (paroxetine) and 36 (citalopram) hr for the other SSRIs. Sertraline and citalopram show linear and fluoxetine, fluvoxamine, and paroxetine nonlinear pharmacokinetics. SSRIs underlie an extensive metabolism with high interindividual variability, whereby cytochrome P450 (CYP) isoenzymes play a major rol…

CYP2D6FluvoxamineCitalopramPharmacologyCitalopramSerotonergicbehavioral disciplines and activitiesFluoxetineSertralinemental disordersmedicineHumansDrug InteractionsPharmacology (medical)Serotonin Uptake InhibitorsPharmacologyClinical Trials as TopicFluoxetineSertralinebusiness.industryParoxetineParoxetineFluvoxaminebusinessSelective Serotonin Reuptake Inhibitorsmedicine.drugPharmacology & Therapeutics
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A common mechanism of action of the selective serotonin reuptake inhibitors citalopram and fluoxetine: Reversal of chronic psychosocial stress-induce…

2010

The transcription factor CREB regulates adaptive responses like memory consolidation, addiction, and synaptic refinement. Recently, chronic psychosocial stress as animal model of depression has been shown to stimulate CREB transcriptional activity in the brain; this stimulation was prevented by treatment with the antidepressant imipramine, which inhibits both noradrenaline and serotonin reuptake. However, it was unknown whether the selective inhibition of serotonin reuptake is sufficient for inhibition of stress-induced CREB activation, as it is for the clinical antidepressant effect. Therefore, the effect of two selective serotonin reuptake inhibitors (SSRIs), citalopram and fluoxetine, wa…

medicine.medical_specialtyTranscription GeneticMice TransgenicCitalopramBiologyCitalopramCREBImipramineDrug Administration ScheduleMice03 medical and health sciences0302 clinical medicineGenes ReporterCREB in cognitionFluoxetineInternal medicinemedicineAnimalsPhosphorylationCyclic AMP Response Element-Binding ProteinTranscription factor030304 developmental biologyPharmacology0303 health sciencesFluoxetineBrain3. Good healthEndocrinologyGene Expression RegulationMechanism of actionbiology.proteinAntidepressantmedicine.symptomSelective Serotonin Reuptake InhibitorsStress Psychological030217 neurology & neurosurgerymedicine.drugEuropean Journal of Pharmacology
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Role of the amygdala in antidepressant effects on hippocampal cell proliferation and survival and on depression-like behavior in the rat

2021

The stimulation of adult hippocampal neurogenesis by antidepressants has been associated with multiple molecular pathways, but the potential influence exerted by other brain areas has received much less attention. The basolateral complex of the amygdala (BLA), a region involved in anxiety and a site of action of antidepressants, has been implicated in both basal and stress-induced changes in neural plasticity in the dentate gyrus. We investigated here whether the BLA modulates the effects of the SSRI antidepressant fluoxetine on hippocampal cell proliferation and survival in relation to a behavioral index of depression-like behavior (forced swim test). We used a lesion approach targeting th…

MaleLong-Term Potentiationlcsh:MedicineHippocampal formationElement-Binding ProteinAmygdala/*drug effects/physiopathologyHippocampusMemory FormationRats Sprague-Dawleyddc:616.890302 clinical medicineMedial Prefrontal CortexElevated Plus-MazeSerotonin Uptake Inhibitors/*pharmacologylcsh:ScienceBasolateral Amygdala0303 health sciencesMultidisciplinaryNeuroscience/Behavioral NeuroscienceDepressionNeurogenesisBLAAmygdalaImmunohistochemistryChronic FluoxetineAdult-RatNeuroscience/Psychologymedicine.anatomical_structureFluoxetine/*pharmacologyDepression/*pathologyAntidepressantAntidepressive Agents Second-GenerationSelective Serotonin Reuptake InhibitorsResearch ArticleEstrèsElevated plus mazemedicine.medical_specialtyAnimal-ModelAntidepressive Agents Second-Generation/*pharmacologyCell SurvivalAmygdala03 medical and health sciencesFluoxetineNeuroplasticityHippocampus/cytology/*drug effectsmedicineAnimalsPsychiatryMaze Learning030304 developmental biologyCell Proliferationbusiness.industryDentate gyrusMental Health/Mood Disorderslcsh:RBasolateral complex of the amygdaleRatsCell Proliferation/*drug effectsDentate Gyruslcsh:QCell Survival/*drug effectsbusinessNeuroscience030217 neurology & neurosurgeryBasolateral amygdala
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Autoinhibition of noradrenaline release from the rat heart as a function of the biophase concentration. Effects of exogenous alpha-adrenoceptor agoni…

1984

1. Rat isolated perfused hearts with the right sympathetic nerves intact were loaded with 3H-(-)-noradrenaline. The nerves were stimulated with trains of 180 pulses at 3 Hz and at 10 min intervals. The overflow of 3H-noradrenaline and 3H-metabolites was determined by liquid scintillation spectrometry. 2. Clonidine (IC50 17 nM), oxymetazoline (IC50 63 nM), and α-methylnoradrenaline (apparent IC50 35 nM, determined in the presence of cocaine and propranolol) decreased the stimulation-evoked overflow of 3H-noradrenaline by 26, 49, and 78%, respectively, but not methoxamine up to 100 μM (propranolol present). Oxymetazoline and α-methyl-noradrenaline did not cause desensitization of the presynap…

Malemedicine.medical_specialtyOxymetazolinePropranololIn Vitro TechniquesMethoxamineReuptakeFeedbackchemistry.chemical_compoundNorepinephrinePhentolamineCocaineInternal medicinemedicineAnimalsNeurotransmitterPhentolaminePharmacologyMyocardiumYohimbineRats Inbred StrainsGeneral MedicineElectric StimulationYohimbineRatsPerfusionEndocrinologychemistryPerfusionAdrenergic alpha-Agonistsmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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Serotonergic modulation of response inhibition and re-engagement? Results of a study in healthy human volunteers

2010

Objective Cognitive functions dependent on the prefrontal cortex, such as the ability to suppress behavior (response inhibition) and initiate a new one (response re-engagement) is important in the activities of daily life. Central serotonin (5-HT) function is thought to be a critical component of these cognitive functions. In recent studies, 5-HT failed to affect stop-signal reaction time (SSRT), a fundamental process in behavioral inhibition. We were interested if response inhibition and re-engagement are influenced through central 5-HT activity as mediated via the 5-HT transporter. Methods Here, using a stop-change task, we investigated the effects of acute and repeated treatment with 10 …

AdultMaleSerotoninCitalopramCitalopramSerotonergicDrug Administration ScheduleDevelopmental psychologyDouble-Blind MethodReaction TimemedicineHumansEscitalopramPharmacology (medical)Prefrontal cortex5-HT receptorCross-Over StudiesDose-Response Relationship DrugCognitionInhibition PsychologicalPsychiatry and Mental healthNeurologyNeurology (clinical)SerotoninReuptake inhibitorPsychologyNeuroscienceSelective Serotonin Reuptake Inhibitorsmedicine.drugHuman Psychopharmacology: Clinical and Experimental
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Influence of concomitant medications on the total clearance and the risk for supra-therapeutic plasma concentrations of Citalopram. A population-base…

2014

Introduction: The main objective of this study was to investigate the influence of the use of multiple medications and other risk factors on citalopram plasma concentrations. Methods: A retrospective cohort study with a naturalistic population of 957 patients for whom routine therapeutic drug monitoring (TDM) of citalopram had been requested between 2006 and 2013 was conducted. Results: Concomitant drugs inhibiting at least 2 different CYP subtypes involved in the metabolism of citalopram decreased statistically significantly the total clearance (Clt). Compared to younger patients over 64-year-old patients had on average a 4.5 times higher risk rate of supra-therapeutic plasma concentration…

DrugAdultMalemedicine.medical_specialtyMetabolic Clearance Ratemedia_common.quotation_subjectPopulationPharmacologyCitalopramCitalopramLogistic regressionbehavioral disciplines and activitiesSex FactorsPharmacokineticsRisk FactorsInternal medicinemental disordersmedicineCytochrome P-450 Enzyme InhibitorsHumansPharmacology (medical)Body Weights and MeasuresDrug Interactionseducationmedia_commonAgedRetrospective StudiesCytochrome P-450 Enzyme Inducerseducation.field_of_studymedicine.diagnostic_testDose-Response Relationship Drugbusiness.industryAge FactorsRetrospective cohort studyGeneral MedicineMiddle AgedPsychiatry and Mental healthTherapeutic drug monitoringConcomitantAntidepressive Agents Second-GenerationFemaleDrug MonitoringbusinessSelective Serotonin Reuptake Inhibitorsmedicine.drugPharmacopsychiatry
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Effect of sertraline treatment on benzodiazepine receptors in the rat brain.

1993

In this paper we describe the modification of benzodiazepine (BDZ) binding sites in the rat brain after different times of treatment with the 5-hydroxytryptamine-(5HT) uptake blocker sertraline. We investigated the effect of 8, 15 and 30 days sertraline treatment (10 mg/kg/day, i.p.) on 3 H-flunitrazepam binding sites. In order to describe the anatomical site of action of the drug, the experiment has been carried out by means of quantitative receptor autoradiography. After 8 days of sertraline treatment, an increase of BDZ receptor density is found in the olfactory tubercle. This effect is reversed at 15 and 30 days. At 15 days of treatment, an increase is found in the anterior cingulate co…

Malemedicine.medical_specialtymedicine.drug_classRats Sprague-DawleyInternal medicineSertralinemedicineLimbic SystemAnimalsReceptorBiological Psychiatry5-HT receptorBrain ChemistryCerebral CortexBenzodiazepineSertralineBehavior AnimalGABAA receptorChemistryOlfactory tubercleBody WeightSeptal nucleiOlfactory PathwaysReceptors GABA-AAntidepressive AgentsRatsPsychiatry and Mental healthmedicine.anatomical_structureEndocrinology1-NaphthylamineNeurologyAnti-Anxiety AgentsCerebral cortexNeurology (clinical)Selective Serotonin Reuptake Inhibitorsmedicine.drugJournal of neural transmission. General section
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